Search results for "Muscarinic acetylcholine receptor"

showing 10 items of 182 documents

Aged Mice Devoid of the M3 Muscarinic Acetylcholine Receptor Develop Mild Dry Eye Disease

2021

The parasympathetic nervous system is critically involved in the regulation of tear secretion by activating muscarinic acetylcholine receptors. Hence, various animal models targeting parasympathetic signaling have been developed to induce dry eye disease (DED). However, the muscarinic receptor subtype (M1–M5) mediating tear secretion remains to be determined. This study was conducted to test the hypothesis that the M3 receptor subtype regulates tear secretion and to evaluate the ocular surface phenotype of mice with targeted disruption of the M3 receptor (M3R−/−). The experimental techniques included quantification of tear production, fluorescein staining of the ocular surface, environmenta…

Male0301 basic medicineM3medicine.disease_causeMiceM<sub>3</sub>0302 clinical medicineMuscarinic acetylcholine receptorTear secretionBiology (General)SpectroscopyCorneal epitheliumMice Knockouttear secretionChemistryEpithelium CornealMuscarinic acetylcholine receptor M3General Medicinedry eye diseaseComputer Science ApplicationsChemistrymedicine.anatomical_structureDry Eye SyndromesGoblet CellsConjunctivamedicine.medical_specialtyConjunctivaQH301-705.5ArticleCatalysisProinflammatory cytokineInorganic Chemistry03 medical and health sciencesmuscarinic receptorInternal medicinecorneamedicineAnimalsPhysical and Theoretical ChemistryQD1-999Molecular BiologyReceptor Muscarinic M3Goblet cellOrganic Chemistryeye diseasesMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologyTears030221 ophthalmology & optometrysense organsReactive Oxygen SpeciesOxidative stressInternational Journal of Molecular Sciences
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Non-neuronal cholinergic system contributes to corticosteroid resistance in chronic obstructive pulmonary disease patients

2016

Background: Inhaled corticosteroid (ICS) with long-acting beta-2 agonists is a well-documented combination therapy for chronic obstructive pulmonary disease (COPD) based on its additive anti-inflammatory properties. By contrast, the recommendation of ICS in combination with long-acting muscarinic antagonist (LAMA) is not evidence-based. In this study, neutrophils obtained from COPD patients were used to compare the anti-inflammatory effects of aclidinium bromide (a long-acting muscarinic antagonist) with corticosteroids and their potential additive effect. Methods: Human sputum and blood neutrophils were isolated from healthy individuals ( n = 37), patients with stable COPD ( n = 52) and th…

Male0301 basic medicineNeutrophilsVesicular Acetylcholine Transport ProteinsAnti-Inflammatory AgentsDrug ResistanceNon-neuronal cholinergic systemPulmonary Disease Chronic Obstructive0302 clinical medicineMuscarinic acetylcholine receptorCOPDDrug SynergismAclidinium bromideMuscarinic acetylcholine receptor M2Middle AgedReceptors MuscarinicBronchodilator AgentsCorticosteroidDrug Therapy CombinationFemaleInflammation Mediatorsmedicine.drugPulmonary and Respiratory Medicinemedicine.medical_specialtyCombination therapymedicine.drug_classCorticosteroid resistanceMuscarinic AntagonistsFluticasone propionateCholine O-Acetyltransferase03 medical and health sciencesAclidinium bromideInternal medicinemedicineHumansCOPDAgedDose-Response Relationship Drugbusiness.industryResearchSputumMuscarinic antagonistmedicine.disease030104 developmental biologyEndocrinology030228 respiratory systemCase-Control StudiesFluticasonebusinessTropanesRespiratory Research
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Pancreatic polypeptide stimulates mouse gastric motor activity through peripheral neural mechanisms

