Search results for "Lymphocyte"

showing 10 items of 2280 documents

GM-CSF restores innate, but not adaptive, immune responses in glucocorticoid-immunosuppressed human blood in vitro.

2003

Abstract Infection remains the major complication of immunosuppressive therapy in organ transplantation. Therefore, reconstitution of the innate immunity against infections, without activation of the adaptive immune responses, to prevent graft rejection is a clinically desirable status in transplant recipients. We found that GM-CSF restored TNF mRNA and protein expression without inducing IL-2 production and T cell proliferation in glucocorticoid-immunosuppressed blood from either healthy donors or liver transplant patients. Gene array experiments indicated that GM-CSF selectively restored a variety of dexamethasone-suppressed, LPS-inducible genes relevant for innate immunity. A possible ex…

Graft RejectionLipopolysaccharidesT-LymphocytesCell Cycle ProteinsCell SeparationOrgan transplantationDexamethasoneMiceCDC2-CDC28 KinasesConcanavalin ATumor Cells CulturedImmunology and AllergySkin TransplantationMiddle AgedCyclin-Dependent KinasesUp-RegulationSurvival Ratemedicine.anatomical_structureImmunity ActiveTumor necrosis factor alphaGlucocorticoidCell DivisionCyclin-Dependent Kinase Inhibitor p27Immunosuppressive Agentsmedicine.drugAdultmedicine.medical_specialtyT cellImmunologyDown-RegulationBiologyProtein Serine-Threonine KinasesImmune systemAdjuvants ImmunologicIn vivomedicineAnimalsHumansDexamethasoneAgedSalmonella Infections AnimalInnate immune systemTumor Suppressor ProteinsCyclin-Dependent Kinase 2Granulocyte-Macrophage Colony-Stimulating FactorImmunity InnateGene Expression RegulationImmunologyLeukocytes MononuclearMice Inbred CBAInterleukin-2Interleukin-1Journal of immunology (Baltimore, Md. : 1950)
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HCV-induced immune responses influence the development of operational tolerance after liver transplantation in humans.

2014

Pathogen-induced immune responses prevent the establishment of transplantation tolerance in experimental animal models. Whether this occurs in humans as well remains unclear. The development of operational tolerance in liver transplant recipients with chronic hepatitis C virus (HCV) infection allows us to address this question. We conducted a clinical trial of immunosuppression withdrawal in HCV-infected adult liver recipients to elucidate (i) the mechanisms through which allograft tolerance can be established in the presence of an ongoing inflammatory response and (ii) whether anti-HCV heterologous immune responses influence this phenomenon. Of 34 enrolled liver recipients, drug withdrawal…

Graft RejectionMaleHepatitis C virusT cellmedicine.medical_treatmentHepacivirusLiver transplantationCD8-Positive T-Lymphocytesmedicine.disease_causeImmune systemInterferonmedicineImmune ToleranceHumansLymphocyte CountImmunosuppression Therapybusiness.industryImmunityImmunosuppressionReceptors Antigen T-Cell gamma-deltaGeneral MedicineMiddle Agedmedicine.diseaseLiver TransplantationTransplantationmedicine.anatomical_structureGene Expression RegulationImmunologyInterferon Type IFemaleViral hepatitisbusinessBiomarkersmedicine.drugScience translational medicine
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Induction and tolerization of anti-male CD8+ cytotoxic T lymphocytes by in vivo immunization with an H-Y-derived peptide

1999

Abstract We have analyzed the immune response induced by a 9mer synthetic peptide derived from the male histocompatibility antigen H-Y and containing D b -binding motifs in C57BL/6 mice. In this study we report that a single, subcutaneous injection of the peptide emulsified in IFA gave rise to the development of male-specific CD8 + T cells which displayed H-Y-specific proliferative response in vitro and showed a Tc1-type pattern of cytokine production (i.e. they secreted IFN-γ and IL-2, but not IL-4 and IL-10). Development of a strong cytotoxic activity required in vitro stimulation with specific peptide and IL-2: under these culture conditions, we were able to generate potent CD8 + CTLs th…

Graft RejectionMaleTime Factorsmedicine.medical_treatmentH-Y AntigenImmunologyPeptideBiologyMajor histocompatibility complexMiceImmune systemAntigenOsmotic PressuremedicineAnimalsImmunology and AllergyCytotoxic T cellH-Y antigenchemistry.chemical_classificationSkin TransplantationGeneral MedicineMolecular biologyMice Inbred C57BLCytokineSolubilitychemistryImmunologybiology.proteinFemalePeptidesCD8T-Lymphocytes CytotoxicHuman Immunology
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Inflammation Causes Resistance to Anti-CD20–Mediated B Cell Depletion

2016

B cells play a central role in antibody-mediated rejection and certain autoimmune diseases. However, B cell-targeted therapy such as anti-CD20 B cell-depleting antibody (aCD20) has yielded mixed results in improving outcomes. In this study, we investigated whether an accelerated B cell reconstitution leading to aCD20 depletion resistance could account for these discrepancies. Using a transplantation model, we found that antigen-independent inflammation, likely through toll-like receptor (TLR) signaling, was sufficient to mitigate B cell depletion. Secondary lymphoid organs had a quicker recovery of B cells when compared to peripheral blood. Inflammation altered the pharmacokinetics (PK) and…

