Search results for "Mali"

showing 10 items of 3900 documents

Campodeidae (Hexapoda: Diplura) from Kyrgyzstan, Central Asia, with the description of a remarkable new genus and species

2021

Samples collected in Central Asia, Kyrgyzstan, have revealed a hitherto unknown diversity of Campodeidae (Diplura) in soil and cave habitats, including a new genus and species, Kyrgyzstancampa sanare Sendra & Ferreira gen. et sp. nov., Turkmenocampa edaphica Sendra & Sánchez-García sp. nov. and a previously recognized soil-dwelling species, Campodea (Dicampa) catalana Denis, 1930. Kyrgyzstancampa sanare Sendra & Ferreira gen. et sp. nov. was collected in the deep zone of an interesting geological and cultural cave, Ak-Turpak Cave, located near the western margin of Kadamjay District, Batken Province. This genus belongs to the subfamily Campodeinae, sharing the morphology of the …

CampodeidaeAsiaArthropodaBiogeographyDipluraHexapodacavesCaveddc:590GenusCampodeidaeAnimaliabiogeographyEcology Evolution Behavior and SystematicsTaxonomygeographygeography.geographical_feature_categorybiologyEcologyArtròpodesBotanyEdaphicEntognathaBiodiversitybiology.organism_classificationQL1-991BiogeografiaQK1-989CampodeaDipluraFauna cavernícolasoil faunaZoology
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Asian cave-adapted diplurans, with the description of two new genera and four new species (Arthropoda, Hexapoda, Entognatha)

2021

Twenty-nine specimens of Diplura collected from eight caves in China and Myanmar contain two new genera, Hubeicampa Sendra & Lips gen. nov. and Mueggejapyx Sendra & Komerički gen. nov., as well as four new species, Anisuracampa ywangana Sendra & Komerički sp. nov., Hubeicampa melissa Sendra & Lips gen. et sp. nov., Pacificampa wudonghuii Sendra sp. nov. and Mueggejapyx brehieri Sendra & Komerički gen. et sp. nov. These cave-adapted taxa showcase an impressive diversity in morphological adaptation (troglomorphy) to cave ecosystems. Their sensorial equipment, setae and receptors in the cupuliform organ have unique forms (H. melissa gen. et sp. nov.), as well as the pretars…

CampodeidaeAsiaArthropodaJapygidaeBiogeographyZoologyDipluraHexapodacavesCaveddc:590lcsh:Botanylcsh:ZoologyCampodeidaeAnimalialcsh:QL1-991biogeographyEcology Evolution Behavior and SystematicsTaxonomygeography.geographical_feature_categorybiologyArtròpodesEntognathaBiodiversitybiology.organism_classificationlcsh:QK1-989GeographyTaxonBiogeografiaEntognathaDipluraJapygidaeFauna cavernícolatroglomorphy
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Malignant transformation of the liver tumour precursor cell line OC/CDE 22 by the four stereoisomeric fjord region 3,4-dihydrodiol 1,2-epoxides of be…

1995

In previous work we established the rat liver oval cell line OC/CDE 22 in order to study in vitro mechanisms of liver cell transformation. We have now exposed OC/CDE 22 cells to each of the four optically active fjord region dihydrodiol epoxides of benzo[c]phenanthrene to investigate their capacity for malignant transformation of liver cells. All four configurational isomers, which are among the most potent carcinogenic metabolites of polycyclic aromatic hydrocarbons tested in murine tumour models, malignantly transform OC/CDE 22 cells at a 2 microM dose level, resulting in a similar colony-forming efficiency in soft agar. Inoculation of the transformed cells into newborn syngeneic rats pro…

Cancer ResearchBenzo(c)phenanthreneMalignant transformationRats Sprague-Dawleychemistry.chemical_compoundLiver Neoplasms ExperimentalTumor Cells CulturedmedicineAnimalsCarcinogenConfluencyCell growthLiver cellStereoisomerismGeneral MedicinePhenanthrenesRatsCell Transformation Neoplasticmedicine.anatomical_structureLiverchemistryBiochemistryCell cultureHepatocyteCarcinogensPrecancerous ConditionsCell DivisionCarcinogenesis
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Abstract 3512: MYCN and survivin cooperatively contribute to malignant transformation of fibroblasts

