Search results for "Lymphocyte"

showing 10 items of 2280 documents

Impact of antigen presentation on TCR modulation and cytokine release: implications for detection and sorting of antigen-specific CD8+ T cells using …

2002

Abstract Soluble MHC class I molecules loaded with antigenic peptides are available either to detect and to enumerate or, alternatively, to sort and expand MHC class I-restricted and peptide-reactive T cells. A defined number of MHC class I/peptide complexes can now be implemented to measure T cell responses induced upon Ag-specific stimulation, including CD3/CD8/ζ-chain down-regulation, pattern, and quantity of cytokine secretion. As a paradigm, we analyzed the reactivity of a Melan-A/MART-1-specific and HLA-A2-restricted CD8+ T cell clone to either soluble or solid-phase presented peptides, including the naturally processed and presented Melan-A/MART-1 peptide AAGIGILTV or the peptide ana…

Cytotoxicity ImmunologicT cellCD8 AntigensImmunologyAntigen presentationReceptors Antigen T-CellDown-RegulationEpitopes T-LymphocyteCD8-Positive T-LymphocytesMHC class IHLA-A2 AntigenmedicineImmunology and AllergyCytotoxic T cellHumansAntigen PresentationPeptide analogbiologyAntigen processingMembrane ProteinsMHC restrictionMolecular biologymedicine.anatomical_structureAmino Acid SubstitutionReceptor-CD3 Complex Antigen T-Cellbiology.proteinMutagenesis Site-DirectedCytokinesCD8Journal of immunology (Baltimore, Md. : 1950)
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Amino acid substitutions at position 97 in HLA-A2 segregate cytolysis from cytokine release in MART-1/Melan-A peptide AAGIGILTV-specific cytotoxic T …

1996

CD8+ T lymphocytes recognize antigenic peptides presented by major histocompatibility complex (MHC) class I molecules. Individual peptide termini appear to be fixed at the C- and N-terminal ends. In contrast, central peptide side chains residues may point in different directions and exhibit limited flexibility, dependent on the MHC class I structural variation. For instance, position 97 in HLA-A201 has been shown to shift individual peptide species into different coordinations, one oriented towards the peptide N terminus, or more towards the C-terminal end. The conformational shape of such non-anchor peptide residues may affect the affinity of MHC/peptide/TCR interaction, resulting in quant…

Cytotoxicity ImmunologicT cellImmunologyPeptide bindingMajor histocompatibility complexMART-1 AntigenAntigens NeoplasmMHC class IHLA-A2 AntigenmedicineTumor Cells CulturedImmunology and AllergyCytotoxic T cellHumansAmino Acid SequencePeptide sequenceMelanomabiologyMHC restrictionMolecular biologyNeoplasm Proteinsmedicine.anatomical_structureBiochemistrybiology.proteinCytokinesCD8Protein BindingT-Lymphocytes CytotoxicEuropean journal of immunology
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Frequency of cytotoxic T lymphocyte precursors to herpes simplex virus type 1 as determined by limiting dilution analysis.

1983

The conditions for establishing a limiting dilution assay to measure cytotoxic T lymphocyte precursors (CTL-P) against herpes simplex virus type 1 (HSV-1) were determined. Analysis by Poisson statistics demonstrated that the estimated frequency of HSV-1-reactive cells in the spleens of normal mice was less than 1/250,000. In contrast, mice immunized previously with infectious HSV-1 demonstrated a CTL-P frequency between 1/3,500 and 1/15,670. The generation of a maximum cytotoxic T lymphocyte response required that mice be primed in vivo with infectious virus. Immunization with inactivated virus either failed to elicit detectable CTL-P frequencies or gave frequencies markedly less than thos…

Cytotoxicity ImmunologicT cellImmunologyPopulationchemical and pharmacologic phenomenaBiologymedicine.disease_causeMicrobiologyVirusMiceImmune systemAntigenmedicineCytotoxic T cellAnimalsAntigens LySimplexviruseducationCytotoxicityCells Culturededucation.field_of_studyVirologyInfectious Diseasesmedicine.anatomical_structureHerpes simplex virusImmunologic TechniquesMice Inbred CBAInterleukin-2ParasitologyImmunizationSpleenT-Lymphocytes CytotoxicViral Infections and ImmunityInfection and immunity
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Precursor frequency can compensate for lower TCR expression in T cell competition during priming in vivo.

