Search results for "Lymphocytes"

showing 10 items of 1818 documents

Development of spontaneous airway changes consistent with human asthma in mice lacking T-bet.

2002

Human asthma is associated with airway infiltration by T helper 2 (TH2) lymphocytes. We observed reduced expression of the TH1 transcription factor, T-bet, in T cells from airways of patients with asthma compared with that in T cells from airways of nonasthmatic patients, suggesting that loss of T-bet might be associated with asthma. Mice with a targeted deletion of the T-bet gene and severe combined immunodeficient mice receiving CD4+cells from T-bet knockout mice spontaneously demonstrated multiple physiological and inflammatory features characteristic of asthma. Thus, T-bet deficiency, in the absence of allergen exposure, induces a murine phenotype reminiscent of both acute and chronic h…

TBX21CD4-Positive T-LymphocytesAdoptive cell transferRatónchemical and pharmacologic phenomenaMice SCIDMicemedicineAnimalsHumansLungAsthmaMice KnockoutMultidisciplinarybusiness.industryRespiratory diseaseGene targetinghemic and immune systemsT lymphocyteAllergensmedicine.diseaseAdoptive TransferAsthmarespiratory tract diseasesDisease Models AnimalCollagen Type IIIKnockout mouseImmunologyGene TargetingCytokinesInterleukin-4Bronchial HyperreactivityInterleukin-5businessT-Box Domain ProteinsBronchoalveolar Lavage FluidTranscription FactorsScience (New York, N.Y.)
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Infectious Tolerance

2002

Regulatory CD4(+)CD25(+) T cells (Treg) are mandatory for maintaining immunologic self-tolerance. We demonstrate that the cell-cell contact-mediated suppression of conventional CD4(+) T cells by human CD25(+) Treg cells is fixation resistant, independent from membrane-bound TGF-beta but requires activation and protein synthesis of CD25(+) Treg cells. Coactivation of CD25(+) Treg cells with Treg cell-depleted CD4(+) T cells results in anergized CD4(+) T cells that in turn inhibit the activation of conventional, freshly isolated CD4(+) T helper (Th) cells. This infectious suppressive activity, transferred from CD25(+) Treg cells via cell contact, is cell contact-independent and partially medi…

TGF-βCD4-Positive T-Lymphocyteshuman regulatory T cellsT-LymphocytesImmunologyCellchemical and pharmacologic phenomenaIn Vitro TechniquesLymphocyte ActivationT-Lymphocytes RegulatoryImmune toleranceInterleukin 21AntigenTransforming Growth Factor betaCD4+CD25+ T cellsCell AdhesionImmune TolerancemedicineHumansImmunology and AllergyCytotoxic T cellIL-2 receptorbiologyBrief Definitive ReportModels ImmunologicalReceptors Interleukin-2hemic and immune systemsT-Lymphocytes Helper-InducerTransforming growth factor betainfectious tolerancemedicine.anatomical_structureT cell inhibitionImmunologyCancer researchbiology.proteinTransforming growth factorJournal of Experimental Medicine
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Regulation of B cell homeostasis and activation by the tumor suppressor gene CYLD

2007

B cell homeostasis is regulated by multiple signaling processes, including nuclear factor-kappaB (NF-kappaB), BAFF-, and B cell receptor signaling. Conditional disruption of genes involved in these pathways has shed light on the mechanisms governing signaling from the cell surface to the nucleus. We describe a novel mouse strain that expresses solely and excessively a naturally occurring splice variant of CYLD (CYLD(ex7/8) mice), which is a deubiquitinating enzyme that is integral to NF-kappaB signaling. This shorter CYLD protein lacks the TRAF2 and NEMO binding sites present in full-length CYLD. A dramatic expansion of mature B lymphocyte populations in all peripheral lymphoid organs occur…

TRAF2Tumor suppressor geneImmunologyCellBiologyArticleDeubiquitinating Enzyme CYLDMiceB cell homeostasismedicineAnimalsHomeostasisImmunology and AllergyB-cell activating factorEmbryonic Stem CellsSequence DeletionB-LymphocytesRELBGenetic VariationExonsArticlesFibroblastsDeubiquitinating Enzyme CYLDAlternative SplicingCysteine Endopeptidasesmedicine.anatomical_structureProtein BiosynthesisCancer researchSignal transductionSignal TransductionJournal of Experimental Medicine
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Modulation of intracellular calcium concentrations and T cell activation by prickly pear polyphenols.