2016

Background Pancreatic polypeptide (PP) is supposed to be one of the major endogenous agonists of the neuropeptide Y4 receptor. Pancreatic polypeptide can influence gastrointestinal motility, acting mainly through vagal mechanisms, but whether PP acts directly on the stomach has not been explored yet. The aims of this study were to investigate the effects of PP on mouse gastric emptying, on spontaneous tone of whole stomach in vitro and to examine the mechanism of action. Methods Gastric emptying was measured by red phenol method after i.p. PP administration (1–3 nmol per mouse). Responses induced by PP (1–300 mmol L−1) on gastric endoluminal pressure were analyzed in vitro in the presence o…

Male0301 basic medicinemedicine.medical_specialtyPhysiologyGastric emptyingMuscarinic AntagonistsBiologyEndocrine and Autonomic SystemMice03 medical and health sciencesOrgan Culture Techniques0302 clinical medicineInternal medicineMuscarinic acetylcholine receptormedicineAnimalsPancreatic polypeptidePeripheral NervesPancreatic polypeptideReceptorAntrumDose-Response Relationship DrugGastric emptyingEndocrine and Autonomic SystemsStomachGastroenterologyMotilityAcetylcholineReceptors Neuropeptide YMice Inbred C57BL030104 developmental biologyEndocrinologymedicine.anatomical_structureMechanism of actionTachykininmedicine.symptomEnteric nervous systemGastrointestinal Motility030217 neurology & neurosurgeryAcetylcholinemedicine.drug
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Characterization of the muscarinic receptor subtype(s) mediating contraction of the guinea-pig lung strip and inhibition of acetylcholine release in …

1997

1 The muscarinic receptor subtypes mediating contraction of the guinea-pig lung strip and inhibition of the release of acetylcholine from cholinergic vagus nerve endings in the guinea-pig trachea in vitro have previously been characterized as M-2-like, i.e. having antagonist affinity profiles that are qualitatively similar but quantitatively dissimilar compared to cardiac M-2 receptors. The present study sought to establish definitely the identity of these receptor subtypes by using the selective muscarinic receptor antagonist, tripitramine. Guinea-pig atria and guinea-pig trachea (postjunctional contractile response) were included for reference.2 It was found that tripitramine antagonized …

MaleAUTORECEPTORSlung strip guinea-pigsubtypes ofatria guinea-pigBenzodiazepinesFUNCTIONAL-CHARACTERIZATIONMuscarinic acetylcholine receptorReceptorLungAIRWAYSeducation.field_of_studyguinea-pigSMOOTH-MUSCLEMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2METHOCTRAMINE-RELATED TETRAAMINESAtrial FunctionReceptors MuscarinicSchild regressionTracheaDepression ChemicalPapersHEARTFemaleAcetylcholineBINDING-PROPERTIESmedicine.drugMuscle Contractionmedicine.medical_specialtyCardiotonic Agentstrachea guinea-piglung stripPopulationGuinea PigsMuscarinic AntagonistsBiologyTritiummuscarinic receptorRABBITInternal medicinemedicineAnimalsNEUROTRANSMITTER RELEASEHeart AtriaeducationAcetylcholine receptorPharmacologyprejunctional muscarinic autoreceptorMuscle SmoothMyocardial ContractionAcetylcholineElectric StimulationEndocrinologyatriaCELLSBritish journal of pharmacology
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Endogenous noradrenaline release from guinea-pig isolated trachea is inhibited by activation of M2 receptors

1992

Overflow of endogenous noradrenaline (NA) from guinea-pig isolated tracheae was evoked by electrical field stimulation (3 Hz, 540 pulses). The muscarinic receptor agonist oxotremorine inhibited the evoked overflow of NA in a concentration-dependent manner (EC50 84 nM). Methoctramine, pirenzepine and p-fluoro-hexahydrosiladiphenidol (each 1 microM) shifted the concentration-response curves of oxotremorine to the right with apparent pA2 values of 7.60, 6.74 and 6.18, respectively. It is concluded that sympathetic nerve terminals in the guinea-pig trachea are endowed with inhibitory muscarinic M2 receptors.

