Search results for "Microscopy"

showing 10 items of 3390 documents

Gastric antisecretory drugs induce leukocyte-endothelial cell interactions through gastrin release and activation of CCK-2 receptors.

2007

Antisecretory drugs are effective antiulcer agents, but its chronic use generates hypergastrinemia and accelerates the development of atrophic gastritis in Helicobacter pylori-positive patients. We have recently shown that gastrin exerts a proinflammatory effect in rats through CCK-2 receptor activation that contributes to the inflammation induced by H. pylori. The present study was designed to examine whether gastrin hypersecretion in response to treatment with antisecretory drugs induces an inflammatory response that could promote mucosal atrophy. The effects of omeprazole or famotidine on leukocyte/endothelial cell interactions in vivo were analyzed in rat mesenteric venules using intrav…

Malemedicine.medical_specialtyProglumidemedicine.drug_classInflammationCD18Cell CommunicationProinflammatory cytokineRats Sprague-DawleyInternal medicineGastrinsmedicineLeukocytesAnimalsOmeprazoleGastrinPharmacologyInflammationMicroscopy Videobusiness.industryEndothelial CellsProton Pump InhibitorsReceptor antagonistAnti-Ulcer AgentsFlow CytometryImmunohistochemistryReceptor Cholecystokinin BRatsFamotidineChemotaxis LeukocyteEndocrinologyGastric MucosaMolecular Medicinemedicine.symptombusinessmedicine.drugThe Journal of pharmacology and experimental therapeutics
researchProduct

Accumulation of hydroxyethyl starch (HES) in the liver of patients with renal failure and portal hypertension

1986

Summary Hydroxyethyl starch (HES) has gained wide clinical acceptance as a colloidal plasma substitute. We were able to study the liver biopsies of two patients with renal failure who developed ascites after repeated infusions of HES. All types of liver cells displayed massive accumulation of HES with the morphologic resemblance to a storage disease. These changes could be distinguished clearly from the lesions of a hereditary disorder by light and electron microscopy. Although it is difficult to establish a causative role for HES in the development of ascites on the bases of morphological changes alone, one should be cautious about giving HES to patients with renal failure until exact data…

Malemedicine.medical_specialtyResuscitationPathologyHydroxyethyl starchHydroxyethyl Starch DerivativesRenal DialysisHypertension PortalAscitesmedicineHumansreproductive and urinary physiologyHepatologybusiness.industryStarchMiddle Agedmedicine.diseaseSurgeryMicroscopy ElectronLiverKidney Failure ChronicPortal hypertensionFemaleHypotensionbiological phenomena cell phenomena and immunitymedicine.symptombusinessPerfusionmedicine.drugJournal of Hepatology
researchProduct

Chronic ethanol feeding induces cellular antioxidants decrease and oxidative stress in rat peripheral nerves. Effect of S-adenosyl-L-methionine and N…

1998

Chronic ethanol feeding promotes oxidative stress in rat peripheral nerve. Malondialdehyde, a lipid peroxidation product, content increases in sciatic nerves of rats fed an ethanol-containing diet, when compared with pair-fed animals. Moreover, glutathione content and glutathione peroxidase activity in this same tissue decrease in ethanol-fed vs. pair-fed rats. S- adenosyl-L-methionine and N-acetyl-L-cysteine, both with possible therapeutic action on alcoholism, were tested in this animal model. Only N-acetyl-L- cysteine was able to normalize malondialdehyde content and to restore glutathione content and glutathione peroxidase activity, to values not significantly different from those of sc…

Malemedicine.medical_specialtyS-AdenosylmethionineEthanol feedingmedicine.disease_causeBiochemistryAntioxidantsLipid peroxidationRats Sprague-Dawleychemistry.chemical_compoundFree radicalPhysiology (medical)Internal medicineMalondialdehydemedicineAnimalsPeripheral Nerveschemistry.chemical_classificationGlutathione PeroxidaseEthanolS-adenosyl-L-methionineEthanolGlutathione peroxidaseN-acetyl-L- cysteineNerveGlutathioneMalondialdehydeGlutathionePeripheralAcetylcysteineRatsMicroscopy ElectronOxidative StressEndocrinologychemistryBiochemistryGlutathione peroxidaseLipid PeroxidationEthanoOxidative stressFree radical biologymedicine
researchProduct

Mitochondrial myopathy--a result of clofibrate/etofibrate treatment? Case report.

