Search results for "Mutagenicity"

showing 10 items of 92 documents

Genotoxic potential of by-products in drinking water in relation to water disinfection: Survey of pre-ozonated and post-chlorinated drinking water by…

2006

Mutagenic potential of drinking water samples derived from ranneywells was studied. 100-100 l of untreated (rough) and ozone-treated as well as chlorinated-disinfected water were dropped on and adsorbed by macroreticular resin columns (Serdolit PAD-III and Amberlite XAD-2). The adsorbed material was desorbed by methanol and dichloromethane. After elimination of the solvents by vacuum distillation the adsorbed material was dissolved in dimethylsulfoxide. The mutagenic activity was tested in the Ames-Salmonella/rat liver microsome system. The tester strains were TA-98 and TA-100. The material adsorbed to Serdolit PAD-III from rough and also disinfected water did not induce mutagenicity in cas…

MaleSalmonella typhimuriumAmberliteIn Vitro TechniquesToxicologymedicine.disease_causeAmes testchemistry.chemical_compoundOxidants PhotochemicalOzoneAdsorptionWater SupplyBy-productmedicineAnimalsDimethyl SulfoxideHistidineDichloromethaneChromatographyMutagenicity TestsSterilizationSterilization (microbiology)RatschemistryEnvironmental chemistryMicrosomes LiverMethanolChlorineGenotoxicityChromatography LiquidDisinfectantsMutagensToxicology
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In vivo antigenotoxic effects of dietary allyl sulfides in the rat

1997

The effects of dietary administration of diallyl sulfide (DAS), diallyl disulfide (DADS) and allyl mercaptan (AM) on the genotoxicity of different chemicals were studied in two experimental systems: (i) measurement of hepatic DNA single-strand breaks induced in rats by aflatoxin B1 (AFB1), N-nitrosodimethylamine (NDMA) or methylnitrosourea (MNU); (ii) mutagenicity of AFB1 or NDMA on Salmonella typhimurium TA100 using hepatic S9 from rats fed allyl sulfides as the activation system. All compounds strongly reduced hepatic DNA breaks induced by AFB1 and NDMA but did not modify the genotoxicity of MNU. In the Ames test, the mutagenicity of NDMA was strongly inhibited by hepatic S9 from rats fed…

MaleSalmonella typhimuriumCancer ResearchAflatoxin B1[SDV]Life Sciences [q-bio]Allyl compoundMutagenSulfidesmedicine.disease_cause030226 pharmacology & pharmacyDimethylnitrosamineAmes test03 medical and health scienceschemistry.chemical_compound0302 clinical medicineN-NitrosodimethylaminemedicineAnimalsAnticarcinogenic AgentsDisulfidesComputingMilieux_MISCELLANEOUSMutagenicity TestsDiallyl disulfidefood and beveragesAntimutagenic AgentsMethylnitrosoureaRats3. Good healthAllyl Compounds[SDV] Life Sciences [q-bio]LiverOncologychemistryBiochemistry030220 oncology & carcinogenesisRATAllyl MercaptanCARCINOGENESEAllyl SulfideGenotoxicityDNA DamageMutagensCancer Letters
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Clavine alkaloids and derivatives as mutagens detected in the Ames test.

1992

Eight cytostatic clavines were investigated for mutagenicity in Salmonella typhimurium (reversion of the his-strains TA98, TA100, TA102 and TA1537), directly and in the presence of a mammalian xenobiotic metabolizing system, S9 (NADPH-fortified postmitochondrial fraction of liver homogenate from Aroclor 1254-treated rats). Four compounds (festuclavine, 17-bromofestuclavine, 1-allylelymoclavine and 1-methyllysergol methyl ether) were direct mutagens, whose activity was enhanced in the presence of S9. The other compounds (1-cyclopentylfestuclavine, 13-bromo-1-cyclopropylmethylfestuclavine, 6-cyano-1-propyl-6-norfestuclavine and 6-allyl-1-propyl-6-norfestuclavine) showed mutagenic effects only…

MaleSalmonella typhimuriumCancer ResearchSalmonellaErgot AlkaloidsReversionEthermedicine.disease_causeAmes testRats Sprague-Dawleychemistry.chemical_compoundmedicineAgroclavineAnimalsPharmacology (medical)BiotransformationPharmacologychemistry.chemical_classificationMutagenicity Testsfood and beveragesAntimicrobialRatsEnzymeOncologyBiochemistrychemistryXenobioticMutagensAnti-cancer drugs
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Mechanism-based predictions of interactions.

