Search results for "Lymphocyte"

showing 10 items of 2280 documents

Tumor cell specific toxicity of Inula helenium extracts.

2006

The aim of the research program was to identify botanical extracts with antineoplastic activity. In this respect extracts prepared from Inula helenium roots showed a remarkable activity. As evidenced by the MTT assay, the Inula helenium extract revealed a highly selective toxicity toward four different tumor cell lines (HT-29, MCF-7, Capan-2 and G1), but a much lower toxicity against healthy human peripheral blood lymphocytes (PBLs) from two donors. The extract-induced death of tumor cells was studied extensively by electron microscopy. There was a remarkable similarity of morphological alterations observed in the four cell lines: patchy chromatin condensations, cytoplasmic vesiculation, sw…

Programmed cell deathCell SurvivalContext (language use)Plant RootsLethal Dose 50Cell Line TumorToxicity Tests AcuteHumansMTT assayLymphocytesAnnexin A5CytotoxicityPharmacologyInulabiologyMutagenicity TestsPlant Extractsbiology.organism_classificationMolecular biologyAntineoplastic Agents PhytogenicApoptosisCell cultureImmunologyInulaHT29 CellsHeleniumPhytotherapy research : PTR
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Molecular mechanisms of rosmarinic acid from Salvia miltiorrhiza in acute lymphoblastic leukemia cells

2015

Abstract Ethnopharmacological relevance Rosmarinic acid (RA), a major hydrosoluble bioactive compound found in the Chinese medicinal herb, Salvia miltiorrhiza Bunge, which has been used in traditional Chinese medicine to treat various diseases, including cancer. However, the mechanisms have not been fully elucidated. Aim of the study Guided by microarray hybridization and Ingenuity Pathway Analysis, we identified modes of action of rosmarinic acid (RA) isolated from S. miltiorrhiza on acute lymphoblastic leukemia cells. Materials and methods Microarray data were verified by independent methods: Real-time RT-PCR (mRNA expression), resazurin assay (cytotoxicity of RA towards parental CCRF-CEM…

Programmed cell deathCell SurvivalDNA damageNecroptosisCellAntineoplastic AgentsApoptosisSalvia miltiorrhizaPharmacologyCell morphologyDepsidesSalvia miltiorrhizaCell Line TumorDrug DiscoveryCell AdhesionmedicineHumansLymphocytesCells CulturedMembrane Potential MitochondrialPharmacologybusiness.industryGene Expression ProfilingCell CycleNF-kappa BPrecursor Cell Lymphoblastic Leukemia-LymphomaCell cycleMolecular biologyDrug Resistance MultipleMolecular Docking Simulationmedicine.anatomical_structureCinnamatesDrug Resistance NeoplasmApoptosisComet AssayReactive Oxygen SpeciesbusinessDNA DamageJournal of Ethnopharmacology
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Toxicity of a dental adhesive compared with ionizing radiation and zoledronic acid

2015

Background: To determine the toxicity of aqueous dilutions of a universal self-priming dental adhesive (DA) and comparing these with those elicited by exposure to ionizing radiation (IR), Zoledronic acid (Z) treatment and the synergic effects of the combined treatment with IR+Z. Material and Methods: The genotoxic effect of DA was determined by the increase in the frequency of micronuclei in cytokinesis-blocked in cultured human lymphocytes before and after exposure to 2Gy of X-rays. The cytotoxic effect was studied by using the MTT cell viability test in normal prostate cell lines (PNT2) after exposure to different X-ray doses (0Gy-20Gy). The cell lines divided into different groups and tr…

Programmed cell deathDental CementsOdontologíaPharmacologymedicine.disease_causeZoledronic AcidIonizing radiationToxicologyPolymethacrylic AcidsRadiation IonizingToxicity TestsmedicineCytotoxic T cellHumansViability assayLymphocytesGeneral DentistryCells CulturedOral Medicine and PathologyDiphosphonatesChemistryResearchImidazoles:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludIn vitroOtorhinolaryngologyToxicityMicronucleus testUNESCO::CIENCIAS MÉDICASSurgeryGenotoxicity
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Natural and induced apoptosis during lymphocyte development in the axolotl