1985

A 66-year-old man had developed a myopathy while undergoing several periods of etofibrate and clofibrate therapy over the past 5 years. Discontinuation of etofibrate treatment failed to reverse his muscle illness which, however, did not progress. A muscle biopsy revealed a chronic myopathy marked by abundant, abnormally structured muscle mitochondria. His mitochondrial myopathy may represent a forme fruste of the Kearns-Sayre syndrome or other types of mitochondrial myopathy, clinically made evident by the etofibrate/clofibrate therapy, or a permanent, adverse side effect of clofibrate treatment. If the latter assumption proves to be correct, it will indicate that clofibrate therapy may ind…

Malemedicine.medical_specialtySide effectKearns-Sayre SyndromePathology and Forensic Medicine03 medical and health sciencesCellular and Molecular NeuroscienceClofibric Acid0302 clinical medicineMitochondrial myopathyMuscular DiseasesInternal medicinemedicineHumansClofibrateMyopathy030304 developmental biologyAgedHypolipidemic Agents0303 health sciencesMuscle biopsyClofibratemedicine.diagnostic_testbusiness.industryMusclesForme frustemedicine.disease3. Good healthDiscontinuationMitochondria MuscleMicroscopy ElectronEndocrinologyEtofibrateNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgerymedicine.drugActa neuropathologica
researchProduct

Principal neurons projecting to the pineal gland in close association with small intensely fluorescent cells in the superior cervical ganglion of rats

1988

The localization in the superior cervical ganglia (SCG) of small, intensely fluorescent (SIF) cells and of principal nerve (PN) cells innervating the pineal gland was examined in adult male Sprague-Dawley rats. PN cells were demonstrated by means of the retrograde neuron-tracing method using the fluorescent tracer Fluoro-Gold (FG) injected into the pineal gland. SIF cells were visualized by the formaldehyde-induced fluorescence method. Twenty-nine percent of the FG-labeled PN cells were found closely associated with SIF cells. In the rostral half of the ganglion, 43% of the SIF cells were situated in juxtaposition to one or several labeled neurons. The possible influence of SIF cells on the…

Malemedicine.medical_specialtySuperior cervical ganglionHistologyStilbamidinesCentral nervous systemEnteroendocrine cellBiologyPineal GlandPathology and Forensic MedicinePineal glandFormaldehydeInternal medicineNeural PathwaysmedicineAnimalsFluorescent DyesNeuronsGanglia SympatheticRats Inbred StrainsCell BiologyRetrograde tracingMolecular biologyRatsGanglionmedicine.anatomical_structureEndocrinologyMicroscopy Fluorescencenervous systemCervical gangliaNeuronCell and Tissue Research
researchProduct

The impact of ovarian stimulation on the expression of candidate reprogramming genes in mouse preimplantation embryos.

2012

Ovarian stimulation with gonadotrophins is an integral part of assisted reproductive technologies in human subfertility/infertility treatment. Recent findings have associated ovarian stimulation with the increased incidence of imprinting disorders in humans as well as defects in genome-wide methylation reprogramming and, in particular, imprinting in mice. Here, we present the first study that determined the impact of ovarian stimulation on the expression of developmentally important reprogramming genes <i>(Apex1, Lig1, Lig3, Mbd2, Mbd3, Mbd4, </i>and<i> Polb)</i> in single early mouse morula embryos (16-cell stage). Using absolute quantification of mRNA by quantitati…

Malemedicine.medical_specialtyTime FactorsGonadotropins EquineDown-RegulationStimulationReproductive technologyBiologyChorionic GonadotropinMBD4AndrologyMiceOogenesisOvulation InductionInternal medicineGeneticsmedicineDNA-(Apurinic or Apyrimidinic Site) LyaseAnimalsHumansHorsesRNA MessengerMolecular BiologyGenetics (clinical)GametogenesisDNA Polymerase betaRegulation of gene expressionReverse Transcriptase Polymerase Chain ReactionEmbryogenesisGene Expression Regulation DevelopmentalEmbryoDNA-Binding ProteinsMice Inbred C57BLEndocrinologyBlastocystMicroscopy Fluorescenceembryonic structuresFemaleReprogrammingTranscription FactorsCytogenetic and genome research
researchProduct

Exercise-induced necrotic muscle damage and enzyme release in the four days following prolonged submaximal running in rats.

1994

Male Wistar rats were made to run uphill on a treadmill 5.5° incline at 17 m min−1 for 4 h, and killed for muscle and serum sampling 2, 4, 12, 24, 48 or 96 h after the exertion. To estimate the degree of muscle damage,β-glucuronidase activity, total protein concentration, water content and morphology were examined in the red parts of quadriceps femoris (MQF) and soleus (MS) muscles, the distal white part of the rectus femoris muscle (MRF) and the superficial part of triceps brachii muscle (MTB). Simultaneous serum samples were assayed for creatine kinase (CK) activity and carbonic anhydrase III (CA III) concentration. Fibre swelling and interstitial oedema were detected in MS at 4 h and in …