MaleAgonistmedicine.medical_specialtymedicine.drug_classGuinea PigsMuscarinic AntagonistsDiaminesIn Vitro TechniquesInhibitory postsynaptic potentialGuinea pigNorepinephrinechemistry.chemical_compoundPiperidinesInternal medicineMuscarinic acetylcholine receptorMethoctraminemedicineOxotremorineAnimalsPharmacologyDose-Response Relationship DrugOxotremorineParasympatholyticsMuscarinic acetylcholine receptor M2PirenzepineReceptors MuscarinicPirenzepineTracheaEndocrinologychemistryFemaleResearch Articlemedicine.drugBritish Journal of Pharmacology
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The effects of several muscarinic antagonists on pre- and postsynaptic receptors in the isolated rabbit heart

1981

In order to reveal possible differences between pre- and postsynaptic muscarine receptors, seven antagonists were tested for their affinities on these receptor sites in the rabbit isolated perfused heart. Methacholine was used as an agonist to inhibit the noradrenaline overflow evoked by electrical stimulation (3 Hz, 3 min) of the sympathetic nerves (presynaptic parameter) and to decrease the systolic tension development of the right atrium (postsynaptic parameter). The affinity of an antagonist was expressed as pA2. A decreasing order of potency was obtained with ipratropium, scopolamine, atropine, trihexyphenidyl, amitriptyline, and gallamine, both for pre- and postsynaptic responses. The…

MaleAgonistmedicine.medical_specialtymedicine.drug_classPopulationStimulationIn Vitro TechniquesNorepinephrinechemistry.chemical_compoundPostsynaptic potentialInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsReceptors CholinergicReceptoreducationPharmacologyeducation.field_of_studyBinding SitesMuscarineDose-Response Relationship DrugGallamine TriethiodideMyocardiumParasympatholyticsGeneral MedicineReceptors MuscarinicReceptors NeurotransmitterEndocrinologychemistryFemaleMethacholineRabbitsmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Effects of scopolamine on dopamine neurons in the substantia nigra : role of the pedunculopontine tegmental nucleus

2009

Previous neurochemical and behavioral studies suggest that muscarinic receptor antagonism has an excitatory effect on the nigrostriatal dopamine (DA) system. Using in vivo extracellular single unit recording, this study examined whether blockade of the muscarinic receptor by scopolamine alters the firing properties of DA neurons in the substantia nigra (SN). Scopolamine was administered either systemically or locally to DA neurons using microiontophoresis. Surprisingly, scopolamine did not cause any significant change in either the firing rate or pattern of the spontaneously active DA neurons. However, systemic injection of scopolamine significantly increased the number of active DA neurons…

MaleDopamineParkinson's diseaseScopolamineAction PotentialsSubstantia nigraMuscarinic AntagonistsStriatumelectrophysiology microiontophoresisSettore BIO/09 - FisiologiaRats Sprague-DawleyCellular and Molecular NeuroscienceDopamineBasal gangliaMuscarinic acetylcholine receptorPedunculopontine Tegmental NucleusmedicineSubstantia nigraAnimalsPedunculopontine Tegmental NucleusNeuronsAnalysis of VarianceDose-Response Relationship DrugChemistryDrug Administration RoutesIontophoresisAcetylcholineRatsSubstantia Nigranervous systemAutoreceptorBasal gangliaNeuroscienceAcetylcholinemedicine.drug
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Muscarinic Properties of Compounds Related to Arecaidine Propargyl Ester

2011

A series of new analogues of the arecaidine propargyl ester (CAS 35516-99-5, APE, 1a) with alcohols consisting of 4 or 5 carbon atoms were investigated at muscarinic receptor subtypes. The muscarinic activity of the quaternary and tertiary salts of the APE-related compounds were assayed on the isolated guinea-pig ileum (M 3 receptor subtype) and guinea-pig left atria (M 2 receptor subtype) as well as on rabbit isolated vas deferens (M 1 receptor subtype). The structural variations made in the APE molecule, replacing the triple bond in the ester side chain with structures such as double bond, an allene moiety, a single bond, a cyclopropyl group or two triple bonds should alter the selectivit…

