Search results for "Lymphocyte"

showing 10 items of 2280 documents

Blockade of interleukin 6 trans signaling suppresses T-cell resistance against apoptosis in chronic intestinal inflammation: Evidence in Crohn diseas…

2000

The pro-inflammatory cytokine interleukin (IL)-6 (refs. 1-5) can bind to cells lacking the IL-6 receptor (IL-6R) when it forms a complex with the soluble IL-6R (sIL-6R) (trans signaling). Here, we have assessed the contribution of this system to the increased resistance of mucosal T cells against apoptosis in Crohn disease (CD), a chronic inflammatory disease of the gastrointestinal tract. A neutralizing antibody against IL-6R suppressed established experimental colitis in various animal models of CD mediated by type 1 T-helper cells, by inducing apoptosis of lamina propria T cells. Similarly, specific neutralization of sIL-6R in vivo by a newly designed gp130-Fc fusion protein caused suppr…

AdultMaleSTAT3 Transcription FactorT-Lymphocytesmedicine.medical_treatmentT cellbcl-X ProteinApoptosisGeneral Biochemistry Genetics and Molecular BiologyMiceCrohn DiseaseAntigenAntigens CDCytokine Receptor gp130medicineAnimalsHumansInterleukin 6Mice Inbred BALB CMembrane GlycoproteinsbiologyInterleukin-6Models ImmunologicalInterleukinGeneral MedicineMiddle AgedReceptors Interleukin-6DNA-Binding ProteinsCytokinemedicine.anatomical_structureProto-Oncogene Proteins c-bcl-2ApoptosisImmunologyTrans-Activatorsbiology.proteinSTAT proteinCancer researchColitis UlcerativeFemaleSignal transductionProtein BindingSignal TransductionNature Medicine
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Synovial fluid-derivedYersinia-reactive T cells responding to human 65-kDa heat-shock protein and heat-stressed antigen-presenting cells

1991

Humoral and cellular immune reactions to heat-shock proteins have been implicated in the pathogenesis of arthritis. Heat-shock proteins occur in bacteria as well as all eukaryotes and have been highly conserved during evolution. Cross-reactivity between bacterial and human heat-shock proteins induced at the site of inflammation may underlie the pathogenesis of some forms of arthritis. In order to test this hypothesis, we raised and cloned a Yersinia-specific T cell line from the synovial fluid lymphocytes of a patient with Yersinia-induced reactive arthritis. From this line we obtained a CD4+ T cell clone that proliferated in response to Yersinia antigens and both to the mycobacterial and t…

AdultMaleSalmonella typhimuriumHot TemperatureT-LymphocytesT cellImmunologyDose-Response Relationship ImmunologicAntigen-Presenting CellsArthritisCross ReactionsBiologyArthritis ReactiveImmune systemTetanus ToxinAntigenHeat shock proteinCandida albicansSynovial FluidEscherichia colimedicineHumansImmunology and AllergySynovial fluidAntigen-presenting cellHeat-Shock ProteinsT lymphocytebeta-Galactosidasemedicine.diseaseYersiniaCell biologymedicine.anatomical_structureImmunologyEuropean Journal of Immunology
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Salmonella-reactive Synovial Fluid T-cell Clones in a Patient with Post-infectious Salmonella Arthritis

1990

From a patient with reactive arthritis following Salmonella typhimurium enteritis, synovial fluid T-lymphocytes were cloned and expanded in vitro. Seven out of 74 clones showed a marked proliferative response to antigens of heat-killed Salmonella typhimurium with autologous T-cell-depleted peripheral blood mononuclear cells as antigen-presenting cells. The Salmonella-reactive clones were of the CD4+ phenotype, antigen-induced proliferation could be inhibited by a monoclonal antibody to HLA class II. One clone recognized both Salmonella and Campylobacter jejuni antigens in the proliferation assay. The multiclonality of Salmonella-reactive synovial fluid T-cells indicates that the microorgani…

