Search results for "Methylenetetrahydrofolate reductase"

showing 10 items of 43 documents

Superficial thrombophlebitis in varicose vein disease: the particular role of methylenetetrahydrofolate reductase.

2010

Background The purpose of this study was to compare the genetic background of superficial (SVT) and deep vein thrombosis (DVT). Methods Factor V (FV)-Leiden (G16891A)-, factor II(G20210A)-mutations, protein C- and S, as well as methylenetetrahydrofolate reductase (MTHFR) polymorphisms at C677T and A1298C, and serum homocysteine levels (hcy) were determined in 29 patients with SVT and 26 with DVT. Findings FV- and –II-mutations were less frequent in patients with SVT (2/3) compared with DVT (9/5), respectively ( P < 0.002 in case of FV). However, the frequency of the MTHFR C677T polymorphism was significantly higher in patients with SVT compared with DVT (CT 12 versus 10, and TT 7 versus …

AdultMalemedicine.medical_specialtyPathologyDeep veinMutation MissenseDiseaseGastroenterologyVaricose VeinsInternal medicineVaricose veinsGenotypemedicineHumansSuperficial thrombophlebitisGenetic Predisposition to DiseaseHomocysteineAgedAged 80 and overMethylenetetrahydrofolate Dehydrogenase (NADP)Venous Thrombosisbiologybusiness.industryFactor VGeneral MedicineMiddle AgedThrombophlebitismedicine.diseaseThrombosisVenous thrombosismedicine.anatomical_structureAmino Acid SubstitutionMethylenetetrahydrofolate reductasebiology.proteinFemaleProthrombinmedicine.symptomCardiology and Cardiovascular MedicinebusinessPhlebology
researchProduct

Gene-diet interaction in plasma lipid response to plant sterols and stanols: A review of clinical trials

2021

Abstract Plant sterols and stanols (PS) are well known for their cholesterol-lowering effect by reducing intestinal absorption of cholesterol. However, genetic factors modulate the low-density lipoprotein cholesterol (LDL-C) response to PS therapy. This review examines clinical trials evaluating the impact of the main genes associated with response of plasma lipid concentrations to PS intake: APOE, CYP7A1, ABCG5/G8, NPC1L1, CETP, APOA4/A5, SCARBI, HMGCR, PPARα, LIPC, MTHFR and LPA. Evidence indicates that carriers of mutant allele of the CYP7A1 c. −204 A > C variant experience a greater plasma cholesterol reduction after PS intake, although there is discrepancy for the rest of genetic varia…

Apolipoprotein Emedicine.medical_specialtyLipid-lowering effectMedicine (miscellaneous)Cholesterol 7 alpha-hydroxylaseInterindividual variabilityIntestinal absorptionchemistry.chemical_compoundAPOA4Internal medicinemedicineTX341-641NutrigeneticsGen-dietNutrition and DieteticsbiologyNutrition. Foods and food supplyCholesterolbusiness.industryPlant sterols/stanolsClinical trialEndocrinologychemistryMethylenetetrahydrofolate reductaseABCG5biology.proteinlipids (amino acids peptides and proteins)businessFood ScienceJournal of Functional Foods
researchProduct

MTHFR 677C → T genotype modulates the effect of a 5-year supplementation with B-vitamins on homocysteine concentration: The SU.FOL.OM3 randomized con…

2018

Aims To study how MTHFR 677C→T genotype modulates the effect of supplementation with B-vitamins on total homocysteine (tHcy) and B-vitamin concentrations. Methods 2381 patients with a personal history of cardiovascular disease were randomly assigned to one of four groups: 1) B-vitamins alone (560 μg of 5-methyl-THF, 3 mg of vitamin B6 and 20 μg of vitamin B12), 2) n-3 fatty acids alone (600 mg of EPA and DHA in a 2:1 ratio), 3) B-vitamins and n-3 fatty acids, and 4) placebo. Participants were followed up for 4.7 years. At baseline and annually thereafter, biological parameters were assessed. Multivariate and linear mixed models were fit to study the interaction between B-vitamins and MTHFR …

B VitaminsMaleHomocysteinePhysiologylcsh:Medicine[SDV.GEN] Life Sciences [q-bio]/Genetics030204 cardiovascular system & hematologyBiochemistryGastroenterologychemistry.chemical_compound0302 clinical medicineBlood plasmaGenotypeMedicine and Health Sciences030212 general & internal medicinelcsh:ScienceHomocysteine[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyMultidisciplinarybiologyOrganic CompoundsFatty AcidsPyridoxineVitaminsMiddle AgedLipidsBody Fluids3. Good healthChemistryBloodCardiovascular DiseasesCreatininePhysical SciencesVitamin B ComplexFemaleAnatomyResearch Articlemedicine.medical_specialtyGenotypePlaceboBlood PlasmaCobalamins03 medical and health sciencesFolic AcidDouble-Blind MethodInternal medicine[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyVitamin B12Methylenetetrahydrofolate Reductase (NADPH2)[SDV.GEN]Life Sciences [q-bio]/GeneticsCreatininePolymorphism Geneticbusiness.industryOrganic Chemistrylcsh:RChemical CompoundsBiology and Life Sciences[SDV.AEN] Life Sciences [q-bio]/Food and NutritionB vitaminschemistry[SDV.SPEE] Life Sciences [q-bio]/Santé publique et épidémiologieMethylenetetrahydrofolate reductaseDietary Supplementsbiology.protein[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologielcsh:Qbusiness[SDV.AEN]Life Sciences [q-bio]/Food and NutritionBiomarkers[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyPLoS ONE
researchProduct

Buerger`s Disease and Hyperhomocysteinemia: Is there a Relationship?

