Search results for "MUTATION"

showing 10 items of 2830 documents

Expression of the pea S -adenosylmethionine decarboxylase gene is involved in developmental and environmental responses

2002

A cDNA, able to complement the S-adenosyl-L-methionine decarboxylase (SAMdC; EC 4.1.1.50)-defective yeast strain Y342, has been isolated from pea (Pisum sativum L.). Expression of the SAMdC gene was characterised during pea development. Northern analysis showed a differential expression of the pea SAMdC gene in vegetative and reproductive tissues. The highest SAMdC mRNA levels were found in undifferentiated callus and tissues with high rates of cell division, and at the onset of fruit development. SAMdC expression was also induced in senescing ovaries, probably in relation to an accumulation of spermine during ovary senescence. Finally, the levels of SAMdC transcripts in leaves and shoots w…

Adenosylmethionine DecarboxylaseDNA ComplementaryCarboxy-lyasesMolecular Sequence DataSpermineSaccharomyces cerevisiaePlant ScienceEnvironmentBiologyGene Expression Regulation EnzymologicPisumchemistry.chemical_compoundOzoneGene Expression Regulation PlantGene expressionGeneticsAmino Acid SequenceGeneSequence Homology Amino AcidReproductionGenetic Complementation TestPeasGene Expression Regulation Developmentalfood and beveragesSequence Analysis DNAbiology.organism_classificationCell biologyPlant LeavesBiochemistrychemistryAdenosylmethionine decarboxylaseFruitCallusMutationSperminePolyamineSequence AlignmentCell DivisionPlant ShootsPlanta
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Mediación en derecho administrativo

2018

Presenciamos un momento de creciente fascinación por la mediación. Sin embargo, el Derecho administrativo se presenta históricamente como un ámbito plagado de obstáculos frente a las ADR y, por tanto, frente a la mediación. Sin embargo, los cambios sociales y la mutación de las estructuras administrativas llevan a buscar nuevos cauces para la resolución de los conflictos con la Administración. La mediación se ha visto fuertemente impulsada por la Unión Europea y en el ámbito interno, el Consejo General del Poder Judicial ha propiciado la incorporación de la mediación administrativa mediante proyectos pilotos que impulsan el consenso.

Administrative lawPolitical scienceLawSocial changeMediationMutation (genetic algorithm)media_common.cataloged_instanceGeneral MedicineEuropean unionmedia_commonRevista Jurídica Digital UANDES
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TCTN3 Mutations Cause Mohr-Majewski Syndrome

2012

Orofaciodigital syndromes (OFDSs) consist of a group of heterogeneous disorders characterized by abnormalities in the oral cavity, face, and digits and associated phenotypic abnormalities that lead to the delineation of 13 OFDS subtypes. Here, by a combined approach of homozygozity mapping and exome ciliary sequencing, we identified truncating TCTN3 mutations as the cause of an extreme form of OFD associated with bone dysplasia, tibial defect, cystic kidneys, and brain anomalies (OFD IV, Mohr-Majewski syndrome). Analysis of 184 individuals with various ciliopathies (OFD, Meckel, Joubert, and short rib polydactyly syndromes) led us to identify four additional truncating TCTN3 mutations in un…

AdolescentFoot Deformities CongenitalMolecular Sequence DataCiliopathiesJoubert syndromeYoung AdultFetusReportCerebellumGLI3medicineGeneticsHumansExomeHedgehog ProteinsGenetics(clinical)Sonic hedgehogChildExomeGenetics (clinical)Adaptor Proteins Signal TransducingCystic kidneyGeneticsBase SequencebiologyHomozygoteIntracellular Signaling Peptides and ProteinsMembrane ProteinsCiliary transition zoneSequence Analysis DNAOrofaciodigital Syndromesmedicine.diseaseCleft PalateCiliopathyPhenotypeMutationbiology.proteinApoptosis Regulatory ProteinsHand Deformities CongenitalSignal TransductionThe American Journal of Human Genetics
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Phenotype determining alleles in GM1 gangliosidosis patients bearing novel GLB1 mutations.

2010

Hofer D, Paul K, Fantur K, Beck M, Roubergue A, Vellodi A, Poorthuis BJ, Michelakakis H, Plecko B, Paschke E. Phenotype determining alleles in GM1 gangliosidosis patients bearing novel GLB1 mutations. GM1 gangliosidosis manifests with progressive psychomotor deterioration and dysostosis of infantile, juvenile, or adult onset, caused by alterations in the structural gene coding for lysosomal acid s-galactosidase (GLB1). In addition, allelic variants of this gene can result in Morquio B disease (MBD), a phenotype with dysostosis multiplex and entire lack of neurologic involvement. More than 100 sequence alterations in the GLB1 gene have been identified so far, but only few could be proven to …

AdolescentGenotypeNonsense mutationBlotting WesternDNA Mutational AnalysisBiologymedicine.disease_causeCell LineGenotypeChlorocebus aethiopsGeneticsmedicineMissense mutationAnimalsHumansAlleleChildGenetics (clinical)AllelesGeneticsMutationGangliosidosis GM1DysostosisInfantmedicine.diseasebeta-GalactosidasePhenotypePhenotypeGLB1Child PreschoolCOS CellsMutationClinical genetics
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Lymphoma cell apoptosis in the liver induced by distant murine cytomegalovirus infection.

