Search results for "Maze Learning"

showing 10 items of 85 documents

Effects of nicotinamide on central cholinergic transmission and on spatial learning in rats

1996

High-dose nicotinamide (1000 mg/kg) leads to a minor increase of plasma choline but to a major increase of the choline concentrations in the intra- and extracellular spaces of the brain. In the hippocampus, the nicotinamide-induced increase in choline was associated with an increase in the release of acetylcholine under stimulated conditions. In young rats, nicotinamide in doses between 10 and 1000 mg/kg did not influence spatial learning, as tested in the Morris water maze. In old rats, low doses of nicotinamide were ineffective whereas the high dose of 1000 mg/kg even impaired spatial learning. The combined administration of choline and nicotinamide had a synergistic effect on brain choli…

MaleNiacinamideAgingClinical BiochemistryHippocampusMorris water navigation taskMotor ActivityPharmacologyToxicologyHippocampusSynaptic TransmissionBiochemistryCholineBehavioral Neurosciencechemistry.chemical_compoundCognitionParasympathetic Nervous SystemmedicineExtracellularAnimalsCholineRats WistarMaze LearningBiological PsychiatryBrain ChemistryPharmacologyNicotinamideBiological activityAcetylcholineRatschemistryBiochemistryCholinergicExtracellular SpaceAcetylcholinemedicine.drugPharmacology Biochemistry and Behavior
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Spatial learning in male mice with different levels of aggressiveness: effects of housing conditions and nicotine administration

2003

The main aim of the present investigation was to evaluate the possible modulation of spatial learning ability by housing conditions and level of aggressiveness in mice, also testing whether differences in locomotion and anxiety could influence this relationship. Additionally, we have examined effects of nicotine in the acquisition and retention of a spatial learning task in groups of mice differing in these variables. NMRI male mice were either group-housed or individually housed for 30 days and then classified into mice with short (SAL) and long (LAL) attack latency after a pre-screening agonistic encounter. Locomotor activity and baseline levels of anxiety of these groups were evaluated i…

MaleNicotinemedicine.medical_specialtyElevated plus mazeTime Factorsmedicine.drug_classSpatial BehaviorEscape responseWater mazeAnxietyMotor ActivitySocial EnvironmentAnxiolyticDevelopmental psychologyDiscrimination LearningNicotineMiceBehavioral NeuroscienceEscape ReactionInternal medicineReaction TimemedicineAnimalsNicotinic AgonistsMaze LearningAnalysis of VarianceBehavior AnimalDose-Response Relationship DrugHousing AnimalAggressionEndocrinologyNicotinic agonistSocial IsolationAnxiogenicAnalysis of variancePsychologymedicine.drugBehavioural Brain Research
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Behavioral effects of combined environmental enrichment and chronic nicotine administration in male NMRI mice

2013

Abstract Environmental enrichment (EE) is an experimental paradigm which provides sensory, social, physical and cognitive stimulation for rodents. Experimental evidence indicates that this type of housing induces different neurobiological and behavioral changes. However, few studies have evaluated the consequences of combined exposure to an enriched environment and nicotine administration during a critical period of development such as adolescence. Taking into account previous studies, it can be hypothesized that a chronic treatment with nicotine would modulate the effects of rearing animals in enriched environments. In the current study, our main aim was to evaluate the effects of EE and c…

MaleNicotinemedicine.medical_specialtyTime FactorsDrinkingExperimental and Cognitive PsychologyEnvironmentInhibitory postsynaptic potentialDrug Administration ScheduleNicotineMiceBehavioral Neurosciencechemistry.chemical_compoundInternal medicineAnimals Outbred StrainsAvoidance LearningmedicineAnimalsNicotinic AgonistsCotinineMaze LearningAnalysis of VarianceEnvironmental enrichmentBehavior AnimalBody WeightInhibition PsychologicalEndocrinologyChronic nicotinechemistryNmri miceAnesthesiaExploratory BehaviorAnxietyCholinergicmedicine.symptomPsychologyCotinineLocomotionmedicine.drugPhysiology & Behavior
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The use of the Emotional-Object Recognition as an assay to assess learning and memory associated to an aversive stimulus in rodents

2016

Abstract Background Emotionally salient experiences induce the formation of explicit memory traces, besides eliciting automatic or implicit emotional memory in rodents. This study aims at investigating the implementation of a novel task for studying the formation of limbic memory engrams as a result of the acquisition- and retrieval- of fear-conditioning – biased declarative memory traces, measured by animal discrimination of an “emotional-object”. Moreover, by using this new method we investigated the potential interactions between stimulation of cannabinoid transmission and integration of emotional information and cognitive functioning. New method The Emotional-Object Recognition task is …