1994

Abstract Exposure to more than one toxic compound is common in real life. The resulting toxic effects are often more than the simple sum of the effects of the individual compounds. It is unlikely that it will ever be possible to test all combinations. It is therefore highly desirable to improve or develop means for reasonably approximating predictions of interactions. In order to be valid and extrapolatable, these predictions are most promising if they are mechanism-based. Examples will be given for possibilities of mechanism-based predictions of interactions which exceed trivialities of simple increases by enzyme induction of enzymatic rates of a given biotransformation pathway leading to …

MaleSalmonella typhimuriumEndogenous FactorsHealth Toxicology and MutagenesisMetaboliteMechanism basedRats sprague dawleyXenobioticsRats Sprague-Dawleychemistry.chemical_compoundStilbenesBenzo(a)pyreneAnimalsIn real lifeDrug InteractionsPhosphorylationEpoxide HydrolasesMutagenicity TestsMechanism (biology)Public Health Environmental and Occupational HealthRatschemistryBiochemistryEnzyme InductionMicrosomes LiverBiochemical engineeringXenobioticMutagenicity TestResearch ArticleEnvironmental Health Perspectives
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Antimutagenic activity of organosulfur compounds from Allium is associated with phase II enzyme induction

2001

In a previous study, we showed that naturally occurring organosulfur compounds (OSCs) from garlic and onion modulated the activation of carcinogen via the alteration of cytochromes P450. The present study was undertaken to determine the incidence of the in vivo induction of phase II enzymes by individual OSCs on the genotoxicity of several carcinogens. Diallyl sulfide (DAS), diallyl disulfide (DADS), dipropyl sulfide (DPS) and dipropyl disulfide (DPDS), were administered by gavage (1mmol/kg) to male SPF Wistar rats for 4 consecutive days. The effects of treatments on phase II enzymes and on the genotoxicity of carcinogens were evaluated with hepatic cytosols and microsomes from OSCs-treated…

MaleSalmonella typhimuriumHealth Toxicology and Mutagenesis[SDV]Life Sciences [q-bio]Allyl compoundAdministration OralSulfidesmedicine.disease_causeAmes testAllium03 medical and health scienceschemistry.chemical_compoundPropane0302 clinical medicineGeneticsmedicineNAD(P)H Dehydrogenase (Quinone)AnimalsDisulfidesRats WistarEpoxide hydrolaseCarcinogenComputingMilieux_MISCELLANEOUS030304 developmental biologyGlutathione TransferaseEpoxide Hydrolases0303 health sciencesDose-Response Relationship DrugChemistryDiallyl disulfideMutagenicity TestsAntimutagenic Agents3. Good healthRatsSpecific Pathogen-Free Organisms[SDV] Life Sciences [q-bio]Allyl CompoundsBiochemistryAntimutagenic AgentsLiver030220 oncology & carcinogenesisEnzyme InductionAntimutagenGenotoxicityMutagensSubcellular Fractions
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Antimutagenic effects and possible mechanisms of action of vitamins and related compounds against genotoxic heterocyclic amines from cooked food.

1999

Possible antimutagenic activity of 26 vitamins and related compounds - ascorbic acid, beta-carotene, cyanocobalamin, folic acid, nicotinic acid, nicotinamide, pantothenic acid, pyridoxale, pyridoxamine, pyridoxine, retinal, retinol, retinoic acid, retinyl acetate, retinyl palmitate, riboflavin, riboflavin 5'-phosphate, flavin adenine dinucleotide (FAD), alpha-tocopherol, alpha-tocopherol acetate, vitamins K(1), K(3), K(4), 1, 4-naphthoquinone, and coenzyme Q(10) - was tested against six heterocyclic amine (HCA) mutagens, i.e., 2-amino-3-methyl-imidazo[4, 5-f]quinoline (IQ), 2-amino-3,4-dimethyl-imidazo[4,5-f]quinoline (MeIQ), 2-amino-3,8-dimethyl-imidazo[4,5-f]quinoxaline (MeIQx), 2-amino-1…

MaleSalmonella typhimuriumHot TemperatureVitamin KHealth Toxicology and MutagenesisRiboflavinFood ContaminationRetinyl acetateIn Vitro TechniquesRats Sprague-Dawleychemistry.chemical_compoundMenadioneRetinyl palmitateGeneticsAnimalsVitamin ABiotransformationFlavin adenine dinucleotidechemistry.chemical_classificationNicotinamideMutagenicity TestsAntimutagenic AgentsVitaminsAscorbic acidRatschemistryBiochemistryHeterocyclic amineFlavin-Adenine DinucleotideMicrosomes LiverQuinolinesFood AnalysisMutagensMutation research
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Propyldazine is mutagenic inSalmonella typhimurium and Escherichia coli: Distinct specificity for strains TA1537 AND TA97

1985

The antihypertensive drug propyldazine (Atensil) was demonstrated to be muta- genic with auxotrophic mutants of Salmonella typhimurium and Escherichia coli. Addition of liver S9 mix (postmitochondrial supernatant fraction supplemented with an NADPH-generating system) had little, if any, effect on the mutagenicity. The mutagenicity showed an unusual pattern of strain specificity. Increased fre- quencies of reversion were observed with all strains whose auxotrophy was caused by frame-shift mutations: the number of revertant colonies per plate from S. typhimurium TA98, TA1538, TA97, and TA1537 was increased up to 5-, 9-, 43-, and 160-fold, respectively, above background. Among the strains that…

MaleSalmonella typhimuriumSalmonellaHealth Toxicology and MutagenesisAuxotrophyReversionMutagenBiologyToxicologymedicine.disease_causeAmes testMicrobiologySpecies SpecificityEscherichia coliGeneticsmedicineAnimalsEscherichia coliBiotransformationGenetics (clinical)DihydralazineStrain (chemistry)Mutagenicity Testsfood and beveragesRats Inbred StrainsHydralazineDihydralazineRatsPyridazinesOncologyMutationMicrosomes LiverMutagensmedicine.drugTeratogenesis, Carcinogenesis, and Mutagenesis
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cis- and trans-1,2-diphenylaziridines: induction of xenobiotic-metabolizing enzymes in rat liver and mutagenicity in Salmonella typhimurium.