2006

ABSTRACTCytomegalovirus (CMV) poses a threat to the therapy of hematopoietic malignancies by hematopoietic stem cell transplantation, but efficient reconstitution of antiviral immunity prevents CMV organ disease. Tumor relapse originating from a minimal residual leukemia poses another threat. Although a combination of risk factors was supposed to enhance the incidence and severity of transplantation-associated disease, a murine model of a liver-adapted B-cell lymphoma has previously shown a survival benefit and tumor growth inhibition by nonlethal subcutaneous infection with murine CMV. Here we have investigated the underlying antitumoral mechanism. Virus replication proved to be required, …

Adoptive cell transferProgrammed cell deathMuromegalovirusLymphoma B-CellCD30Lymphomamedicine.medical_treatmentImmunologyApoptosisHematopoietic stem cell transplantationBiologyCD8-Positive T-Lymphocytesmedicine.disease_causeLymphoma T-CellMicrobiologyVirusHerpesviridaeMiceVirologyCell Line TumormedicineAnimalsPoint MutationBone Marrow TransplantationMice Inbred BALB CHerpesviridae Infectionsmedicine.diseaseVirologyAdoptive TransferLymphomaLeukemiaLiverMice Inbred DBAInsect ScienceNIH 3T3 CellsPathogenesis and ImmunityFemaleJournal of virology
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Evolution of lactase persistence: an example of human niche construction

2011

Niche construction is the process by which organisms construct important components of their local environment in ways that introduce novel selection pressures. Lactase persistence is one of the clearest examples of niche construction in humans. Lactase is the enzyme responsible for the digestion of the milk sugar lactose and its production decreases after the weaning phase in most mammals, including most humans. Some humans, however, continue to produce lactase throughout adulthood, a trait known as lactase persistence. In European populations, a single mutation (−13910*T) explains the distribution of the phenotype, whereas several mutations are associated with it in Africa and the Middle …

Adult0106 biological sciencesAsiaNatural selectionmedicine.medical_treatmentLactoseBiology010603 evolutionary biology01 natural sciencesGene Expression Regulation EnzymologicGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesGene FrequencyLactase persistencemedicineAnimalsHumansComputer SimulationNeolithicAlleleDomesticationLactaseddc:599.9030304 developmental biology2. Zero hungerGenetics0303 health sciencesGenetic VariationLactaseArticlesBiological EvolutionEuropeDomestic animalsDairyingLactase persistenceNiche constructionGenetics PopulationMilkAfricaTraitLocal environmentCattleNiche constructionGeneral Agricultural and Biological SciencesSingle mutationPhilosophical Transactions of the Royal Society B: Biological Sciences
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CAF-like state in primary skin fibroblasts with constitutional BRCA1 epimutation sheds new light on tumor suppressor deficiency-related changes in he…

2016

Constitutive epimutations of tumor suppressor genes are increasingly considered as cancer predisposing factors equally to sequence mutations. In light of the emerging role of the microenvironment for cancer predisposition, initiation, and progression, we aimed to characterize the consequences of a BRCA1 epimutation in cells of mesenchymal origin. We performed a comprehensive molecular and cellular comparison of primary dermal fibroblasts taken from a monozygous twin pair discordant for recurrent cancers and BRCA1 epimutation, whose exceptional clinical case we previously reported in this journal. Comparative transcriptome analysis identified differential expression of extracellular matrix-r…

Adult0301 basic medicineCancer ResearchTwinsHaploinsufficiencyKetone BodiesExtracellular matrixTranscriptome03 medical and health sciencesCell Line TumormedicineHumansGenes Tumor SuppressorMolecular BiologyPDPNCells CulturedOligonucleotide Array Sequence AnalysisSkinExtracellular Matrix ProteinsbiologyBRCA1 ProteinCell growthGenes HomeoboxCancerDNA MethylationFibroblastsmedicine.diseaseGene Expression Regulation Neoplastic030104 developmental biologyCulture Media ConditionedMutationDNA methylationImmunologyCancer researchbiology.proteinCytokinesCancer-Associated FibroblastsFemaleNeoplasm Recurrence LocalACTA2TranscriptomeResearch PaperEpigenetics
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A recurrent TP63 mutation causing EEC3 and Rapp–Hodgkin syndromes

