Search results for "Lymphocyte"

showing 10 items of 2280 documents

Diminished Contact Hypersensitivity Response in IL‐4 Deficient Mice at a Late Phase of the Elicitation Reaction

1997

Contact hypersensitivity (CHS) is thought to depend on the activation of T cells of Th1 and/or Tc1 type. The role of Th2/Tc2 cells in the contact allergic reaction is not clear. The aim of this study was to analyse the functional contribution of Th2/Tc2 cells in CHS using the interleukin-4 (IL-4) deficient mouse model. Interleukin-4 deficient (IL4T) and control (wt) mice were sensitized by epicutaneous application of 2,4-dinitrofluorobenzene. The ear swelling response measured 24 h after challenge was similar in IL4T and control mice. However, from 48 h onwards, ear swelling values were significantly reduced in IL4T mice. The stimulatory capacity of freshly isolated as well as 3-day culture…

MaleImmunologyPopulationCellCell CountBiologyDermatitis ContactLymphocyte ActivationFlow cytometryMiceT-Lymphocyte SubsetsmedicineAnimalseducationInterleukin 4SkinMice Inbred BALB Ceducation.field_of_studymedicine.diagnostic_testEpidermis (botany)EffectorT-cell receptorContact hypersensitivityReceptors Antigen T-Cell gamma-deltaDendritic CellsGeneral MedicineFlow CytometryMolecular biologyMice Mutant StrainsMice Inbred C57BLmedicine.anatomical_structureLangerhans CellsImmunologyFemaleInterleukin-4EpidermisScandinavian Journal of Immunology
researchProduct

In vitro anticholinergic drugs affect CD8+ peripheral blood T-cells apoptosis in COPD

2011

Novel pharmacological strategies are aimed at the resolution of systemic inflammation in COPD potentiating peripheral blood T-cell (PBT-cell) apoptosis. Although muscarinic acetylcholine receptors (mAChRs) M(3) and choline-acetyltransferase (ChAT) participate in the airway inflammation of COPD, their role in PBT-cell apoptosis remains unexplained. We evaluated in PBT-cells from COPD patients, smoker (S) and control (C) subjects: (1) apoptosis (by annexin V binding), (2) mAChR M(3) and ChAT expression, acetylcholine (ACh)-binding; (3) choline levels in serum and PBT-cells extracts. We tested the effects of Tiotropium (Spiriva(®)) and hemicholinium-3 (HCh-3) on apoptosis, NFκB pathway, caspas…

MaleImmunologyScopolamine DerivativesApoptosisCD8-Positive T-LymphocytesPharmacologySystemic inflammationCholinergic AntagonistsCholineCholine O-AcetyltransferasePulmonary Disease Chronic ObstructiveAnnexinMuscarinic acetylcholine receptormedicineHumansImmunology and AllergyLymphocyte CountTiotropium BromideCaspaseAgedReceptor Muscarinic M3Caspase 8COPDbiologyCaspase 3Systemic inflammation Non-neuronal components of cholinergic system Caspases NF B pathwaybusiness.industryNF-kappa BHematologyTiotropium bromideMiddle Agedmedicine.diseaserespiratory tract diseasesEnzyme ActivationApoptosisbiology.proteinFemalemedicine.symptombusinessAcetylcholineProtein BindingSignal Transductionmedicine.drugImmunobiology
researchProduct

Protective role of nuclear factor of activated T cells 2 in CD8+ long-lived memory T cells in an allergy model

2007

Background The transcriptional regulation of cytokines released and controlled by memory T cells is not well understood. Defective IFN-γ production in allergic asthma correlates in human beings with the risk of wheezing in childhood. Objective To understand the role of the transcription factor nuclear factor of activated T cells 2 (NFATc2) in memory and effector T cells in the airways in experimental allergic asthma. Methods We used murine models of allergic asthma and adoptive cell transfer of fluorescence-activated sorted cells in a disease model. Results Mice lacking NFATc2 developed an increase in airway hyperresponsiveness (AHR), remodeling, and serum IgE levels on ovalbumin sensitizat…