2006

The factors controlling clonal dominance of cytotoxic T lymphocyte (CTL) responses are currently not well understood. To study the functional impact of the strength of the interaction of a T cell with an antigen-presenting cell in this context, we established a new mouse model comprised of two T cell receptor (TCR)-transgenic strains expressing the identical TCR in differing amounts, hence providing two CTL clones with different avidities but identical specificity and affinity. Utilizing this new model, we show that upon antigen challenge higher-avidity CTL expand at the expense of moderate-avidity CTL in vivo if present in equal numbers. Beyond this, moderate-avidity T cells can also contr…

Cytotoxicity ImmunologicT cellImmunologyReceptors Antigen T-CellPriming (immunology)chemical and pharmacologic phenomenaMice TransgenicStreptamerImmunodominanceBiologyLymphocyte ActivationMiceAntigenmedicineImmunology and AllergyCytotoxic T cellAnimalsAntigen PresentationStem CellsT-cell receptorhemic and immune systemsFlow CytometryAdoptive TransferCell biologyMice Inbred C57BLCTL*medicine.anatomical_structureImmunologyT-Lymphocytes CytotoxicEuropean journal of immunology
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Enhanced susceptibility to cytotoxic T lymphocytes without increase of MHC class I antigen expression after conditional overexpression of heat shock …

1999

Antigenic peptides have been found associated with heat shock proteins (HSP) including cytoplasmic HSP70 and heat shock cognate protein 70 as well as the endoplasmic reticulum-resident glucose-regulated protein 94. Recently, HSP70 transfection has been reported to increase MHC class I cell surface expression and antigen presentation on mouse melanoma B16 cells (Wells et al., Int. Immunol. 1998. 10: 609). To analyze the effect of HSP70 on MHC class I cell surface expression and lysability of target cells we transfected a human melanoma cell line with the rat Hsp70-1 gene using the Tet-On system for conditional overexpression of HSP70. Induction of HSP70 did not increase cell surface expressi…

Cytotoxicity ImmunologicT-LymphocytesImmunologyAntigen presentationCD1BiologyMajor histocompatibility complexMajor Histocompatibility ComplexMiceMHC class ITumor Cells CulturedImmunology and AllergyCytotoxic T cellAnimalsHumansHSP70 Heat-Shock ProteinsMelanomaAntigen PresentationAntigen processingMHC class I antigenGene Transfer TechniquesMHC restrictionMolecular biologyRatsGene Expression Regulation Neoplasticbiology.proteinEuropean journal of immunology
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Fine specificity and cytolytic activity of continuously growing alloreactive cytotoxic T lymphocyte clones.

1980

The role of Lyt 1+ T-cell-derived secondard CTL inducing factor allowed the cloning of alloreactive cytotoxic T lymphocytes (CTL) by the limiting dilution approach. Several monoclonal cell lines were established in vitro. The lytic activity of some of the cell lines exceeded that of CTL from bulk cultures; that is, 50% of the target cells were lysed at an effector to target cell ratio of 0.04:1. The fine specificity of individual CTL clones is discussed.

Cytotoxicity ImmunologicT-LymphocytesImmunologyCellchemical and pharmacologic phenomenaCell LineMiceAntibody SpecificitymedicineCytotoxic T cellAnimalsAntigens ViralMice Inbred BALB CChemistryEffectorH-2 Antigenshemic and immune systemsGeneral MedicineVirologyMolecular biologyClone CellsCTL*Cytolysismedicine.anatomical_structureLytic cycleCell cultureMonoclonalMice Inbred CBALymphocyte Culture Test MixedSpleenScandinavian journal of immunology
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Cyclosporin A mediates immunosuppression of primary cytotoxic T cell responses by impairing the release of interleukin 1 and interleukin 2

1981

The site of action of the immunosuppressive drug cyclosporin A in in vitro cytotoxic allograft responses has been localized. General cytotoxic effects of the drug on proliferating T cells became apparent at concentrations of 500-1000 ng/ml, while selective effects were observed at concentrations of 10-100 ng/ml. The selective effects included a blockade of interleukin 2 release from activated T helper cells on the one hand and inhibition of interleukin 1 release from splenic adherent cells on the other. While cyclosporin A did not interfere with the intracellular events required for the activation and subsequent clonal expansion of alloreactive T cells, the lack of interleukin 1 and interle…

Cytotoxicity ImmunologicT-LymphocytesImmunologyCyclosporinsPharmacologyBiologyLymphocyte ActivationMiceInterleukin 21Cyclosporin aAnimalsImmunology and AllergyInterleukin 5Interleukin 4Interleukin 3Mice Inbred BALB CProteinsInterleukinInterleukin 33Protein BiosynthesisMice Inbred CBAInterleukin 12Interleukin-2Lymphocyte Culture Test MixedImmunosuppressive AgentsInterleukin-1European Journal of Immunology
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Herpes-Simplex-virus-specific, H-2Dk-restricted T lymphocytes bear receptors for H-2Dd alloantigen.

