Search results for "Muscle Contraction"

showing 10 items of 569 documents

Plantar flexor activation capacity and H reflex in older adults: adaptations to strength training.

2002

The purpose of this study was to investigate whether the voluntary neural drive and the excitability of the reflex arc could be modulated by training, even in old age. To this aim, the effects of a 16-wk strengthening program on plantar flexor voluntary activation (VA) and on the maximum Hoffman reflex (Hmax)-to-maximum M wave (Mmax) ratio were investigated in 14 elderly men (65–80 yr). After training, isometric maximum voluntary contraction (MVC) increased by 18% ( P < 0.05) and weight-lifting ability by 24% ( P < 0.001). Twitch contraction time decreased by 8% ( P < 0.01), but no changes in half relaxation time and in peak twitch torque were observed. The VA, assessed by twitch …

MaleVolitionAgingPhysiologydegenerationadaptationIsometric exerciseNerve conduction velocityhuman experimentH-ReflexTriceps surae muscleTwitch interpolationadaptation; aged; aging; article; controlled study; degeneration; excitability; exercise; Hoffmann reflex; human; human experiment; male; muscle contraction; muscle isometric contraction; muscle relaxation; muscle twitch; nerve conduction; normal human; plantaris muscle; priority journal; reflex arc; strength; torque; training; voluntary movement; Aging; Exercise; Triceps surae; Twitch interpolationAged 80 and overtrainingvoluntary movementReflex arcarticlereflex arcmusculoskeletal systemAdaptation Physiologicalmuscle twitchmedicine.anatomical_structuremuscle relaxationpriority journalCardiologyTriceps suraemedicine.symptomstrengthMuscle contractionMuscle Contractionmedicine.medical_specialtyWeight Liftingplantaris muscleStrength trainingPhysical exercisePhysical medicine and rehabilitationPhysiology (medical)Internal medicineexcitabilitymedicineHumanscontrolled studyhumannormal humanMuscle SkeletalExercisenerve conductionAgedmuscle isometric contractionbusiness.industrybody regionsHoffmann reflexTorqueH-reflexbusinessJournal of applied physiology (Bethesda, Md. : 1985)
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Effect of xanthine oxidase-generated extracellular superoxide on skeletal muscle force generation

2009

Skeletal muscle contractions increase superoxide anion in skeletal muscle extracellular space. We tested the hypotheses that 1) after an isometric contraction protocol, xanthine oxidase (XO) activity is a source of superoxide anion in the extracellular space of skeletal muscle and 2) the increase in XO-derived extracellular superoxide anion during contractions affects skeletal muscle contractile function. Superoxide anion was monitored in the extracellular space of mouse gastrocnemius muscles by following the reduction of cytochrome c in muscle microdialysates. A 15-min protocol of nondamaging isometric contractions increased the reduction of cytochrome c in microdialysates, indicating an …

MaleXanthine Oxidasemedicine.medical_specialtyPhysiologyOxypurinolfree radicalsSuperoxide dismutaseExtensor digitorum longus muscleMice03 medical and health sciencesGastrocnemius musclechemistry.chemical_compound0302 clinical medicineSuperoxidescontractile functionIsometric ContractionPhysiology (medical)Internal medicinemedicineAnimalsMuscle Skeletal030304 developmental biologySoleus muscle0303 health sciencesexercisebiologyMuscle fatigueSuperoxide DismutaseChemistrySuperoxideCytochromes cSkeletal muscleArticlesmusculoskeletal systemElectric StimulationMice Inbred C57BLmedicine.anatomical_structureEndocrinologyBiochemistryModels AnimalMuscle Fatiguebiology.proteinmedicine.symptomExtracellular Space030217 neurology & neurosurgeryMuscle ContractionMuscle contractionAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology
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Complex I dysfunction and tolerance to nitroglycerin: an approach based on mitochondrial-targeted antioxidants.

