Search results for "Lymphocyte"

showing 10 items of 2280 documents

Interferon-regulatory factor 4 is essential for the developmental program of T helper 9 cells.

2010

Summary Interferon-regulatory factor 4 (IRF4) is essential for the development of T helper 2 (Th2) and Th17 cells. Herein, we report that IRF4 is also crucial for the development and function of an interleukin-9 (IL-9)-producing CD4 + T cell subset designated Th9. IRF4-deficient CD4 + T cells failed to develop into IL-9-producing Th9 cells, and IRF4-specific siRNA inhibited IL-9 production in wild-type CD4 + T cells. Chromatin-immunoprecipitation (ChIP) analyses revealed direct IRF4 binding to the Il9 promoter in Th9 cells. In a Th9-dependent asthma model, neutralization of IL-9 substantially ameliorated asthma symptoms. The relevance of these findings is emphasized by the fact that the ind…

ImmunologyBiologyPathogenesisInterleukin 21MiceDownregulation and upregulationmedicineImmunology and AllergyAnimalsHumansInterleukin 9RNA Small InterferingMOLIMMUNOPromoter Regions GeneticCells CulturedMice KnockoutInterleukin-9Cell DifferentiationT helper cellT-Lymphocytes Helper-InducerAsthmaMice Inbred C57BLInfectious Diseasesmedicine.anatomical_structureCELLIMMUNOImmunologyInterferon Regulatory FactorsFunction (biology)Platelet factor 4IRF4Protein BindingImmunity
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Nitric Oxide Promotes Resistance to Tumor Suppression by CTLs

2006

Abstract Many human tumors express inducible NO synthetase (NOS2), but the roles of NO in tumor development are not fully elucidated. An important step during tumor development is the acquisition of apoptosis resistance. We investigated the dose-dependent effects of endogenously produced NO on apoptosis using ecdysone-inducible NOS2 cell lines. Our results show that short-term NOS2 expression enhances CD95-mediated apoptosis and T cell cytotoxicity dose dependently. Furthermore, we could show that during chronic exposure to NO, besides the primary cytotoxic NO effect, there is selection of cell clones resistant to NO that show cross-resistance to CD95-induced apoptosis and the killing by CT…

ImmunologyCellNitric Oxide Synthase Type IIApoptosisBiologyEndoplasmic ReticulumNitric OxideCell LineMalignant transformationParacrine signallingImmune systemNeoplasmsmedicineHumansImmunology and AllergyCytotoxic T cellfas ReceptorAutocrine signallingMitochondriamedicine.anatomical_structureGene Expression RegulationApoptosisCell cultureMitochondrial MembranesImmunologyCancer researchSignal TransductionT-Lymphocytes CytotoxicThe Journal of Immunology
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Recognition of human renal cell carcinoma and melanoma by HLA-A2-restricted cytotoxic T lymphocytes is mediated by shared peptide epitopes and up-reg…

1996

Cytotoxic T lymphocytes (CTL) have previously been isolated from peripheral blood of patients with renal cell carcinoma (RCC). The CD8-positive CTL line MZ1257-CTL-5 (CTL-5) has been shown to lyse autologous cultured RCC cells in an HLA-A2 restricted fashion. Allogeneic, HLA-A2-matched RCC and melanoma cell lines were also lysed by CTL-5, suggesting that melanoma and renal cancer share antigenic determinants. The aim of the study was to determine whether RCC and melanoma share peptide epitopes that are recognized by CTL-5 in the context of HLA-A2 molecules. Peptides were acideulated from various cell lines, separated by reversed phase high performance liquid chromatography (RP-HPLC), and as…

ImmunologyCellurologic and male genital diseasesEpitopeEpitopesInterferon-gammaAntigenMHC class IHLA-A2 AntigenmedicineTumor Cells CulturedCytotoxic T cellHumansInterferon gammaCarcinoma Renal CellMelanomaB-LymphocytesbiologyMelanomaGeneral Medicinemedicine.diseaseMolecular biologyUp-RegulationCTL*medicine.anatomical_structurebiology.proteinPeptidesmedicine.drugT-Lymphocytes CytotoxicScandinavian journal of immunology
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Enrichment of Immediate-Early 1 (m123/pp89) Peptide-Specific CD8 T Cells in a Pulmonary CD62LloMemory-Effector Cell Pool during Latent Murine Cytomeg…