2004

Opuntia ficus indica (prickly pear) polyphenolic compounds (OFPC) triggered an increase in [Ca2+]i in human Jurkat T-cell lines. Furthermore, OFPC-induced rise in [Ca2+]i was significantly curtailed in calcium-free buffer (0% Ca2+) as compared to that in 100% Ca2+ medium. Preincubation of cells with tyrphostin A9, an inhibitor of Ca2+ release-activated Ca2+ (CRAC) channels, significantly diminished the OFPC-induced sustained response on the increases in [Ca2+]i. Lanthanum and nifedipine, the respective inhibitors of voltage-dependent and L-type calcium channels, failed to curtail significantly the OFPC-induced calcium response. As OFPC still stimulated increases in [Ca2+]i in 0% Ca2+ medium…

ThapsigarginT-LymphocytesClinical Biochemistrychemistry.chemical_elementGene ExpressionBiologyCalciumLymphocyte ActivationCalcium in biologyMembrane Potentialschemistry.chemical_compoundJurkat CellsPhenolsmedicineHumansCalcium SignalingRNA MessengerMolecular BiologyCalcium signalingCalcium metabolismFlavonoidsVoltage-dependent calcium channelDose-Response Relationship DrugPlant ExtractsReverse Transcriptase Polymerase Chain ReactionEndoplasmic reticulumOpuntiaPolyphenolsCell BiologyGeneral MedicineTyrphostinsMechanism of actionBiochemistrychemistryBiophysicsInterleukin-2ThapsigarginCalciummedicine.symptomMolecular and cellular biochemistry
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Thymoma and paraneoplastic myasthenia gravis

2010

Paraneoplastic autoimmune diseases associate occasionally with small cell lung cancers and gynecologic tumors. However, myasthenia gravis (MG) occurs in at least 30% of all patients with thymomas (usually present at MG diagnosis). These epithelial neoplasms almost always have numerous admixed maturing polyclonal T cells (thymocytes). This thymopoiesis-and export of mature CD4(+)T cells-particularly associates with MG, though there are rare/puzzling exceptions in apparently pure epithelial WHO type A thymomas. Other features potentially leading to inefficient self-tolerance induction include defective epithelial expression of the autoimmune regulator (AIRE) gene and/or of major histocompatib…

ThymomaThymomaT-LymphocytesGenes MHC Class IIImmunologyCellThymus Glandmedicine.disease_causeAutoantigensAutoimmunityhemic and lymphatic diseasesMyasthenia GravisHumansImmunology and AllergyMedicineLymphopoiesisPolyendocrinopathies AutoimmuneAutoantibodiesMHC class IIbiologybusiness.industryLymphopoiesisFOXP3Epithelial Cellsmedicine.diseaseAutoimmune regulatorMyasthenia gravismedicine.anatomical_structureImmunoglobulin GImmunologybiology.proteinbusinessParaneoplastic Syndromes Nervous SystemTranscription FactorsAutoimmunity
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Secretion of neutral and acid DNases in cultivated human lymphocytes after incubation with DNA; possible consequences for inhalation anesthesia.

1995

Abstract After incubation with DNA human lymphocytes release neutral and acid DNase activities into the culture medium; the release depends on DNA concentration and time of cultivation. The electrophoretic mobility of the released neutral DNase activity is in accordance with DNase I and the electrophoretic mobility of the released acid DNase activity with DNase II. The released DNase activities do not originate from dead cells and are not influenced by blast cell formation. The anesthetic halothane can inhibit the released neutral and acid DNase activities. Inhalation anesthesia can possibly disturb the correlation between DNA and DNases in human blood.

Time FactorsBiologyGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundPrecursor cellmedicineDeoxyribonuclease IHumansSecretionLymphocytesIncubationCells CulturedDeoxyribonucleasesEndodeoxyribonucleasesInhalationDose-Response Relationship DrugDNAHydrogen-Ion ConcentrationDose–response relationshipKineticschemistryBiochemistryAnestheticHalothaneAnesthesia InhalationHalothaneDNAmedicine.drugZeitschrift fur Naturforschung. C, Journal of biosciences
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T-cell-mediated cytotoxicity against herpes simplex virus-infected target cells

1977

THE control of herpes simplex virus (HSV) infection by immunological mechanisms seems to be complex and is poorly understood. Neutralising antibodies to HSV plus complement seem to have no effect on the propagation of HSV infection, because HSV spreads to adjacent cells by passing through intercellular bridges1–3. Anti-HSV antibodies plus complement, however, destroy virus-infected cells, but cannot prevent the spread of HSV, suggesting that the virus must be transferred to neighbouring cells before immune lysis occurs1,5. Therefore if lymphocyte-mediated cytolytic mechanisms are instrumental in blocking the spread of HSV in vivo, they ought to destroy infected cells at a very early stage i…

Time FactorsCell SurvivalT-Lymphocytesvirusesmedicine.disease_causeVirusMicrobiologyMiceImmune systemmedicineAnimalsSimplexvirusCytotoxic T cellCells CulturedAntibody-dependent cell-mediated cytotoxicityMultidisciplinarybiologyMacrophagesHerpes SimplexCytotoxicity Tests ImmunologicVirologyCTL*Herpes simplex virusMice Inbred CBAbiology.proteinAntibodyT cell mediated cytotoxicityNature
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Perpetual proliferation of LYT-1 cells requires repetitive signals for IL-2 receptor induction by antigen-presenting cells.