2006

Nitroglycerin (GTN) tolerance was induced in vivo (rats) and in vitro (rat and human vessels). Electrochemical detection revealed that the incubation dose of GTN (5×10 −6 mol/L) did not release NO or modify O 2 consumption when administered acutely. However, development of tolerance produced a decrease in both mitochondrial O 2 consumption and the K m for O 2 in animal and human vessels and endothelial cells in a noncompetitive action. GTN tolerance has been associated with impairment of GTN biotransformation through inhibition of aldehyde dehydrogenase (ALDH)-2, and with uncoupling of mitochondrial respiration. Feeding rats with mitochondrial-targeted antioxidants (mitoquinone [MQ]) and i…

MaleantioxidantAntioxidantPhysiologyUbiquinonemedicine.medical_treatmentMuscle RelaxationVasodilator AgentsAldehyde dehydrogenasePharmacologyMitochondrionmedicine.disease_causeAntioxidantsMuscle Smooth VascularRats Sprague-Dawleychemistry.chemical_compoundNitroglycerinDrug toleranceoxidative stressCyclic GMPchemistry.chemical_classificationbiologyAldehyde Dehydrogenase MitochondrialDrug ToleranceGlutathioneMitochondriamitochondriaBiochemistrycardiovascular systemCardiology and Cardiovascular Medicinecirculatory and respiratory physiologyMuscle ContractionendotheliumIn Vitro TechniquesMitochondrial ProteinsOrganophosphorus CompoundsOxygen ConsumptionRespirationmedicineAnimalsHumansReactive oxygen speciesElectron Transport Complex IDose-Response Relationship DrugEndothelial CellsGlutathioneAldehyde DehydrogenasenitroglycerinRatsOxidative Stresschemistrybiology.proteinReactive Oxygen SpeciesOxidative stressCirculation research
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Effects of Constant and Doublet Frequency Electrical Stimulation Patterns on Force Production of Knee Extensor Muscles

2016

International audience; This study compared knee extensors' neuromuscular fatigue in response to two 30-minute stimulation patterns: constant frequency train (CFT) and doublet frequency train (DFT). Fifteen men underwent two separate sessions corresponding to each pattern. Measurements included torque evoked by each contraction and maximal voluntary contractions (MVC) measured before and immediately after the stimulation sessions. In addition, activation level and torque evoked during doublets (Pd) and tetanic contractions at 80-Hz (P80) and 20-Hz (P20) were determined in six subjects. Results indicated greater mean torque during the DFT stimulation session as compared with CFT. But, no dif…

MalecontractionsMuscle PhysiologyCritical Care and Emergency MedicineContraction (grammar)Knee JointRefractory Period ElectrophysiologicalPhysiologyRefractory periodKneeslcsh:MedicineStimulationKnee JointsIsometric exerciseElectromyographyPathology and Laboratory MedicineEndoplasmic Reticulum0302 clinical medicineNuclear magnetic resonanceMedicine and Health Scienceslcsh:ScienceSpinal Cord InjuryMusculoskeletal SystemTrauma MedicinePhysicsSecretory PathwayMultidisciplinarymedicine.diagnostic_test[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]PhysicsClassical MechanicsAnatomyNeurologyCell ProcessesPhysical SciencesMuscle FatigueLegsspinal-cord-injuryAnatomyCellular Structures and Organellesmedicine.symptomadaptationsTraumatic InjuryindividualsResearch ArticleMuscle ContractionMuscle contractionAdulthuman skeletal-muscleSurgical and Invasive Medical ProcedurestrainsMotion03 medical and health sciencesSigns and SymptomsDiagnostic MedicineIsometric ContractionSarcoplasmic Reticula[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]medicineHumansFunctional electrical stimulationMuscle SkeletalpotentiationFunctional Electrical StimulationMuscle fatigueElectromyographylcsh:RLimbs (Anatomy)Biology and Life SciencesCell Biology030229 sport sciencesElectric StimulationJoints (Anatomy)Torquerecruitmentresponseslcsh:Qfatigue030217 neurology & neurosurgery
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Role of cholinergic neurons in the motor effects of glucagon-like peptide-2 in mouse colon

2010

Glucagon-like peptide-2 (GLP-2) reduces mouse gastric tone and small intestine transit, but its action on large intestine motility is still unknown. The purposes of the present study were 1) to examine the influence of GLP-2 on spontaneous mechanical activity and on neurally evoked responses, by recording intraluminal pressure from mouse isolated colonic segments; 2) to characterize GLP-2 mechanism of action; and 3) to determine the distribution of GLP-2 receptor (GLP-2R) in the mouse colonic muscle coat by immunohistochemistry. Exogenous GLP-2 (0.1–300 nM) induced a concentration-dependent reduction of the spontaneous mechanical activity, which was abolished by the desensitization of GLP-…