AdultMaleSalmonella typhimuriumSalmonellaT-LymphocytesT cellImmunologyArthritismedicine.disease_causeCampylobacter jejuniPeripheral blood mononuclear cellMicrobiologyRheumatologyAntigenSalmonellaSynovitisSynovial FluidmedicineHumansImmunology and AllergySynovial fluidBlood CellsbiologyArthritisGeneral Medicinebiology.organism_classificationmedicine.diseaseVirologyClone Cellsmedicine.anatomical_structureSalmonella InfectionsCell DivisionScandinavian Journal of Rheumatology
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Generation of cytotoxic T-cell responses with synthetic melanoma-associated peptidesin vivo: Implications for tumor vaccines with melanoma-associated…

1996

Peptide epitopes derived from differentiation antigens of the melanocyte lineage have been identified in human melanomas and normal cultured melanocytes as targets for MHC-restricted cytotoxic T lymphocytes (CTL). Characterization of multiple CTL-defined antigenic determinants and the presence of corresponding precursor CTL open perspectives for the development of antigen-based vaccines. In the present study, we determined the CTL reactivity against melanoma-associated peptides derived from Melan A/MART-1, tyrosinase and gp100/Pmel17 in 10 HLA-A2+ melanoma patients and 10 healthy individuals. Then, we examined the immunological effects and toxicity of intradermal inoculation of synthetic me…

AdultMaleSignal peptideCancer ResearchInjections IntradermalMolecular Sequence DataTyrosinase Peptide10050 Institute of Pharmacology and Toxicology610 Medicine & healthchemical and pharmacologic phenomenaPeptideEpitopeImmune systemAntigenAntigens NeoplasmHumansCytotoxic T cellMedicine1306 Cancer ResearchHypersensitivity DelayedAmino Acid SequenceMelanomaCells CulturedAgedchemistry.chemical_classificationVaccinesbusiness.industryMiddle AgedNeoplasm ProteinsCTL*OncologychemistryImmunology570 Life sciences; biology2730 OncologyFemalebusinessMelanoma-Specific AntigensT-Lymphocytes CytotoxicInternational Journal of Cancer
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Genotoxicity of selected pesticides in the mouse bone-marrow micronucleus test and in the sister-chromatid exchange test with human lymphocytes in vi…

1996

Selected pesticides (aldicarb, 1,3-dichloropropene, methidathion, parathion, triadimefon, vinclozolin) were tested for their clastogenic and aneugenic activities in the mouse bone-marrow micronucleus (MN) test in vivo and for their sister-chromatid exchange-inducing activities in human lymphocytes in vitro in the presence and absence of an exogenous metabolizing system from rat-liver S9. 1,3-Dichloropropene significantly increased the frequencies of micronucleated polychromatic erythrocytes (PCE) in bone-marrow cells of female mice from 3.3 MN/1000 PCE to 15.3 MN/1000 PCE (187 mg per kg body weight). 1,3-Dichloropropene (100 microM) induced 16.0 SCE/metaphase after 24 h of incubation as com…

AdultMaleSister chromatid exchangeMethidathion010501 environmental sciencesBiologyToxicologymedicine.disease_cause01 natural sciencesMice03 medical and health scienceschemistry.chemical_compoundClastogenBone MarrowHydrocarbons ChlorinatedmedicineAnimalsHumansLymphocytesVinclozolinPesticidesOxazolesMetaphaseCells CulturedMicronuclei Chromosome-Defective030304 developmental biology0105 earth and related environmental sciences0303 health sciencesMicronucleus TestsParathionOrganothiophosphorus CompoundsGeneral MedicineTriazolesMolecular biology3. Good healthAllyl CompoundschemistryImmunologyMicronucleus testFemaleMicronucleusSister Chromatid ExchangeAldicarbGenotoxicityToxicology Letters
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NF-κB protects Behçet's disease T cells against CD95-induced apoptosis up-regulating antiapoptotic proteins

