Search results for "MUCOSA"

showing 10 items of 1066 documents

Oleuropein protects against dextran sodium sulfate-induced chronic colitis in mice.

2013

The anti-inflammatory effect of oleuropein (1), the major phenolic secoiridoid in Olea europaea, was evaluated in an experimental model of chronic colitis in mice. Animals were exposed to four repeated cycles of dextran sodium sulfate in drinking water followed by a 7-day rest period. Animals receiving a standard diet supplemented with 0.25% of 1 (equivalent to 500 mg/kg/day) for 56 days exhibited a decrease of inflammatory symptoms, as reflected by improvement of disease activity index and histopathological changes. It was found that 1 decreased inflammatory cell recruitment and the release of inflammatory cytokines interleukin (IL)-1β and IL-6 with increased IL-10 levels in colon tissue. …

T-LymphocytesInterleukin-1betaIridoid GlucosidesAnti-Inflammatory AgentsPharmaceutical ScienceNitric Oxide Synthase Type IIPharmacologyp38 Mitogen-Activated Protein KinasesAnalytical Chemistrychemistry.chemical_compoundMiceOleuropeinOleaDrug DiscoveryAnimalsIridoidsIntestinal MucosaChronic colitisPyransPharmacologybiologyMolecular StructureExperimental modelInterleukin-6Organic ChemistryDextran Sulfatebiology.organism_classificationColitisInterleukin-10Mice Inbred C57BLComplementary and alternative medicinechemistryOleaCyclooxygenase 2Molecular MedicineDextran sodium sulfateJournal of natural products
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Host immune response to Cryptosporidium parvum infection

2010

Species of the genus Cryptosporidium are protozoan parasites (Apicomplexa) that cause gastroenteritis in animals and humans. Of these Cryptosporidium parvum and Cryptosporidium hominis are the major causative agents of human cryptosporidiosis. Whereas infection is self-limiting in the immunocompetent hosts, immunocompromised individuals develop a chronic, life-threatening disease. As specific therapeutic or preventive interventions are not yet available, better understanding of the immune response to the parasite is required. This minireview briefly summarizes the factors involved in the innate and acquired immune response in this pathogen-host interaction with an emphasis on more recent da…

T-Lymphocytesanimal diseasesAIDS-Related Opportunistic InfectionsImmunologyAntibodies ProtozoanCryptosporidiosisAdaptive ImmunityBiologyNitric OxideImmunocompromised HostMiceImmune systemIntestinal mucosaImmunityparasitic diseasesAnimalsHumansIntestinal MucosaCryptosporidium parvumB-LymphocytesPhagocytesAIDS-Related Opportunistic InfectionsComplement System ProteinsDendritic CellsGeneral MedicineAcquired immune systembiology.organism_classificationVirologyImmunity InnateKiller Cells NaturalDisease Models AnimalInfectious DiseasesCryptosporidium parvumImmunologyCytokinesParasitologyImmunocompetenceImmunocompetenceCryptosporidium hominisExperimental Parasitology
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Influence of Chronic Alcohol Intake on Intestinal Taurine and Antipyrine Transport in Pregnant Rats

1998

Taurine is a nonessential amino acid that plays a critical role in development. However, biosynthetic capacity is almost negligible in the fetus and must be supplied by the mother. Therefore, when maternal taurine status is depressed during gestation, fetal tissue taurine concentrations can also be compromised. In the present study, the effect of chronic alcohol intake on the intestinal transport of taurine during pregnancy has been investigated by an in vitro technique that allows measurement of the unidirectional influx of the amino acid across the intact rat mid jejunum. The influence of alcohol intake on the passive component of the intestinal transport was also investigated with antipy…

Taurinemedicine.medical_specialtyFetusEthanolLiquid dietMedicine (miscellaneous)ToxicologyIntestinal absorptionSmall intestineJejunumPsychiatry and Mental healthchemistry.chemical_compoundmedicine.anatomical_structureEndocrinologychemistryIntestinal mucosaInternal medicinemedicineAlcoholism: Clinical and Experimental Research
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In-Vitro Investigation of Fatigue and Fracture Behavior of Transmucosal versus Submerged Bone Level Implants Used in Fixed Prosthesis

2021

Background: The present in vitro study aimed to investigate the fatigue performance of different dental fixtures in two different emergence profiles. Biological failures are frequently reported because the problem canonly be solved by replacing a failing implant with a new one. Clinicians addressed minor mechanical failures, such as bending, loosening or the fracture of screws, abutment, or the entire prosthesis, by simply replacing or fixing them. Methods: Transmucosal and submerged bone-level dental implants underwent fatigue strength tests (statical and dynamical performance) by a standardized test: UNI EN ISO 14801:2016. Two types of emergence profiles (Premium sub-crestal straight impl…

