Search results for "Macrophage"

showing 10 items of 781 documents

Posttranslational modifications by ADAM10 shape myeloid antigen-presenting cell homeostasis in the splenic marginal zone

2021

The spleen contains phenotypically and functionally distinct conventional dendritic cell (cDC) subpopulations, termed cDC1 and cDC2, which each can be divided into several smaller and less well-characterized subsets. Despite advances in understanding the complexity of cDC ontogeny by transcriptional programming, the significance of posttranslational modifications in controlling tissue-specific cDC subset immunobiology remains elusive. Here, we identified the cell-surface–expressed A-disintegrin-and-metalloproteinase 10 (ADAM10) as an essential regulator of cDC1 and cDC2 homeostasis in the splenic marginal zone (MZ). Mice with a CD11c-specific deletion of ADAM10 (ADAM10(ΔCD11c)) exhibited a …

MaleLangerinLymphoid TissueNotch signaling pathwayAntigen-Presenting CellsCD11cSpleenADAM10 ProteinMicePhosphatidylinositol 3-KinasesmedicineAnimalsHomeostasisMyeloid CellsProtein kinase BPI3K/AKT/mTOR pathwayCell ProliferationMultidisciplinarybiologyMacrophagesMembrane ProteinsCell DifferentiationDendritic CellsBiological SciencesCD11c AntigenCell biologyMice Inbred C57BLmedicine.anatomical_structurebiology.proteinFemaleAmyloid Precursor Protein SecretasesSignal transductionProtein Processing Post-TranslationalSpleenConventional Dendritic CellSignal TransductionProceedings of the National Academy of Sciences
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Correlation between expression of cyclooxygenase-2 and the presence of inflammatory cells in human primary hepatocellular carcinoma: Possible role in…

2005

im: To investigate the association of cyclooxygenase-2 (COX-2) expression with angiogenesis and the number and type of inflammatory cells (macrophages/Kupffer cells; mast cells) within primary hepatocellular carcinoma (HCC) tissues and adjacent non-tumorous (NT) tissues. Methods: Immunohistochemistry for COX-2, CD34, CD68 and mast cell tryptase (MCT) was performed on 14 well-characterized series of liver-cirrhosis-associated HCC patients. COX-2 expression and the number of inflammatory cells in tumor lesions and surrounding liver tissues of each specimen were compared. Moreover, COX-2, CD34 staining and the number of inflammatory cells in areas with different histological degrees within eac…

MaleLiver CancerPathologymedicine.medical_specialtyCarcinoma HepatocellularEndotheliumMacrophageAngiogenesisKupffer CellsNeovascularizationCarcinomamedicineHumansMast CellsHCCAgedInflammationbiologyNeovascularization Pathologicbusiness.industryMacrophagesLiver NeoplasmsGastroenterologyMembrane ProteinsGeneral MedicineCOX-2Middle Agedmedicine.diseasedigestive system diseasesAngiogenesimedicine.anatomical_structureMembrane proteinCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesHepatocellular carcinomabiology.proteinTumor promotionFemaleCyclooxygenaseEndothelium Vascularmedicine.symptombusiness
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Oncogene transformation can induce tolerogenicity in murine macrophages after down-regulation of immunogenicity without altering major histocompatibi…

1993

In vitro studies on cell lines may allow analyses of the mechanisms of immunogenicity and tolerogenicity in cells. We used a model of oncogenic transformation of an established murine macrophage cell line and report here that one v-mos-transformed clone expressing unaltered high amounts of MHC class I and II antigens does not induce proliferation of unprimed T cells in primary mixed lymphocyte reactions, in sharp contrast to its non-transformed parental cells. Interestingly, this clone induces specific unresponsiveness, as revealed by the lack of responsiveness of MHC-specific T cells when subsequently exposed to the pertinent MHC alloantigens in immunogenic form but unaltered MHC-third par…

MaleLymphocyteT-LymphocytesImmunologyClone (cell biology)Down-RegulationBiologyMajor histocompatibility complexImmune toleranceCell LineMiceTransformation GeneticAntigenHistocompatibility AntigensMHC class ImedicineImmune ToleranceAnimalsRNA MessengerGenes mosMice Inbred BALB CMice Inbred C3HImmunogenicityMacrophagesGeneral MedicineCell biologyClone CellsMice Inbred C57BLmedicine.anatomical_structureCell cultureImmunologybiology.proteinFemaleLymphocyte Culture Test MixedCell DivisionInterleukin-1Scandinavian journal of immunology
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Circulating CSF-1 Promotes Monocyte and Macrophage Phenotypes that Enhance Lupus Nephritis

2009

Macrophages mediate kidney disease and are prominent in a mouse model (MRL- Fas lpr ) of lupus nephritis. Colony stimulating factor-1 (CSF-1) is the primary growth factor for macrophages, and CSF-1 deficiency protects MRL- Fas lpr mice from kidney disease and systemic illness. Whether this renoprotection derives from a reduction of macrophages and whether systemic CSF-1, as opposed to intrarenal CSF-1, promotes macrophage-dependent lupus nephritis remain unclear. Here, we found that increasing systemic CSF-1 hastened the onset of lupus nephritis in MRL- Fas lpr mice. Using mutant MRL- Fas lpr strains that express high, moderate, or no systemic CSF-1, we detected a much higher tempo of kidne…

