Search results for "Microarray analysi"

showing 10 items of 109 documents

Enhanced production of CCL18 by tolerogenic dendritic cells is associated with inhibition of allergic airway reactivity

2012

Background IL-10–treated dendritic cells (DCs) have been shown to inhibit T-cell responses through induction of anergy and regulatory T cells in various model systems, including allergic inflammation, but the factors being involved in this inhibition are still unclear. Objective This study set out to analyze such factors produced or induced by IL-10–treated DCs by using gene expression profiling and to explore their function. Methods CD4 + T cells from allergic donors were stimulated with autologous monocyte-derived allergen-pulsed mature DCs or IL-10–treated DCs. After 24 hours, the transcriptional profile was analyzed by using Affymetrix technology. Results were validated by using quantit…

CD4-Positive T-LymphocytesChemokinemedicine.medical_treatmentImmunologyT-Lymphocytes RegulatoryAllergic inflammationMiceMice Inbred NODImmune ToleranceRespiratory HypersensitivitymedicineAnimalsHumansImmunology and AllergyCCL17dendritic cellsCells CulturedT(H)1/T(H)2 cellsMice KnockoutbiologyCCL18FOXP3regulationDendritic cellMicroarray AnalysisallergyCoculture TechniquesInterleukin-10Disease Models Animalhumanized miceCytokineChemokines CCImmunologyHumanized mousebiology.proteinChemokinesTranscriptomeJournal of Allergy and Clinical Immunology
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Nupr1-Aurora Kinase A Pathway Provides Protection against Metabolic Stress-Mediated Autophagic-Associated Cell Death

2012

Abstract Purpose: The limited supply of oxygen and nutrients is thought to result in rigorous selection of cells that will eventually form the tumor. Experimental Design: Nupr1 expression pattern was analyzed in human tissue microarray (TMA) and correlated with survival time of the patient. Microarray analysis was conducted on MiaPaCa2 cells subjected to metabolic stress in Nupr1-silenced conditions. DNA repair and cell cycle–associated gene expression was confirmed by real-time quantitative PCR (qRT-PCR). Nupr1 and AURKA protective role were analyzed using RNA interference (RNAi) silencing or overexpression. DNA damage and autophagy were analyzed by Western blot analysis and immunofluoresc…

Cancer ResearchProgrammed cell deathCell SurvivalDNA damageDNA repairAdenocarcinomaProtein Serine-Threonine KinasesBiologyAurora KinasesStress PhysiologicalCell Line TumorAutophagyBasic Helix-Loop-Helix Transcription FactorsHumansGene silencingAurora Kinase ARegulation of gene expressionGene knockdownMicroarray analysis techniquesAURKA GeneMolecular biologyCell HypoxiaNeoplasm ProteinsCell biologyGene Expression Regulation NeoplasticGlucoseOncologyRNA InterferenceCarcinoma Pancreatic DuctalClinical Cancer Research
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Human Embryonic Stem Cell Derived Hepatocyte-Like Cells as a Tool for In Vitro Hazard Assessment of Chemical Carcinogenicity

2011

Hepatocyte-like cells derived from the differentiation of human embryonic stem cells (hES-Hep) have potential to provide a human relevant in vitro test system in which to evaluate the carcinogenic hazard of chemicals. In this study, we have investigated this potential using a panel of 15 chemicals classified as noncarcinogens, genotoxic carcinogens, and nongenotoxic carcinogens and measured whole-genome transcriptome responses with gene expression microarrays. We applied an ANOVA model that identified 592 genes highly discriminative for the panel of chemicals. Supervised classification with these genes achieved a cross-validation accuracy of > 95%. Moreover, the expression of the response g…

Carcinogenicity TestsCellular differentiationCell Culture TechniquesGene Expressionsystems toxicologyComputational biologyBiologyToxicologymedicine.disease_causeHazardous SubstancesTranscriptomecomputational biologyCytochrome P-450 Enzyme SystemNaturvetenskapmedicinecarcinogenicityHumansMicroscopy Phase-ContrastEmbryonic Stem CellsCarcinogenAnalysis of VarianceDose-Response Relationship DrugReverse Transcriptase Polymerase Chain ReactionMicroarray analysis techniquesGene Expression ProfilingReproducibility of Resultsrisk assessmentCell DifferentiationMicroarray AnalysisImmunohistochemistryEmbryonic stem cellMolecular biologyGene expression profilingCell culturetoxicogenomicsCarcinogensHepatocytesNatural SciencesCarcinogenesisToxicological Sciences
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Dystrophin-deficiency increases the susceptibility to doxorubicin-induced cardiotoxicity

2007

Background and aim: The clinical use of doxorubicin (DOX) and other anthracyclines is limited by a dosage-dependent cardiotoxicity, which can lead to cardiomyopathy. The role of the individual genetic makeup in this disorder is poorly understood. Alterations in genes encoding cardiac cytoskeleton or sarcolemma proteins may increase the susceptibility to doxorubicin-related cardiotoxicity. Methods: Female dystrophin-deficient mice (MDX) and age-matched wild-type mice underwent chronic treatment with doxorubicin. Cardiac function and tissue damage were assessed by echocardiography and histopathology, respectively. Gene expression changes were investigated using microarrays. Results: DOX treat…