1986

trans-Stilbene imine (trans-1,2-diphenylaziridine) is the nitrogen analog of trans-stilbene oxide, a potent inducer of several microsomal and cytosolic xenobiotic-metabolizing enzymes. Although the acute toxicity of cis- and trans-stilbene imines prevents their application at the usual dose for trans-stilbene oxide (400 mg/kg/day), it is apparent that the imines nevertheless potently induce several xenobiotic-metabolizing enzymes in rat liver. The IP administration of trans-stilbene imine resulted in statistically significant increases in the activities of aminopyrine N-demethylase, microsomal epoxide hydrolase, glutathione transferase (toward 1-chloro-2,4-dinitrobenzene, 1,2-dichloro-4-nit…

MaleSalmonella typhimuriumStereochemistryHealth Toxicology and MutagenesisImineAziridines10050 Institute of Pharmacology and Toxicology610 Medicine & healthMutagenToxicologymedicine.disease_causeAmes testchemistry.chemical_compound2307 Health Toxicology and MutagenesismedicineAnimalsToxicology and MutagenesisEnzyme inducerchemistry.chemical_classificationbiologyAzirinesMutagenicity Tests3005 ToxicologyRats Inbred StrainsStereoisomerismGeneral MedicineCis trans isomerizationRatsEnzymechemistryBiochemistryLiverHealthMicrosomal epoxide hydrolaseEnzyme InductionMicrosomebiology.protein570 Life sciences; biologyMutagensArchives of toxicology
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Biphenyl and fluorinated derivatives: liver enzyme-mediated mutagenicity detected in Salmonella typhimurium and Chinese hamster V79 cells.

1992

Abstract Hepatocarcinogenic polychlorinated and polybrominated biphenyls usually show negative results in in vitro mutagenicity assays. Problems in their testing result from their low water solubility and their slow rate of metabolism. We therefore investigated better soluble model compounds, namely biphenyl and its 3 possible monofiuorinated derivatives. In the direct test, these compounds proved tobe nonmutagenic in Salmonella typhimurium TA98 and TA100 (reversion to histidine prototrophy) and in Chinese hamster V79 cells (acquisition of resistance to 6-thioguanine). However, when the exposure was carried out in the presence of NADPH-fortified postmitochondrial fraction of liver homogenat…

MaleSalmonella typhimuriumendocrine systemChinese hamsterAmes testCell LineToxicologychemistry.chemical_compoundStructure-Activity RelationshipCricetulusCricetinaeAnimalsBiotransformationBiphenylbiologyChemistryMutagenicity TestsBiphenyl CompoundsRats Inbred StrainsGeneral MedicineMetabolismbiology.organism_classificationEnterobacteriaceaeIn vitroRatsBiochemistryMicrosomal epoxide hydrolaseMicrosomes LiverPolybrominated BiphenylsMutation research
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Mutagenicity spectra in Salmonella typhimurium strains of glutathione, L-cysteine and active oxygen species

1989

Glutathione and L-cysteine, in the presence of rat kidney post-mitochondrial supernatant (S9) fraction, and various forms of active oxygen were investigated for mutagenicity in seven his- strains of Salmonella typhimurium. Glutathione and L-cysteine showed qualitatively and quantitatively virtually identical mutagenic activities. The number of mutants induced in strain TA97 was 3-4 times higher than in TA100, the strain in which the mutagenicity was originally detected. Mutagenic effects were also observed in strains TA92, TA102 and TA104, but not in TA1535 and TA1537. Hydrogen peroxide, superoxide and glucose/glucose oxidase in the presence and absence of kidney S9 fraction showed pronounc…

MaleSalmonella typhimuriumendocrine systemHealth Toxicology and MutagenesisIn Vitro TechniquesKidneyToxicologyAmes testSuperoxide dismutasechemistry.chemical_compoundSuperoxidesGeneticsAnimalsCysteineBiotransformationGenetics (clinical)chemistry.chemical_classificationReactive oxygen speciesbiologyMutagenicity TestsSuperoxide DismutaseSuperoxidefungifood and beveragesKidney metabolismRats Inbred StrainsHydrogen PeroxideGlutathioneCatalaseGlutathioneRatsOxygenchemistryS9 fractionBiochemistryCatalasebiology.proteinMutagensMutagenesis
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