Search results for "Lymphocyte"

showing 10 items of 2280 documents

Regulatory T Cells and IL-10 Independently Counterregulate Cytotoxic T Lymphocyte Responses Induced by Transcutaneous Immunization

2011

Background: The imidazoquinoline derivate imiquimod induces inflammatory responses and protection against transplanted tumors when applied to the skin in combination with a cognate peptide epitope (transcutaneous immunization, TCI). Here we investigated the role of regulatory T cells (Treg) and the suppressive cytokine IL-10 in restricting TCI-induced cytotoxic T lymphocyte (CTL) responses. Methodology/Principal Findings: TCI was performed with an ointment containing the TLR7 agonist imiquimod and a CTL epitope was applied to the depilated back skin of C57BL/6 mice. Using specific antibodies and FoxP3-diphteria toxin receptor transgenic (DEREG) mice, we interrogated inhibiting factors after…

Mouselcsh:MedicineEpitopes T-LymphocyteAdaptive ImmunityT-Lymphocytes RegulatoryImmune toleranceMiceMedicineCytotoxic T celllcsh:ScienceImmune ResponseSkinMice KnockoutB-LymphocytesMultidisciplinaryImiquimodFOXP3hemic and immune systemsForkhead Transcription FactorsAnimal ModelsFlow CytometryInterleukin-10Interleukin 10medicine.anatomical_structureAminoquinolinesCytokinesIntercellular Signaling Peptides and ProteinsImmunotherapyResearch ArticleHeparin-binding EGF-like Growth FactorT cellImmune CellsImmunologychemical and pharmacologic phenomenaImmune SuppressionImmunomodulationImmune systemModel OrganismsImmune ToleranceAnimalsBiologyB cellbusiness.industrylcsh:RImmunityMice Inbred C57BLCTL*Immune SystemImmunologyImmunologic Techniqueslcsh:QImmunizationbusinessT-Lymphocytes CytotoxicPLoS ONE
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Unraveling the T-B tangle in anti-CD20 multiple sclerosis therapy.

2019

Significance Multiple sclerosis (MS) is an inflammatory demyelinating disease of the central nervous system. CD8+ T cells have been strongly implicated in MS pathogenesis, but it is unclear whether myelin is a CD8+ T cell autoantigenic target in MS. This study demonstrated that while myelin-specific CD8+ T cells are present at similar frequencies in untreated MS patients and healthy subjects, the proportion of memory and CD20-expressing myelin-specific CD8+ T cells was increased in MS patients, suggesting prior antigen encounter. This activated phenotype was reversible as the memory and CD20-expressing populations of certain myelin-specific CD8+ T cells were reduced following anti-CD20 trea…

Multiple SclerosisCentral nervous systemAxonal lossDiseaseCD8-Positive T-LymphocytesCD8+ T cellsanti-CD20 therapy03 medical and health sciences0302 clinical medicineImmune systemImmunology and InflammationAntigenmedicineHumansMyelin SheathMultidisciplinarybusiness.industryMultiple sclerosisExperimental autoimmune encephalomyelitisBiological Sciencesmedicine.diseaseAntigens CD20medicine.anatomical_structureImmunizationImmunologybusinessmyelin antigen030217 neurology & neurosurgery030215 immunologyProceedings of the National Academy of Sciences of the United States of America
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Treatment of multiple sclerosis with rituximab: A multicentric Italian–Swiss experience

2020

Background: Rituximab, an anti-CD20 monoclonal antibody leading to B lymphocyte depletion, is increasingly used as an off-label treatment option for multiple sclerosis (MS). Objective: To investigate the effectiveness and safety of rituximab in relapsing–remitting (RR) and progressive MS. Methods: This is a multicenter, retrospective study on consecutive MS patients treated off-label with rituximab in 22 Italian and 1 Swiss MS centers. Relapse rate, time to first relapse, Expanded Disability Status Scale (EDSS) progression, incidence of adverse events, and radiological outcomes from 2009 to 2019 were analyzed. Results: A total of 355/451 enrolled subjects had at least one follow-up visit an…

Multiple Sclerosismedicine.drug_classLymphocyte depletionrelapsing–remittingMonoclonal antibodyPrimary progressive03 medical and health sciences0302 clinical medicineMultiple Sclerosis Relapsing-Remittingreal lifemedicineHumansImmunologic Factors030212 general & internal medicineSecondary progressiveRetrospective Studiesprimary progressivebusiness.industryMultiple sclerosisRituximab; multiple sclerosis; primary progressive; real life; relapsing–remitting; secondary progressiveTreatment optionsmedicine.diseaseNeurologyRelapsing remittingItalymultiple sclerosiImmunologysecondary progressiveRituximabSettore MED/26 - NeurologiaNeurology (clinical)businessRituximab030217 neurology & neurosurgerySwitzerlandmedicine.drug
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Two Antigenic Peptides from Genes m123 and m164 of Murine Cytomegalovirus Quantitatively Dominate CD8 T-Cell Memory in the H-2 d Haplotype