Cardiac function curveProgrammed cell deathPathologymedicine.medical_specialtyHeart DiseasesCytoskeleton organizationCardiomyopathyGene Expression030204 cardiovascular system & hematologyDystrophinMice03 medical and health sciences0302 clinical medicineRisk FactorsmedicineAnimalsDoxorubicinUltrasonography030304 developmental biology0303 health sciencesCardiotoxicityAntibiotics AntineoplasticSarcolemmabiologybusiness.industryGenetic VariationMicroarray Analysismedicine.disease3. Good healthDoxorubicinDisease Progressionbiology.proteinCancer researchFemaleDisease SusceptibilityCardiology and Cardiovascular MedicineDystrophinbusinessmedicine.drugEuropean Journal of Heart Failure
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Multiple sclerosis patient-derived CSF induces transcriptional changes in proliferating oligodendrocyte progenitors.

2014

Background: Cerebrospinal fluid (CSF) is in contact with brain parenchyma and ventricles, and its composition might influence the cellular physiology of oligodendrocyte progenitor cells (OPCs) thereby contributing to multiple sclerosis (MS) disease pathogenesis. Objective: To identify the transcriptional changes that distinguish the transcriptional response induced in proliferating rat OPCs upon exposure to CSF from primary progressive multiple sclerosis (PPMS) or relapsing remitting multiple sclerosis (RRMS) patients and other neurological controls. Methods: We performed gene microarray analysis of OPCs exposed to CSF from neurological controls, or definitive RRMS or PPMS disease course. R…

Cell physiologyAdultPathologymedicine.medical_specialtyTranscription GeneticGalectin 3GalectinsImmunocytochemistryBiologyArticleCerebrospinal fluidMultiple Sclerosis Relapsing-RemittingNeural Stem CellsmedicineAnimalsHumansProgenitor cellCells CulturedCell ProliferationCerebrospinal FluidMultiple sclerosisBrainHuman brainBlood ProteinsMultiple Sclerosis Chronic Progressivemedicine.diseaseMicroarray AnalysisNeural stem cellOligodendrocyteRatsUp-RegulationOligodendrogliamedicine.anatomical_structureNeurologyNeurology (clinical)Multiple sclerosis (Houndmills, Basingstoke, England)
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Multifaceted effects of oligodendroglial exosomes on neurons: impact on neuronal firing rate, signal transduction and gene regulation.

2014

Exosomes are small membranous vesicles of endocytic origin that are released by almost every cell type. They exert versatile functions in intercellular communication important for many physiological and pathological processes. Recently, exosomes attracted interest with regard to their role in cell–cell communication in the nervous system. We have shown that exosomes released from oligodendrocytes upon stimulation with the neurotransmitter glutamate are internalized by neurons and enhance the neuronal stress tolerance. Here, we demonstrate that oligodendroglial exosomes also promote neuronal survival during oxygen–glucose deprivation, a model of cerebral ischaemia. We show the transfer from…

Cell typeCell signalingEndocytic cycleBlotting WesternAction PotentialsCell CommunicationNeurotransmissionBiologyExosomesReal-Time Polymerase Chain ReactionExosomeSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyMiceAnimalsPhosphorylationCells CulturedNeuronsSuperoxide DismutaseGlutamate receptorCatalaseMicroarray AnalysisPart III: Intercellular communication—basic insightImmunohistochemistryMicrovesiclesCell HypoxiaCell biologyMice Inbred C57BLOligodendrogliaGlucoseGene Expression RegulationSignal transductionGeneral Agricultural and Biological SciencesSignal TransductionPhilosophical transactions of the Royal Society of London. Series B, Biological sciences
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Laser capture microdissection and genetic analysis of carbon-labeled Kupffer cells.

2009

AIM: To develop a method of labeling and micro-dissecting mouse Kupffer cells within an extraordinarily short period of time using laser capture microdissection (LCM). METHODS: Tissues are complex structures comprised of a heterogeneous population of interconnected cells. LCM offers a method of isolating a single cell type from specific regions of a tissue section. LCM is an essential approach used in conjunction with molecular analysis to study the functional interaction of cells in their native tissue environment. The process of labeling and acquiring cells by LCM prior to mRNA isolation can be elaborate, thereby subjecting the RNA to considerable degradation. Kupffer cell labeling is ach…

Cell typeKupffer CellsCellPopulationReceptor Macrophage Colony-Stimulating FactorAsialoglycoprotein ReceptorBiologyMicemedicineAnimalsRNA MessengerReceptors ImmunologiceducationMicrodissectionLaser capture microdissectionOligonucleotide Array Sequence Analysiseducation.field_of_studyMicroarray analysis techniquesGene Expression ProfilingLasersKupffer cellGastroenterologyGeneral MedicineOriginal ArticlesMolecular biologyCarbonGene expression profilingMice Inbred C57BLmedicine.anatomical_structureFemaleMicrodissectionWorld journal of gastroenterology
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Expression profiling of prospero in the Drosophila larval chemosensory organ: Between growth and outgrowth