1999

Lymphocytes apoptosis was characterized in a urodele amphibian, the axolotl, by morphology using electron microscopy and by flow cytometry after propidium iodide staining, as well as by biochemical criteria with the detection of DNA ladders after glucocorticoid treatment. The morphological and biochemical features observed in treated axolotls are in accordance with the criteria of apoptosis found in different models of mammalian lymphocyte programmed cell death. The onset of natural apoptosis was then detected by DNA fragmentation in thymus and in spleen during lymphocyte development and ontogenesis. A typical DNA ladder characteristic of apoptosis is detectable in the thymus as early as 5 …

Programmed cell deathHydrocortisoneT-LymphocytesLymphocyteImmunologyApoptosisBiologyAmbystomaFlow cytometryEnterotoxinschemistry.chemical_compoundAxolotlmedicineSuperantigenAnimalsLymphocytesPropidium iodideSuperantigensmedicine.diagnostic_testCell Differentiationbiology.organism_classificationMolecular biologymedicine.anatomical_structurechemistryApoptosisLarvaDNA fragmentationDevelopmental BiologyDevelopmental & Comparative Immunology
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Involvement of NO in contact hypersensitivity.

1998

The NO synthases (NOS) generate NO from L-arginine. High concentrations of NO have been shown to be responsible for tissue injury and cell death, while low concentrations of NO induce vasodilatation and other signaling effects. We have investigated the involvement of NO in contact hypersensitivity (CHS) reactions. CHS induced by treatment of BALB/c mice with the contact allergen 2,4-dinitrofluorobenzene (DNFB) was significantly reduced by the NOS inhibitor N-methyl-L-arginine (L-NMA), but not by the stereoisomer D-NMA, as shown by reduced ear swelling responses and evaluation of ear tissue sections. The CHS response was also reduced by aminoguanidine, which is known to preferentially inhibi…

Programmed cell deathLangerhans cellArginineInjections IntradermalT-LymphocytesImmunologyNitric Oxide Synthase Type IIBiologyArginineDermatitis ContactNitric OxideGuanidineschemistry.chemical_compoundMicemedicineImmunology and AllergyAnimalsRNA MessengerEnzyme InhibitorsSkinMice Inbred BALB Cintegumentary systemEpidermis (botany)Histocompatibility Antigens Class IIGeneral MedicineAllergensMolecular biologyPimagedineNitric oxide synthasemedicine.anatomical_structurechemistryLangerhans Cellsbiology.proteinDinitrofluorobenzeneSignal transductionNitric Oxide SynthaseKeratinocyteHaptensInternational immunology
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Apoptosis of T cells and the control of inflammatory bowel disease: therapeutic implications.

2007

Inflammatory bowel diseases (IBDs) such as Crohn’s disease and ulcerative colitis are the result of an imbalanced mucosal T cell response. Despite the identification of a genetic susceptibility region in the NOD2/CARD15 (nucleotide-binding oligomerisation domain 2/caspase recruitment domain 15) gene, the aetiology is still unclear. Thus, the hunt for disease-initiating factors such as defects in the mucosal barrier or pathogenic microorganisms is ongoing. By contrast, the immunopathogenesis in IBDs is better understood. The identification of cytokines that are involved in T cell and monocyte signalling led to specific therapeutic concepts. Recent data have clearly shown that the most powerf…

Programmed cell deathNecrosisCell Survivalmedicine.medical_treatmentT cellT-LymphocytesApoptosisImmune systemCrohn DiseaseNOD2AzathioprinemedicineHumansIntestinal MucosaMesalamineImmunity Mucosalbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaAnti-Inflammatory Agents Non-SteroidalGastroenterologyRecent Advances in Basic ScienceInflammatory Bowel DiseasesInterleukin-12Immunosuppressive drugmedicine.anatomical_structureApoptosisImmunologyTumor necrosis factor alphamedicine.symptombusinessImmunosuppressive AgentsSignal TransductionGut
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Selective targeting of activated T cells in chronic intestinal inflammation

