Search results for "MLP"

showing 10 items of 22 documents

Region of interest detection using MLP

2014

A novel technique to detect regions of interest in a time series as deviation from the characteristic behavior is proposed. The deterministic form of a signal is obtained using a reliably trained MLP neural network with detailed complexity management and cross-validation based generalization assurance. The proposed technique is demonstrated with simulated and real data. peerReviewed

MLPneural networks
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Mobile Edge Computing: Architetture ed Analisi della Live Migration

2020

Con l'ormai prossima rete mobile 5G entreranno a far parte della nostra quotidianità nuovi servizi applicativi, mai prima possibili, grazie all'avvicinamento di risorse di calcolo e di memoria nei pressi dell'utente in mobilità. Un’architettura abilitante i futuri servizi è quella di Mobile Edge Computing (MEC) in cui cloud di capacità inferiori rispetto a quelli presenti nella core della rete sono dislocati nei pressi della stazioni radio e metteranno a disposizione risorse di calcolo tali da permettere, tramite la tecnica di offloading, la fruizione di servizi quali realtà aumentata, gaming online, contenuti streaming ad alta risoluzione ed operazioni di data analytics. Ogni nuovo paradig…

Pre-CopyMLP networkDockerVirtual MachineKata-Containerworking set sizeMECLive MigrationContainer
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Mutation analysis of the SPG4 gene in Italian patients with pure and complicated forms of spastic paraplegia

2010

Mutations in the SPG4 gene are the most common causes of hereditary spastic paraplegia (HSP) accounting for up to 40% of autosomal dominant (AD) forms and 12-18% of sporadic cases. The phenotype associated with HSP due to mutations in the SPG4 gene tends to be pure. There is increasing evidence, however, of patients with complicated forms of spastic paraplegia in which SPG4 mutations were identified. A cohort of 38 unrelated Italian patients with spastic paraplegia, of which 24 had a clear dominant inheritance and 14 were apparently sporadic, were screened for mutations in the SPG4 gene.We identified 11 different mutations, six of which were novel (p.Glu143GlyfsX8, p.Tyr415X, p.Asp548Asn, c…

MaleSpastinDNA Mutational AnalysisHereditary spastic paraplegiaEXON DELETIONSGene mutationmedicine.disease_causeSpastinFAMILIESCohort StudiesExonGenotypeSpasticMutation frequencyChild3' Untranslated RegionsChromatography High Pressure LiquidAdenosine TriphosphatasesGeneticsMutationHereditary spastic paraplegia SPG4Reverse Transcriptase Polymerase Chain ReactionMutation analysiExonsMiddle AgedMLPAPhenotypeMutation analysisItalyNeurologySettore MED/26 - NeurologiaFemaleAdultAdolescentGenotypeHereditary spastic paraplegia3 ' UTR3′ UTRMutation MissenseFREQUENTSPG4CLASSIFICATIONYoung AdultmedicineHumansAgedParaplegiaSPECTRUMbusiness.industrymedicine.diseaseNeurology (clinical)businessCOLLECTIONEXPRESSION ANALYSISGene Deletion
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Somatic copy number alterations are associated with EGFR amplification and shortened survival in patients with primary glioblastoma.

2019

Glioblastoma (GBM) is the most common malignant primary tumor of the central nervous system. With no effective therapy, the prognosis for patients is terrible poor. It is highly heterogeneous and EGFR amplification is its most frequent molecular alteration. In this light, we aimed to examine the genetic heterogeneity of GBM and to correlate it with the clinical characteristics of the patients. For that purpose, we analyzed the status of EGFR and the somatic copy number alterations (CNAs) of a set of tumor suppressor genes and oncogenes. Thus, we found GBMs with high level of EGFR amplification, low level and with no EGFR amplification. Highly amplified tumors showed histological features of…