2010

AbstractBackgroundThe antenno-maxilary complex (AMC) forms the chemosensory system of theDrosophilalarva and is involved in gustatory and olfactory perception. We have previously shown that a mutant allele of the homeodomain transcription factor Prospero (prosVoila1,V1), presents several developmental defects including abnormal growth and altered taste responses. In addition, many neural tracts connecting the AMC to the central nervous system (CNS) were affected. Our earlier reports on larval AMC did not argue in favour of a role ofprosin cell fate decision, but strongly suggested thatproscould be involved in the control of other aspect of neuronal development. In order to identify these fu…

Central Nervous SystemMESH : Transcription FactorsMESH: DrosophilaOF-FUNCTION SCREEN;MUSCA-DOMESTICA L;HOUSE-FLY LARVA;FINE-STRUCTURE;AXON GUIDANCE;TRANSCRIPTION FACTOR;PATTERN-FORMATION;GENETIC-ANALYSIS;NERVOUS-SYSTEMGenes InsectMESH: Genes InsectAXON GUIDANCEMUSCA-DOMESTICA L0302 clinical medicineMESH: Gene Expression Regulation DevelopmentalCluster AnalysisDrosophila ProteinsMESH: AnimalsTRANSCRIPTION FACTORMESH: Nerve Tissue ProteinsMESH : Nerve Tissue ProteinsOF-FUNCTION SCREENOligonucleotide Array Sequence AnalysisGenetics0303 health sciencesMESH : Central Nervous SystemMicrobiology and ParasitologyMESH : Genes InsectGene Expression Regulation DevelopmentalNuclear ProteinsMESH: Transcription FactorsNull alleleMicrobiologie et ParasitologieMESH : Oligonucleotide Array Sequence Analysis[ SDV.BBM.GTP ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]Larva[SDV.BBM.GTP] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]DrosophilaDrosophila ProteinResearch ArticleBiotechnologylcsh:QH426-470MESH: Drosophila Proteinslcsh:BiotechnologyNerve Tissue ProteinsBiotechnologiesBiology03 medical and health sciencesMESH: Gene Expression ProfilingGENETIC-ANALYSIS[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]lcsh:TP248.13-248.65GeneticsAnimalsMESH : Cluster AnalysisMESH: Central Nervous SystemAlleleMESH : DrosophilaAlleles030304 developmental biologyMESH : LarvaMicroarray analysis techniquesHOUSE-FLY LARVAGene Expression ProfilingMESH : Gene Expression ProfilingMESH: AllelesWild typeMESH : Nuclear ProteinsProsperobiology.organism_classificationMESH : Drosophila ProteinsMESH: Cluster AnalysisNERVOUS-SYSTEMGene expression profilinglcsh:GeneticsMESH: Oligonucleotide Array Sequence AnalysisHomeoboxMESH : AnimalsMESH : Gene Expression Regulation DevelopmentalMESH : AllelesMESH: Nuclear ProteinsMESH: Larva030217 neurology & neurosurgeryTranscription FactorsPATTERN-FORMATIONFINE-STRUCTURE
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Solution Processed Micro- and Nano-Bioarrays for Multiplexed Biosensing

2012

This Feature article reports on solution dispensing methodologies which enable the realization of multiplexed arrays at the micro- and nanoscale for relevant biosensing applications such as drug screening or cellular chips.

ChemistryNanotechnologyBiosensing TechniquesElectrochemical TechniquesEquipment DesignHardware_PERFORMANCEANDRELIABILITYMicroarray AnalysisMultiplexingHigh-Throughput Screening AssaysAnalytical ChemistrySolution processedNano-Hardware_INTEGRATEDCIRCUITSAnimalsHumansNanotechnologyBiochipBiosensorAnalytical Chemistry
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MicroRNA Profile in Response to Doxorubicin Treatment in Breast Cancer

2015

UNLABELLED Chemotherapy treatment is the standard in triple negative breast cancers, a cancer subgroup which lacks a specific target. The mechanisms leading to the response, as well as any markers that allow the differentiation between responder and non-responder groups prior to treatment are unknown. In parallel, miRNAs can act as oncogenes or tumor suppressors and there is evidence of their involvement in promoting resistance to anticancer drugs. Therefore we hypothesized that changes in miRNA expression after doxorubicin treatment may also be relevant in treatment response. OBJECTIVE To study miRNAs that are differentially expressed in response to doxorubicin treatment. METHODS One lumin…

ChemotherapyMicroarray analysis techniquesmedicine.medical_treatmentCancerCell BiologyBiologyBioinformaticsmedicine.diseaseBiochemistryBreast cancermicroRNACancer researchmedicineDoxorubicinViability assayMolecular BiologyGenemedicine.drugJournal of Cellular Biochemistry
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