MaleInterleukin 2Adoptive cell transferImmunologyMice SCIDCD8-Positive T-LymphocytesInterferon-gammaMiceInterleukin 21T-Lymphocyte SubsetsHypersensitivitymedicineAnimalsImmunology and AllergyCytotoxic T cellInterleukin-7 receptorLungMice KnockoutMice Inbred BALB CReceptors Interleukin-7NFATC Transcription Factorsbusiness.industryInterleukin-17Cell Differentiationrespiratory systemAdoptive TransferMolecular biologyGrowth InhibitorsUp-Regulationrespiratory tract diseasesInterleukin-2 Receptor beta SubunitInterleukin 10ImmunologyFemaleInterleukin 17Bronchial HyperreactivitybusinessImmunologic MemoryCD8medicine.drugJournal of Allergy and Clinical Immunology
researchProduct

The effect of cyclosporin A, FK506 and rapamycin on the murine contact sensitivity reaction

1998

We have evaluated the effects of three potent immunosuppressive agents, cyclosporin A (CsA), FK506 and rapamycin, on the murine contact sensitivity (CS) reaction to the hapten trinitrochlorobenzene. Development of CS reaction requires participation of three distinct T cell subsets: alphabeta+, CD4+ T lymphocytes, which are the classical effector cell of the CS reaction, gammadelta+ T lymphocytes, and alphabeta+, double-negative (CD4- CD8-) T lymphocytes that express the B220 molecule and produce IL-4. We found that all three drugs inhibit the development of the CS reaction, but they affect different target cells. In fact, rapamycin and FK-506 block both alphabeta+, CD4+ and gammadelta+ T ly…

MaleInterleukin 2Cellular immunityReceptors Antigen T-Cell alpha-betamedicine.medical_treatmentT cellImmunologyPicryl ChloridePolyenesBiologyDermatitis ContactLymphocyte ActivationTacrolimusMiceImmune systemAntigenT-Lymphocyte SubsetsCyclosporin amedicineAnimalsImmunology and AllergySirolimusReceptors Antigen T-Cell gamma-deltaOriginal ArticlesT lymphocyteCytokinemedicine.anatomical_structureImmunologyCyclosporineMice Inbred CBAImmunosuppressive Agentsmedicine.drugClinical and Experimental Immunology
researchProduct

Therapeutic implications of low lymphocyte count in non-ST segment elevation acute coronary syndromes

2009

Abstract Background Low lymphocyte count (LLC), a surrogate for inflammation, has emerged as a potential risk factor for cardiovascular outcomes, especially new ischemic events. To identify patients with non-ST segment elevation acute coronary syndromes (NSTEACS) who benefit from an invasive revascularization strategy remains a challenge. We sought to determine if patients with high-risk NSTEACS who exhibited LLC have a greater reduction in long-term post-discharge myocardial infarction (MI) when managed under a revascularization invasive strategy (RIS) as compared with conservative strategy (CS). Methods Nine hundred seventy two consecutive patients with high-risk NSTEACS were treated unde…

MaleInvasive strategymedicine.medical_specialtyLymphocytemedicine.medical_treatmentMyocardial InfarctionRevascularizationElectrocardiographyRisk FactorsLymphopeniaInternal medicineMyocardial RevascularizationInternal MedicineHumansMedicineST segmentLymphocyte CountMyocardial infarctionAcute Coronary SyndromeLow lymphocyte countAgedProportional Hazards ModelsRetrospective Studiesbusiness.industryProportional hazards modelPrognosismedicine.diseaseTreatment Outcomemedicine.anatomical_structureQuartileCardiologyFemalebusinessEuropean Journal of Internal Medicine
researchProduct

Conditions for the Enhancing Effect of Protease Inhibitors on the Concanavalin A Induced Thymidine Response of Murine Lymphocytes