MaleMacrophage colony-stimulating factorMice Inbred MRL lprLupus nephritisMice TransgenicInflammationKidneyMonocytesMiceimmune system diseasesmedicineAnimalsHumansskin and connective tissue diseasesCell ProliferationInflammationMice Inbred BALB CMice Inbred C3HSystemic lupus erythematosusbiologyCD68business.industryMacrophage Colony-Stimulating FactorMacrophagesMonocyteGeneral MedicineMonocyte proliferationmedicine.diseaseLupus NephritisMice Inbred C57BLDisease Models AnimalBasic ResearchPhenotypemedicine.anatomical_structureIntegrin alpha MNephrologyImmunologybiology.proteinFemalemedicine.symptombusinessJournal of the American Society of Nephrology
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Human CD8+ T-cells Recognizing Peptides from Mycobacterium tuberculosis (Mtb) Presented by HLA-E Have an Unorthodox Th2-like, Multifunctional, Mtb In…

2015

Mycobacterial antigens are not exclusively presented to T-cells by classical HLA-class Ia and HLA-class II molecules, but also through alternative antigen presentation molecules such as CD1a/b/c, MR1 and HLA-E. We recently described mycobacterial peptides that are presented in HLA-E and recognized by CD8+ T-cells. Using T-cell cloning, phenotyping, microbiological, functional and RNA-expression analyses, we report here that these T-cells can exert cytolytic or suppressive functions, inhibit mycobacterial growth, yet express GATA3, produce Th2 cytokines (IL-4,-5,-10,-13) and activate B-cells via IL-4. In TB patients, Mtb specific cells were detectable by peptide-HLA-E tetramers, and IL-4 and…

MaleMacrophageQH301-705.5ImmunologyAntigen presentationBacterial ProteinMycobacterium tuberculosiHuman leukocyte antigenGATA3 Transcription FactorCD8-Positive T-LymphocytesMicrobiologyMicrobiologyMycobacterium tuberculosisImmune systemTh2 CellsGeneticHLA-EBacterial ProteinsVirologyGeneticsCytotoxic T cellHumansBiology (General)Th2 CellCytokineMolecular BiologyAntigen PresentationbiologyMacrophagesHistocompatibility Antigens Class ICD8-Positive T-LymphocyteMycobacterium tuberculosisRC581-607biology.organism_classificationBacterial Proteins; CD8-Positive T-Lymphocytes; Cytokines; Female; GATA3 Transcription Factor; Histocompatibility Antigens Class I; Humans; Macrophages; Male; Mycobacterium tuberculosis; Peptides; Th2 Cells; Antigen Presentation; Microbiology; Parasitology; Virology; Immunology; Genetics; Molecular BiologyPhenotypeVirology3. Good healthPeptideCytokinesParasitologyFemaleImmunologic diseases. AllergyPeptidesCD8HumanResearch ArticlePLoS Pathogens
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GM-CSF Programs Hematopoietic Stem and Progenitor Cells During Candida albicans Vaccination for Protection Against Reinfection

2021

More mechanistic studies are needed to reveal the hidden details of in vivo-induced trained immunity. Here, using a Candida albicans live vaccine mouse model we show that vaccination protects mice against a secondary infection and increases the number of bone marrow, and especially, splenic trained monocytes. Moreover, vaccination expands and reprograms hematopoietic stem and progenitor cells (HSPCs) early during infection and mobilize them transiently to the spleen to produce trained macrophages. Trained HSPCs are not only primed for myeloid cell production but also reprogramed to produce a greater amount of proinflammatory cytokines in response to a second challenge. Additionally, their a…

MaleMacrophagesImmunologyVaccinationHSPCsGranulocyte-Macrophage Colony-Stimulating FactorGM-CSFmyelopoiesisRC581-607Hematopoietic Stem CellscandidiasisMice Inbred C57BLMicetrained immunityReinfectionCandida albicansImmunology and AllergyAnimalsCytokinesFemaleFungal VaccinesImmunologic diseases. AllergyOriginal ResearchFrontiers in Immunology
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Gene toxicity studies on titanium dioxide and zinc oxide nanomaterials used for UV-protection in cosmetic formulations

2010

Titanium dioxide and zinc oxide nanomaterials, used as UV protecting agents in sunscreens, were investigated for their potential genotoxicity in in vitro and in vivo test systems. Since standard OECD test methods are designed for soluble materials and genotoxicity testing for nanomaterials is still under revision, a battery of standard tests was used, covering different endpoints. Additionally, a procedure to disperse the nanomaterials in the test media and careful characterization of the dispersed test item was added to the testing methods. No genotoxicity was observed in vitro (Ames' Salmonella gene mutation test and V79 micronucleus chromosome mutation test) or in vivo (mouse bone marrow…