Maleendocrine systemmedicine.medical_specialtyCarbacholColonPhysiologymedicine.drug_classBlotting WesternBiologyApaminSettore BIO/09 - FisiologiaMicechemistry.chemical_compoundenteric nervous systemcolonic motilityPhysiology (medical)Internal medicineGlucagon-Like Peptide 2Receptors GlucagonmedicineAnimalsCholinergic neuronNeuronsAnalysis of VarianceDose-Response Relationship DrugHepatologydigestive oral and skin physiologyGastroenterologyMuscle Smoothgastrointestinal hormoneMotor neuronReceptor antagonistImmunohistochemistryCholine acetyltransferaseElectric StimulationacetylcholineEndocrinologymedicine.anatomical_structurechemistryGlucagon-Like Peptide-2 ReceptorCholinergicGastrointestinal Motilityhormones hormone substitutes and hormone antagonistsAcetylcholineMuscle Contractionmedicine.drugAmerican Journal of Physiology-Gastrointestinal and Liver Physiology
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Interplay between PACAP and NO in mouse ileum

2003

Abstract We investigated the possibility that pituitary adenylate cyclase activating peptide (PACAP) has a role in the control of contractility in the mouse ileum. PACAP-(1-27) produced tetrodotoxin (TTX)-insensitive, concentration-dependent reduction of the amplitude of the spontaneous contractions of longitudinal muscle up to their complete disappearance. This effect was inhibited by PACAP-(6-38), PACAP receptor antagonist, and by apamin, blocker of small-conductance Ca2+-activated K+-channels. Nω-nitro- l -arginine methyl ester (L-NAME), nitric oxide (NO) synthase inhibitor, reduced the PACAP-inhibitory response, and the joint application of apamin plus L-NAME produced additive effects. …

Maleendocrine systemmedicine.medical_specialtyMuscle RelaxationMouse ileumStimulationIn Vitro TechniquesApaminSettore BIO/09 - FisiologiaContractilityMiceCellular and Molecular Neurosciencechemistry.chemical_compoundSmooth muscleIleumInternal medicinemedicineAnimalsMyocyteNitric Oxide DonorsPharmacologyDose-Response Relationship DrugNeuropeptidesNitric oxideSmooth muscle contractionMice Inbred C57BLPituitary adenylate cyclase-activating peptideEndocrinologyApaminchemistryTetrodotoxinPituitary Adenylate Cyclase-Activating PolypeptideSodium nitroprussidePACAP (pituitary adenylate cyclase activating peptide)hormones hormone substitutes and hormone antagonistsmedicine.drugNeuropharmacology
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Differences in the nitric oxide/soluble guanylyl cyclase signalling pathway in the myocardium of neonatal and adult rats

2000

Abstract The effects of a nitric oxide-donor, S -nitroso- N -acetylpenicillamine, and a direct activator of soluble guanylyl cyclase, 3-(5′-hydroxymethyl-2′-furyl)-1-benzyl indazole (YC-1), on force of contraction ( F c ) and L-type Ca 2+ currents ( I Ca(L) ) were investigated in myocardial preparations from neonatal and adult rats. Since hearts from adult and neonatal animals contained 160 and 47 mg/100 g wet weight myoglobin, respectively, its possible interaction with both drugs was also investigated. Both S -nitroso- N -acetylpenicillamine (100 μM) and YC-1 (30 μM) were ineffective in myocardial preparations from adult rats but reduced the magnitude of I Ca(L) and F c in preparations fr…

Maleinorganic chemicalsmedicine.medical_specialtyContraction (grammar)Calcium Channels L-Typechemistry.chemical_elementCalciumNitric OxideNitric oxideRats Sprague-Dawleychemistry.chemical_compoundInternal medicinemedicineAnimalsCyclic GMPPharmacologychemistry.chemical_classificationDose-Response Relationship DrugMyoglobinMyocardiumPenicillamineAge FactorsMyocardial ContractionIn vitroRatsEndocrinologyEnzymeAnimals NewbornchemistryMyoglobinGuanylate Cyclasemedicine.symptomSoluble guanylyl cyclaseSignal TransductionMuscle contractionEuropean Journal of Pharmacology
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Antagonism of calcium by zinc in guinea-pig isolated taenia caeci and trachealis muscle

