Search results for "Lymphocytes"

showing 10 items of 1818 documents

HLA-B27-restricted T cells from patients with ankylosing spondylitis recognize peptides from B*2705 that are similar to bacteria-derived peptides

2003

SUMMARY Ankylosing spondylitis (AS) is an inflammatory systemic disease affecting the spine, sacroiliacal and peripheral joints. Although the aetiology of AS remains unknown, the strong association with the HLA-B27 allele might reflect directly a detrimental effect of the HLA-B27 molecule itself, resulting from its potential capability to present ‘arthritogenic’ peptides to CD8+ T cells. Because some forms of SpA are triggered by enterobacterial infection, such arthritogenic peptides might originate from autologous and/or bacterial proteins triggering cross-reactive CD8+ T cell clones. Intriguingly, two peptides from the second extracellular domain of HLA-B*2705 share sequence homologies wi…

musculoskeletal diseasesAdultCytotoxicity ImmunologicMaleT cellReceptors Antigen T-Cell alpha-betaImmunologyComplementarity determining regionCD8-Positive T-LymphocytesAutoantigensEpitopeCell LineEpitopesAntigenClinical StudiesImmunology and AllergyMedicineHumansSpondylitis AnkylosingCells CulturedHLA-B27 AntigenAgedAged 80 and overHLA-B27Antigens Bacterialbusiness.industryTumor Necrosis Factor-alphaELISPOTT lymphocyteMiddle AgedComplementarity Determining Regionsmedicine.anatomical_structureImmunologyFemalebusinessPeptidesCD8
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HLA-B27-restricted CD8 T cells derived from synovial fluids of patients with reactive arthritis and ankylosing spondylitis.

1993

Ankylosing spondylitis and seronegative spondylarthropathies such as Reiter's syndrome and reactive arthritis are strongly associated with HLA-B27. However, the mechanisms by which HLA-B27 is involved in disease susceptibility and pathogenesis are unknown. If the disease association is a consequence of HLA-B27's physiological function in antigen presentation, the disease should be mediated by cytotoxic T lymphocytes (CTLs) that recognise bacterial or self peptides presented by HLA-B27. Proof of this arthritogenic peptide model requires isolation of B27-restricted CD8 T cells from arthritic joints of patients with spondylarthropathies. An important question is whether "arthritogenic" bacteri…

musculoskeletal diseasesAdultMaleSalmonella typhimuriumYersinia InfectionsCD8 AntigensAntigen presentationArthritisArthritis ReactiveSynovial FluidmedicineCytotoxic T cellHumansReactive arthritisSpondylitis Ankylosingskin and connective tissue diseasesSpondylarthropathiesHLA-B27 AntigenYersinia enterocoliticaAnkylosing spondylitisHLA-B27business.industryGeneral MedicineChlamydia Infectionsmedicine.diseaseClone CellsImmunologySalmonella InfectionsbusinessCD8T-Lymphocytes CytotoxicLancet (London, England)
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HLA-B27-restricted cytotoxic T lymphocyte responses to arthritogenic enterobacteria or self-antigens are dominated by closely related TCRBV gene segm…

1996

Identification of the T-cell receptors (TCR) used by synovial cytotoxic T lymphocytes (CTL) of patients with reactive arthritis (ReA) may be crucial to better understanding the pathogenetic mechanism underlying the HLA-B27 association of spondylarthropathies. The authors, therefore, sequenced 25 TCRB chains from HLA-B27-restricted CD8+ CTL clones and two clonal lines specific for self- or Yersinia enterocolitica antigen isolated from synovial fluids of 3 HLA-B27+ patients with ReA and PBL of one healthy HLA-B27+ individual. Fourteen non-HLA-B27-restricted CTL served as controls. Both autoreactive and Y. enterocolitica specific HLA-B27-restricted CTL used a highly limited set of VB genes wit…

musculoskeletal diseasesAdultMaleSalmonella typhimuriumYersinia InfectionsReceptors Antigen T-Cell alpha-betaImmunologyMolecular Sequence Datachemical and pharmacologic phenomenaChlamydia trachomatisBiologyCD8-Positive T-LymphocytesArthritis ReactiveAutoantigensPolymerase Chain ReactionProhibitinsSynovial FluidCytotoxic T cellHumansAmino Acid SequenceGene Rearrangement beta-Chain T-Cell Antigen Receptorskin and connective tissue diseasesReceptorSpondylarthropathiesGeneHLA-B27 AntigenYersinia enterocoliticaHLA-B27Antigens BacterialT-cell receptorhemic and immune systemsGeneral MedicineDNAChlamydia InfectionsCTL*ImmunologySalmonella InfectionsCD8T-Lymphocytes CytotoxicScandinavian journal of immunology
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Bacteria-specific cytotoxic CD8+ T cells: a missing link in the pathogenesis of the HLA-B27-associated spondylarthropathies.