MaleNociceptionLimbic memory engramCB1 receptorMorpholinesConditioning ClassicalExplicit emotional memoryNaphthalenesSpatial memory03 medical and health sciences0302 clinical medicineExplicit memoryAvoidance LearningSemantic memoryAnimalsVisual short-term memoryRats WistarMaze LearningGeneralLiterature_REFERENCE(e.g.dictionariesencyclopediasglossaries)Episodic memoryMethods used to study memoryAnalgesicsAnalysis of VarianceNeuroscience (all)Long-term memoryGeneral NeuroscienceRecognition PsychologyFearElectric Stimulation030227 psychiatryBenzoxazinesRatsObject discriminationExploratory BehaviorMemory consolidationCuesPsychology030217 neurology & neurosurgeryLocomotionCognitive psychology
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Effects of nicotine on spatial learning in C57BL mice

2000

In the present study, the effects of nicotine on spatial memory in C57BL/6J mice was evaluated. Mice were trained in a water maze during four daily sessions of three trials each. In the first experiment, nicotine (0.7 and 0.35 mg/kg) or saline was administered once daily for 4 days, 15 min before the start of daily training: an impairment of performance of the water maze was observed in the group treated with 0.7 mg/kg of nicotine. In the second experiment, nicotine (0.7 and 0.35 mg/kg) or saline was administered from the 5 days prior to the beginning of the task and during the 4 days of acquisition. The results indicated an improvement in the rate of learning in the 9-day nicotine treated …

MalePharmacologyNicotineDose-Response Relationship Drugbusiness.industrymedicine.medical_treatmentWater mazeDrug Administration ScheduleMice Inbred C57BLNicotineMicePsychiatry and Mental healthEscape ReactionOrientationAnesthesiaMental RecallReaction TimeSpatial learningAnimalsMedicineOnce dailyMaze LearningbusinessSalinemedicine.drugBehavioural Pharmacology
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Behavioural and neurotoxic long-lasting effects of MDMA plus cocaine in adolescent mice

2008

The poly-drug pattern is the most common among MDMA users, with cocaine being a frequently associated drug. The aim of the present work was to evaluate the behavioural and neurotoxic long-term effects of exposure during adolescence to MDMA alone or plus cocaine. Mice of 28 to 30 days of age received a treatment of two daily injections of an identical dose of MDMA (5, 10 or 20 mg/kg), alone or plus cocaine (25 mg/kg), for 3 days (6 administrations). Three weeks after receiving MDMA, an increase in the time dedicated by the animals to social contacts with their conspecifics was observed, whilst their behaviour in the elevated plus maze showed no differences from that of non-treated mice. Afte…

MaleSerotoninElevated plus mazemedicine.drug_classDopamineN-Methyl-34-methylenedioxyamphetamineMotor ActivityPharmacologyAnxiolyticBody TemperatureMicechemistry.chemical_compoundCocaineDopaminemental disordersmedicineAnimalsMaze LearningSocial BehaviorNeurotransmitterPharmacologyBehavior AnimalLocal anestheticDopaminergicBrainMDMACorpus StriatumchemistrySerotoninPsychologypsychological phenomena and processesmedicine.drugEuropean Journal of Pharmacology
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Behavioral deficits induced by lead exposure are accompanied by serotonergic and cholinergic alterations in the prefrontal cortex

2013

The effects of long-term lead (Pb) exposure producing a blood Pb concentration of lower than 20 μg/dL, i.e. below that associated with overt neurological deficits in occupationally exposed individuals, was studied in adult rats. In order to assess gender differences, we performed parallel behavioral experiments in male and female rats. Exposure to Pb acetate (50 ppm in drinking water) for 6 months induced motor and cognitive alterations, however these effects were gender- and task-dependent. Chronic lead exposure impaired spatial learning assessed in the Morris water maze test (MWM) in both genders, whereas it only induced hyperactivity in the open field and impaired motor coordination in t…

MaleSerotoninmedicine.medical_specialtyDopamineMicrodialysisPrefrontal CortexMorris water navigation taskPoison controlMotor ActivitySerotonergicOpen fieldCellular and Molecular NeuroscienceInternal medicinemedicineAnimalsRats WistarMaze LearningPrefrontal cortexEnvironmental ExposureCell BiologyAcetylcholineRatsEndocrinologyLeadCholinergicFemaleSerotoninPsychologyNeuroscienceAcetylcholinemedicine.drugNeurochemistry International
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Effect of intermittent exposure to ethanol and MDMA during adolescence on learning and memory in adult mice

2012

Abstract Background Heavy binge drinking is increasingly frequent among adolescents, and consumption of 3,4-methylenedioxymethamphetamine (MDMA) is often combined with ethanol (EtOH). The long-lasting effects of intermittent exposure to EtOH and MDMA during adolescence on learning and memory were evaluated in adult mice using the Hebb-Williams maze. Methods Adolescent OF1 mice were exposed to EtOH (1.25 g/kg) on two consecutive days at 48-h intervals over a 14-day period (from PD 29 to 42). MDMA (10 or 20 mg/kg) was injected twice daily at 4-h intervals over two consecutive days, and this schedule was repeated six days later (PD 33, 34, 41 and 42), resulting in a total of eight injections. …

