Search results for "Myocardium"

showing 10 items of 365 documents

Usefulness of high-sensitivity troponin T for the evaluation of patients with acute chest pain and no or minimal myocardial damage

2012

Although high-sensitivity troponins allow early diagnosis of acute myocardial infarction, their role for identification of acute coronary syndrome in patients with normal conventional troponin remains unclear.A total of 446 patients presenting to the emergency department with chest pain and normal troponin (common practice assays) in 2 serial samples were included. Both samples were also centrally analyzed for high-sensitivity troponin T (hs-TnT) (Roche Diagnostics, Basel, Switzerland). Detection (3 ng/L) and 99th percentile (≥ 14 ng/L) cutoffs of the maximum hs-TnT levels (hs-TnTmax) were considered. The end points were acute coronary syndrome diagnosis and the composite of in-hospital rev…

MaleChest Painmedicine.medical_specialtyAcute coronary syndromemedicine.medical_treatmentMyocardial InfarctionChest painRevascularizationTroponin TPredictive Value of TestsInternal medicinemedicineAcute chest painHumansMyocardial infarctionAcute Coronary SyndromeAgedbiologyTroponin Tbusiness.industryMyocardiumEmergency departmentMiddle Agedmedicine.diseaseAcute PainTroponinbiology.proteinCardiologyFemalemedicine.symptomEmergency Service HospitalCardiology and Cardiovascular MedicinebusinessAlgorithmsAmerican Heart Journal
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GAS CHROMATOGRAPHIC ESTIMATION OF ACETYLCHOLINE IN THE RABBIT HEART USING A NITROGEN SELECTIVE DETECTOR

1973

The distribution of ACh in the rabbit heart was investigated by a modified gas chromatographic estimation method. ACh was extracted with perchloric acid, precipitated as reineckate and demethylated with sodium benzenethiolate. The tertiary amines derived from ACh and other choline esters were concentrated by a microdistillation procedure. Gas chromatography was performed using a nitrogen selective detector. In the range of concentrations between 0.4 and 2.5 nmol ACh per tissue sample the coefficient of variation was 5.2 per cent. The recovery of ACh added to heart extracts was 101 per cent. Evidence for the identity of the choline ester isolated from rabbit hearts and authentic ACh was obta…

MaleChromatography GasHeart VentriclesCoefficient of variationSodiumchemistry.chemical_elementBlood PressureBiochemistryCholineCellular and Molecular Neurosciencechemistry.chemical_compoundMethodsmedicineAnimalsBioassayCholineHeart AtriaPerchloric acidButyrylcholineChromatographyChemistryMyocardiumAcetylcholineRatsButyratesBiological AssayFemaleRabbitsGas chromatographyPropionatesAcetylcholinemedicine.drugJournal of Neurochemistry
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Acetylcholine overflow from isolated perfused hearts of various species in the absence of cholinesterase inhibition

1977

1. The content of acetylcholine in the tissue and effluent of isolated hearts of various birds and mammals was determined in the absence of inhibition of cholinesterase. 2. Stimulation of both vagus nerves for 15 min at 20 Hz caused marked negative chronotropic effects in all species. Spontaneous or stimulation-induced overflow of acetylcholine into the effluents was not detected in mammals. In the avian heart, the order of spontaneous overflow was: duck = chicken > pigeon, whereas the order of evoked overflow was: chicken > pigeon > duck. The acetylcholine overflow from the cat heart was below the limit of estimation (3 pmol g−1 min−1). In the chicken heart, the evoked overflow per min (28…

MaleChronotropicmedicine.medical_specialtyPhysostigmineanimal structuresAcetylcholine synthesisPhysostigmineGuinea PigsStimulationIn Vitro TechniquesBiologyBirdsParasympathetic nervous systemSpecies SpecificityInternal medicinemedicineAnimalsColumbidaeCholinesteraseMammalsPharmacologyMyocardiumVagus NerveGeneral MedicineAcetylcholineElectric StimulationCholinesterase inhibitionDucksEndocrinologymedicine.anatomical_structurenervous systemCatscardiovascular systembiology.proteinFemaleCholinesterase InhibitorsRabbitsChickensAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Fatty acid oxidation and related gene expression in heart depleted of carnitine by mildronate treatment in the rat.