2000

ABSTRACTInterstitial cytomegalovirus (CMV) pneumonia is a clinically relevant complication in recipients of bone marrow transplantation (BMT). Recent data for a model of experimental syngeneic BMT and concomitant infection of BALB/c mice with murine CMV (mCMV) have documented the persistence of tissue-resident CD8 T cells after clearance of productive infection of the lungs (J. Podlech, R. Holtappels, M.-F. Pahl-Seibert, H.-P. Steffens, and M. J. Reddehase, J. Virol. 74:7496–7507, 2000). It was proposed that these cells represent antiviral “standby” memory cells whose functional role might be to help prevent reactivation of latent virus. The pool of pulmonary CD8 T cells was composed of two…

ImmunologyCytomegalovirusPeptideCD8-Positive T-LymphocytesBiologyEffector cellMicrobiologyImmediate-Early ProteinsMiceInterleukin 21Latent VirusAntigenVirologyAnimalsCytotoxic T cellAntigens ViralLungAntigenic peptidechemistry.chemical_classificationMice Inbred BALB Cvirus diseasesVirologyVirus LatencyCytomegalovirus infectionchemistryInsect ScienceCytomegalovirus InfectionsImmunologyPathogenesis and ImmunityFemaleImmunologic MemoryJournal of Virology
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Cyclic adenosine monophosphate and IL-10 coordinately contribute to nTreg cell-mediated suppression of dendritic cell activation

2010

In humans and mice naturally occurring regulatory T cells (nTregs) are crucial for the maintenance of peripheral tolerance by controlling not only potentially autoreactive T cells but virtually all cells of the adaptive and innate immune system. Here we show that co-culture of murine dendritic cells (DC) and nTregs results in an immediate increase of cAMP in DC, responsible for a rapid down-regulation of co-stimulatory molecules (CD80, CD86). In addition, the inhibitory surface molecule B7-H3 on DC is up-regulated. Subsequently, nTreg-derived IL-10 inhibits the cytokine production (IL-6, IL-12) of suppressed DC therewith preserving their silent phenotype. Hence, our data indicate that nTreg…

ImmunologyDown-RegulationCell CommunicationBiologyT-Lymphocytes RegulatoryImmune toleranceMiceImmune systemCyclic AMPImmune ToleranceAnimalsCD86Innate immune systemInterleukin-6Peripheral toleranceDendritic CellsDendritic cellInterleukin-12Coculture TechniquesInterleukin-10Cell biologyInterleukin 10B7-1 AntigenB7-2 AntigenCD80Signal TransductionCellular Immunology
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Inhibition of human allergic T-helper type 2 immune responses by induced regulatory T cells requires the combination of interleukin-10-treated dendri…

2006

Summary Background In grass pollen-allergic individuals, T cell anergy can be induced by IL-10-treated dendritic cells (IL-10-DC) resulting in the suppression of T helper type 1 (Th1) as well as Th2 cells. This study was performed to analyse whether such IL-10-DC-treated T cells are able to act as regulatory T cells (Treg) suppressing the function of other T cells in the periphery. As transforming growth factor (TGF)-β is also a potential inducer of Treg, we additionally analysed the inhibitory capacity of TGF-β-treated T cells in this system. Materials and Methods Freshly isolated CD4+ or CD4+CD25− T cells from grass pollen-allergic donors were stimulated with autologous mature monocyte-de…

ImmunologyEnzyme-Linked Immunosorbent AssayCell CommunicationBiologyPoaceaeT-Lymphocytes RegulatoryInterleukin 21Interferon-gammaTh2 CellsAntigens CDTransforming Growth Factor betaHypersensitivityImmunology and AllergyCytotoxic T cellHumansCTLA-4 AntigenIL-2 receptorAntigen-presenting cellCells CulturedInterleukin 3Cell ProliferationDendritic cellDendritic CellsAllergensNatural killer T cellFlow CytometryAntigens DifferentiationCell biologyInterleukin-10ImmunologyInterleukin 12PollenImmunizationInterleukin-4Interleukin-5Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology
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Impact of MHC class I alleles on the M. tuberculosis antigen-specific CD8+ T-cell response in patients with pulmonary tuberculosis

2007

Challenged by scattered understanding of protective immunity to Mycobacterium tuberculosis (MTB), we have mapped peptide epitopes to human leukocyte antigen (HLA)-A*0101, A*0201, A*1101, A*2402, B*0702, B*0801 and B*1501 of the secreted mycobacterial antigen Ag85B, a vaccine candidate that may be associated with immune protection. Affinity (ED(50)) and half-life (t(1/2), off-rate) analysis for individual peptide species on HLA-A and HLA-B molecules revealed binding ranges between 10(-3) and 10(-7) M. After selection of the best matches, major histocompatibility complex class I/peptide tetramer complexes were constructed to measure the CD8+ T-cell responses directly ex vivo in peripheral blo…

ImmunologyGenes MHC Class IPeptide bindingHuman leukocyte antigenCD8-Positive T-LymphocytesMajor histocompatibility complexEpitopeMycobacterium tuberculosisMHC class IGeneticsHumansCytotoxic T cellTuberculosis PulmonaryAllelesCells CulturedGenetics (clinical)HLA-A AntigensbiologyMycobacterium tuberculosisFlow Cytometrybiology.organism_classificationVirologyMolecular biologyHLA-B Antigensbiology.proteinEpitope MappingCD8Genes & Immunity
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Expression of cell adhesion molecules in inflammatory myopathies.