2001

ABSTRACT The importance of CD8 T cells for the control of cytomegalovirus (CMV) infection has raised interest in the identification of immunogenic viral proteins as candidates for vaccination and cytoimmunotherapy. The final aim is to determine the viral “immunome” for any major histocompatibility complex class I molecule by antigenicity screening of proteome-derived peptides. For human CMV, there is a limitation to this approach: the T cells used as responder cells for peptide screening are usually memory cells that have undergone in vivo selection. On this basis, pUL83 (pp65) and pUL123 (IE1 or pp68 to -72) were classified as immunodominant proteins. It is an open question whether this li…

MuromegalovirusAdoptive cell transferAntigenicityImmunologyCD8-Positive T-LymphocytesBiologymedicine.disease_causeMajor histocompatibility complexMicrobiologyImmediate-Early ProteinsViral Matrix ProteinsMiceOpen Reading FramesViral ProteinsImmune systemVirologymedicineAnimalsCytotoxic T cellAntigens ViralMice Inbred BALB CH-2 AntigensCytomegalovirusHerpesviridae InfectionsPhosphoproteinsVirologyPeptide FragmentsHaplotypesInsect ScienceProteomeImmunologybiology.proteinPathogenesis and ImmunityFemaleImmunologic MemorySpleenCD8Journal of Virology
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Control of murine cytomegalovirus in the lungs: Relative but not absolute immunodominance of the immediate-early 1 nonapeptide during the antiviral c…

1998

Effective control by the immune system is a hallmark of cytomegalovirus (CMV) infection. Accordingly, human CMV disease is a medical problem restricted to the immunologically immature or immunocompromised host (for a review, see reference 21). Murine models have implicated natural killer (NK) cells and CD8 T cells in the control of CMV infection. While NK cells mediate early protection in genetically resistant mouse inbred strains (4, 5, 31, 51), CD8 T cells establish enduring protective memory and function as principal antiviral effectors in susceptible strains (31). Specifically, in the BALB/c strain, major histocompatibility complex (MHC) class I-restricted antiviral CD8 T cells resolve …

MuromegalovirusAdoptive cell transferImmunologyViral Pathogenesis and ImmunityBone Marrow CellsImmunodominanceVirus ReplicationMajor histocompatibility complexMicrobiologyImmediate-Early ProteinsMiceImmune systemAntigenVirologyMHC class IAnimalsCytotoxic T cellLungAntigen PresentationMice Inbred BALB CbiologyImmunodominant EpitopesAntigen processingvirus diseasesHerpesviridae InfectionsVirologyKineticsInsect ScienceImmunologyTrans-Activatorsbiology.proteinFemaleT-Lymphocytes Cytotoxic
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Adoptive CD8 T Cell Control of Pathogens Cannot Be Improved by Combining Protective Epitope Specificities

2008

Adoptive transfer of CD8 T cells has the potential to cure infectious or malignant diseases that are refractory to conventional chemotherapy. A practically important but still unanswered question is whether mixtures of protective CD8 T cells with different epitope specificities mediate more efficient effector cell functions than do the monospecific individual CD8 T cell populations. In this study, we have addressed this issue for models of viral and bacterial infection. CD8 T cell-mediated cytotoxicity in vitro and protection in vivo were assessed to test whether CD8 T cell lines cooperate in target cell lysis and control of infection, respectively. Our data clearly show that mixtures of cy…

MuromegalovirusAdoptive cell transferT cellEpitopes T-LymphocyteBacteremiaStreptamerCD8-Positive T-LymphocytesBiologyEpitopeMicemedicineAnimalsImmunology and AllergyCytotoxic T cellViremiaAntigen-presenting cellT lymphocyteAdoptive TransferListeria monocytogenesVirologyDisease Models AnimalInfectious Diseasesmedicine.anatomical_structureCytomegalovirus InfectionsImmunologyFemaleCD8The Journal of Infectious Diseases
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In vivo impact of cytomegalovirus evasion of CD8 T-cell immunity: Facts and thoughts based on murine models

2010

Cytomegaloviruses (CMVs) co-exist with their respective host species and have evolved to avoid their elimination by the hosts' immune effector mechanisms and to persist in a non-replicative state, known as viral latency. There is evidence to suggest that latency is nevertheless a highly dynamic condition during which episodes of viral gene desilencing, which can be viewed as incomplete reactivations, cause intermittent antigenic activity that stimulates CD8 memory-effector T cells and drives their clonal expansion. These T cells are supposed to terminate reactivation before completion of the productive viral cycle. In this view, CMVs do not "evade" their respective host's immune response bu…