0301 basic medicineMaleCancer ResearchBiopsyL-amp GB EGFR-low amplified glioblastomamedicine.disease_causewt wildtypeMYBPC3 myosin-binding protein C0302 clinical medicineHIC1 hypermethylated in cancer 1Gene duplicationIn Situ Hybridization FluorescenceIDH2 isocitrate dehydrogenase 2MutationRB-pat RB signaling pathwayEGFRvIII epidermal growth factor receptor variant number IIIPAH phenylalanine hydroxylaseGBM glioblastoma IDH-wildtype (glioblastoma multiforme primary glioblastoma).ANOVA ANalysis Of VArianceN-amp GB EGFR-no amplified glioblastomaMiddle AgedCDKN2A cyclin-dependent kinase inhibitor 2Alcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensPrognosisPrimary tumorImmunohistochemistryH-amp GB EGFR-high amplified glioblastomaErbB ReceptorsTKR-pat tyrosine-kinase receptors signaling pathway030220 oncology & carcinogenesisDisease ProgressionCDK6 cyclin-dependent kinase 6CDH1 Cadherin 1FemaleCREM cAMP response element modulatorIHC immunohistochemistryAdultOriginal articleDNA Copy Number VariationsCDKN1B cyclin-dependent kinase inhibitor 1BBiologyRARB retinoic acid receptor betaCNS central nervous systemlcsh:RC254-282IDH1 isocitrate dehydrogenase 1BCL2 B-cell cll/ lymphoma 2CNAs copy number algerationsWHO World Health Organization03 medical and health sciencesYoung Adultp53-pat p53 signaling pathwaymedicineBiomarkers TumorTMA tissue microarrayPTENHumansProtein kinase BPI3K/AKT/mTOR pathwaySurvival analysisAgedGenetic heterogeneityGene AmplificationGFAP glial fibrillary acidic proteinMLPA multiplex ligation-dependent probe amplificationmedicine.diseaseFISH fluorescence in situ hibridizationSurvival AnalysisCDKN2B cyclin-dependent kinase inhibitor 2BPTEN phosphatase and tensin homologEGFR epidermal growth factor receptorCNV-load load of copy number variations030104 developmental biologyMutationPARK2 parkinCancer researchbiology.proteinTCGA The Cancer Genome AtlasLARGE1 acetylglucosaminyltransferase-like protein 1GlioblastomaCHD7 Chromodomain Helicase DNA Binding Protein 7DAPI 4′6-diamidino-2-phenylindoleNeoplasia (New York, N.Y.)
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Tetrazīna fragmentu saturošu linkeru sintēze proteīnu modificēšanai

2022

Maģistra darbā ir veikta jaunu, alifātisku un PEG fragmentus saturošu, tetrazīna linkeru sintēze proteīnu biokonjugācijai. Darba ietvaros iegūti un izolēti 3 linkeri, kas paredzēti neselektīvai Lys aminoskābju atlikumu modificēšanai, un arī iegūti vēl 3 linkeri, kas paredzēti reģiospecifiskai proteīna N-gala modificēšanai. Darba ietvaros arī izolēts viens linkeris, kas paredzēts His modificēšanai. Visi sintezētie linkeri tika testēti modificējot Borrelia Burgdorferi MlpC lipoproteīnu, kā arī veikta paraugu MALDI-TOF MS analīze, kurā no septiņiem linkeriem, seši ir uzrādījuši konjugāta veidošanos ar MlpC.

LINKERIBIOKONJUGĀCIJATETRAZĪNIPROTEĪNSMLPCĶīmija
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Agencia Local de Empleo MLPE de Estrasburgo: ¡Todos los jóvenes tienen futuro! (Francia)

2012

International audience

Francia[SHS.ANTHRO-SE] Humanities and Social Sciences/Social Anthropology and ethnologyAgencia Local de EmpleojóvenesMLPE[SHS.ANTHRO-SE]Humanities and Social Sciences/Social Anthropology and ethnologyIntelligentia Territorial[SHS]Humanities and Social Sciences[ SHS.ANTHRO-SE ] Humanities and Social Sciences/Social Anthropology and ethnology[ SHS ] Humanities and Social SciencesEstrasburgo[SHS] Humanities and Social SciencesfuturoComputingMilieux_MISCELLANEOUS
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Analisi di delezioni esoniche del gene PAH in pazienti affetti da iperfenilalaninemia

2009

MLPA PAHSettore BIO/13 - Biologia Applicata
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Impacto de la variación en el número de copias y del estado de metilación de genes supresores de tumor en las vías de progresión del meningioma

