Search results for "Models"

showing 10 items of 8211 documents

Involvement of distal airways in a chronic model of experimental asthma.

2005

Summary Background Bronchial asthma is characterized by chronic airway inflammation and airway remodelling which occurs in both proximal and distal airways. These changes are associated with development of airway hyper-responsiveness and airflow limitation. Objective This study was aimed to analyse whether chronic inhalative allergen challenges in mice lead to morphological and physiological changes comparable with this phenotype. Methods For this purpose, BALB/c mice were systemically sensitized to ovalbumin (OVA) followed by aerosol allergen challenges on 2 consecutive days per week for 12 weeks. Results In chronically challenged mice, tissue inflammation in proximal as well as distal air…

Pathologymedicine.medical_specialtyAllergyOvalbuminImmunologyInflammationBronchiMiceTransforming Growth Factor betaAdministration InhalationmedicineImmunology and AllergyAnimalsRespiratory systemAsthmaMice Inbred BALB CMucous MembraneInhalationbusiness.industryRespiratory diseaserespiratory systemAllergensmedicine.diseaseAsthmarespiratory tract diseasesDisease Models Animalmedicine.anatomical_structureImmunologyChronic DiseaseDisease ProgressionCytokinesFemalemedicine.symptomBronchial HyperreactivityAirwaybusinessBronchoalveolar Lavage FluidRespiratory tractClinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology
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Presence of endothelial progenitor cells, distinct from mature endothelial cells, within human CD146+ blood cells.

2006

SummaryCD146 is an adhesion molecule present on endothelial cells throughout the vascular tree. CD146 is also expressed by circulating endothelial cells (CECs) widely considered to be mature endothelial cells detached from injured vessels. The discovery of circulating endothelial progenitor cells (EPCs) originating from bone marrow prompted us to investigate whether CD146 circulating cells could also contains EPCs. We tested this hypothesis using an approach combining elimination of CECs by an adhesion step, followed by immunomagnetic sorting of remaining CD146+ cells from the non adherent fraction of cord blood mononuclear cells. When cultured under endothelial-promoting conditions, these …

Pathologymedicine.medical_specialtyAngiogenesisCD 146CD34progenitor endothelial cellsMyocardial InfarctionNeovascularization PhysiologicAntigens CD34CD146 AntigenMice SCIDMicecirculating endothelial cellAntigens CDSettore BIO/10 - BiochimicamedicineAnimalsHumansCell LineageProgenitor cellCells CulturedCell Proliferationbusiness.industryStem CellsangiogenesiEndothelial CellsCell DifferentiationHematologyFetal BloodMolecular biologyEndothelial stem cellDrug CombinationsKineticsmedicine.anatomical_structurePhenotypeCord bloodModels Animalcardiovascular systemCD146Leukocyte Common AntigensProteoglycansBone marrowCollagenLamininStem cellbusinessThrombosis and haemostasis
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Effect of antiangiogenic treatment on peritoneal endometriosis-associated nerve fibers

2012

Objective To investigate the effect of antiangiogenic treatment on experimental endometriotic lesion nerve fibers. Design Heterologous mouse model of endometriosis. Setting University Institute IVI, University Hospital La Fe. Animal(s) Ovariectomized nude mice (n = 16) receiving human endometrial fragments from oocyte donors (n = 4). Intervention(s) Endometrium fragments stuck in the peritoneum of 5-week-old female nude mice treated with vehicle (n = 8) and antiangiogenic agent cabergoline (n = 8; Cb 2, 0.05 mg/kg/day) for 14 days. Main Outcome Measure(s) Immunofluorescence analysis of von-Willebrand factor (vWF) and vascular smooth muscle cells (αSMA) for evaluating the number of immature …

Pathologymedicine.medical_specialtyCabergolineTime FactorsAngiogenesisOvariectomyEndometriosisEndometriosisFluorescent Antibody TechniqueMice NudeAngiogenesis InhibitorsNerve fiberPeritoneal DiseasesEndometriumEndometriumMicechemistry.chemical_compoundNerve FibersPeritoneumvon Willebrand FactorAnimalsHumansMedicineMast CellsErgolinesNeovascularization Pathologicbusiness.industryMacrophagesObstetrics and GynecologyMast cellmedicine.diseaseImmunohistochemistryActinsVascular endothelial growth factorDisease Models Animalmedicine.anatomical_structureReproductive MedicinechemistryMicrovesselsImmunologyFemalebusinessBiomarkersBlood vesselFertility and Sterility
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Exploratory study on the effects of biodegradable nanoparticles with drugs on malignant B cells and on a human/mouse model of Burkitt lymphoma.