Graft RejectionMalemedicine.drug_classInflammation030230 surgeryMonoclonal antibodyArticleLymphocyte DepletionMice03 medical and health sciences0302 clinical medicinemedicineAnimalsImmunologic FactorsImmunology and AllergyPharmacology (medical)ReceptorB cellInflammationB-LymphocytesMice Inbred BALB CTransplantationbiologybusiness.industryGraft SurvivalAlloimmunityImmunoglobulins IntravenousAntigens CD20Mice Inbred C57BLTransplantationmedicine.anatomical_structureImmunologybiology.proteinHeart TransplantationFemaleRituximabAntibodymedicine.symptomRituximabbusiness030215 immunologymedicine.drugAmerican Journal of Transplantation
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A distinct subset of HLA-DR+-regulatory T cells is involved in the induction of preterm labor during pregnancy and in the induction of organ rejectio…

2010

Regulatory T cells (Tregs) are known to suppress alloimmune responses during pregnancy and post organ transplantation. We demonstrate that a distinct subset of FoxP3(+)DR(+)-Tregs among the total CD4(+)CD127(low+/-)CD25(+)-Treg cell pool is critically involved in preterm labor induction and kidney transplant rejection as well. Compared to healthy pregnancies and non-rejecting kidney recipients, we found that the percentage of the FoxP3(+)DR(+)-Treg subset was not reduced, but that the level of HLA-DR expression of such Tregs was strongly diminished in preterm laboring women and in patients with acute renal allograft rejection. In addition, both patient collectives showed a significantly red…

Graft RejectionMalemedicine.medical_specialtyImmunologychemical and pharmacologic phenomenaT-Lymphocytes RegulatoryOrgan transplantationImmune toleranceInterleukin-7 Receptor alpha SubunitObstetric Labor PrematurePregnancyT-Lymphocyte SubsetsHLA-DRImmune ToleranceImmunology and AllergyMedicineHumansKidney transplantationbusiness.industryInterleukin-2 Receptor alpha SubunitFOXP3hemic and immune systemsForkhead Transcription FactorsHLA-DR Antigensmedicine.diseaseKidney TransplantationTransplant rejectionCD4 Lymphocyte CountTransplantationTolerance inductionImmunologyPremature BirthFemalebusinessClinical immunology (Orlando, Fla.)
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Pivotal Advance: Up-regulation of acetylcholine synthesis and paracrine cholinergic signaling in intravascular transplant leukocytes during rejection…

2009

Abstract A new role and source of the old mediator acetylcholine is described, which is produced by graft monocytes and attenuates monocytic ATP-signaling. During acute rejection, large numbers of leukocytes accumulate in the blood vessels of experimental renal allografts. About 70% of them are activated, cytotoxic monocytes that appear to be involved in allograft destruction. ACh exerts anti-inflammatory effects upon monocytes/macrophages and has been proposed to be a key player in neuroimmunological interactions. Its short half-life, however, makes it unlikely that neuronal ACh affects blood leukocytes. Renal transplantation was performed in the allogeneic DA to LEW and in the isogeneic L…

Graft RejectionPathologymedicine.medical_specialtyIsograftImmunologyBiologyReceptors NicotinicParacrine signallingAdenosine TriphosphateIn vivoParacrine CommunicationmedicineImmunology and AllergyCytotoxic T cellAnimalsTransplantation HomologousLymphocytesCation Transport ProteinsMonocyteCell BiologyKidney TransplantationAcetylcholineRatsUp-RegulationTransplantationTransplantation Isogeneicmedicine.anatomical_structureRats Inbred LewImmunohistochemistryCholinergicSignal TransductionJournal of leukocyte biology
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The effect of ischemia/reperfusion on the kidney graft.

2014

Ischemia/reperfusion injury is an unavoidable companion after kidney transplantation and influences short-term as well as long-term graft outcome. Clinically ischemia/reperfusion injury is associated with delayed graft function, graft rejection, and chronic graft dysfunction. Ischemia/reperfusion affects many regulatory systems at the cellular level as well as in the renal tissue that eventually result in a distinct inflammatory reaction of the kidney graft.Underlying factors include energy metabolism, cellular changes of the mitochondria and cellular membranes, initiation of different forms of cell death-like apoptosis and necrosis together with a recently discovered mixed form termed necr…

Graft RejectionTransplantationmedicine.medical_specialtyKidneyGraft dysfunctionGraft rejectionbusiness.industryT-LymphocytesIschemiamedicine.diseaseKidney TransplantationDelayed Graft FunctionImmunity InnateSurgerysurgical procedures operativemedicine.anatomical_structureImmunityReperfusion InjurymedicineImmunology and AllergyAnimalsHumansbusinessReperfusion injuryKidney transplantationCurrent opinion in organ transplantation
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Preclinical report on allogeneic uterus transplantation in non-human primates