2004

The metabolic and genic effects induced by a 20-fold lowering of carnitine content in the heart were studied in mildronate-treated rats. In the perfused heart, the proportion of palmitate taken up then oxidized was 5-10% lower, while the triacylglycerol (TAG) formation was 100% greater than in controls. The treatment was shown to increase the maximal capacity of heart homogenates to oxidize palmitate, the mRNA level of carnitine palmitoyltransferase I (CPT-I) isoforms, the specific activity of CPT-I in subsarcolemmal mitochondria and the total carnitine content of isolated mitochondria. Concomitantly, the increased mRNA expression of lipoprotein lipase, fatty acid translocase and enzymes of…

MaleClinical BiochemistryPalmitic AcidBlood lipidsBiologyMitochondrionIn Vitro TechniquesMitochondria HeartOxygen ConsumptionCarnitinemedicineAnimalsCarnitineRNA MessengerRats WistarMolecular BiologyBeta oxidationHeart metabolismTriglycerideschemistry.chemical_classificationLipoprotein lipaseCarnitine O-PalmitoyltransferaseEsterificationMyocardiumFatty AcidsFatty acidBiological TransportCardiovascular AgentsCell BiologyGeneral MedicineRatsPerfusionLipoprotein LipasechemistryBiochemistryGene Expression RegulationCarnitine palmitoyltransferase IOxidation-Reductionmedicine.drugMethylhydrazinesMolecular and cellular biochemistry
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Downregulation and Nuclear Relocation of MLP During the Progression of Right Ventricular Hypertrophy Induced by Chronic Pressure Overload

2000

Abstract The cardiac LIM domain protein MLP plays a crucial role in the architecture and mechanical function of cardiac myocytes. Mice lacking the MLP gene develop cardiac hypertrophy, dilated cardiopathy and heart failure. We investigated whether downregulation of MLP is induced by pressure overload and contributes to the physiopathology of cardiac hypertrophy and failure. We studied this mechanism in rat right ventricles submitted to pulmonary arterial hypertension, because it is known that this ventricle is very vulnerable to the deleterious effects of pressure overload. During the progression of cardiac hypertrophy to failure over a 31 days period there was a dramatic decrease by 50% of…

MaleCytoplasmmedicine.medical_specialtyTime FactorsTranscription GeneticHeart VentriclesDown-RegulationMuscle ProteinsCardiomegalyCytosolMyofibrilsDownregulation and upregulationRight ventricular hypertrophyInternal medicinePressureAnimalsVentricular FunctionMedicineMyocyteRNA MessengerRats WistarLungMolecular BiologyCell NucleusHomeodomain ProteinsPressure overloadReverse Transcriptase Polymerase Chain Reactionbusiness.industryMyocardiumLIM Domain Proteinsmedicine.diseaseImmunohistochemistryPulmonary hypertensionRatsMicroscopy Electronmedicine.anatomical_structureVentricleHeart failureCardiologyCardiology and Cardiovascular MedicinebusinessMyofibrilJournal of Molecular and Cellular Cardiology
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Deep Learning Techniques for Automatic MRI Cardiac Multi-Structures Segmentation and Diagnosis: Is the Problem Solved?