MaleSerotoninmedicine.medical_specialtyMDMA34-Dihydroxyphenylacetic acidDopamineN-Methyl-34-methylenedioxyamphetamineCognitive NeuroscienceBinge drinkingStriatumHippocampuslcsh:RC346-429MiceBehavioral Neurosciencechemistry.chemical_compoundSerotonin AgentsMemoryDopamineSerotonin AgentsInternal medicinemental disordersmedicineAnimalsLearningHippocampus (mythology)Maze Learninglcsh:Neurology. Diseases of the nervous systemBiological PsychiatryBehavior AnimalEthanolResearchMDMAGeneral MedicineHydroxyindoleacetic AcidCorpus StriatumEndocrinologychemistryAnesthesia34-Dihydroxyphenylacetic AcidHebb Williams mazeSerotoninPsychologypsychological phenomena and processesmedicine.drugBehavioral and Brain Functions
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TLR4 response mediates ethanol-induced neurodevelopment alterations in a model of fetal alcohol spectrum disorders

2017

Background Inflammation during brain development participates in the pathogenesis of early brain injury and cognitive dysfunctions. Prenatal ethanol exposure affects the developing brain and causes neural impairment, cognitive and behavioral effects, collectively known as fetal alcohol spectrum disorders (FASD). Our previous studies demonstrate that ethanol activates the innate immune response and TLR4 receptor and causes neuroinflammation, brain damage, and cognitive defects in the developmental brain stage of adolescents. We hypothesize that by activating the TLR4 response, maternal alcohol consumption during pregnancy triggers the release of cytokines and chemokines in both the maternal …

MaleSerum0301 basic medicineChemokineDevelopmental Disabilitiesmedicine.medical_treatmentlcsh:RC346-429MiceMyelin0302 clinical medicineNeuroinflammationPregnancyTLR4Maternal BehaviorFetal alcohol spectrum disordersMice KnockoutMicrogliabiologyGeneral NeuroscienceAge FactorsBrainCerebral cortexBehavior impairmentsmedicine.anatomical_structureCytokineNeurologyPrenatal Exposure Delayed EffectsCytokinesFemalemedicine.symptomMyelin ProteinsAmniotic fluidmedicine.medical_specialtyOffspringImmunologyNerve Tissue ProteinsBrain damage03 medical and health sciencesCellular and Molecular NeuroscienceInternal medicineAvoidance LearningmedicineAnimalsMaze Learninglcsh:Neurology. Diseases of the nervous systemNeuroinflammationEthanolbusiness.industryResearchBody WeightCentral Nervous System DepressantsMice Inbred C57BLToll-Like Receptor 4Disease Models AnimalMicroscopy Electron030104 developmental biologyEndocrinologyAnimals NewbornPrenatal ethanol exposureImmunologybiology.proteinTLR4business030217 neurology & neurosurgeryJournal of Neuroinflammation
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Short-term adaptation of conditioned fear responses through endocannabinoid signaling in the central amygdala

2010

International audience; Both, the cannabinoid receptor type 1 (CB1) and the central nucleus of the amygdala (CeA) are known to play crucial roles in the processing of fear and anxiety, whereby they appear to be especially involved in the control of fear states. However, in contrast to many other brain regions including the cortical subregions of the amygdala, the existence of CB1 in the CeA remains enigmatic. Here we show that CB1 is expressed in the CeA of mice and that CB1 in the CeA mediates short-term synaptic plasticity, namely depolarization-induced suppression of excitation (DSE) and inhibition (DSI). Moreover, the CB1 antagonist AM251 increased both excitatory and inhibitory postsyn…

MaleTime FactorsAction PotentialsAnxietyExtinction PsychologicalGABA AntagonistsPropanolaminesMice0302 clinical medicinePiperidinesReceptor Cannabinoid CB1Adaptation PsychologicalConditioning PsychologicalMoodFear conditioningHabituationStress DisordersMice Knockout0303 health sciencesBehavior AnimalCentral nucleus of the amygdalaValineFearExtinctionAmygdalaPyridazinesPsychiatry and Mental healthmedicine.anatomical_structureOriginal ArticlePsychologypsychological phenomena and processesSignal TransductionSensory Receptor CellsNeurophysiologyIn Vitro TechniquesInhibitory postsynaptic potentialAmygdala03 medical and health sciencesQuinoxalinesCannabinoid Receptor ModulatorsmedicineAnimalsMaze Learning030304 developmental biologyPharmacologyFear processing in the brainLearning & MemoryCannabinoidsExtinction (psychology)Phosphinic AcidsElectric StimulationMice Inbred C57BLGene Expression Regulationnervous systemSynaptic plasticityPyrazolesNeuroscienceExcitatory Amino Acid Antagonists030217 neurology & neurosurgeryEndocannabinoidsConditioning
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