2009

Thromboangiitis obliterans, also known as Buerger's disease, is a cause of juvenile lower limb ischaemia. Buerger's disease is idiopathic and one of diagnostic criteria is the absence of atherosclerotic risk factors other than smok- ing. A possible involvement of thrombophilia has been investigated and the role of hyperhomocysteinemia is still matter of discussion. We describe 9 patients with Buerger's disease followed-up for the past 3 years. We found a significant in- crease in circulating homocysteine levels (mean: 31.6 in patients vs 8.2 μmol/L in control subjects). We also analyzed the C677T mutation of MTHFR; 5/9 Buerger's patients were heterozygotes and 4/9 homozygotes for the mutati…

Buerger's diseasemedicine.medical_specialtyHyperhomocysteinemiaPathologyHomocysteinebiologybusiness.industryDiseasemedicine.diseaseThrombophiliaGastroenterologyPathogenesischemistry.chemical_compoundchemistrythrombophilia hyperhomocysteinemia MTHFR Buerger's diseaseInternal medicineMethylenetetrahydrofolate reductasemedicinebiology.proteinEndothelial dysfunctionbusinessThe Open Atherosclerosis & Thrombosis Journal
researchProduct

The Correlation Between Abnormal Uterine Artery Flow in the First Trimester and Genetic Thrombophilic Alteration: A Prospective Case-Controlled Pilot…

2020

Introduction. Evaluation of the first trimester uterine artery flow can predict the development of obstetrical complications. A genotype, making women prone to microthrombi. constitutes the main known susceptibility factor for anomalous development of placenta. Our aim was to study whether polymorphisms of 10 genes leading to blood clotting abnormalities are related to abnormal uterine artery blood flow in the first trimester, and may predict placenta-related diseases. Material and methods. In primary analyses we included 19 singleton pregnancies with abnormal blood flow in the uterine arteries during the first trimester of gestation, and 24 matched control with normal flow patterns. All pa…

Clinical BiochemistryPhysiology030204 cardiovascular system & hematologyArticlePreeclampsiaCorrelation03 medical and health sciences0302 clinical medicinegenetic polymorphismsPlacentamedicine.arteryGenotypemedicineUterine arterylcsh:R5-920030219 obstetrics & reproductive medicineabnormal uterine artery flowbiologybusiness.industryBlood flowmedicine.diseasemedicine.anatomical_structureMethylenetetrahydrofolate reductasebiology.proteinGestationHuman medicinebusinesslcsh:Medicine (General)Diagnostics
researchProduct

Concentración plasmática de homocisteína: relación con los niveles plasmáticos de ácido fólico y con el polimorfismo 677C → T de la 5,10-metilenotetr…

2002

Antecedentes: El aumento moderado de la homocisteína plasmática en niños se ha relacionado con infartos cerebrales y trombosis venosas y con los antecedentes familiares de enfermedad coronaria prematura (ECP). La determinación de homocisteína en la infancia y el estudio de los factores que determinan su concentración podría ser importante para la prevención primaria de la ECP. Objetivo: Detectar algún caso de hiperhomocistinemia y valorar su relación con la concentración plasmática de ácido fólico y el polimorfismo 677C → T de la 5,10-metilenotetrahidrofolato reductasa (MTHFR). Métodos: Se ha estudiado mediante la regresión lineal múltiple la relación entre la concentración plasmática de ho…

Folic acidMethylenetetrahydrofolate reductasePediatrics Perinatology and Child HealthHomocysteinePediatricsRJ1-570Anales de Pediatría
researchProduct

Does Down's syndrome support the homocysteine theory of atherogenesis?

