Search results for "Mito"

showing 10 items of 2513 documents

Fantāzijas tēlu grafiskā interpretācija tekstilā

2018

Darbā “Fantāzijas tēlu grafiskā interpretācija tekstilā” tiek pētīts fantāzijas tēls, tā interpretācijas tekstilā, grafiskā izpildījumā. Izzinātas grafiskās variācijas vēsturiskā aspektā, kā arī veidots ieskats mitoloģijā kā vienā no fantāzijas tēlu rašanās avotiem. Autore uzmanību pievērš fantāzijas tēlu radošajam virzienam, izpētot un analizējot mūsdienās radītos tēlus. Tiek pētīti to izveides galvenie priekšnosacījumi. Apkopotā informācija tiek izmantota jaunu fantāzijas tēlu izveidei, kas kā grafiski attēli izmantoti tekstila apģērbu apdrukā. Darbā autore meklē fantāzijas tēlu saikni un galvenās iezīmēs no viduslaikiem līdz mūsdienām. Tiek meklētas kopsakarības, iedvesmas avoti un līdzī…

apdrukaMākslas zinātnedizainsmitoloģijafantāzijas tēlstekstils
researchProduct

Autophagy

2012

Klionsky, Daniel J. et al.

autophagy assays[SDV]Life Sciences [q-bio]AutolysosomeAutophagosome maturationautophagosomeBioinformaticsstressChaperone-mediated autophagyModelsLC3MESH: Animalsguidelinesautolysosome autophagosome flux LC3 lysosome phagophore stress vacuoleSettore BIO/06 - Anatomia Comparata E CitologiaComputingMilieux_MISCELLANEOUSSettore BIO/17Autophagy databaseautolysosome3. Good healthddc:540lysosomeEnergy and redox metabolism Mitochondrial medicine [NCMLS 4]methods [Biological Assay]Biological AssaySettore BIO/17 - ISTOLOGIANeuroniMAP1LC3BHumanautophagygenetics [Autophagy]AutofagiaMESH: Autophagy*/genetics[SDV.BC]Life Sciences [q-bio]/Cellular BiologyAutofagia; Neuroni; istologiaBiologyModels BiologicalLC3; autolysosome; autophagosome; flux; lysosome; phagophore; stress; vacuoleddc:570AutophagyAnimalsHumansAutophagy-Related Protein 7[SDV.BC] Life Sciences [q-bio]/Cellular BiologyBiological Assay/methodsMolecular BiologyBiologyAutophagy; guidelines; autophagy assaysistologiaphagophoreMESH: HumansAnimals; Biological Assay; Humans; Models Biological; AutophagyvacuoleAnimal[ SDV.BC ] Life Sciences [q-bio]/Cellular BiologyMESH: Models BiologicalPathogenesis and modulation of inflammation Infection and autoimmunity [N4i 1]Cell BiologyBiologicalAutophagy/geneticsfluxAutophagosome membraneAutophagy Protein 5Human medicineMESH: Biological Assay/methods*Neuroscienceautolysosome; autophagosome; flux; LC3; lysosome; phagophore; stress; vacuoleAutophagy
researchProduct

Glutathione in Cancer Cell Death

2011

Glutathione (L-γ-glutamyl-L-cysteinyl-glycine; GSH) in cancer cells is particularly relevant in the regulation of carcinogenic mechanisms; sensitivity against cytotoxic drugs, ionizing radiations, and some cytokines; DNA synthesis; and cell proliferation and death. The intracellular thiol redox state (controlled by GSH) is one of the endogenous effectors involved in regulating the mitochondrial permeability transition pore complex and, in consequence, thiol oxidation can be a causal factor in the mitochondrion-based mechanism that leads to cell death. Nevertheless GSH depletion is a common feature not only of apoptosis but also of other types of cell death. Indeed rates of GSH synthesis and…

autophagyCancer ResearchProgrammed cell deathCell growthapoptosisReviewGlutathioneMitochondrionBiologylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenslcsh:RC254-282necrosisCell biologychemistry.chemical_compoundcell deathOncologyMitochondrial permeability transition porechemistryApoptosisCancer cellcancerglutathioneIntracellularCancers
researchProduct

Mitochondria and T2D: Role of Autophagy, ER Stress, and Inflammasome.