2018

Delineation of the left ventricular cavity, myocardium, and right ventricle from cardiac magnetic resonance images (multi-slice 2-D cine MRI) is a common clinical task to establish diagnosis. The automation of the corresponding tasks has thus been the subject of intense research over the past decades. In this paper, we introduce the “Automatic Cardiac Diagnosis Challenge” dataset (ACDC), the largest publicly available and fully annotated dataset for the purpose of cardiac MRI (CMR) assessment. The dataset contains data from 150 multi-equipments CMRI recordings with reference measurements and classification from two medical experts. The overarching objective of this paper is to measure how f…

MaleDatabases FactualHeart DiseasesComputer science[SDV]Life Sciences [q-bio]Lleft and right ventricles030218 nuclear medicine & medical imagingTask (project management)Cardiac segmentation and diagnosis03 medical and health sciences0302 clinical medicineDeep LearningImage Interpretation Computer-AssistedmedicineMedical imagingHumansSegmentationElectrical and Electronic EngineeringRadiological and Ultrasound Technologymedicine.diagnostic_testbusiness.industryMyocardiumDeep learningMagnetic resonance imagingPattern recognitionHeartImage segmentationMagnetic Resonance ImagingComputer Science ApplicationsCardiac Imaging Techniquesmedicine.anatomical_structureVentricleFemaleArtificial intelligencebusinessCardiac magnetic resonanceLeft and right ventricles030217 neurology & neurosurgerySoftwareMRIIEEE transactions on medical imaging
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Higher Free Fatty Acid Uptake in Visceral Than in Abdominal Subcutaneous Fat Tissue in Men

2010

Visceral adipose tissue has been shown to have high lipolytic activity. The aim of this study was to examine whether free fatty acid (FFA) uptake into visceral adipose tissue is enhanced compared to abdominal subcutaneous tissue in vivo. Abdominal adipose tissue FFA uptake was measured using positron emission tomography (PET) and [F-18]-labeled 6-thia-hepta-decanoic acid ([F-18]FTHA) and fat masses using magnetic resonance imaging (MRI) in 18 healthy young adult males. We found that FFA uptake was 30% higher in visceral compared to subcutaneous adipose tissue (0.0025 +/- 0.0018 vs. 0.0020 +/- 0.0016 mu mol/g/min, P = 0.005). Visceral and subcutaneous adipose tissue FFA uptakes were strongly…

MaleEndocrinology Diabetes and MetabolismMedicine (miscellaneous)Adipose tissueWhite adipose tissueFatty Acids NonesterifiedGLUCOSE0302 clinical medicineEndocrinologyReference ValuesIN-VIVOchemistry.chemical_classification0303 health sciencesINSULIN-RESISTANCENutrition and DieteticsFatty AcidsMagnetic Resonance Imagingmedicine.anatomical_structureADIPOSE-TISSUEPROLONGED EXERCISESKELETAL-MUSCLESubcutaneous tissueAdultmedicine.medical_specialtyIntra-Abdominal Fat030209 endocrinology & metabolismIntra-Abdominal FatMETABOLISMYoung Adult03 medical and health sciencesInsulin resistanceInternal medicinemedicineHumansTotal TissueINCREASED PHYSICAL-ACTIVITY030304 developmental biologyBLOOD-FLOWbusiness.industryFatty acidSkeletal muscleBiological Transportmedicine.diseaseSubcutaneous Fat AbdominalEndocrinologychemistryPositron-Emission TomographyRadiopharmaceuticalsbusinessMYOCARDIUMObesity
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Phospholipase D in heart: basal activity and stimulation by phorbol esters and aluminum fluoride

1992

Evidence for a general role of phospholipase D in signal transduction is accumulating. In the present study, the activity of the enzyme was investigated in heart tissue under basal conditions and after addition of phorbol esters or aluminum fluoride (AlF inf4 sup− ; 10 mM NaF plus 10 μM AlCl3). Atria of rats and chickens were incubated with [3H]-myristic acid in order to label preferentially phosphatidylcholine. Under basal conditions, the tissues generated choline and phosphatidic acid (PtdOH), the primary catalytic products of phospholipase D. When 0.5 or 2.0% ethanol was present, [3H]-phosphatidyl-ethanol (PETH) was rapidly formed at the expense of [3H]-PtdOH. This transphosphatidylation…