2014

Abstract The oncogenes MYCN and survivin (BIRC5) maintain aggressiveness of diverse cancers including sarcomas. To investigate whether these oncogenes cooperate in initial malignant transformation, we transduced them into Rat-1 fibroblasts. Indeed, survivin enhanced MYCN-driven contact-uninhibited and anchorage-independent growth in vitro. Importantly, upon subcutaneous transplantation into mice, cells overexpressing both instead of either one of the oncogenes generated tumors with shortened latency, marked anaplasia and an increased proliferation-to-apoptosis ratio resulting in accelerated growth. Mechanistically, the increased tumorigenicity was associated with an enhanced Warburg effect …

Cancer ResearchCancerBiologymedicine.diseaseWarburg effectIn vitroMalignant transformationTransplantationOncologyImmunologySurvivinmedicineCancer researchmedicine.symptomneoplasmsAnaplasiaCancer Research
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Cytokines in cancer therapy

1989

The treatment options for patients with cancer are presently limited to surgical and radiotherapeutic strategies for localized disease and the systemic use of cytotoxic drugs for disseminated disease. So far, chemotherapy remains the mainstay for the treatment of metastatic cancer. Treatment results, however, have been stagnant particularly for the more frequent cancers such as lung cancer, breast cancer and colorectal cancer. Current research is seeking new concepts of cancer treatment, based upon a more profound understanding of tumor cell biology. The oncogenetic defect in neoplastic cells is a genetic alteration in a primordial cancer cell, which subsequently leads to clonal expansion a…

Cancer ResearchColorectal cancerCancerGeneral MedicineBiologymedicine.diseaseSomatic evolution in cancerMalignant transformationCell therapyBreast cancerOncologyCancer cellmedicineCancer researchNeoplastic transformationJournal of Cancer Research and Clinical Oncology
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Coordinate mutation and transformation of mouse fibroblasts: induction by nitroquinoline oxide and modulation by caffeine

1981

Mutation and malignant transformation were followed in the same cells. Mouse fibroblasts (C3H 10T 1/2) were mutated and transformed by 4-nitroquinoline-1-oxide with similar, approximately linear dose-responses. The presence of caffeine immediately after exposure to 4-nitroquinoline-1-oxide potently inhibited mutation and transformation at high but not at low doses of 4-nitroquinoline-1-oxide. Whilst the coordinate induction of mutation and transformation could be explained by both a common target (DNA) or a common reactive species hitting several targets, the identical modulation by a DNA repair inhibitor of both end points suggests fundamental similarities in the nature of the lesions lead…

Cancer ResearchDNA repairDrug ResistanceBiologyMalignant transformationMicechemistry.chemical_compoundCaffeinemedicineAnimalsA-DNAOuabainFibroblastCells CulturedMice Inbred C3HNitroquinolinesDrug SynergismGeneral MedicineMolecular biology4-Nitroquinoline-1-oxideTransformation (genetics)Cell Transformation Neoplasticmedicine.anatomical_structurechemistryMutationMutation (genetic algorithm)Cancer researchCaffeineDNACarcinogenesis
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GSK3β as a novel promising target to overcome chemoresistance in pancreatic cancer

2021

Pancreatic cancer is an aggressive malignancy with increasing incidence and poor prognosis due to its late diagnosis and intrinsic chemoresistance. Most pancreatic cancer patients present with locally advanced or metastatic disease characterized by inherent resistance to chemotherapy. These features pose a series of therapeutic challenges and new targets are urgently needed. Glycogen synthase kinase 3 beta (GSK3β) is a conserved serine/threonine kinase, which regulates key cellular processes including cell proliferation, DNA repair, cell cycle progression, signaling and metabolic pathways. GSK3β is implicated in non-malignant and malignant diseases including inflammation, neurodegenerative …

Cancer ResearchDNA repairDruggabilityDiseaseMalignancyPancreatic cancerHumansMedicinePharmacology (medical)GSK3BCell ProliferationPharmacologyGlycogen Synthase Kinase 3 betabusiness.industryKinaseGSK3βCancerTumor chromatin profilingOncogenesPancreatic cancermedicine.diseaseAnticancer drug combinationsPancreatic NeoplasmsInfectious DiseasesOncologyDrug Resistance NeoplasmCancer researchbusinessChemoresistanceDrug Resistance Updates
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Characterization of bep1 and bep4 antigens involved in cell interactions during Paracentrotus lividus development

1992

Abstract We have identified and partially characterised two antigens, extracted with 3% butanol, from Paracentrotus lividus embryos dissociated at the blastula stage, and encoded by the cDNA clones previously described as bep1 and bep4 (bep-butanol extracted proteins). The cDNA fragments containing the specific central portions of bep1 and bep4 were expressed as MS2 polymerase fusion proteins in Escherichia coli. These two fusion proteins, called 1C1 (bep1) and 4A1 (bep4), were injected subcutaneously into rabbits and the corresponding polyclonal antibodies generated. Western blot analysis of proteins, extracted with 3% butanol, from sea urchin embryos at the blastula stage (b.e.p.), establ…