2006

Abstract Down syndrome (DS) is generally considered as an “atheroma-free model”. In this preliminary study, we investigated homocysteine, folate and Vitamin B 12 levels in 13 DS patients (male, average age 60 years) and 20 age-matched individuals. We also studied lipid fractions, and polymorphisms for Cystothionine β-synthase (CBS), 5,10-methyl-tetrahydro-folate reductase (MTHFR) and apolipoprotein E (Apo-E) genes. However, DS patients with the MTHFR TT genotype showed an increased of plasma homocysteine (tHcy). Our results indicate that this group of “healthy old” Down syndrome patients, although showing some classical biochemical risk factors for atherosclerosis, did not suffer clinical c…

GeneticsApolipoprotein EAgingDown syndromemedicine.medical_specialtyHealth (social science)biologyApolipoprotein BHomocysteinebusiness.industrymedicine.diseasechemistry.chemical_compoundEndocrinologychemistryMethylenetetrahydrofolate reductaseInternal medicineGenotypebiology.proteinMedicineVitamin B12Geriatrics and GerontologybusinessTrisomyGerontologyArchives of Gerontology and Geriatrics
researchProduct

Genotype and Allele Frequencies of Drug-Metabolizing Enzymes and Drug Transporter Genes Affecting Immunosuppressants in the Spanish White Population

2013

Interpatient variability in drug response can be widely explained by genetically determined differences in metabolizing enzymes, drug transporters, and drug targets, leading to different pharmacokinetic and/or pharmacodynamic behaviors of drugs. Genetic variations affect or do not affect drug responses depending on their influence on protein activity and the relevance of such proteins in the pathway of the drug. Also, the frequency of such genetic variations differs among populations, so the clinical relevance of a specific variation is not the same in all of them. In this study, a panel of 33 single nucleotide polymorphisms in 14 different genes (ABCB1, ABCC2, ABCG2, CYP2B6, CYP2C19, CYP2C…

GenotypeCYP2B6Nod2 Signaling Adaptor ProteinOrganic Anion TransportersSingle-nucleotide polymorphismCYP2C19PharmacologyPolymorphism Single NucleotideWhite PeopleCytochrome P-450 Enzyme SystemGene FrequencyGenetic variationGenotypeHumansPharmacology (medical)ATP Binding Cassette Transporter Subfamily B Member 1GlucuronosyltransferaseAllele frequencyCYP2C9Methylenetetrahydrofolate Reductase (NADPH2)PharmacologyGeneticsbiologyMethyltransferasesMultidrug Resistance-Associated Protein 2Tissue DonorsTransplant RecipientsSpainInactivation MetabolicUDP-Glucuronosyltransferase 1A9biology.proteinSLCO1B1Immunosuppressive AgentsTherapeutic Drug Monitoring
researchProduct

No major role for periconceptional folic acid use and its interaction with theMTHFR C677Tpolymorphism in the etiology of congenital anorectal malform…

2014

Background: Both genetic and nongenetic factors are suggested to be involved in the etiology of congenital anorectal malformations (ARM). Maternal periconceptional use of folic acid supplements were inconsistently suggested to play a role in the prevention of ARM. Therefore, we investigated independent associations and interactions of maternal periconceptional folic acid supplement use and the infant and maternal MTHFR (methylenetetrahydrofolate reductase) C677T polymorphisms with the risk of ARM and subgroups of ARM. Methods: A case-control study was conducted among 371 nonsyndromic ARM cases and 714 population-based controls born between 1990 and 2012 using maternal questionnaires and DNA…

GynecologyEmbryologymedicine.medical_specialtyeducation.field_of_studyPregnancybiologybusiness.industryObstetricsPopulationGeneral MedicineOdds ratiomedicine.diseaseConfidence intervalMethylenetetrahydrofolate reductasePediatrics Perinatology and Child HealthPediatric surgerymedicineEtiologybiology.proteinImperforate anuseducationbusinessDevelopmental BiologyBirth Defects Research Part A: Clinical and Molecular Teratology
researchProduct

Thrombophilic genetic factors PAI-1 4G-4G and MTHFR 677TT as risk factors of alcohol, cryptogenic liver cirrhosis and portal vein thrombosis, in a Ca…

2015

The thrombophilic genetic factors (THRGFs), PAI-1 4G-4G, MTHFR 677TT, V Leiden 506Q and Prothrombin 20210A, were studied as risk factors in 865 Caucasian patients with liver cirrhosis, consecutively enrolled from June 2008 to January 2014. A total of 582 HCV, 80 HBV, 94 alcohol, (82 with more than one etiologic factor) and 191 cryptogenic patients with liver cirrhosis had been consecutively enrolled; 243 patients showed portal vein thrombosis (PVT). At least one of the above THRGFs was present in 339/865 patients (39.2%). PAI-1 4G-4G and MTHFR 677TT were the most frequent THRGFs, statistically significant in patients with alcohol, cryptogenic liver cirrhosis, and PVT: respectively 24 and 28…

Liver CirrhosisMalemedicine.medical_specialtyAlcoholic liver diseaseCirrhosisAlcohol DrinkingAlcoholThrombophiliaLogistic regressionGastroenterologyPolymorphism Single NucleotideWhite Peoplechemistry.chemical_compoundRisk FactorsInternal medicineGenotypePlasminogen Activator Inhibitor 1GeneticsmedicineHumansGenetic Predisposition to DiseaseGenetic Association StudiesMethylenetetrahydrofolate Reductase (NADPH2)Venous ThrombosisbiologyPortal VeinGeneral Medicinemedicine.diseasedigestive system diseasesPortal vein thrombosischemistryMethylenetetrahydrofolate reductasebiology.proteinFemaleGene
researchProduct