2010

The aim of this study was to determine if Rituximab coated Biodegradable Nanoparticles (BNPs) loaded with Chlorambucil and Hydroxychloroquine could induce apoptosis of B-Chronic Lymphocytic Leukemia (B-CLL), MEC-1 and BJAB cells in vitro and evaluate their toxic and therapeutic effects on a Human/Mouse Model of Burkitt Lymphoma at an exploratory, proof of concept scale. We found that Rituximab-Chlorambucil-Hydroxychloroquine BNPs induce a decrease in cell viability of malignant B cells in a dose-dependent manner. The mediated cytotoxicity resulted from apoptosis, and was confirmed by monitoring the B-CLL cells after Annexin V/propidium iodide staining. Additional data revealed that these BN…

Pathologymedicine.medical_specialtyCell Survivalhuman/mouse model of Burkitt lymphoma.human lymphomamodel SCID mouseAntineoplastic Agentschemistry.chemical_compoundAntibodies Monoclonal Murine-DerivedMicerituximabimmune system diseasesAnnexinhemic and lymphatic diseasesnanoparticles; rituximab; human lymphoma; model SCID mouseTumor Cells CulturedMedicineAnimalsHumansPharmacology (medical)Propidium iodideGeneral Pharmacology Toxicology and PharmaceuticsCytotoxicityB-LymphocytesChlorambucilDose-Response Relationship Drugbusiness.industrymalignant B cellnanoparticleDrug SynergismGeneral MedicineBiodegradable nanoparticles with drugmedicine.diseaseBurkitt LymphomaLymphomaMice Inbred C57BLLeukemiaDisease Models AnimalDrug CombinationschemistryApoptosisMonoclonalCancer researchNanoparticlesChlorambucilbusinessRituximabmedicine.drugHydroxychloroquine
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Animal models of non-alcoholic steatohepatitis: of mice and man.

2010

The epidemic occurrence of obesity has led to a rapid increase in the incidence of non-alcoholic fatty liver disease (NAFLD) in industrial countries. The disease spectrum includes hepatic steatosis, lobular inflammation with steatohepatitis (NASH) and varying degrees of liver fibrosis, which can progress to cirrhosis. Hepatocellular carcinoma can develop in patients with NASH, even in the absence of cirrhosis. The majority of patients with primary NASH exhibit risk factors that define the metabolic syndrome including insulin resistance and visceral obesity. However, only a minority of patients with NAFLD progress to end-stage liver disease and, so far, predictors to identify these patients …

Pathologymedicine.medical_specialtyCirrhosisDiseaseBioinformaticsLiver diseaseMiceMethionineGenetic predispositionMedicineAnimalsHumansbusiness.industryFatty liverGastroenterologyGeneral Medicinemedicine.diseaseDietary Fatsdigestive system diseasesCholine DeficiencyFatty LiverDisease Models AnimalLiverSteatosisMetabolic syndromeSteatohepatitisbusinessDigestive diseases (Basel, Switzerland)
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Simultaneous confocal laser endomicroscopy and chromoendoscopy with topical cresyl violet

2009

Background Confocal laser endomicroscopy (CLE) has been shown to reliably predict histology during ongoing endoscopy. To unmask lesions for CLE, chromoendoscopy has been mandated. Usually fluorescein then serves as a contrast agent for CLE, but it does not allow direct nuclear visualization, must be injected, leads to a transient skin discoloration, and may have allergic side effects. Objective To establish a single topical dye, cresyl violet (CV), for simultaneous chromoendoscopy and in vivo CLE of the lower GI tract. Design Animal preclinical study, prospective clinical trial. Setting Mainz University Clinic (tertiary care center). Patients, Methods, and Interventions To establish the sta…

Pathologymedicine.medical_specialtyColonAdministration TopicalConfocalPilot ProjectsEndoscopy Gastrointestinallaw.inventionChromoendoscopyDiagnosis DifferentialMicechemistry.chemical_compoundCresyl violetIleumIn vivoConfocal microscopylawOxazinesEndomicroscopyAnimalsHumansMedicineRadiology Nuclear Medicine and imagingIntestinal MucosaFluoresceinColoring AgentsMicroscopy Confocalbusiness.industryGastroenterologyReproducibility of ResultsHistologyMiddle AgedBenzoxazinesIleal NeoplasmsDisease Models AnimalchemistryColonic NeoplasmsFemalebusinessCarcinoma in SituGastrointestinal Endoscopy
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Assessment of Tumor Development and Wound Healing Using Endoscopic Techniques in Mice

2010

Mouse models of intestinal inflammation and colon cancer are valuable tools to gain insights into the pathogenesis of the corresponding human diseases. Recently, in vivo mouse endoscopy has been developed, allowing not only the high-resolution monitoring and scoring of experimental disease development, but also enables the investigator to perform manipulations, including local injection of reagents or the taking of biopsies for molecular and histopathologic analyses. Chromoendoscopic staining with methylene blue enables visualization of the crypt structure and allows discrimination between inflammatory and neoplastic changes. The development of endoscopic techniques in live mice opened new …