2005

Objective To determine whether prolongation of the inflammatory reaction in patients with Behcet's disease (BD) is related to apoptosis resistance and is associated with the up-regulation of antiapoptotic factors. Methods The percentage of cell death was evaluated by flow cytometry in peripheral blood mononuclear cells from 35 patients with BD and 30 healthy volunteers. The expression levels of antiapoptotic factors and NF-κB regulatory proteins were measured using Western blotting and immunohistochemical analyses. To down-regulate NF-κB nuclear translocation, BD T lymphocytes were exposed in vitro to thalidomide and subjected to transfection with NF-κB small interfering RNA. Results Althou…

AdultMaleSmall interfering RNAProgrammed cell deathT-LymphocytesT cellImmunologyCASP8 and FADD-Like Apoptosis Regulating Proteinbcl-X ProteinApoptosisCaspase 3TransfectionCaspase 8RheumatologyHumansImmunology and AllergyMedicinePharmacology (medical)fas ReceptorRNA Small InterferingCells CulturedDose-Response Relationship Drugbusiness.industryBehcet SyndromeIntracellular Signaling Peptides and ProteinsNF-kappa BTransfectionFlow CytometryFas receptorThalidomideUp-Regulationmedicine.anatomical_structureGene Expression RegulationProto-Oncogene Proteins c-bcl-2ApoptosisImmunologyLeukocytes MononuclearCancer researchFemalebusinessArthritis & Rheumatism
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Brief Report: Inhibition of interleukin-6 function corrects Th17/Treg cell imbalance in patients with rheumatoid arthritis

2012

OBJECTIVE: From an immunologic standpoint, the mechanisms by which treatment with tocilizumab (TCZ), a humanized anti-interleukin-6 (anti-IL-6) receptor antibody, results in improvement in rheumatoid arthritis (RA) patients are still not fully understood. In vitro studies and studies in mouse models have demonstrated the critical role of IL-6 in Th17 cell differentiation. Th17 lymphocytes have been shown to be strongly involved in RA pathogenesis, and the purpose of this study was to investigate the effect of IL-6 blockade on the balance between Th17 cells and Treg cells in patients with active RA. METHODS: Patients with active RA for whom TCZ had been prescribed by a rheumatologist were en…

AdultMaleT cellImmunologyArthritisCell Countchemical and pharmacologic phenomenaInflammationAntibodies Monoclonal HumanizedSeverity of Illness IndexT-Lymphocytes RegulatoryArthritis Rheumatoid03 medical and health scienceschemistry.chemical_compound0302 clinical medicineTocilizumabRheumatologymedicineHumansImmunology and AllergyPharmacology (medical)IL-2 receptorInterleukin 6030304 developmental biology030203 arthritis & rheumatology0303 health sciencesbiologyInterleukin-6business.industryFOXP3hemic and immune systemsMiddle Agedmedicine.diseaseReceptors Interleukin-63. Good healthPhenotypemedicine.anatomical_structurechemistryCase-Control StudiesRheumatoid arthritisImmunologybiology.proteinTh17 CellsFemalemedicine.symptombusinessArthritis & Rheumatism
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Bacteria-specific T-cell clones are selective in their reactivity towards different enterobacteria or H. pylori and increased in inflammatory bowel d…

1996

In the present study the authors investigated the T-cell response to different enterobacteria or Helicobacter pylori and tested the hypothesis that the frequency of bacteria-specific T cells is increased in the intestine of patients with active inflammatory bowel disease (IBD), i.e. Crohn's disease (CD) and ulcerative colitis (UC). The analysis of a large panel of T-cell clones (Tc) (n = 888) from peripheral blood, non-inflamed and inflamed intestine from IBD patients and control individuals shows that both peripheral blood and intestinal T-cell clones were selectively stimulated by either Salmonella typhimurium, Yersinia enterocolitica 03, Escherichia coli or Helicobacter pylori sonicates,…

