Search results for "Micronuclei"

showing 3 items of 13 documents

Gene toxicity studies on titanium dioxide and zinc oxide nanomaterials used for UV-protection in cosmetic formulations

2010

Titanium dioxide and zinc oxide nanomaterials, used as UV protecting agents in sunscreens, were investigated for their potential genotoxicity in in vitro and in vivo test systems. Since standard OECD test methods are designed for soluble materials and genotoxicity testing for nanomaterials is still under revision, a battery of standard tests was used, covering different endpoints. Additionally, a procedure to disperse the nanomaterials in the test media and careful characterization of the dispersed test item was added to the testing methods. No genotoxicity was observed in vitro (Ames' Salmonella gene mutation test and V79 micronucleus chromosome mutation test) or in vivo (mouse bone marrow…

MaleMaterials scienceBiomedical EngineeringBone Marrow CellsNanotechnologyCosmeticsGene mutationToxicologymedicine.disease_causeCell LineNanomaterialsMicechemistry.chemical_compoundSalmonellaIn vivoCricetinaeAdministration InhalationMacrophages AlveolarmedicineAnimalsRats WistarMicronuclei Chromosome-DefectiveTitaniumChromatographyMutagenicity TestsBody WeightIn vitroNanostructuresRatschemistryData Interpretation StatisticalMicronucleus testTitanium dioxideZinc OxideMicronucleusSunscreening AgentsGenotoxicityNanotoxicology
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Destabilized green fluorescent protein detects rapid removal of transcription blocks after genotoxic exposure

2007

High stabilities of reporter proteins and their messenger RNAs (mRNAs) interfere with the detection of rapid transient changes in gene expression, such as transcriptional blocks posed by genotoxic DNA lesions. We have modified a green fluorescent protein (GFP) gene within the episomal pMARS vector by addition of a fragment encoding for mouse ornithine decarboxylase (ODC) proline-glutamate-serine-threonine-rich (PEST) sequence in order to target the protein to the proteasomes and achieved an unprecedentedly fast GFP turnover in permanently transfected human cells. As early as 1 h after inhibition of protein synthesis by cycloheximide, the number of fluorescent cells decreased more than 5-fo…

Transcription GeneticMutagenicity TestsUltraviolet RaysDNA repairGreen Fluorescent ProteinsfungiCycloheximideBiologyMolecular biologyGeneral Biochemistry Genetics and Molecular BiologyGreen fluorescent proteinchemistry.chemical_compoundSpectrometry FluorescencechemistryTranscription (biology)Gene expressionProtein biosynthesisHumansGeneMicronuclei Chromosome-DefectiveDNADNA DamageHeLa CellsBiotechnologyBioTechniques
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DNA damage by bromate: Mechanism and consequences

2005

Abstract Exposure of mammalian cells to bromate (BrO3−) generates oxidative DNA modifications, in particular 7,8-dihydro-8-oxo-guanine (8-oxoG). The damaging mechanism is quite unique, since glutathione, which is protective against most oxidants and alkylating agents, mediates a metabolic activation, while bromate itself does not react directly with DNA. Neither enzymes nor transition metals are required as catalysts in the activation. The ultimate DNA damaging species has not yet been established, but experiments under cell-free conditions suggest that neither molecular bromine nor reactive oxygen species such as superoxide, hydrogen peroxide or singlet oxygen are involved. Rather bromine …

GuanineCell SurvivalDNA damageHypochloriteToxicologymedicine.disease_causeMicechemistry.chemical_compoundCricetulusCell Line TumorCricetinaemedicineAnimalsHydrogen peroxideMicronuclei Chromosome-Defectivechemistry.chemical_classificationReactive oxygen speciesMicronucleus TestsDose-Response Relationship DrugBromatesSinglet oxygenSuperoxideBromatechemistryBiochemistryReactive Oxygen SpeciesOxidative stressDNA DamageMutagensToxicology
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