Pathologymedicine.medical_specialtyColorectal cancerBiopsyDiseasePathogenesisMiceMicromanipulation03 medical and health sciences0302 clinical medicineIn vivoIntestinal inflammationmedicineAnimalsHumans030304 developmental biologyWound Healing0303 health sciencesMiniaturizationHepatologymedicine.diagnostic_testbusiness.industryDisease mechanismsGastroenterologyEndoscopyColitismedicine.disease3. Good healthEndoscopyEndoscopes GastrointestinalDisease Models AnimalColonic Neoplasms030211 gastroenterology & hepatologybusinessWound healingGastroenterology
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Identification of novel mutations in the ABCA12 gene, c.1857delA and c.5653–5655delTAT, causing harlequin ichthyosis

2013

Abstract Harlequin ichthyosis (HI) is a severe autosomal recessive developmental disorder of the skin that is frequently but not always fatal in the first few days of life. In HI, mutations in both ABCA12 gene alleles must have a severe impact on protein function and most mutations are truncating. The presence of at least one nontruncating mutation (predicting a residual protein function) usually causes a less severe congenital ichthyosis (lamellar ichthyosis or congenital ichthyosiform erythroderma). Here we report on a girl with severe HI diagnosed by prenatal ultrasound at 33 5/7 week gestation. Ultrasound findings included ectropion, eclabium, deformed nose, hands and feet, joint contra…

Pathologymedicine.medical_specialtyCongenital ichthyosiform erythrodermaDNA Mutational AnalysisBiologyModels BiologicalPolymorphism Single NucleotideUltrasonography PrenatalExonFatal OutcomePregnancyCongenital ichthyosisGeneticsmedicineHumansABCA12Sequence DeletionGeneticsInfant NewbornEctropionGeneral MedicineLamellar ichthyosisHarlequin Ichthyosismedicine.diseaseEclabiumbiology.proteinATP-Binding Cassette TransportersFemalemedicine.symptomIchthyosis LamellarGene
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Endothelial nitric oxide synthase upregulation in the guinea pig organ of Corti after acute noise trauma.

2004

Endothelial nitric oxide synthase (eNOS) upregulation was identified 60 h after acute noise trauma in morphologically intact cells of the reticular lamina in the organ of Corti of the guinea pig in the second turn of the cochlea. Using gold-coupled anti-eNOS antibodies and electron microscopy, it was shown that eNOS expression was upregulated in all cell areas and cell types except inner hair cells. Furthermore, eNOS was found in the organelle-free cytoplasm and in mitochondria of various cell types. The density of eNOS in mitochondria was considerably higher compared with the surrounding cytoplasm. Since eNOS activity is regulated by calcium, the eNOS detection was combined with calcium pr…

Pathologymedicine.medical_specialtyCytoplasmNitric Oxide Synthase Type IIIGuinea Pigschemistry.chemical_elementCalciumMicrotubulesDownregulation and upregulationMicroscopy Electron TransmissionEnosStress PhysiologicalHair Cells AuditorymedicineAnimalsCalcium SignalingMolecular BiologyOrgan of CortiCytoskeletonbiologyGeneral NeuroscienceNitric Oxide Synthase Type IIIbiology.organism_classificationImmunohistochemistryCell biologyMitochondriaUp-RegulationNitric oxide synthaseActin CytoskeletonDisease Models Animalmedicine.anatomical_structureDrosophila melanogasterchemistryAcoustic StimulationHearing Loss Noise-InducedCytoplasmOrgan of Cortibiology.proteinCalciumNeurology (clinical)Nitric Oxide SynthaseNoiseIntracellularDevelopmental BiologyBrain research
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Microvascular in vivo assessment of reperfusion injury: significance of prostaglandin E1 and I2 in postischemic “no-reflow” and “reflow-paradox”

2004

Microvascular ischemia-reperfusion (I/R) injury is characterized by failure of capillary perfusion ("no-reflow") and reoxygenation-associated phenomena ("reflow-paradox"), including activation of leukocyte-endothelium interaction with cytotoxic mediator-induced loss of endothelial integrity. The objectives of this study were to elucidate the impact of both prostaglandins E(1) (PGE(1)) and I(2) (PGI(2)) in microvascular reperfusion injury, with special focus on the distinct pathophysiology of no-reflow- and reflow-paradox phenomena.By use of the hamster dorsal skinfold preparation and in vivo fluorescence microscopy, the microcirculation of a striated skin muscle was assessed before 4 h of p…

Pathologymedicine.medical_specialtyEndotheliummedicine.medical_treatmentIschemiaPharmacologyMicrocirculationCapillary Permeabilitychemistry.chemical_compoundIn vivoCricetinaemedicineAnimalsVascular Diseasescardiovascular diseasesAlprostadilMuscle SkeletalProstaglandin E1SkinMicroscopyMesocricetusbusiness.industryMicrocirculationmedicine.diseaseEpoprostenolPathophysiologyCapillariesChemotaxis Leukocytemedicine.anatomical_structurechemistryReperfusion InjuryModels Animalcardiovascular systemSurgeryEndothelium VascularbusinessReperfusion injuryPlatelet Aggregation InhibitorsProstaglandin EJournal of Surgical Research
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