Cancer ResearchEmbryo Nonmammaliananimal structuresRecombinant Fusion ProteinsEmbryonic DevelopmentFluorescent Antibody TechniqueParacentrotus lividusCell–cell interactionWestern blotComplementary DNAbiology.animalmedicineAnimalsMolecular BiologySea urchinCell Aggregationbiologymedicine.diagnostic_testMembrane ProteinsCell Biologybiology.organism_classificationBlastulaMolecular biologyFusion proteinPolyclonal antibodiesSea Urchinsembryonic structuresbiology.proteinDevelopmental BiologyDifferentiation
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The Compass-like Locus, Exclusive to the Ambulacrarians, Encodes a Chromatin Insulator Binding Protein in the Sea Urchin Embryo

2013

Chromatin insulators are eukaryotic genome elements that upon binding of specific proteins display barrier and/or enhancer-blocking activity. Although several insulators have been described throughout various metazoans, much less is known about proteins that mediate their functions. This article deals with the identification and functional characterization in Paracentrotus lividus of COMPASS-like (CMPl), a novel echinoderm insulator binding protein. Phylogenetic analysis shows that the CMPl factor, encoded by the alternative spliced Cmp/Cmpl transcript, is the founder of a novel ambulacrarian-specific family of Homeodomain proteins containing the Compass domain. Specific association of CMPl…

Cancer ResearchEmbryo Nonmammalianchromatin insulators genome evolution alternative splicing sea urchin embryolcsh:QH426-470RepressorSettore BIO/11 - Biologia MolecolareRegulatory Sequences Nucleic AcidHistonesGene clusterGeneticsAnimalsPromoter Regions GeneticEnhancerMolecular BiologyPhylogenyGenetics (clinical)Ecology Evolution Behavior and SystematicsGeneticsMessenger RNAbiologyBinding proteinGene Expression Regulation DevelopmentalFusion proteinChromatinNucleosomesChromatinlcsh:GeneticsEnhancer Elements GeneticNucleoproteinsHistoneSea UrchinsParacentrotusbiology.proteinInsulator ElementsCarrier ProteinsResearch ArticleProtein BindingPLoS Genetics
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Pimasertib Versus Dacarbazine in Patients With Unresectable NRAS-Mutated Cutaneous Melanoma: Phase II, Randomized, Controlled Trial with Crossover

2020

This study investigated the efficacy and safety of pimasertib (MEK1/MEK2 inhibitor) versus dacarbazine (DTIC) in patients with untreated NRAS-mutated melanoma. Phase II, multicenter, open-label trial. Patients with unresectable, stage IIIc/IVM1 NRAS-mutated cutaneous melanoma were randomized 2:1 to pimasertib (60 mg

Cancer ResearchGastroenterologypimasertiblaw.invention0302 clinical medicineRandomized controlled triallawClinical endpoint030212 general & internal medicineMalignant melanomaHazard ratioProgression-free survivalSciences bio-médicales et agricoleslcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens3. Good healthDacarbazineOncology[SDV.SP.PHARMA] Life Sciences [q-bio]/Pharmaceutical sciences/Pharmacology030220 oncology & carcinogenesismedicine.symptomPimasertibmedicine.drugQuality of lifemedicine.medical_specialtyNauseaDacarbazinemalignant melanomadacarbazine[SDV.CAN]Life Sciences [q-bio]/CancerN-(2 3-dihydroxypropyl)-1-((2-fluoro-4-iodophenyl)amino)isonicotinamideNeutropeniaN-(23-dihydroxypropyl)-1-((2-fluoro-4-iodophenyl)amino)isonicotinamidelcsh:RC254-282Article03 medical and health sciences[SDV.CAN] Life Sciences [q-bio]/CancerInternal medicinemedicineProgression-free survivalAdverse effectbusiness.industry[SDV.MHEP.DERM] Life Sciences [q-bio]/Human health and pathology/Dermatologymedicine.diseaseadverse eventsCancérologiequality of lifeAdverse events[SDV.SP.PHARMA]Life Sciences [q-bio]/Pharmaceutical sciences/Pharmacologybusinessprogression-free survival[SDV.MHEP.DERM]Life Sciences [q-bio]/Human health and pathology/Dermatology
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