1983

Incorporation of [<sup>3</sup>H]-thymidine – [<sup>3</sup>H]-TdR – into concanavalin A (Con A) stimulated murine splenocytes and thymocytes was found to be enhanced by addition of certain concentrations of phenyl-methyl-sulfonylfluoride (PMSF), di-isopropylfluorophosphate (DFP), N-α-tosyl-<i>L</i>-lysyl-<i>L</i>-chloromethylketone (TLCK), and soybean trypsin inhibitor (SBTI). No enhancement could be observed when mononuclear cells of the peripheral blood were used, and a medium enhancement when thymocytes were applied. Furthermore, no enhancing effect of the protease inhibitors (PI) on the Con A response of murine splenocytes could be observed…

MaleIsoflurophateTime Factorsmedicine.medical_treatmentImmunologyBiologyLymphocyte ActivationTosyllysine Chloromethyl KetoneMicechemistry.chemical_compoundPhagocytosisConcanavalin AmedicineAnimalsImmunology and AllergyProtease InhibitorsMice Inbred BALB CProteaseDose-Response Relationship DrugMurine splenocytesDrug SynergismGeneral MedicineMolecular biologyKineticschemistryBiochemistryConcanavalin AImmunologybiology.proteinThymidineSpleenThymidineInternational Archives of Allergy and Immunology
researchProduct

Extended analysis of a genome-wide association study in primary sclerosing cholangitis detects multiple novel risk loci

2012

Background & Aims: A limited number of genetic risk factors have been reported in primary sclerosing cholangitis (PSC). To discover further genetic susceptibility factors for PSC, we followed up on,a second tier of single nucleotide polymorphisms (SNPs) from a genome-wide association study (GWAS). Methods: We analyzed 45 SNPs in 1221 PSC cases and 3508 controls. The association results from the replication analysis and the original GWAS (715 PSC cases and 2962 controls) were combined in a meta-analysis comprising 1936 PSC cases and 6470 controls. We performed an analysis of bile microbial community composition in 39 PSC patients by 16S rRNA sequencing. Results: Seventeen SNPs representing 1…

MaleLinkage disequilibriumendocrine system diseasesGenome-wide association studyPrimary biliary cirrhosisGenotypeBLOOD-GROUPBileChildPOPULATIONAged 80 and overGeneticseducation.field_of_studyPrimary sclerosing cholangitisdigestive oral and skin physiologyMiddle AgedFucosyltransferasesChild PreschoolDISEASESFemaleNeprilysinReceptors Tumor Necrosis Factor Member 14B-LYMPHOCYTEAdultRiskGenome-wide association studyAdolescentGenotypeSUSCEPTIBILITY LOCIFUT2Cholangitis SclerosingPopulationT-LYMPHOCYTE ATTENUATORSingle-nucleotide polymorphismBiologyPolymorphism Single Nucleotidedigestive systemArticlePrimary sclerosing cholangitisGenetic predispositionmedicineImmunogeneticsHumansGenetic Predisposition to DiseaseeducationMETAANALYSISAgedNON-SECRETOR STATUSHepatologymedicine.diseaseGENEdigestive system diseasesSingle nucleotide polymorphismGenetic LociImmunology
researchProduct

Oncogene transformation can induce tolerogenicity in murine macrophages after down-regulation of immunogenicity without altering major histocompatibi…

1993

In vitro studies on cell lines may allow analyses of the mechanisms of immunogenicity and tolerogenicity in cells. We used a model of oncogenic transformation of an established murine macrophage cell line and report here that one v-mos-transformed clone expressing unaltered high amounts of MHC class I and II antigens does not induce proliferation of unprimed T cells in primary mixed lymphocyte reactions, in sharp contrast to its non-transformed parental cells. Interestingly, this clone induces specific unresponsiveness, as revealed by the lack of responsiveness of MHC-specific T cells when subsequently exposed to the pertinent MHC alloantigens in immunogenic form but unaltered MHC-third par…