2020

Type 2 diabetes (T2D) is one of the main current threats to human health. Both T2D and its numerous clinical complications are related to mitochondrial dysfunction and oxidative stress. Over the past decade, great progress has been made in extending our knowledge about the signaling events regulated by mitochondria. However, the links among mitochondrial impairment, oxidative stress, autophagy, endoplasmic reticulum (ER) stress, and activation of the inflammasome still need to be clarified. In light of this deficit, we aim to provide a review of the existing literature concerning the complicated crosstalk between mitochondrial impairment, autophagy, ER stress, and the inflammasome in the mo…

autophagyMitochondrial DiseasesInflammasomesEndocrinology Diabetes and Metabolism030209 endocrinology & metabolismMitochondrionmedicine.disease_causeInflammasome03 medical and health sciences0302 clinical medicineEndocrinologyinflammasomemedicineAutophagyAnimalsHumansbusiness.industryEndoplasmic reticulumAutophagyMolecular pathogenesisInflammasomeType 2 diabetesEndoplasmic Reticulum StressCell biologyMitochondriamitochondriaCrosstalk (biology)Oxidative StressDiabetes Mellitus Type 2Unfolded protein responsetype 2 diabetesbusinessOxidative stressmedicine.drugTrends in endocrinology and metabolism: TEM
researchProduct

Heavy Metals and Metalloids as Autophagy Inducing Agents: Focus on Cadmium and Arsenic

2012

In recent years, research on the autophagic process has greatly increased, invading the fields of biology and medicine. Several markers of the autophagic process have been discovered and various strategies have been reported studying this molecular process in different biological systems in both physiological and stress conditions. Furthermore, mechanisms of metalloid- or heavy metal-induced toxicity continue to be of interest given the ubiquitous nature and distribution of these contaminants in the environment where they often play the role of pollutants of numerous organisms. The aim of this review is a critical analysis and correlation of knowledge of autophagic mechanisms studied under …

autophagycadmiumchemistry.chemical_elementReviewMitochondrionBiologyBioinformaticssea urchin embryosstressstreHeat shock proteinSettore BIO/06 - Anatomia Comparata E CitologiaProtein kinase Alcsh:QH301-705.5Transcription factorchemistry.chemical_classificationReactive oxygen speciesCadmiumAutophagyapoptosisarsenicGeneral MedicineapoptosiCell biologylcsh:Biology (General)chemistryApoptosisCells
researchProduct

Role of Reduced Lipoic Acid in the Redox Regulation of Mitochondrial Aldehyde Dehydrogenase (ALDH-2) Activity

2007

Chronic therapy with nitroglycerin results in a rapid development of nitrate tolerance, which is associated with an increased production of reactive oxygen species. We have recently shown that mitochondria are an important source of nitroglycerin-induced oxidants and that the nitroglycerin-bioactivating mitochondrial aldehyde dehydrogenase is oxidatively inactivated in the setting of tolerance. Here we investigated the effect of various oxidants on aldehyde dehydrogenase activity and its restoration by dihydrolipoic acid. In vivo tolerance in Wistar rats was induced by infusion of nitroglycerin (6.6 microg/kg/min, 4 days). Vascular reactivity was measured by isometric tension studies of iso…

biologyChemistryAldehyde dehydrogenaseDehydrogenaseCell BiologyOxidative phosphorylationMitochondrionBiochemistrychemistry.chemical_compoundLipoic acidBiochemistryDihydrolipoic acidcardiovascular systembiology.proteinBranched-chain alpha-keto acid dehydrogenase complexMolecular BiologyPeroxynitriteJournal of Biological Chemistry
researchProduct

Long-term response of osteogenic cells on micron and submicron-scale-structured hydrophilic titanium surfaces: sequence of cell proliferation and cel…