2016

The ectrodactyly, ectodermal dysplasia, and cleft lip/palate syndrome 3 (EEC3; OMIM #604292), the Rapp-Hodgkin syndrome (RHS), and various other syndromes are caused by mutations in the TP63 gene, which encodes a p53-like transcription factor. Here, we report on a woman aged 37 years and her daughter aged 3 years with the previously reported c.1028G>A (p.Arg343Gln) mutation in exon 8 of TP63. The mother lacked ectrodactyly, indicating a diagnosis of RHS, whereas the girl presented with all three major features (ectrodactyly, ectodermal dysplasia, clefting) and different minor features (including small and brittle nails, and recurrent conjunctivitis believed to be because of stenotic and blo…

Adult0301 basic medicineHeterozygoteEctodermal dysplasiamedicine.medical_specialtyEctrodactylyFoot Deformities CongenitalCleft Lipmedia_common.quotation_subjectmedicine.disease_causePathology and Forensic MedicineFingers030207 dermatology & venereal diseases03 medical and health sciencesExon0302 clinical medicineEctodermal DysplasiaTP63medicineHumansAlleleAllelesGenetics (clinical)media_commonDaughterMutationbusiness.industryTumor Suppressor ProteinsFaciesExonsGeneral Medicinemedicine.diseaseDermatologyPenetrancePedigreeCleft PalatePhenotype030104 developmental biologyAmino Acid SubstitutionChild PreschoolMutationPediatrics Perinatology and Child HealthFemaleAnatomybusinessHand Deformities CongenitalTranscription FactorsClinical Dysmorphology
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Novel hereditary angioedema linked with a heparan sulfate 3-O-sulfotransferase 6 gene mutation

2020

Background Hereditary angioedema (HAE) is a potentially fatal disorder resulting in recurrent attacks of severe swelling. It may be associated with a genetic deficiency of functional C1 inhibitor or with normal C1 inhibitor (HAEnCI). In families with HAEnCI, HAE-linked mutations in the F12, PLG, KNG1, ANGPT1, or MYOF genes have been identified. In many families with HAEnCI the genetic cause of the disease is currently unknown. Objective The aim of this study was to identify a novel disease-linked mutation for HAEnCI. Methods The study methods comprised whole exome sequencing, Sanger sequencing analysis, pedigree analysis, bioinformatic analysis of the mutation, and biochemical analysis of p…

Adult0301 basic medicineImmunologyMutantGene mutationBiologyC1-inhibitor03 medical and health sciencessymbols.namesakechemistry.chemical_compound0302 clinical medicineExome SequencingmedicineHumansImmunology and AllergyExome sequencingAged 80 and overSanger sequencingGeneticsAngioedemas HereditaryHeparan sulfateMiddle Agedmedicine.disease030104 developmental biology030228 respiratory systemchemistryMutationMutation (genetic algorithm)Hereditary angioedemasymbolsbiology.proteinFemaleSulfotransferasesJournal of Allergy and Clinical Immunology
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Phase II Study of Taselisib (GDC-0032) in Combination with Fulvestrant in Patients with HER2-Negative, Hormone Receptor–Positive Advanced Breast Canc…

2018

AbstractPurpose: This single-arm, open-label phase II study evaluated the safety and efficacy of taselisib (GDC-0032) plus fulvestrant in postmenopausal women with locally advanced or metastatic HER2-negative, hormone receptor (HR)-positive breast cancer.Patients and Methods: Patients received 6-mg oral taselisib capsules daily plus intramuscular fulvestrant (500 mg) until disease progression or unacceptable toxicity. Tumor tissue (if available) was centrally evaluated for PIK3CA mutations. Adverse events (AE) were recorded using NCI-CTCAE v4.0. Tumor response was investigator-determined using RECIST v1.1.Results: Median treatment duration was 4.6 (range: 0.9–40.5) months. All patients expe…

Adult0301 basic medicineOncologyCancer Researchmedicine.medical_specialtyDrug-Related Side Effects and Adverse ReactionsClass I Phosphatidylinositol 3-KinasesReceptor ErbB-2Phases of clinical researchBreast NeoplasmsDisease-Free SurvivalArticle03 medical and health sciences0302 clinical medicineBreast cancerInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansAdverse effectFulvestrantAgedAged 80 and overResponse rate (survey)Fulvestrantbusiness.industryImidazolesCancerMiddle Agedmedicine.diseaseOxazepines030104 developmental biologyReceptors EstrogenOncologyHormone receptor030220 oncology & carcinogenesisMutationToxicityFemalebusinessmedicine.drugClinical Cancer Research
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