Malemedicine.medical_specialtyTime FactorsPhysiologyClinical BiochemistryInflammationPhysical exerciseRectus femoris muscleMotor ActivityNecrosisBody WaterPhysiology (medical)Internal medicinemedicineAnimalsExertionRats WistarCreatine KinaseCarbonic AnhydrasesGlucuronidaseSoleus musclebiologyMusclesTriceps brachii muscleSkeletal muscleAnatomyRatsMicroscopy ElectronEndocrinologymedicine.anatomical_structurebiology.proteinPhysical EnduranceCreatine kinasemedicine.symptomPflugers Archiv : European journal of physiology
researchProduct

Effect of large positioning holes on capsule fixation of plate-haptic intraocular lenses

2000

Abstract Purpose To compare the centration and fixation of silicone plate-haptic intraocular lenses (IOLs) with different-sized positioning holes. Setting Eye Clinic of the Johannes Gutenberg-University Mainz, Mainz, Germany. Methods In a prospective randomized study, 51 Chiroflex C10 and 56 Chiroflex C11 IOLs were implanted under standardized conditions by the same surgeon. The IOL position was documented at the end of surgery and by retroillumination on the first day and 5 months postoperatively. The positioning-hole area was evaluated by ultrasound biomicroscopy (50 MHz) 5 months postoperatively. Results One day postoperatively, no IOL in either group was decentered more than 1.0 mm. Aft…

Malemedicine.medical_specialtygenetic structuresmedicine.medical_treatmentEye diseaseLens Capsule CrystallineUltrasound biomicroscopyIntraocular lensProsthesis Designlaw.inventionFixation (surgical)Foreign-Body MigrationLens Implantation IntraocularlawOphthalmologyHumansMedicineProspective StudiesCapsulorhexisAgedUltrasonographyAged 80 and overLenses Intraocularbusiness.industrySuture TechniquesCapsuleMiddle Agedmedicine.diseaseCapsulorhexiseye diseasesSensory SystemsSurgeryLens (optics)OphthalmologyCapsulotomyFemaleSurgerysense organsbusinessCell DivisionJournal of Cataract and Refractive Surgery
researchProduct

Betulinic Acid Protects from Ischemia-Reperfusion Injury in the Mouse Retina

2021

Ischemia/reperfusion (I/R) events are involved in the pathophysiology of numerous ocular diseases. The purpose of this study was to test the hypothesis that betulinic acid protects from I/R injury in the mouse retina. Ocular ischemia was induced in mice by increasing intraocular pressure (IOP) to 110 mm Hg for 45 min, while the fellow eye served as a control. One group of mice received betulinic acid (50 mg/kg/day p.o. once daily) and the other group received the vehicle solution only. Eight days after the I/R event, the animals were killed and the retinal wholemounts and optic nerve cross-sections were prepared and stained with cresyl blue or toluidine blue, respectively, to count cells in…

Malemedicine.medical_specialtyretinagenetic structuresQH301-705.5ischemia-reperfusion injuryarteriolesVideo microscopyProtective AgentsArticlechemistry.chemical_compoundMicebetulinic acidInternal medicineBetulinic acidmedicineAnimalsBiology (General)AxonGanglion cell layerreactive oxygen speciesRetinaAnti-Inflammatory Agents Non-SteroidalRetinalGeneral Medicinemedicine.diseaseeye diseasesMice Inbred C57BLmedicine.anatomical_structureEndocrinologychemistryReperfusion InjuryOptic nervesense organsPentacyclic TriterpenesReperfusion injuryCells
researchProduct

C1-ESTERASE INHIBITOR REVERSES FUNCTIONAL CONSEQUENCES OF SUPERIOR MESENTERIC ARTERY ISCHEMIA/REPERFUSION BY LIMITING REPERFUSION INJURY AND RESTORIN…

2006

Activated complement contributes significantly to reperfusion injury after ischemia. This study explores functional consequences of C1-esterase inhibitor (C1-INH) treatment after superior mesenteric artery occlusion (SMAO)/ reperfusion using intravital microscopy. Thirty anesthetized, spontaneously breathing, male Sprague-Dawley rats underwent SMAO for 60 min followed by reperfusion (4 h). C1-esterase inhibitor (100 and 200 IU/kg body weight) or saline (0.9%) was given as a single bolus before reperfusion. Sham-operated animals (n = 10) without SMAO served as controls.Systemichemodynamicsweremonitoredcontinuously,arterial bloodgasesanalyzedintermittently, andleukocyte/ endothelial interacti…

Malemedicine.medical_treatmentIschemiaPharmacologyCritical Care and Intensive Care MedicineRats Sprague-DawleyBolus (medicine)Mesenteric Artery Superiormedicine.arterymedicineAnimalsSuperior mesenteric arterySalinebusiness.industryMicrocirculationMetabolic acidosismedicine.diseaseRatsRegional Blood FlowMesenteric ischemiaReperfusion InjuryAnesthesiaEmergency MedicinebusinessComplement C1 Inhibitor ProteinReperfusion injuryIntravital microscopyShock
researchProduct