MaleDouble bondStereochemistryArecolineGuinea PigsCholinergic AgentsIn Vitro Techniqueschemistry.chemical_compoundVas DeferensIleumDrug DiscoveryMuscarinic acetylcholine receptorAnimalsMoietySingle bondReceptorReceptor Muscarinic M3chemistry.chemical_classificationReceptor Muscarinic M2Receptor Muscarinic M1HeartMuscle SmoothArecaidineTriple bondMyocardial ContractionReceptors MuscarinicchemistryPropargylFemaleRabbitsMuscle ContractionArzneimittelforschung
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Prejunctional M1 and postjunctional M3 muscarinic receptors in the circular muscle of the guinea-pig ileum.

1995

The effects of subtype-selective muscarinic receptor antagonists on electrically evoked release of acetylcholine and muscle contraction were compared in circular muscle preparations of the guinea-pig ileum. Incubation of the preparation with [3H]choline resulted in the formation of [3H]acetylcholine. Electrical stimulation caused the release of [3H]acetylcholine which was abolished by tetrodotoxin and omission of calcium from the medium. 5-Hydroxytryptamine (10 microM) and the nicotinic agonist 1,1-dimethyl-4-phenyl-piperazinium (300 microM) did not change acetylcholine release. The muscarinic antagonists pirenzepine (M1 selective), AF-DX 116 (M2 selective) and hexahydrosiladifenidol (M3 se…

MaleGuinea PigsNeuromuscular JunctionMuscarinic AntagonistsPharmacologyIn Vitro TechniquesCholinePiperidinesIleumMuscarinic acetylcholine receptorMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptor M4medicineAnimalsPharmacologyChemistryMuscarinic acetylcholine receptor M3ParasympatholyticsMuscarinic acetylcholine receptor M2Muscle SmoothGeneral MedicineMuscarinic acetylcholine receptor M1AnatomyPirenzepinePirenzepineReceptors MuscarinicAcetylcholineFemaleAcetylcholinemedicine.drugMuscle ContractionNaunyn-Schmiedeberg's archives of pharmacology
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Anti-inflammatory and antioxidant effects of muscarinic acetylcholine receptor (mAChR) activation in the rat hippocampus

2019

AbstractRecently we found that acute treatment with Oxotremorine (Oxo), a non-selective mAChRs agonist, up-regulates heat shock proteins and activates their transcription factor heat shock factor 1 in the rat hippocampus. Here we aimed to investigate: a) if acute treatment with Oxo may regulate pro-inflammatory or anti-inflammatory cytokines and oxidative stress in the rat hippocampus; b) if chronic restraint stress (CRS) induces inflammatory or oxidative alterations in the hippocampus and whether such alterations may be affected by chronic treatment with Oxo. In the acute experiment, rats were injected with single dose of Oxo (0.4 mg/kg) and sacrificed at 24 h, 48 h and 72 h. In the CRS ex…

MaleHydrocortisonemedicine.medical_treatmentInterleukin-1betaNeuroimmunologyAnti-Inflammatory Agentslcsh:MedicinePharmacologymedicine.disease_causeHippocampusSettore BIO/09 - FisiologiaAntioxidantsSuperoxide Dismutase-1Muscarinic acetylcholine receptorPhosphorylationlcsh:Sciencechemistry.chemical_classificationMultidisciplinarybiologyneurodegenerationAlzheimer's diseaseReceptors MuscarinicNeuroprotective AgentsCytokineSignal Transductionmedicine.drugRestraint PhysicalAgonistmedicine.drug_classScopolaminemuscarinic acetylcholine receptorMuscarinic AgonistsArticleOxotremorine anti-inflammatory cytokinesSuperoxide dismutaseHeat shock proteinOxotremorinemedicineAnimalsRats WistarInflammationReactive oxygen speciesInterleukin-6Superoxide DismutaseOxotremorinelcsh:RTranscription Factor RelARatsOxidative Stresschemistrybiology.proteinlcsh:QReactive Oxygen SpeciesProtein Processing Post-TranslationalOxidative stressScientific Reports
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