1994

The term seronegative spondylarthropathies is used for an entity of rheumatic syndromes of peripheral joints and the spine (ankylosing spondylitis, reactive arthritis, Reiter's syndrome, arthritis in psoriasis and in inflammatory bowel disease) which are strongly associated with the MHC class I molecule HLA-B27. However, the mechanisms whereby HLA-B27 confers disease susceptibility have so far remained unknown. There is strong evidence that gut inflammation and infection with gram-negative bacteria play a role in the induction of B27-associated disease. HLA-B27, like other MHC class I molecules, physiologically binds antigenic peptides in its binding groove and presents them to CD8+ T lymph…

musculoskeletal diseasesCytotoxicity ImmunologicAnkylosisEpitopeEpitopesAntigenEnterobacteriaceaeMHC class IMedicineCytotoxic T cellAnimalsHumansSpondylarthropathiesHLA-B27 AntigenHLA-B27Antigens Bacterialbiologybusiness.industryArthritisSynovial MembraneGeneral MedicineDisease Models AnimalImmunologybiology.proteinBacterial antigenbusinessCD8Protein BindingT-Lymphocytes CytotoxicAnnals of medicine
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Enterobacterial Antigens with Tropism for Joint Structures and HLA-B27=Restricted Cytotoxic T-Cells in Reactive Arthritis

1995

In reactive arthritis (ReA), sterile synovitis is an immunological sequela following gastrointestinal or urogenital infection with facultatively intracellular bacteria (Yersinia, Salmonella, Shigella, Chlamydia). It is widely accepted now that the development of arthritis is closely related to the persistance of bacteria or bacterial antigens in extraarticular mucosal or lymphoid tissues (i.e. gut mucosa, gut associated lymphoid tissue, genitourinary mucosa); however, it is still unclear which host mechanisms are responsible for the poorer elimination of arthritis-causing microorganisms in those ReA patients. Bacterial components are also camed to the joints where they can be demonstrated i…

musculoskeletal diseasesGut-associated lymphoid tissueImmunologyYersiniaArthritis ReactiveTropismMicrobiologyImmune systemEnterobacteriaceaeRheumatologyAntigenSynovitismedicineAnimalsHumansImmunology and AllergySynovial fluidHLA-B27 AntigenAntigens BacterialbiologyGeneral Medicinebiology.organism_classificationmedicine.diseasemedicine.anatomical_structureImmunologyJointsBacterial antigenSynovial membraneT-Lymphocytes CytotoxicScandinavian Journal of Rheumatology
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Identification of the Yersinia enterocolitica urease beta subunit as a target antigen for human synovial T lymphocytes in reactive arthritis.

1993

The local T-cell response to bacterial antigens is involved in the pathogenesis of reactive arthritis (ReA). Here, we have identified a 19-kDa antigen of Yersinia enterocolitica O:9 recognized by Yersinia-specific synovial fluid CD4+ T cells in two patients with Yersinia-induced ReA. N-terminal amino acid sequencing of this protein revealed that it was identical to the 19-kDa urease beta subunit of Y. enterocolitica O:9. This protein has previously been shown to be arthritogenic in preimmunized rats after intra-articular injection. Analysis of the T-cell response to this protein showed that it contains several T-cell epitopes, one of which cross-reacts with other enterobacteria not able to …

musculoskeletal diseasesProtein subunitT-LymphocytesImmunologyMolecular Sequence DataBiologyLymphocyte ActivationMicrobiologyEpitopeMicrobiologyAntigenProhibitinsSynovial FluidSynovial fluidHumansAmino Acid SequenceYersinia enterocoliticaHLA-DR AntigenYersinia enterocoliticaAntigens BacterialSequence Homology Amino AcidArthritisT lymphocyteHLA-DR Antigensbiology.organism_classificationbacterial infections and mycosesUreaseInfectious DiseasesParasitologyBacterial antigenResearch Article
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An update on the relationships between rheumatoid arthritis and atherosclerosis

2010

Rheumatoid arthritis is a chronic inflammatory disease. Cardiovascular events are the most important cause of mortality and morbidity in patients with rheumatoid arthritis. Beyond the traditional cardiovascular risk factors, chronic systemic inflammation has been shown to be a crucial factor in atherosclerosis development and progression from endothelial dysfunction to plaque rupture and thrombosis. Many studies have shown that atherosclerosis is not a passive event like accumulation of lipids in the vessel walls; by contrast, it represents an active inflammation of the vessels. Inflammatory cells such as macrophages, monocytes and T cells play important roles in the development of both rhe…

musculoskeletal diseasesT-LymphocytesAtherosclerosis Rheumatoid arthritis Inflammation Cardiovascular risk10265 Clinic for Endocrinology and DiabetologyInflammation610 Medicine & healthComorbiditySystemic inflammationModels BiologicalMonocytes2705 Cardiology and Cardiovascular MedicineArthritis RheumatoidRisk FactorsImmunopathologymedicineHumansEndothelial dysfunctionAutoimmune diseaseInflammationbusiness.industryVascular diseaseMacrophagesmedicine.diseaseAtherosclerosisThrombosisOxidative StressCardiovascular DiseasesRheumatoid arthritisImmunologyDisease Progressionmedicine.symptomInsulin ResistanceCardiology and Cardiovascular Medicinebusiness
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Pathogenesis of polymyalgia rheumatica