TechnologyQH301-705.5QC1-999medicine.medical_treatmentProsthesis03 medical and health sciences0302 clinical medicinemedicineimplant fractureUltimate failureGeneral Materials ScienceBiology (General)QD1-999InstrumentationMathematicsFluid Flow and Transfer ProcessesOrthodonticstransmucosal implantTPhysicsProcess Chemistry and TechnologyGeneral EngineeringNonparametric statistics030206 dentistryfatigue testEngineering (General). Civil engineering (General)Fatigue limitComputer Science ApplicationsChemistryCoronal planeFracture (geology)ImplantTA1-2040submerged bone-level implant neckAbutment (dentistry)030217 neurology & neurosurgeryApplied Sciences
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Nickel induces intracellular calcium mobilization and pathophysiological responses in human cultured airway epithelial cells.

2009

Abstract Environmental exposure to nickel is associated to respiratory disorders and potential toxicity in the lung but molecular mechanisms remain incompletely explored. The extracellular Ca 2+ -sensing receptor (CaSR) is widely distributed and may be activated by divalent cations. In this study, we investigated the presence of CaSR in human cultured airway epithelial cells and its activation by nickel. Nickel transiently increased intracellular calcium (−log EC 50  = 4.67 ± 0.06) in A549 and human bronchial epithelial cells as measured by epifluorescence microscopy. Nickel (20 μM)-induced calcium responses were reduced after thapsigargin or ryanodine exposure but not by Ca 2+ -free medium…

ThapsigarginInterleukin-1betachemistry.chemical_elementRespiratory MucosaBiologyCalciumToxicologyCalcium in biologychemistry.chemical_compoundNickelExtracellularHumansRNA Small InterferingCells CulturedA549 cellRyanodine receptorRyanodineTumor Necrosis Factor-alphaInositol trisphosphateEpithelial CellsGeneral MedicineEnvironmental exposureIntercellular Adhesion Molecule-1Cell biologychemistryMicroscopy FluorescenceType C PhospholipasesImmunologyGTP-Binding Protein alpha Subunits Gq-G11ThapsigarginCalciumReceptors Calcium-SensingChemico-biological interactions
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Electron Microscopic Contrast of the Cytoskeleton and Junctional Complexes of Intestinal Epithelial Cells by Ethanolic Phosphotungstic Acid

2000

After glutaraldehyde fixation and treatment with ethanolic phosphotungstic acid (E-PTA) before plastic embedding, sections of rat large intestine showed a characteristic electron contrasting pattern in epithelial cells. The axis of microvilli, terminal web, a thin band below the luminal plasma membrane, centrioles and junctional complexes (tight junctions, adherens junctions, and desmosomes) appeared highly contrasted. In addition to protein components of microfilaments and intermediate filaments, proteins from the junctional complexes could also be implicated in the contrasting reaction with E-PTA. Mitochondrial membranes, chromatin masses, and nucleoli of enterocytes showed considerable e…

Tissue FixationBiologyMicrofilamentSpecimen HandlingAdherens junctionTerminal webGlycocalyxchemistry.chemical_compoundAnimalsIntestine LargePhosphotungstic acidIntestinal MucosaRats WistarCytoskeletonIntermediate filamentCytoskeletonEthanolMicrovilliStaining and LabelingTissue EmbeddingTight junctionEpithelial CellsPhosphotungstic AcidAgricultural and Biological Sciences (miscellaneous)ChromatinMitochondriaRatsCell biologySolutionsMicroscopy ElectronIntercellular JunctionschemistrySolventsAnatomyCell NucleolusEuropean Journal of Morphology
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T-bet and mucosal Th1 responses in the gastrointestinal tract

2002

T cells play an essential role in regulating mucosal immune responses in the gastrointestinal tract. Recent observations on T helper cell differentiation and activation by regulatory transcription factors-especially T-bet-in chronic inflammatory diseases have provided new perspectives for understanding mucosal immunity. Here we summarise recent advances in the field of transcription factors and discuss the implications of these findings for future therapeutic approaches in inflammatory bowel diseases. In particular, we have focused on the role of T-bet in controlling mucosal Th1 responses in the gastrointestinal tract.