1984

Abstract T cell lines with specificity for bovine insulin and ovalbumin were maintained by serial stimulation with antigen presented on irradiated syngeneic spleen cells, alternating 3 days later with subculture in IL-2 containing medium (CM). When the cultures were repetitively split in CM, with concomitant dilution of antigen-presenting cells, a gradual loss of proliferative capacity of the cells in the presence of CM was observed. Absorption studies revealed a 20-fold reduction of IL-2 receptors on the surface of T blasts assayed 12 days after antigenic stimulation as compared with day 5 blasts. This decrement in the number of IL-2 acceptor sites reflected an actual decrease in cell surf…

Time FactorsCell divisionOvalbuminT cellT-LymphocytesImmunologyReceptors Antigen T-CellLymphocyte ActivationAbsorptionCell LineMiceAntigenmedicineImmunology and AllergyCytotoxic T cellAnimalsIL-2 receptorAntigensReceptors ImmunologicReceptorAntigen-presenting cellCD40biologyReceptors Interleukin-2HematologyMolecular biologymedicine.anatomical_structureImmunologybiology.proteinInterleukin-2SpleenImmunobiology
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Dendritic cell aggresome-like-induced structure formation and delayed antigen presentation coincide in influenza virus-infected dendritic cells.

2005

Abstract Influenza virus infection induces maturation of murine dendritic cells (DCs), which is most important for the initiation of an immune response. However, in contrast to EL-4 and MC57 cells, DCs present viral CTL epitopes with a delay of up to 10 h. This delay in Ag presentation coincides with the up-regulation of MHC class I molecules as well as costimulatory molecules on the cell surface and the accumulation of newly synthesized ubiquitinated proteins in large cytosolic structures, called DC aggresome-like-induced structures (DALIS). These structures were observed previously after LPS-induced maturation of DCs, and it was speculated that they play a role in the regulation of MHC cl…

Time FactorsImmunologyAntigen presentationCellAntigen-Presenting CellsEpitopes T-Lymphocytechemical and pharmacologic phenomenaBone Marrow CellsVirusCell LineMiceImmune systemCell Line TumorMHC class ImedicineImmunology and AllergyAnimalsHumansReceptors ImmunologicCells CulturedAntigen PresentationMice Inbred C3HbiologyUbiquitinViral Core ProteinsRNA-Binding ProteinsCell DifferentiationDendritic cellDendritic CellsNucleocapsid ProteinsVirologyToll-Like Receptor 2Cell biologyNucleoproteinMice Inbred C57BLToll-Like Receptor 4Aggresomemedicine.anatomical_structureNucleoproteinsInfluenza A virusbiology.proteinCytoplasmic StructuresT-Lymphocytes CytotoxicJournal of immunology (Baltimore, Md. : 1950)
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Cross-circulation and Cell Distribution Kinetics in Parabiotic Mice

2011

Blood-borne nucleated cells participate not only in inflammation, but in tissue repair and regeneration. Because progenitor and stem cell populations have a low concentration in the blood, the circulation kinetics and tissue distribution of these cells is largely unknown. An important approach to tracking cell lineage is the use of fluorescent tracers and parabiotic models of cross-circulation. Here, we investigated the cross-circulation and cell distribution kinetics of C57/B6 GFP(+)/wild-type parabionts. Flow cytometry analysis of the peripheral blood after parabiosis demonstrated no evidence for a "parabiotic barrier" based on cell size or surface characterstics; all peripheral blood cel…

Time FactorsPhysiologyParabiosisT-LymphocytesClinical BiochemistryGreen Fluorescent ProteinsParabiosisMice TransgenicBiologyArticleFlow cytometryMiceNucleated cellWeight LossmedicineAnimalsPeripheral blood cellWhole bloodmedicine.diagnostic_testBehavior AnimalCell BiologyMolecular biologyMice Inbred C57BLLymphatic systemGene Expression RegulationImmunologyLymphStem cell
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