MaleMaterials scienceBiomedical EngineeringBone Marrow CellsNanotechnologyCosmeticsGene mutationToxicologymedicine.disease_causeCell LineNanomaterialsMicechemistry.chemical_compoundSalmonellaIn vivoCricetinaeAdministration InhalationMacrophages AlveolarmedicineAnimalsRats WistarMicronuclei Chromosome-DefectiveTitaniumChromatographyMutagenicity TestsBody WeightIn vitroNanostructuresRatschemistryData Interpretation StatisticalMicronucleus testTitanium dioxideZinc OxideMicronucleusSunscreening AgentsGenotoxicityNanotoxicology
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MyD88 is dispensable for resistance toParacoccidioides brasiliensisin a murine model of blood-borne disseminated infection

2008

We have studied the role of MyD88, an adaptor protein of Toll-like receptors (TLRs), in murine defenses against Paracoccidioides brasiliensis in a model of blood-borne disseminated infection. Wild-type (WT) and MyD88-deficient mice infected intravenously with P. brasiliensis yeast cells showed an equivalent fungal burden, as well as similar levels of proinflammatory IL-1beta, IL-6, IL-12p70, tumor necrosis factor (TNF)-alpha and MIP-2, T-helper type 1 (Th1) (IFN-gamma) and Th2 cytokines (IL-4) in tissue homogenates. In vitro production of TNF-alpha, IFN-gamma and IL-12p70, by antigen-stimulated splenocytes from infected animals, was also similar in both types of mice; this production of Th1…

MaleMicrobiology (medical)medicine.medical_treatmentImmunologyNerve Tissue ProteinsMicrobiologyParacoccidioidesMicrobiologyProinflammatory cytokineMicePeritoneal cavitymedicineAnimalsHumansImmunology and AllergyLectins C-TypeMice KnockoutParacoccidioides brasiliensisbiologyMembrane ProteinsParacoccidioidesGeneral MedicineTh1 Cellsbiology.organism_classificationToll-Like Receptor 2Mice Inbred C57BLToll-Like Receptor 4Disease Models AnimalTLR2Infectious Diseasesmedicine.anatomical_structureCytokineMyeloid Differentiation Factor 88Macrophages PeritonealTLR4CytokinesTumor necrosis factor alphaParacoccidioidomycosisFungemiaFEMS Immunology & Medical Microbiology
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Blockade of MIF-CD74 Signalling on Macrophages and Dendritic Cells Restores the Antitumour Immune Response Against Metastatic Melanoma.

2018

Mounting an effective immune response against cancer requires the activation of innate and adaptive immune cells. Metastatic melanoma is the most aggressive form of skin cancer. While immunotherapies have shown a remarkable success in melanoma treatment, patients develop resistance by mechanisms that include the establishment of an immune suppressive tumor microenvironment. Thus, understanding how metastatic melanoma cells suppress the immune system is vital to develop effective immunotherapies against this disease. In this study, we find that macrophages (MOs) and dendritic cells (DCs) are suppressed in metastatic melanoma and that the Ig-CDR-based peptide C36L1 is able to restore MOs and …

MaleModels MolecularImmunologyMelanoma Experimentalchemical and pharmacologic phenomenaModels Biologicalimmune responseMiceStructure-Activity RelationshipT-Lymphocyte SubsetsAnimalsdendritic cellsNeoplasm MetastasisReceptors ImmunologicMacrophage Migration-Inhibitory FactorsMelanomaOriginal ResearchMacrophagesHistocompatibility Antigens Class IIImmunitypeptide-based immunotherapyAntigens Differentiation B-LymphocyteCD74macrophage migration inhibitory factorPeptidesProtein BindingSignal Transductionmetastatic melanomaFrontiers in immunology
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Early Cellular Reactions in Mechanically Stimulated Gingival Connective Tissue

2001

Aim: The aim of this study was to describe early cellular reactions occuring in mechnically stimulated gingival connective tissue. Material and Method: Elastic bands were inserted between the maxillary first and second molars of male rats aged 8 weeks, which were pulse-labeled with 3H-thymidine and subsequently killed in groups together with labeled control animals after periods of 1–168 hours. Autoradiographs were prepared from plastic mesiodistal sections and parameters of cell proliferation determined in gingival connective tissue sampling areas coronal to the interdental bone crest and in the central zone of the body of the papilla between the second and third molars. The incidence of m…

MaleMolarPathologymedicine.medical_specialtyTooth Movement TechniquesEndotheliumGingivaConnective tissueOrthodonticsStimulationMandibular second molarmedicineAnimalsEndotheliumFibroblastbusiness.industryMacrophagesRats Inbred StrainsFibroblastsMolarRatsVasodilationMajor duodenal papillaEndothelial stem cellmedicine.anatomical_structureConnective TissueOral SurgerybusinessCell DivisionJournal of Orofacial Orthopedics/Fortschritte der Kieferorthop�die
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