1980

Cytotoxic T lymphocytes generated in the course of an HSV-infection of CBA (H-2k) mice not only lyse syngeneic, virus-infected target cells but also cross-react with noninfected taraget cells expressing the Dd alloantigen. On the effector cell level, this alloreactivity is mediated by virus-specific CTL's that are restricted to H-2Dk determinants. On the prekiller cell level, the anti-HSV-reactive T cells exhibiting cross-reactivity for Dd alloantigen could be positively selected on H-2d spleen-cell monolayers. After differentiation into cytolytic effector cells, target cells expressing Dd alloantigens and syngeneic HSV-infected target were lysed with equal efficiency. The results imply tha…

Cytotoxicity ImmunologicT-LymphocytesImmunologyReceptors Antigen T-Cellchemical and pharmacologic phenomenaMice Inbred StrainsBiologymedicine.disease_causeVirusEpitopesMiceGeneticsmedicineCytotoxic T cellAnimalsSimplexvirusReceptorEffectorH-2 Antigenshemic and immune systemsbiology.organism_classificationMolecular biologyCytolysisCTL*RickettsiaHerpes simplex virusImmunologyImmunogenetics
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T cell-mediated cytotoxic immune responsiveness of chimeric mice bearing a thymus graft fully allogeneic to the graft of lymphoid stem cells

1980

Fully allogeneic, chimeric mice were established by adult thymectomy of (A × B) F1animals, grafting parental A-type thymus under the kidney capsula, followed by lethal (900 rd) irradiation and reconstitution with B parental-type bone marrow cells treated with xenogeneic anti-T cell antiserum plus complement. Following in vivo sensitization with inactivated Sendai virus (SV) suspensions, no virus-specific T cells could be detected within the spleen cells of the mice. Upon stimulation with third-party allogeneic cells in a primary mixed lymphocyte culture, spleen cells of all animals generated alloreactive cytotoxic T lymphocytes (CTL). More interestingly, upon secondary in vitro stimulation …

Cytotoxicity ImmunologicT-LymphocytesT cellImmunologySpleenThymus GlandBiologyMiceImmune systemBone MarrowmedicineAnimalsImmunology and AllergyCytotoxic T cellLymphocytesMice Inbred BALB CChimeraMolecular biologyParainfluenza Virus 1 HumanMice Inbred C57BLCTL*medicine.anatomical_structureRadiation ChimeraImmunologyMice Inbred CBALymphoid Progenitor CellsBone marrowStem cellEuropean Journal of Immunology
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Targeting positive regulatory domain I-binding factor 1 and X box-binding protein 1 transcription factors by multiple myeloma-reactive CTL.

2005

Abstract Growing evidence indicates that multiple myeloma (MM) and other malignancies are susceptible to CTL-based immune interventions. We studied whether transcription factors inherently involved in the terminal differentiation of mature B lymphocytes into malignant and nonmalignant plasma cells provide MM-associated CTL epitopes. HLA-A*0201 (A2.1) transgenic mice were used to identify A2.1-presented peptide Ag derived from the plasma cell-associated transcriptional regulators, positive regulatory domain I-binding factor 1 (PRDI-BF1) and X box-binding protein 1 (XBP-1). A2.1-restricted CTL specific for PRDI-BF1 and XBP-1 epitopes efficiently killed a variety of MM targets. PRDI-BF1- and X…

Cytotoxicity ImmunologicX-Box Binding Protein 1Cellular differentiationImmunologyEpitopes T-LymphocyteMice TransgenicRegulatory Factor X Transcription FactorsBiologyEpitopeMiceImmune systemCell Line TumorHLA-A2 AntigenImmunology and AllergyAnimalsHumansTranscription factorAntigen PresentationB-LymphocytesCell DeathT-cell receptorCell DifferentiationCytotoxicity Tests ImmunologicX-Box Binding Protein 1Molecular biologyPeptide FragmentsCell biologyDNA-Binding ProteinsMice Inbred C57BLRepressor ProteinsCTL*Self ToleranceNIH 3T3 CellsPositive Regulatory Domain I-Binding Factor 1Multiple MyelomaCD8T-Lymphocytes CytotoxicTranscription FactorsJournal of immunology (Baltimore, Md. : 1950)
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