Search results for "Lymphocyte Subsets"

showing 10 items of 212 documents

The protein tyrosine kinase Tec regulates a CD44highCD62L- Th17 subset.

2010

Abstract The generation of Th17 cells has to be tightly controlled during an immune response. In this study, we report an increase in a CD44highCD62L− Th17 subset in mice deficient for the protein tyrosine kinase Tec. CD44highCD62L− Tec−/− CD4+ T cells produced enhanced IL-17 upon activation, showed increased expression levels of IL-23R and RORγt, and IL-23–mediated expansion of Tec−/− CD4+ T cells led to an increased production of IL-17. Tec−/− mice immunized with heat-killed Streptococcus pneumoniae displayed increased IL-17 expression levels in the lung postinfection with S. pneumoniae, and this correlated with enhanced pneumococcal clearance and reduced lung inflammation compared with T…

Encephalomyelitis Autoimmune ExperimentalTECeducationImmunologyImmunoblottingInflammationEnzyme-Linked Immunosorbent AssayCell SeparationBiologyMiceImmune systemIn vivoRAR-related orphan receptor gammaT-Lymphocyte SubsetsmedicineImmunology and AllergyAnimalsCell LineageL-SelectinMice KnockoutReverse Transcriptase Polymerase Chain ReactionCD44Interleukin-17hemic and immune systemsCell DifferentiationPneumoniaT-Lymphocytes Helper-InducerProtein-Tyrosine KinasesFlow CytometryMolecular biologyHyaluronan ReceptorsCancer researchbiology.proteinCytokinesmedicine.symptomSignal transductiontissuesTyrosine kinaseSignal TransductionJournal of immunology (Baltimore, Md. : 1950)
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Regulatory T cells--the renaissance of the suppressor T cells.

2007

Immune reactions are stringently regulated and balanced by complex interactions of stimulating and suppressing mechanisms. Dysfunctions of this sophisticated immune regulatory network can lead to a variety of diseases such as autoimmunity, allergy, cancer, and pregnancy disorders. The rediscovery of suppressor T cells a decade ago--now designated as T regulatory cells--set off a huge avalanche of research activities leading to a multitude of preclinical and clinical studies. Herein, we give a comprehensive review about this research on T regulatory cells and the relevance of this suppressive T cell population for the development of innovative immune therapeutic strategies.

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentT cellPopulationAutoimmunitymedicine.disease_causeInfectionsT-Lymphocytes RegulatoryAutoimmunitylaw.inventionMiceImmune systemlawPregnancyT-Lymphocyte SubsetsTransplantation ImmunologyNeoplasmsmedicineSuppressor Factors ImmunologicAnimalsHumanseducationeducation.field_of_studybusiness.industryModels ImmunologicalGeneral MedicineT lymphocyteImmunotherapyInflammatory Bowel DiseasesTransplantationDisease Models Animalmedicine.anatomical_structureDiabetes Mellitus Type 1ImmunologySuppressorFemaleImmunotherapybusinessAnnals of medicine
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TGF-beta as a T cell regulator in colitis and colon cancer

2005

TGF-beta is a pleiotropic cytokine with powerful immunosuppressive functions. Mice deficient for TGF-beta1 show a dramatic phenotype with severe multiorgan inflammation and die shortly after birth. Recent investigations have highlighted the role of TGF-beta in suppression of T cell mediated autoimmune inflammation and anti-tumor immunity. In addition to its direct anti-inflammatory effects on T cells, TGF-beta has been implicated as central regulator of regulatory T cells. TGF-beta not only mediates the suppression of effector T cells by Tregs, recent evidence also reveals a role for TGF-beta along with TCR stimulation in the peripheral induction of regulatory T cells from naïve CD4+CD25- c…

Endocrinology Diabetes and MetabolismT cellImmunologyBiologyGeneral Biochemistry Genetics and Molecular BiologyTCIRG1Interleukin 21T-Lymphocyte SubsetsTransforming Growth Factor betamedicineAnimalsHumansImmunology and AllergyCytotoxic T cellIL-2 receptorIntestinal MucosaAntigen-presenting cellZAP70Cell DifferentiationColitisNatural killer T cellDisease Models Animalmedicine.anatomical_structureColonic NeoplasmsImmunologyInflammation MediatorsCytokine & Growth Factor Reviews
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TGF-beta regulates airway responses via T cells.