MuromegalovirusCancer ResearchT cellAntigen presentationReceptors Antigen T-CellCytomegalovirusCD8-Positive T-LymphocytesBiologyMiceImmune systemAntigenVirologyVirus latencymedicineAntigenic variationAnimalsCytotoxic T cellViral InterferenceImmune EvasionAntigen PresentationHistocompatibility Antigens Class IHerpesviridae Infectionsmedicine.diseaseVirologyVirus LatencyDisease Models AnimalInfectious Diseasesmedicine.anatomical_structureCytomegalovirus InfectionsImmunologyVirus ActivationVirus Research
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The Putative Natural Killer Decoy Early Genem04(gp34) of Murine Cytomegalovirus Encodes an Antigenic Peptide Recognized by Protective Antiviral CD8 T…

2000

ABSTRACTSeveral early genes of murine cytomegalovirus (MCMV) encode proteins that mediate immune evasion by interference with the major histocompatibility complex class I (MHC-I) pathway of antigen presentation to cytolytic T lymphocytes (CTL). Specifically, them152gene product gp37/40 causes retention of MHC-I molecules in the endoplasmic reticulum (ER)-Golgi intermediate compartment. Lack of MHC-I on the cell surface should activate natural killer (NK) cells recognizing the “missing self.” The retention, however, is counteracted by them04early gene product gp34, which binds to folded MHC-I molecules in the ER and directs the complex to the cell surface. It was thus speculated that gp34 mi…

MuromegalovirusGenes ViralImmunologyAntigen presentationchemical and pharmacologic phenomenaGenome ViralCD8-Positive T-LymphocytesMajor histocompatibility complexMicrobiologyImmediate-Early ProteinsGene productMiceViral ProteinsImmune systemAntigenPeptide LibraryVirologyAnimalsCytotoxic T cellHistocompatibility Antigen H-2DAntigens ViralCells CulturedGlycoproteinsMice Inbred BALB CMembrane GlycoproteinsbiologyHistocompatibility Antigens Class IH-2 AntigensVirologyKiller Cells NaturalCTL*Insect Sciencebiology.proteinPathogenesis and ImmunityFemaleCarrier ProteinsPeptidesCD8T-Lymphocytes CytotoxicJournal of Virology
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Processing and Presentation of Murine Cytomegalovirus pORFm164-Derived Peptide in Fibroblasts in the Face of All Viral Immunosubversive Early Gene Fu…

2002

ABSTRACTCD8 T cells are the principal effector cells in the resolution of acute murine cytomegalovirus (mCMV) infection in host organs. This undoubted antiviral and protective in vivo function of CD8 T cells appeared to be inconsistent with immunosubversive strategies of the virus effected by early (E)-phase genesm04,m06, andm152. The so-called immune evasion proteins gp34, gp48, and gp37/40, respectively, were found to interfere with peptide presentation at different steps in the major histocompatibility complex (MHC) class I pathway of antigen processing and presentation in fibroblasts. Accordingly, they were proposed to prevent recognition and lysis of infected fibroblasts by cytolytic T…

MuromegalovirusImmunologyAntigen presentationMajor histocompatibility complexMicrobiologyImmediate-Early ProteinsMiceOpen Reading FramesViral ProteinsImmune systemAntigenVirologyMHC class IAnimalsCytotoxic T cellAntigens ViralGenes Immediate-EarlyCells CulturedAntigen PresentationMice Inbred BALB CMembrane GlycoproteinsbiologyAntigen processingFibroblastsVirologyPeptide FragmentsCTL*Insect Sciencebiology.proteinPathogenesis and ImmunityFemaleT-Lymphocytes CytotoxicJournal of Virology
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The Immune Evasion Paradox: Immunoevasins of Murine Cytomegalovirus Enhance Priming of CD8 T Cells by Preventing Negative Feedback Regulation▿

2008

ABSTRACTCytomegaloviruses express glycoproteins that interfere with antigen presentation to CD8 T cells. Although the molecular modes of action of these “immunoevasins” differ between cytomegalovirus species, the convergent biological outcome is an inhibition of the recognition of infected cells. In murine cytomegalovirus, m152/gp40 retains peptide-loaded major histocompatibility complex class I molecules in acis-Golgi compartment, m06/gp48 mediates their vesicular sorting for lysosomal degradation, and m04/gp34, although not an immunoevasin in its own right, appears to assist in the concerted action of all three molecules. Using the Ld-restricted IE1 epitope YPHFMPTNL in the BALB/c mouse m…

MuromegalovirusImmunologyAntigen presentationPriming (immunology)Genome ViralBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexVirus ReplicationMicrobiologyEpitopeImmediate early proteinImmediate-Early ProteinsEpitopesMiceViral ProteinsImmune systemAntigenVirologyCytotoxic T cellAnimalsAntigen PresentationMice Inbred BALB CHerpesviridae InfectionsKiller Cells NaturalInsect ScienceImmunologybiology.proteinPathogenesis and ImmunityFemaleLymph NodesImmunologic MemorySpleen
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