1995

We examined the expression of cell adhesion molecules in 25 cases of inflammatory myopathies. Inflammatory myopathies showed upregulation of adhesion molecules. ICAM-1 was strongly expressed on endothelial cells as well as on fibroblasts and infiltrating leukocytes while the expression of VCAM-1, similar in its distribution, was much weaker. A few muscle fibers in polymyositis revealed sarcolemmal labeling for ICAM-1. ELAM-1 showed only weak expression on vessels. The inflammatory cellular infiltrates contained varying amounts of cells bearing the VCAM-1 ligand VLA-4 and the ELAM-1 ligand SLeX as well as large amounts of cells expressing LFA-1 alpha and beta, ligands of ICAM-1.

ImmunologyIntercellular Adhesion Molecule-1Lewis X AntigenVascular Cell Adhesion Molecule-1InflammationNectinReceptors Very Late AntigenE-selectinmedicineImmunology and AllergyHumansCell adhesionbiologyMyositisCell adhesion moleculeChemistrySoluble cell adhesion moleculesIntercellular Adhesion Molecule-1Lymphocyte Function-Associated Antigen-1Cell biologyNeurologycardiovascular systembiology.proteinNeural cell adhesion moleculeNeurology (clinical)medicine.symptomE-SelectinCell Adhesion MoleculesJournal of neuroimmunology
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Mast cells are crucial for early inflammation, migration of Langerhans cells, and CTL responses following topical application of TLR7 ligand in mice.

2007

Abstract Until recently, IgE-activated mast cells have been regarded merely as effector cells of adaptive immune responses, involved in allergic reactions and mucosal immunity to parasites. Herein, we report that murine dermal mast cells, activated by local administration of a cream containing the synthetic TLR7 ligand imiquimod, are essential to initiate an early inflammatory reaction. The mast-cell–derived cytokines TNF-α and IL-1β play an important role in this process. Furthermore, TLR7-activated mast cells are also able to promote the emigration of Langerhans cells, which partly depends on the expression of mast-cell–derived IL-1β. We have previously shown that TLR7 ligation enhances t…

ImmunologyInterleukin-1betaInflammationImmunoglobulin ELigandsBiochemistryMiceImmune systemAdjuvants ImmunologicCell MovementmedicineCytotoxic T cellAnimalsMast CellsAntigensSkinInflammationImmunity CellularMice Inbred BALB CVaccinesImiquimodMembrane GlycoproteinsbiologyTumor Necrosis Factor-alphaDegranulationCell BiologyHematologyTLR7Immunoglobulin EAcquired immune systemImmunity InnateInterleukin 33Toll-Like Receptor 7Langerhans CellsImmunologybiology.proteinAminoquinolinesImmunizationmedicine.symptomAgranulocytosisT-Lymphocytes CytotoxicBlood
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CD4-mediated functional activation of human CD4+CD25+ regulatory T cells

2007

Naturally occurring CD4(+)CD25(+)FoxP3(+) regulatory T cells (CD25(+) Tregs) constitute a specialized population of T cells that is essential for the maintenance of peripheral self-tolerance. The immune regulatory function of CD25(+) Tregs depends upon their activation. We found that anti-CD4 antibodies activate the suppressive function of human CD25(+) Tregs in a dose-dependent manner. We demonstrate that CD4-activated CD25(+) Tregs suppress the proliferation of CD4(+) and CD8(+) T cells, their IL-2 and IFN-gamma production as well as the capacity of CD8(+) T cells to re-express CD25. By contrast, anti-CD4 stimulation did not induce suppressive activity in conventional CD4(+) T cells. Thes…

ImmunologyInterleukin-2 Receptor alpha SubunitAntibodies MonoclonalFOXP3hemic and immune systemschemical and pharmacologic phenomenaCD8-Positive T-LymphocytesBiologyFlow CytometryLymphocyte ActivationT-Lymphocytes RegulatoryCoculture TechniquesImmune toleranceCell biologyInterleukin 21Immune systemCD4 AntigensImmunologyImmune ToleranceHumansImmunology and AllergyCytotoxic T cellIL-2 receptorCell activationCD8European Journal of Immunology
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