2003

Abstract Allergic asthma is characterized by airway hyperreactivity, inflammation, and a Th2-type cytokine profile favoring IgE production. Beneficial effects of TGF-β and conflicting results regarding the role of Th1 cytokines have been reported from murine asthma models. In this study, we examined the T cell as a target cell of TGF-β-mediated immune regulation in a mouse model of asthma. We demonstrate that impairment of TGF-β signaling in T cells of transgenic mice expressing a dominant-negative TGF-β type II receptor leads to a decrease in airway reactivity in a non-Ag-dependent model. Increased serum levels of IFN-γ can be detected in these animals. In contrast, after injection of OVA …

Epitopes T-LymphocyteNitric Oxide Synthase Type IIImmunoglobulin EMiceAntibody SpecificityCell MovementT-Lymphocyte SubsetsTransforming Growth Factor betaImmunology and AllergyInterferon gammaLungInterleukin-13biologymedicine.diagnostic_testrespiratory systemImmunohistochemistrymedicine.anatomical_structureInterleukin 13Alum Compoundsmedicine.symptomBronchial HyperreactivityBronchoalveolar Lavage Fluidmedicine.drugGenetically modified mousemedicine.medical_specialtyOvalbuminT cellImmunologyCD2 AntigensInflammationMice Inbred StrainsMice TransgenicProtein Serine-Threonine KinasesInterferon-gammaInternal medicineAdministration InhalationmedicineAnimalsHumansAerosolsInflammationbusiness.industryReceptor Transforming Growth Factor-beta Type IITransforming growth factor betaImmunoglobulin ETh1 Cellsrespiratory tract diseasesEndocrinologyBronchoalveolar lavageImmunologybiology.proteinNitric Oxide SynthasebusinessReceptors Transforming Growth Factor betaJournal of immunology (Baltimore, Md. : 1950)
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TGFbeta regulates the CD4+CD25+ T-cell pool and the expression of Foxp3 in vivo.

2004

Factors influencing the development of CD4+CD25+ T-cells in vivo are poorly understood. In order to investigate the contribution of TGFbeta1 to the development and function of CD4+CD25+ T-cells, we generated a gain of function mutation resulting in the overexpression of an active form of TGFbeta1 in T-cells under control of the human CD2 promoter. In peripheral lymphoid organs and in the thymus, the frequency of CD4+CD25+ T-cells was increased in transgenic mice. This appeared to be due to an autocrine effect of TGFbeta on T-cells, since concomitant impairment of TGFbeta-signaling in double transgenic mice resulted in a phenotype similar to wild type. In contrast, in single transgenic mice …

Genetically modified mouseTransgeneT cellImmunologyCD2 AntigensMice TransgenicBiologyMiceIn vivoT-Lymphocyte SubsetsTransforming Growth Factor betamedicineImmunology and AllergyAnimalsAutocrine signallingTranscription factorWild typeFOXP3Forkhead Transcription FactorsReceptors Interleukin-2General MedicineMolecular biologyCell biologyInterleukin-10DNA-Binding Proteinsmedicine.anatomical_structureCD4 AntigensInternational immunology
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A distinct subset of HLA-DR+-regulatory T cells is involved in the induction of preterm labor during pregnancy and in the induction of organ rejectio…

2010

Regulatory T cells (Tregs) are known to suppress alloimmune responses during pregnancy and post organ transplantation. We demonstrate that a distinct subset of FoxP3(+)DR(+)-Tregs among the total CD4(+)CD127(low+/-)CD25(+)-Treg cell pool is critically involved in preterm labor induction and kidney transplant rejection as well. Compared to healthy pregnancies and non-rejecting kidney recipients, we found that the percentage of the FoxP3(+)DR(+)-Treg subset was not reduced, but that the level of HLA-DR expression of such Tregs was strongly diminished in preterm laboring women and in patients with acute renal allograft rejection. In addition, both patient collectives showed a significantly red…

Graft RejectionMalemedicine.medical_specialtyImmunologychemical and pharmacologic phenomenaT-Lymphocytes RegulatoryOrgan transplantationImmune toleranceInterleukin-7 Receptor alpha SubunitObstetric Labor PrematurePregnancyT-Lymphocyte SubsetsHLA-DRImmune ToleranceImmunology and AllergyMedicineHumansKidney transplantationbusiness.industryInterleukin-2 Receptor alpha SubunitFOXP3hemic and immune systemsForkhead Transcription FactorsHLA-DR Antigensmedicine.diseaseKidney TransplantationTransplant rejectionCD4 Lymphocyte CountTransplantationTolerance inductionImmunologyPremature BirthFemalebusinessClinical immunology (Orlando, Fla.)
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Human cytomegalovirus (HCMV)-specific CD4+ T lymphocyte response in AIDS patients with no past or current HCMV disease following HAART.

2003

Abstract Background: The incidence of Human Cytomegalovirus (HCMV) end-organ disease has dramatically decreased since the implementation of highly active antiretroviral therapies (HAARTs), but the precise immune mechanism whereby HCMV is controlled remains to be elucidated. Objectives: To investigate the effect of (HAART) on CD4 + T-cell immunity to HCMV in AIDS patients with no past or current HCMV disease. Study design: Seventeen patients were prospectively examined for CD4 + (CD45RO + and CD45 RA + ) T-cell counts (flow cytometry), HIV RNA load (Amplicor HIV test), HCMV leukoDNAemia and HCMV DNA in urine (nested PCR), lymphoproliferative response (LPR) to HCMV, phytohemagglutinin (PHA) a…