Transcription GeneticCellular differentiationGene Expressionchemical and pharmacologic phenomenaInflammationLeading ArticleBiologyInterferon-gammaMiceImmune systemImmunopathologymedicineAnimalsHumansT-helper cell differentiationImmunity MucosalTranscription factorImmunity CellularGastrointestinal tractT-cell receptorGastroenterologyCell DifferentiationTh1 CellsInflammatory Bowel DiseasesGastric MucosaImmunologyCytokinesmedicine.symptomT-Box Domain ProteinsDigestive SystemInterleukin-1Transcription FactorsGut
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The (2-phenyl-2-trimethylsilyl)ethyl-(PTMSEL)-linker in the synthesis of glycopeptide partial structures of complex cell surface glycoproteins.

2003

The (2-phenyl-2-trimethylsilyl)ethyl-(PTMSEL) linker represents a novel fluoride-sensitive anchor for the solid-phase synthesis of protected peptides and glycopeptides. Its cleavage is achieved under almost neutral conditions using tetrabutylammonium fluoride trihydrate in dichloromethane thus allowing the construction of complex molecules sensitive to basic and acidic media commonly required for the cleavage of standard linker systems. The advantages of the PTMSEL linker are demonstrated in the synthesis of glycopeptides from the liver intestine (LI)-cadherin and the mucin MUC1, bearing carbohydrate moieties such as N-linked chitobiose or O-linked sialyl-T(N)-residues. The synthesis of the…

Trimethylsilyl CompoundsStereochemistryDiketopiperazinesChitobioseCleavage (embryo)DisaccharidesCatalysisPiperazineschemistry.chemical_compoundFluoridesSolid-phase synthesisMoleculeHumansIntestinal MucosaProtein secondary structureDichloromethaneAspartic AcidMethylene ChlorideMembrane GlycoproteinsOrganic ChemistryGlycopeptidesMucinsGeneral ChemistryCadherinsCombinatorial chemistryGlycopeptideIntestinesQuaternary Ammonium CompoundschemistryLiverModels ChemicalSialic AcidsLinkerChemistry (Weinheim an der Bergstrasse, Germany)
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Progression of colorectal cancers correlates with overexpression and loss of polarization of expression of the htid-1 tumor suppressor.

2007

Recently, we identified htid-1, the human counterpart of the Drosophila tumor suppressor gene lethal(2)tumorous imaginal discs [l(2)tid], as a direct molecular ligand of the adenomatous polyposis coli (APC) tumor suppressor. The gene encodes three cytosolic (Tid50, Tid48 and Tid46) and three mitochondrial (Tid43, Tid40 and Tid38) proteins. In the colorectal epithelium the cytosolic forms hTid50/hTid48 interact under physiological conditions with the N-terminal region of APC. This complex which associates with additional proteins such as Hsp70, Hsc70, Actin, Dvl and Axin defines a novel physiological state of APC unrelated to beta-catenin degradation. Here we show that the expression of the …

Tumor suppressor geneProtein familyAdenomatous polyposis coliColorectal cancerAntibodies NeoplasmRNA SplicingAdenomatous Polyposis Coli ProteinGeneticsmedicineHumansHSP70 Heat-Shock ProteinsRNA NeoplasmIntestinal MucosaDNA PrimersGeneticsOncogenebiologyTumor Suppressor ProteinsWnt signaling pathwayCell DifferentiationGeneral MedicineCell cycleHSP40 Heat-Shock Proteinsmedicine.diseaseGene Expression Regulation NeoplasticChaperone (protein)biology.proteinCancer researchDisease ProgressionColorectal NeoplasmsInternational journal of molecular medicine
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Epidemiología de la patología de la mucosa oral más frecuente en niños

2005

El odontólogo responsable de tratar al niño debe tener en cuenta la posibilidad de encontrar cualquier condición patológica a nivel de la mucosa oral sobre todo a edades tempranas. Por ello, hemos tratado de aunar en nuestro estudio tanto la información más actualizada como nuestra propia experiencia para intentar ofrecer los datos de mayor interés, desde el punto de vista epidemiológico, que nos permita diagnosticar la patología de la mucosa oral más frecuente en la población infantil. Los estudios epidemiológicos realizados estos últimos años han mostrado la dispar apreciación de los diferentes investigadores y una gran variabilidad en las prevalencias de las lesiones mucosas orales en la…

UNESCO::CIENCIAS MÉDICASOdontologíapoblación infantil:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludLesiones de la mucosa oralepidemiologíadiagnóstico diferencial
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