2009

Programmed cell death (apoptosis) has been implicated in normal biological processes as well as in the pathology of human diseases.1 The characterisation of genes involved in apoptosis has been pursued intensively and led to the identification of two major classes of genes: the bcl-2 family and the caspase family. Caspases are proteases that cleave their target substrates at specific peptide sequences and during apoptosis the activation of caspases takes place in a cascade fashion, leading to nuclear engulfment and cell death. Thus, caspases represent key functional components of the apoptosis pathway in human cells. Resistance against apoptosis is a key phenomenon in various chronic inflam…

Programmed cell deathRecombinant Fusion ProteinsT-LymphocytesT cellApoptosisLymphocyte ActivationProinflammatory cytokineImmune systemmedicineAnimalsHumansIntestinal MucosaCaspasebiologyCaspase 3Intrinsic apoptosisGastroenterologyColitisCell biologymedicine.anatomical_structureApoptosisChronic DiseaseModels Animalbiology.proteinInterleukin-2Tumor necrosis factor alphaGut
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Selective loss of regulatory T cells in thymomas

2004

Myasthenia gravis (MG) is the prime autoimmune manifestation of thymomas. We investigated the generation of T cells with a regulatory phenotype (T(R)) in thymomas with and without associated MG. In patients with MG(+) thymomas, maturation and export of T(R) cells but not of other T-cell subsets was significantly reduced. We conclude that imbalance between effector and regulatory T cells in thymomas may be involved in modulation of onset and/or severity of MG.

Programmed cell deathThymomabusiness.industryEffectorCellular differentiationchemical and pharmacologic phenomenaT lymphocytemedicine.diseasePhenotypeClonal deletionMyasthenia gravissurgical procedures operativeNeurologyhemic and lymphatic diseasesImmunologyCancer researchMedicineNeurology (clinical)businessneoplasmsAnnals of Neurology
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Processing without proteolytic cleavage is required for recognition of insulin by T cells.

1990

Beef insulin as well as a chymotryptic A-chain fragment [BI-A1-14(SSO3-)3] need uptake by antigen-presenting cells (APC) for efficient presentation in combination with major histocompatibility complex class II molecules to insulin-specific T cells. This could be shown by the inability of aldehyde-fixed APC to present these antigens to T cells. Furthermore, presentation of the insulin fragment as well as presentation of ovalbumin (OVA) was inhibited by treatment of APC with chloroquine, cerulenin or tunicamycin. This was not the case for a processing-independent OVA peptide. Treatment of APC during antigen pulsing with various protease inhibitors, active on all classes of proteases, did not …

ProteasesOvalbuminmedicine.medical_treatmentT-LymphocytesImmunologyAntigen presentationAntigen-Presenting CellsBiologyIn Vitro TechniquesEpitopeCell Linechemistry.chemical_compoundMiceAntigenEndopeptidasesmedicineImmunology and AllergyAnimalsInsulinProtease InhibitorsAntigen-presenting cellProteaseInsulinTunicamycinChloroquineTunicamycinEndocytosischemistryBiochemistryEuropean journal of immunology
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Detection of proteolytic (C 3-cleaving) activity on mouse mastocytoma (P815) cells and other mouse cell lines by formation of cell contact with C 3-c…

1979

Mouse mastocytoma cells (P 815) formed rosettes with normal mouse spleen lymphocytes which had been coated with uncleaved human C 3; this interaction was clearly dependent on the amount of C 3. Lymphocytes treated with C 3 b or buffer alone were ineffective. Formation of cell contact could be inhibited by the presence of protease inhibitors such as diisopropyl fluorophosphate, phenyl methyl sulfonyl fluoride and tosyllysyl chloromethyl ketone. Seve n out of 13 different cell lines behaved like P 815 cells. The results strongly suggested that a proteolytic activity on mouse tumor cells led to a cooperation with uncleaved C 3 on a carrier cell to connect these two cells. We interpreted these …

ProteasesRosette FormationImmunologyCellMast-Cell SarcomaCell CountBiologyCleavage (embryo)Cell LineCell membraneMicemedicineAnimalsImmunology and AllergyProtease InhibitorsLymphocytesCell MembraneMastocytomaComplement C3medicine.diseaseMolecular biologymedicine.anatomical_structureCell cultureMast cell sarcomaDiisopropyl fluorophosphateSpleenmedicine.drugEuropean Journal of Immunology
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