Human cytomegalovirusAdultCD4-Positive T-LymphocytesMalevirusesCytomegalovirusmedicine.disease_causeLymphocyte ActivationHerpesviridaeVirusInterferon-gammaBetaherpesvirinaeT-Lymphocyte SubsetsVirologyImmunopathologyAntiretroviral Therapy Highly ActivemedicineHumansViremiaAcquired Immunodeficiency SyndromebiologyAIDS-Related Opportunistic Infectionsvirus diseasesHIVbiochemical phenomena metabolism and nutritionMiddle AgedViral Loadbiology.organism_classificationmedicine.diseaseVirologyCD4 Lymphocyte CountInterleukin-10Infectious DiseasesImmunologyCytomegalovirus InfectionsDNA ViralCytokinesRNA ViralCytokine secretionFemaleViral diseaseInterleukin-4Lymphoproliferative responseJournal of clinical virology : the official publication of the Pan American Society for Clinical Virology
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Impact of CMV and EBV seropositivity on CD8 T lymphocytes in an old population from West-Sicily.

2007

Abstract Herpes viruses (particularly CMV and to some extent EBV) might play a role in accelerating the deterioration of immune functions with age. Indeed, it has been demonstrated that chronic infection with CMV causes an expansion of specific CD8 T lymphocytes and that this is related to a shrinkage of the T cell repertoire in very elderly people, predicting mortality. We have analysed CD8 T cells in young and old healthy Sicilians who were both CMV- and EBV-seropositive. Our data confirm expansions of T cells specific for the HLA-A2-restricted pp65 (495–503) CMV epitope up to nearly 14% of total peripheral CD8 cells in certain elderly individuals (range 0–14%). However, the mean percenta…

Human cytomegalovirusAdultMaleAgingEpstein-Barr Virus InfectionsHerpesvirus 4 HumanPopulationCytomegalovirusEpitopes T-LymphocyteBiologyCD8-Positive T-LymphocytesAntibodies ViralBiochemistryEpitopeVirusImmunophenotypingElderlyEndocrinologyImmune systemEBVT-Lymphocyte SubsetsHLA-A2 AntigenGeneticsmedicineCytotoxic T cellHumanseducationMolecular BiologySicilyAgedSettore MED/04 - Patologia GeneraleAged 80 and overeducation.field_of_studyCMVCD8Immune senescenceCell BiologyImmunosenescenceMiddle Agedmedicine.diseaseVirologyImmunologyCytomegalovirus InfectionsFemaleCD8Experimental gerontology
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γδT cells elicited by CMV reactivation after allo-SCT cross-recognize CMV and leukemia.

2013

Human cytomegalovirus (CMV) infections and relapse of disease remain major problems after allogeneic stem cell transplantation (allo-SCT), in particular in combination with CMV-negative donors or cordblood transplantations. Recent data suggest a paradoxical association between CMV reactivation after allo-SCT and reduced leukemic relapse. Given the potential of Vδ2-negative γδT cells to recognize CMV-infected cells and tumor cells, the molecular biology of distinct γδT-cell subsets expanding during CMV reactivation after allo-SCT was investigated. Vδ2(neg) γδT-cell expansions after CMV reactivation were observed not only with conventional but also cordblood donors. Expanded γδT cells were ca…

Human cytomegalovirusCancer ResearchAdoptive cell transferT cellT-LymphocytesCytomegalovirusBiologyAntigenT-Lymphocyte SubsetsmedicineHomologous chromosomeHumansTransplantation HomologousLeukemiavirus diseasesReceptors Antigen T-Cell gamma-deltaHematologymedicine.diseaseTransplantationLeukemiamedicine.anatomical_structureOncologyImmunologyVirus ActivationStem cellStem Cell TransplantationLeukemia
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Are human Vδ2(pos) T cells really resistant to aging and Human Cytomegalovirus infection?()

2019

In their recent paper, Weili Xu et al. [1] described the different behaviors of Vδ1pos and Vδ2pos T cell subsets in response to lifelong stress and claimed that Vδ2pos T cells are not affected by aging and Human Cytomegalovirus (HCMV) infection. While we agree that these two γδ T cell subsets diverge both in phenotype/function and in tissue distribution, we are somewhat surprised that authors did not take into account the several previously published and contradictory experimental evidence in regards to senescence of Vδ2pos T cells [2,3]. These latter studies reported that HCMV infection not only induces a clonal expansion of a distinct Vγ9neg/Vδ2pos T cell subset, but also determines a con…

Human cytomegalovirusCytomegalovirus InfectionLetterCongenital cytomegalovirus infectionCytomegaloviruCytomegalovirusT-Lymphocyte SubsetReceptors Antigen T-Cell gamma-deltaGeneral MedicineBiologymedicine.diseaseVirologyGeneral Biochemistry Genetics and Molecular BiologyT-Lymphocyte SubsetsCytomegalovirus InfectionsHost-Pathogen InteractionsmedicineHumansCytomegalovirus infectionsLymphocyte subsetsHumanDisease Resistance
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