2015

El meningioma es el tumor del sistema nervioso central más frecuente en la edad adulta, con una incidencia superior al 30% y una evidente predominancia femenina. La sintomatología deriva fundamentalmente de la localización del tumor, que crece desde las células de la cubierta aracnoidea. El tratamiento es principalmente quirúrgico, y su radicalidad se relaciona clásicamente con la aparición de recidivas tumorales, que es la principal complicación que sufren los afectados. Macroscópicamente, son tumores habitualmente bien delimitados y encapsulados, de crecimiento lento, y microscópicamente la OMS los clasifica en 16 subtipos histológicos. Son tumores positivos para vimentina y EMA, y en men…

:CIENCIAS MÉDICAS ::Patología::Neuropatología [UNESCO]:CIENCIAS DE LA VIDA::Biología humana ::Genética humana [UNESCO]:CIENCIAS DE LA VIDA::Biología celular::Citogenética [UNESCO]citogenéticaUNESCO::CIENCIAS DE LA VIDA::Biología celular::Cultivo celularFFPEUNESCO::CIENCIAS MÉDICAS ::Patología::NeuropatologíameningiomaUNESCO::CIENCIAS DE LA VIDA::Biología celular::CitogenéticaMLPAepigenética:CIENCIAS DE LA VIDA::Biología celular::Cultivo celular [UNESCO]genes supresores de tumorUNESCO::CIENCIAS MÉDICAS ::Patología::HistopatologíaUNESCO::CIENCIAS DE LA VIDA::Biología humana ::Genética humana:CIENCIAS MÉDICAS ::Patología::Histopatología [UNESCO]
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Carrier screening for spinal muscular atrophy in Italian population

2014

Spinal muscular atrophy (SMA) is an autosomal-recessive neuromuscular disorder characterized by motor neuron degeneration in the anterior horn of the spinal cord and brain stem, resulting in progressive muscle weakness and atrophy. The responsible survival motor neuron gene (SMN1; HGNC: 11117) is localized in 5q11.2-13.3. Screening for carriers of SMA is necessary for effective clinical/prenatal diagnosis and genetic counselling. In this study, the copy number of SMN1 gene was determined from a southern Italian population to estimate carrier frequency. This is the first report addressing the estimation of SMA carrier frequency in an Italian population. Our results show that the SMA carrier …

HeterozygoteGenetic counselingGene DosagePhysiologycarrier screeningPrenatal diagnosisSMN1BiologyCarrier testingMuscular Atrophy SpinalAtrophyGene FrequencySettore BIO/13 - Biologia ApplicataPrevalenceGeneticsmedicineHumansGenetic Testingspinal muscular atrophysurvival motor neuron gene (SMN1); spinal muscular atrophy; carrier screening; MLPAExonsSpinal muscular atrophyMotor neuronSMA*medicine.diseaseSurvival of Motor Neuron 1 ProteinMLPAmedicine.anatomical_structureItalysurvival motor neuron gene (SMN1)Journal of Genetics
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Extracellular Vesicles From Liver Progenitor Cells Downregulates Fibroblast Metabolic Activity and Increase the Expression of Immune-Response Related…

2021

Extracellular vesicles (EVs) mediate cell-to-cell crosstalk whose content can induce changes in acceptor cells and their microenvironment. MLP29 cells are mouse liver progenitor cells that release EVs loaded with signaling cues that could affect cell fate. In the current work, we incubated 3T3-L1 mouse fibroblasts with MLP29-derived EVs, and then analyzed changes by proteomics and transcriptomics. Results showed a general downregulation of protein and transcript expression related to proliferative and metabolic routes dependent on TGF-beta. We also observed an increase in the ERBB2 interacting protein (ERBIN) and Cxcl2, together with an induction of ribosome biogenesis and interferon-relate…

MLP29ChemistryMLP29 cell crosstalk exosomes extracellular vesicles (EVs) fibroblast immune responseCell BiologyexosomesCell fate determinationcell crosstalkMicrovesiclesfibroblastimmune responseCell biologyCell and Developmental BiologyCXCL2Crosstalk (biology)Immune systemmedicine.anatomical_structurelcsh:Biology (General)Downregulation and upregulationmedicineProgenitor cellextracellular vesicles (EVs)Fibroblastlcsh:QH301-705.5Original ResearchDevelopmental Biology
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