2018

Polymyalgia rheumatica (PMR) is a chronic, inflammatory disorder of unknown cause, almost exclusively occurring in people aged over 50 and often associated with giant cell arteritis. The evidence that PMR occurs almost exclusively in individuals aged over 50 may indicate that age-related immune alterations in genetically predisposed subjects contribute to development of the disease. Several infectious agents have been investigated as possible triggers of PMR even though the results are inconclusive. Activation of the innate and adaptive immune systems has been proved in PMR patients as demonstrated by the activation of dendritic cells and monocytes/macrophages and the altered balance betwee…

musculoskeletal diseaseslcsh:Internal medicineGiant Cell ArteritisAdaptive immunityeducationlcsh:MedicineDiseaseT-Lymphocytes RegulatoryPathogenesisPolymyalgia rheumatica03 medical and health sciences0302 clinical medicineImmune systemRheumatologyPathogenesiHumansMedicinelcsh:RC31-1245Giant Cell ArteritiB cellAgedInnate immunity030203 arthritis & rheumatologyB-LymphocytesEvidence-Based MedicineInnate immune systembusiness.industrylcsh:RPolymyalgia rheumaticaB-LymphocyteCell DifferentiationBiomarkerPathogenesis.medicine.diseaseAcquired immune systemImmunity InnateSettore MED/16 - ReumatologiaGiant cell arteritismedicine.anatomical_structure030220 oncology & carcinogenesisImmunologyTh17 CellsbusinessBiomarkersHumanReumatismo
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Correlative histologic and arthroscopic evaluation in rheumatoid knee joints.

1992

The correlation between arthroscopic observations and histologic changes in rheumatoid arthritis is still controversial. Synovial samples of 21 knee joints in rheumatoid arthritis patients were comparatively investigated by endoscopy and histology. Biopsies were scored by an endoscopist and subsequently dissected. Different histochemical and immunocytochemical staining techniques were used to define inflammatory activity. Arthroscopic and histological values were compared by rating scales and variance analysis. Our study indicates that synovial biopsy is of diagnostic value in rheumatoid arthritis. However, its usefulness depends on the histochemical methods used. The results revealed highl…

musculoskeletal diseasesmedicine.medical_specialtyPathologyKnee Jointmedicine.medical_treatmentSynovectomyArthritis RheumatoidArthroscopySynovitisInternal medicinemedicineHumansLymphocytesHyperplasiamedicine.diagnostic_testbusiness.industryMacrophagesArthroscopySynovial MembraneHistologyHepatologymedicine.diseaseImmunohistochemistryEndoscopymedicine.anatomical_structureRheumatoid arthritisSurgerySynovial membranebusinessGranulocytesSurgical endoscopy
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Oxymetazoline modulates proinflammatory cytokines and the T‐cell stimulatory capacity of dendritic cells

2007

The nasal decongestant oxymetazoline (OMZ) is frequently used in the topical treatment of rhinitis/sinusitis. As proinflammatory cytokines play a critical role in the development and maintenance of local inflammation, the aim of this study was to investigate the influence of OMZ on immune cells in order to diminish the mucosal infiltration of the nose. Peripheral blood mononuclear cells (PBMC) from buffy coats of healthy volunteers were isolated and stimulated in the presence or absence of OMZ. In addition, monocyte-derived dendritic cells (DC) were generated and different concentrations of OMZ were added. DC phenotype and their T-cell stimulatory properties were analysed. The vasoactive su…

oxymetazolinemedicine.medical_treatmentT cellT-LymphocytesInflammationEnzyme-Linked Immunosorbent AssayDermatologyimmunomodulationLymphocyte ActivationBiochemistryProinflammatory cytokinerhinitismedicineHumansAntigen-presenting cellMolecular BiologyCells CulturedDose-Response Relationship Drugbusiness.industryImmunomagnetic SeparationDendritic cellDendritic CellsFlow CytometryNasal decongestantNasal DecongestantsCytokinemedicine.anatomical_structureImmunologyproinflammatory cytokinesLeukocytes MononuclearCytokinesTumor necrosis factor alphaOriginal Articlemedicine.symptombusinessExperimental Dermatology
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