AdultMaleT cellT-LymphocytesImmunologymedicine.disease_causeLymphocyte ActivationInflammatory bowel diseaseMicrobiologyAntigenEnterobacteriaceaeSpecies SpecificitymedicineSuperantigenHumansIntestinal MucosaEscherichia coliAgedbiologyHelicobacter pyloriGeneral MedicineHelicobacter pyloriMiddle Agedmedicine.diseasebiology.organism_classificationInflammatory Bowel DiseasesUlcerative colitisClone Cellsmedicine.anatomical_structureImmunologybiology.proteinLeukocytes MononuclearFemaleAntibodyScandinavian journal of immunology
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Cerebrospinal fluid T-regulatory cells recognize Borrelia burgdorferi NAPA in chronic Lyme borreliosis.

2013

The NapA protein of B. burgdorferi is essential for the persistence of spirochetes in ticks. One of the most intriguing aspects of NapA is its potential to interfere with the host immune system. Here, we investigated the role of the acquired immune responses induced by NapA in the cerebrospinal fluids (CSF) of patients with chronic Lyme borreliosis. We evaluated the cytokine profile induced in microglia cells and CSF T cells following NapA stimulation. We report here that NapA induced a regulatory T (Treg) response in the CSF of patients with chronic Lyme borreliosis and it is able to expand this suppressive response by promoting the production of TGF-β and IL-10 by microglia cells. Collect…

AdultMaleT regChemokineT-LymphocytesT cells; T reg; Borrelia; Lyme; Adult; Bacterial Proteins; Cerebrospinal Fluid; Chemokines CXC; Chronic Disease; Female; Humans; Interleukin-10; Lyme Disease; Male; Microglia; Middle Aged; T-Lymphocytes Regulatory; Transforming Growth Factor betaImmunologyT cellsT-Lymphocytes RegulatoryImmune systemLyme diseaseBacterial ProteinsTransforming Growth Factor betaImmunology and AllergyMedicineHumansBorrelia burgdorferiCerebrospinal FluidPharmacologyNAPACXCLyme DiseasebiologyMicrogliabusiness.industryBorreliaTransforming growth factor betaMiddle Agedbiology.organism_classificationmedicine.diseaseRegulatoryInterleukin-10Interleukin 10medicine.anatomical_structureImmunologyChronic Diseasebiology.proteinLymeFemaleMicrogliaChemokinesbusinessChemokines CXC
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The human hepatic asialoglycoprotein receptor is a target antigen for liver-infiltrating T cells in autoimmune chronic active hepatitis and primary b…

1990

Autoantibodies to the human hepatic asialoglycoprotein receptor have been found in nearly 50% of the sera of patients with autoimmune chronic active hepatitis and in 15% of patients with primary biliary cirrhosis. In this study we demonstrate that the human hepatic asialoglycoprotein receptor is also a target antigen for T cell-mediated immune responses. Peripheral blood lymphocytes of 37% (7 of 19) of patients with autoimmune chronic active hepatitis and 33% (2 of 6) of patients with primary biliary cirrhosis showed a proliferative response to highly purified human hepatic asialoglycoprotein receptor, whereas no proliferation was found with peripheral blood lymphocytes of patients with chr…

AdultMaleT-LymphocytesAsialoglycoprotein ReceptorLymphocyte Activationdigestive systemAutoantigensAutoimmune DiseasesPrimary biliary cirrhosisAntigenmedicineHumansHepatic Asialoglycoprotein ReceptorReceptors ImmunologicHepatitis ChronicHepatitisAutoimmune diseaseHepatologymedicine.diagnostic_testbusiness.industryLiver Cirrhosis BiliaryHLA-DR AntigensMiddle Agedmedicine.diseaseLiverLiver biopsyImmunologyAsialoglycoprotein receptorbusinessViral hepatitisHepatology (Baltimore, Md.)
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