MaleLymphocyteT-LymphocytesImmunologyClone (cell biology)Down-RegulationBiologyMajor histocompatibility complexImmune toleranceCell LineMiceTransformation GeneticAntigenHistocompatibility AntigensMHC class ImedicineImmune ToleranceAnimalsRNA MessengerGenes mosMice Inbred BALB CMice Inbred C3HImmunogenicityMacrophagesGeneral MedicineCell biologyClone CellsMice Inbred C57BLmedicine.anatomical_structureCell cultureImmunologybiology.proteinFemaleLymphocyte Culture Test MixedCell DivisionInterleukin-1Scandinavian journal of immunology
researchProduct

Human CD8+ T-cells Recognizing Peptides from Mycobacterium tuberculosis (Mtb) Presented by HLA-E Have an Unorthodox Th2-like, Multifunctional, Mtb In…

2015

Mycobacterial antigens are not exclusively presented to T-cells by classical HLA-class Ia and HLA-class II molecules, but also through alternative antigen presentation molecules such as CD1a/b/c, MR1 and HLA-E. We recently described mycobacterial peptides that are presented in HLA-E and recognized by CD8+ T-cells. Using T-cell cloning, phenotyping, microbiological, functional and RNA-expression analyses, we report here that these T-cells can exert cytolytic or suppressive functions, inhibit mycobacterial growth, yet express GATA3, produce Th2 cytokines (IL-4,-5,-10,-13) and activate B-cells via IL-4. In TB patients, Mtb specific cells were detectable by peptide-HLA-E tetramers, and IL-4 and…

MaleMacrophageQH301-705.5ImmunologyAntigen presentationBacterial ProteinMycobacterium tuberculosiHuman leukocyte antigenGATA3 Transcription FactorCD8-Positive T-LymphocytesMicrobiologyMicrobiologyMycobacterium tuberculosisImmune systemTh2 CellsGeneticHLA-EBacterial ProteinsVirologyGeneticsCytotoxic T cellHumansBiology (General)Th2 CellCytokineMolecular BiologyAntigen PresentationbiologyMacrophagesHistocompatibility Antigens Class ICD8-Positive T-LymphocyteMycobacterium tuberculosisRC581-607biology.organism_classificationBacterial Proteins; CD8-Positive T-Lymphocytes; Cytokines; Female; GATA3 Transcription Factor; Histocompatibility Antigens Class I; Humans; Macrophages; Male; Mycobacterium tuberculosis; Peptides; Th2 Cells; Antigen Presentation; Microbiology; Parasitology; Virology; Immunology; Genetics; Molecular BiologyPhenotypeVirology3. Good healthPeptideCytokinesParasitologyFemaleImmunologic diseases. AllergyPeptidesCD8HumanResearch ArticlePLoS Pathogens
researchProduct

Antigen-presenting cells of haematopoietic origin prime cytomegalovirus-specific CD8 T-cells but are not sufficient for driving memory inflation duri…

2011

Expansion of the CD8 T-cell memory pool, also known as ‘memory inflation’, for certain but not all viral epitopes in latently infected host tissues is a special feature of the immune response to cytomegalovirus. The Ld-presented murine cytomegalovirus (mCMV) immediate–early (IE) 1 peptide is the prototype of an epitope that is associated with memory inflation. Based on the detection of IE1 transcripts in latently infected lungs it was previously proposed that episodes of viral gene expression and antigenic activity due to desilencing of a limited number of viral genes may drive epitope-specific memory inflation. This would imply direct antigen presentation through latently infected host tis…

MaleMice Inbred BALB CMuromegalovirusbiologyAntigen presentationAntigen-Presenting CellsPriming (immunology)CD8-Positive T-LymphocytesVirologyEpitopeImmediate-Early ProteinsVirus LatencyEpitopesMiceImmune systemAntigenVirologyImmunologyMHC class Ibiology.proteinAnimalsCytotoxic T cellFemaleAntigen-presenting cellImmunologic MemoryJournal of General Virology
researchProduct