1989

1 In guinea-pig isolated taenia caeci and trachealis bathed in a K+-rich, Ca2+-free medium, CaCl2 (0.01-10 mM) produced a concentration-dependent contraction. Zn2+ (0.01-1 mM), Cd2+ (0.01-1 mM), verapamil (0.01-100 microM) and trifluoperazine (1-100 microM) were effective antagonists of CaCl2-induced responses. 2 Zn2+ and Cd2+ in concentrations from 0.01 to 1 mM were without effect on the tone of taenia and trachea in normal Tris solution. Conversely, Zn2+ and Cd2+, in concentrations of 1 mM, caused contraction of these tissues in a K+-rich, Ca2+-free medium. Zn2+ (1 mM)-induced contractions of taenia and trachea were completely inhibited by verapamil (10 microM). 3 In taenia and trachea sk…

Maleinorganic chemicalsmedicine.medical_specialtyContraction (grammar)Guinea Pigschemistry.chemical_elementTrifluoperazineIn Vitro TechniquesCalciumGuinea pigInternal medicinemedicineAnimalsCecumPharmacologyChemistryCell MembraneMuscle SmoothAcetylcholineTrifluoperazineTracheaZincEndocrinologyVerapamilTrachealis muscleVerapamilCalciumFemalemedicine.symptomAcetylcholineCadmiumResearch Articlemedicine.drugMuscle contraction
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Effects of alkylxanthines on contractility of diaphragm fibres isolated from normal and sensitized guinea-pigs.

1993

Abstract This study investigates the effects of alkylxanthines on twitch tension generated by electrical stimulation (supramaximal pulses, 0·2 ms duration, 1 Hz) of diaphragm muscle fibres isolated from normal and actively-sensitized guinea-pigs. Caffeine, theophylline and theobromine increased, in a concentration-dependent manner (50–500 μm), twitch tension in normal and sensitized diaphragm. Caffeine (500 μm) enhanced contractility to a greater extent than theophylline or theobromine. Twitch potentiation by caffeine (500 μm) was significantly greater in sensitized diaphragm. Verapamil (0·1–100 μm) did not alter twitch contractions in the absence or presence of alkylxanthines in normal or …

Malemedicine.medical_specialtyAdenosineDiaphragmGuinea PigsPharmaceutical ScienceIn Vitro TechniquesDantroleneDantroleneContractilitychemistry.chemical_compoundTheophyllineInternal medicineCaffeinemedicineAnimalsTheophyllineRespiratory systemRats WistarPharmacologyMuscle SmoothSerum Albumin Bovinemusculoskeletal systemElectric StimulationDiaphragm (structural system)Bronchodilator AgentsCulture MediaRatsEndocrinologychemistryVerapamilXanthinesEnprofyllineTheobromineCalciumFemaleImmunizationmedicine.symptomCaffeineExtracellular Spacemedicine.drugMuscle contractionMuscle ContractionThe Journal of pharmacy and pharmacology
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A1 receptors mediate adenosine inhibitory effects in mouse ileum via activation of potassium channels.

2008

Abstract Aims We investigated the effects induced by exogenous adenosine on the spontaneous contractile activity of the longitudinal muscle of a mouse ileum, the receptor subtypes activated, the involvement of enteric nerves and whether opening of K + channels was a downstream event leading to the observed effects. Main methods Mechanical responses of the mouse ileal longitudinal muscle to adenosine were examined in vitro as changes in isometric tension. Key findings Adenosine caused a concentration-dependent reduction of the spontaneous contraction amplitude of the ileal longitudinal muscle up to its complete disappearance. This effect induced was markedly reduced by an A 1 receptor antago…

Malemedicine.medical_specialtyAdenosinePotassium ChannelsAdenosine A2 Receptor AgonistsMouse ileumBlotting WesternAdenosine A3 Receptor AntagonistsAdenosine A1 Receptor AntagonistsApaminSettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular BiologyAdenosine A1 receptorchemistry.chemical_compoundMiceAdenosine A3 Receptor AgonistsIleumInternal medicineNeural PathwaysmedicinePotassium Channel BlockersPurinergic P1 Receptor AgonistsAnimalsGeneral Pharmacology Toxicology and PharmaceuticsP1 purinoceptorDose-Response Relationship DrugChemistryReceptor Adenosine A1Mechanical activityMuscle SmoothGeneral MedicinePurinergic signallingIberiotoxinAdenosine A3 receptorAdenosineAdenosine receptorAdenosine A1 Receptor AgonistsAdenosine A2 Receptor AntagonistsMice Inbred C57BLEndocrinologyPurinergic P1 Receptor AntagonistsAdenosine A2B receptormedicine.drugMuscle ContractionLife sciences
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