MaleG proteinPhospholipaseBiologyCholineFluorideschemistry.chemical_compoundGTP-Binding ProteinsPhosphatidylcholinePhorbol EstersPhospholipase DAnimalsPhospholipase D activityRats WistarAluminum CompoundsProtein kinase AChromatography High Pressure LiquidProtein Kinase CProtein kinase CPharmacologyEthanolPhospholipase DMyocardiumHeartGeneral MedicinePhosphatidic acidMolecular biologyRatsBiochemistrychemistryChickensAluminumSignal TransductionNaunyn-Schmiedeberg's Archives of Pharmacology
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Phospholipase D in rat myocardium: formation of lipid messengers and synergistic activation by G-protein and protein kinase C.

1998

Activation of phospholipase D (PLD) and phosphoinositide-specific phospholipase C (PI-PLC) by fluoride, to stimulate heterotrimeric G-proteins, and by phorbol esters, to stimulate protein kinase C (PKC), was studied in rat atria. Fluoride and 4beta-phorbol-12beta,13alpha-dibutyrate (PDB), in contrast to 4beta-phorbol-13alpha-acetate (PAc), activated PLD, catalyzing the formation of [3H]-phosphatidylethanol ([3H]-PETH), [3H]-phosphatidic acid ([3H]-PA), choline and sn-1,2-diacylglycerol (DAG). Basal PLD activity was resistant to drastic changes in Ca2+ and to Ro 31-8220, a PKC inhibitor, but was decreased by genistein, an inhibitor of tyrosine kinase, and increased by vanadate, a tyrosine ph…

MaleG proteinProtein tyrosine phosphataseBiologyBiochemistrySecond Messenger Systemschemistry.chemical_compoundPhosphoinositide Phospholipase CGTP-Binding ProteinsPhorbol EstersPhospholipase DAnimalsRats WistarProtein kinase CPhorbol 1213-DibutyrateProtein Kinase CDiacylglycerol kinasePharmacologyPhospholipase CPhospholipase DMyocardiumPhosphatidylinositol Diacylglycerol-LyaseTyrosine phosphorylationDrug SynergismLipid MetabolismLipidsRatsEnzyme ActivationBiochemistrychemistryType C PhospholipasesSecond messenger systemlipids (amino acids peptides and proteins)Biochemical pharmacology
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Silibinin improves hepatic and myocardial injury in mice with nonalcoholic steatohepatitis

2012

Abstract Background Nonalcoholic fatty liver disease is a chronic metabolic disorder with significant impact on cardiovascular and liver mortality. Aims In this study, we examined the effects of silibinin on liver and myocardium injury in an experimental model of nonalcoholic fatty liver disease. Methods A four-week daily dose of silibinin (20 mg/kg i.p.) was administrated to db/db mice fed a methionine–choline deficient diet. Hepatic and myocardial histology, oxidative stress and inflammatory cytokines were evaluated. Results Silibinin administration decreased HOMA-IR, serum ALT and markedly improved hepatic and myocardial damage. Silibinin reduced isoprostanes, 8-deoxyguanosine and nitrit…

MaleGene ExpressionIsoprostanesmedicine.disease_causeGastroenterologyAntioxidantsMicechemistry.chemical_compoundMethionineNon-alcoholic Fatty Liver DiseaseNonalcoholic fatty liver diseasePhosphorylationGastroenterologyAlanine TransaminaseGlutathioneCholine DeficiencyMitochondrial respiratory chainLiverHeart Inflammation NAFLD Oxidative stress SilibininCytokinesmedicine.symptomSilymarinmedicine.medical_specialtySilibininInflammationStatistics NonparametricProinflammatory cytokineInsulin resistanceInternal medicinemedicineAnimalsNitritesAnalysis of VarianceNitratesHepatologySettore BIO/16 - Anatomia Umanabusiness.industryMyocardiumJNK Mitogen-Activated Protein KinasesGlutathionemedicine.diseaseDietFatty LiverOxidative StressEndocrinologychemistrySilybinInsulin ResistancebusinessOxidative stressDigestive and Liver Disease
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