2013

Study question Is it possible to perform allogeneic uterus transplantation (UTx) with a donation from a live donor in a non-human primate species and what immunosuppression is needed to prevent rejection? Summary answer Allogeneic UTx in the baboon is a donor- and recipient-safe surgical procedure; immunosuppression with induction therapy and a triple protocol should be used. What is known already UTx may become a treatment for absolute uterine factor infertility. Autologous UTx models have been developed in non-human primates with reports on long-term survival of the uterine grafts. STUDY DESIGN, SIZEAND DURATION: This experimental study included 18 female baboons as uterus donors and 18 f…

Graft Rejectionmedicine.medical_specialtymedicine.medical_treatmentUterusTacrolimusMaintenance ChemotherapyAdrenal Cortex Hormonesmedicine.arteryUterus transplantationmedicineLiving DonorsAnimalsTransplantation HomologousExternal iliac veinAntilymphocyte SerumImmunosuppression TherapyUterine DiseasesHysterectomybusiness.industryRehabilitationGraft SurvivalUterusObstetrics and GynecologyImmunosuppressionInduction ChemotherapyMycophenolic AcidInternal iliac arteryTacrolimusSurgeryTransplantationDisease Models Animalmedicine.anatomical_structureReproductive MedicineFeasibility StudiesDrug Therapy CombinationFemalebusinessInfertility FemalePapio
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Immunogenicity of enhanced green fluorescent protein (EGFP) in BALB/c mice: identification of an H2-Kd-restricted CTL epitope

2000

Enhanced green fluorescent protein (EGFP) is a novel marker gene product, which is readily detectable using techniques of fluorescence microscopy, flow cytometry, or macroscopic imaging. In the present studies, we have examined the immunogenicity of EGFP in murine models. A stable transfectant of the transplantable CMS4 sarcoma of BALB/c origin expressing EGFP, CMS4-EGFP-Zeo, was generated. Splenocytes harvested from mice immunized with a recombinant adenovirus expressing EGFP (Ad-EGFP) were restimulated in vitro with CMS4-EGFP-Zeo. Effector lymphocytes displayed strong cytotoxicity against CMS4-EGFP-Zeo, but not against mock-transfected CMS4-Zeo tumor cells. A number of candidate H2-Kd-bin…

Green Fluorescent ProteinsBiologyCancer VaccinesEpitopeBALB/cFlow cytometryGreen fluorescent proteinMiceAntigenAntigens NeoplasmGeneticsmedicineAnimalsMolecular BiologyMice Inbred BALB Cmedicine.diagnostic_testImmunogenicityfungiH-2 Antigensbiology.organism_classificationMolecular biologyTumor antigenIn vitroLuminescent ProteinsModels AnimalMolecular MedicineT-Lymphocytes CytotoxicGene Therapy
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Cross-reactivity between tumor MHC class I-restricted antigens and an enterococcal bacteriophage

2020

International audience; Intestinal microbiota have been proposed to induce commensal-specific memory T cells that cross-react with tumor-associated antigens. We identified major histocompatibility complex (MHC) class I-binding epitopes in the tail length tape measure protein (TMP) of a prophage found in the genome of the bacteriophage Enterococcus hirae Mice bearing E. hirae harboring this prophage mounted a TMP-specific H-2Kb-restricted CD8+ T lymphocyte response upon immunotherapy with cyclophosphamide or anti-PD-1 antibodies. Administration of bacterial strains engineered to express the TMP epitope improved immunotherapy in mice. In renal and lung cancer patients, the presence of the ent…

H-2 AntigenProgrammed Cell Death 1 ReceptorCD8-Positive T-LymphocytesEpitopeEpitopesFecesMice0302 clinical medicineEnterococcus hiraeNeoplasmsMonoclonalBacteriophages0303 health sciencesMultidisciplinarybiologyAntibodies MonoclonalViral Tail ProteinsAlkylating3. Good healthmedicine.anatomical_structure030220 oncology & carcinogenesisCross ReactionEpitopeImmunotherapyHumanT cellAntineoplastic Agents[SDV.CAN]Life Sciences [q-bio]/CancerCross ReactionsMajor histocompatibility complexAntibodiesMicrobiology03 medical and health sciencesAnimals; Antibodies Monoclonal; Antigens Neoplasm; Antineoplastic Agents Alkylating; Bacteriophages; CD8-Positive T-Lymphocytes; Cross Reactions; Cyclophosphamide; Enterococcus hirae; Epitopes; Feces; Gastrointestinal Microbiome; H-2 Antigens; Histocompatibility Antigens Class I; Humans; Immunotherapy; Mice; Neoplasms; Programmed Cell Death 1 Receptor; Viral Tail Proteins[SDV.CAN] Life Sciences [q-bio]/CancerAntigenAntigens NeoplasmMHC class ImedicineAnimalsHumansAntigensBacteriophageAntineoplastic Agents AlkylatingCyclophosphamideProphage030304 developmental biologyEnterococcus hiraeAnimalHistocompatibility Antigens Class IH-2 AntigensCD8-Positive T-Lymphocytebiology.organism_classificationGastrointestinal Microbiomebiology.proteinNeoplasmFeceCD8
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