2010

Objective: Modifications of surface topography and surface chemistry are key factors for guiding target cells during dental implant healing. Recent in vitro studies confirmed promotion of early osteogenic cell differentiation on submicron scaled surfaces in particular when hydrophilized. However, no long-term observations on both osteogenic cell proliferation as well as on cell maturation have been reported for respectively modified surfaces. Aim of this study was to monitor osteogenic cell proliferation and expression of specific osteogenic cell differentiation markers on a protein level over an extended period of 3 weeks with respect to surface modifications. Material and methods: Modifie…

biologyChemistryCell growthCellular differentiationCellCell Maturationmedicine.anatomical_structureOsteocalcinbiology.proteinmedicineBiophysicsAlkaline phosphataseOsteopontinOral SurgeryMitosisClinical Oral Implants Research
researchProduct

Prebiotic Xylo-oligosaccharides Targeting Faecalibacterium prausnitzii Prevent High Fat Diet-induced Hepatic Steatosis in Rats

2020

Understanding the importance of gut microbiota (GM) in non-alcoholic fatty liver disease (NAFLD) has raised the hope for therapeutic microbes. We have shown that high hepatic fat associated with low abundance of Faecalibacterium prausnitzii in humans and further, administration of F. prausnitzii prevented NAFLD in mice. Here, we aimed to target F. prausnitzii by prebiotic xylo-oligosaccharides (XOS) to treat NAFLD. First, the effect of XOS on F. prausnitzii growth was assessed in vitro. Then, XOS was supplemented or not with high (HFD) or low (LFD) fat-diet for 12-weeks in Wistar rats (n=10/group). XOS increased F. prausnitzii growth having only minor impact on the GM composition. When supp…

biologyChemistryPrebioticmedicine.medical_treatmentdigestive oral and skin physiologyFatty livergastroenterologynutritional and metabolic diseasesfood and beveragesFaecalibacterium prausnitziiHigh fat dietMetabolismGut floraMitochondrionmedicine.diseasebiology.organism_classificationdigestive system3. Good healthmedicineFood scienceSteatosis
researchProduct

Organometallic complexes with biological molecules: V.In vivo cytotoxicity of diorganotin(IV)-amoxicillin derivatives in mitotic chromosomes ofrutilu…

1995

In order to test in vivo cytotoxicity of diorganotin(IV)-amoxicillin (amox) derivatives, mitotic chromosomes of Rutilus rubilio (Pisces, Cyprinidae) have been analyzed using two different chromosome-staining techniques. Results gathered after exposure of fish to the free amox.3H 2 O, R 2 SnClamox.2H 2 O, and R 2 Snamox 2 .2H 2 O (R = methyl, butyl and phenyl ; amox - = 6-[D(-)-β-amino-p-hydroxyphenylacetamido]penicillinate) suggest that methyl derivatives seem to exert a lower cytotoxicity than butyl and phenyl ones and that R 2 Snamox 2 .2H 2 O derivatives are more toxic than R 2 Snclamox.2H 2 O at both 10 -5 and 10 -7 mol dm -3 concentrations. The following structural lesions have been id…

biologyChemistryStereochemistryChromosomeGeneral Chemistrybiology.organism_classificationmedicine.disease_causeChromosome aberrationInorganic ChemistryMoleCyprinidaemedicineRutilusCytotoxicityMitosisGenotoxicityApplied Organometallic Chemistry
researchProduct

A frozen analogue approach to aminopyridinylimidazoles leading to novel and promising p38 MAP kinase inhibitors.

2012

In this study we report the design, synthesis, and biological evaluation of constrained aminopyridinylimidazoles as p38α MAP kinase inhibitors. The frozen analogue approach focused on the pyridinyl unit, using purine bioisosteres as constrained structure analogues. The identification of the most potent bioisostere was followed by a further derivatization to address hydrophobic region II. In combination with C-2 modifications of the imidazole core, we were able to design highly active inhibitors on the p38α MAP kinase. The inhibitor design presented herein represents a promising and highly efficient advancement of recent stages of development in this class of p38 MAP kinase inhibitors. In co…

biologyChemistryStereochemistryPyridinesp38 mitogen-activated protein kinasesEntropyImidazolesMolecular ConformationCombinatorial chemistryp38 Mitogen-Activated Protein KinasesMolecular conformationMolecular Docking Simulationchemistry.chemical_compoundStructure-Activity RelationshipPurinesMitogen-activated protein kinaseDrug DesignDrug Discoverybiology.proteinMolecular MedicineStructure–activity relationshipBioisostereBiological evaluationJournal of medicinal chemistry
researchProduct