Search results for "Myofibril"

showing 10 items of 50 documents

Neonatal form of nemaline myopathy, muscle immaturity, and a microvascular injury.

1990

An infant with a neonatal form of nemaline myopathy showed ultrastructural features of muscle immaturity. Immaturity was characterized by an abnormal presence of myotubes, as well as cells in clusters within a common basement membrane and a great number of satellite cells adhering to very small muscle fibers. In addition, degenerative changes and a severe microvascular lesion were observed. The pathologic findings in the muscle of this patient were those of neonatal nemaline myopathy complicating severe microvascular injury, possibly induced by an unknown toxic agent. ( J Child Neurol 1990;5:122-126).

Pathologymedicine.medical_specialtyMuscle HypotoniaBiopsyIschemiaBiologyMuscle Smooth VascularLesion03 medical and health sciences0302 clinical medicineNemaline myopathyMyofibrilsIschemia030225 pediatricsBiopsymedicineHumansBasement membraneInclusion BodiesAsphyxia NeonatorumRespiratory Distress Syndrome Newbornmedicine.diagnostic_testMyogenesisMicrocirculationMusclesInfant NewbornDisseminated Intravascular Coagulationmedicine.diseaseMicroscopy ElectronMuscular Atrophymedicine.anatomical_structurePediatrics Perinatology and Child HealthMuscle HypotoniaFemaleNeurology (clinical)medicine.symptomMyofibril030217 neurology & neurosurgeryJournal of child neurology
researchProduct

The disruption of myofibre structures in skeletal muscle after forced lengthening contractions

1998

Specific antibodies against structural proteins (actin, desmin, dystrophin, fibronectin) of muscle fibres were used to study the effect of forced lengthening contractions on muscle microarchitecture. Tibialis anterior (TA) muscle of male Wistar rats were subjected to 240 forced lengthening contractions. At consecutive time points (0, and 6 h, 2, 4, and 7 days) after stimulation, the TA muscle was excised for biochemical and histological assays. β-Glucuronidase activity, a quantitative indicator of muscle damage, showed increased values 2–7 days after the lengthening, peaking on day 4 (11.7-fold increase). A typical course of histopathological changes (myofibre swelling, necrosis and regener…

Pathologymedicine.medical_specialtyNecrosisSkeletal muscleAnatomyBiologyPathology and Forensic Medicinemedicine.anatomical_structurePhysiology (medical)medicinebiology.proteinDesminmedicine.symptomMyofibrilIntermediate filamentDystrophinCytoskeletonActinPathophysiology
researchProduct

Filamin C accumulation is a strong but nonspecific immunohistochemical marker of core formation in muscle.

2002

Filamin C is the muscle isoform of a group of large actin-crosslinking proteins. On the one hand, filamin C is associated with the Z-disk of the myofibrillar apparatus and binds to myotilin; on the other hand, it interacts with the sarcoglycan complex at the sarcolemma. Filamin C may be involved in reorganizing the cytoskeleton in response to signalling events and in muscle it may, in addition, fulfill structural functions at the Z-disk. An examination of biopsies from patients with multi-minicore myopathy, central core myopathy and neurogenic target fibers with core-like target formations (TF) revealed strong reactivity of all the cores and target formations with two different anti-filamin…

Pathologymedicine.medical_specialtyanimal structuresBiopsyFilaminsmacromolecular substancesBiologyFilamin03 medical and health sciences0302 clinical medicineContractile ProteinsMuscular DiseasesReference ValuesmedicineMyotilinHumansProtein IsoformsCytoskeletonMyopathyMicroscopy ImmunoelectronMuscle Skeletal030304 developmental biology0303 health sciencesSarcolemmaMicrofilament Proteinsmedicine.diseaseImmunohistochemistryCell biologybody regionsNeurologyDesminNeurology (clinical)medicine.symptomMyofibrilCarrier Proteins030217 neurology & neurosurgeryCentral core diseaseBiomarkersJournal of the neurological sciences
researchProduct

Primary desminopathies.

2007

•  Introduction •  Desmin is an essential component of the extrasarcomeric cytoskeleton in striated muscle cells •  Distal myopathy,cardiac arrhythmias,cardiomyopathy:classical criteria of primary desminopathies •  Sub-sarcolemmal and cytoplasmic desmin-positive protein aggregates:the morphological hallmark of primary and secondary desminopathies •  The spectrum of pathogenic desmin gene mutations •  The molecular pathogenesis of primary desminopathies: some answers gained,but even more questions raised •  Diagnostic work-up to distinguish primary from secondary desminopathies •  Treatment and clinical management of primary desminopathy patients Abstract Mutations of the human desmin gene o…

Pathologymedicine.medical_specialtyintermediate filamentsCardiomyopathyReviewsgranulofilamentous materialdesmininclusion bodiesmacromolecular substancesBiologymyofibrillar myopathyprotein aggregationdesmin-related myopathySarcolemmaMuscular DiseasesmedicineMyocyteAnimalsHumansIntermediate filamentMyopathyMuscle SkeletalCytoskeletonGenetic heterogeneityCardiac muscleCell Biologymedicine.diseasemusculoskeletal systemmutationsmedicine.anatomical_structuredesminopathyMutationMolecular MedicineDesminmedicine.symptomMyofibrilJournal of cellular and molecular medicine
researchProduct

Creatine kinase is the main target of reactive oxygen species in cardiac myofibrils.

1996

Abstract Reactive oxygen species (ROS) have been reported to alter cardiac myofibrillar function as well as myofibrillar enzymes such as myosin ATPase and creatine kinase (CK). To understand their precise mode and site of action in myofibrils, the effects of the xanthine/xanthine oxidase (X/XO) system or of hydrogen peroxide (H 2 O 2 ) have been studied in the presence and in the absence of phosphocreatine (PCr) in Triton X-100–treated cardiac fibers. We found that xanthine oxidase (XO), with or without xanthine, induced a decrease in maximal Ca 2+ -activated tension. We attributed this effect to the high contaminating proteolytic activity in commercial XO preparations, since it could be p…

Xanthine OxidasebiologyFree RadicalsPhysiologyMyosin ATPaseSuperoxideHydrogen PeroxideMyosinsXanthineMyocardial ContractionPhosphocreatineRatschemistry.chemical_compoundchemistryBiochemistryMyofibrilsbiology.proteinAnimalsCreatine kinasePMSFCardiology and Cardiovascular MedicineMyofibrilXanthine oxidaseReactive Oxygen SpeciesCreatine KinaseCirculation research
researchProduct

Influence of Oral Contraceptive Use on Adaptations to Resistance Training

2019

Introduction: The majority of young women use oral contraceptives (OCs). Use of OCs has been associated with lower myofibrillar protein and tendon collagen synthesis rates, but it is unknown whether OCs will limit the adaptive response of myotendinous tissue to resistance training. Design and Methods: Fourteen healthy untrained young regular OC users (24 +/- 1 years, fat% 32 +/- 1, 35 +/- 2 ml.min(-1).kg(-1)) and 14 NOC users (non-OC, controls) (24 +/- 1 years, fat% 32 +/- 2, 34 +/- 2 ml.min(-1).kg(-1)) performed a 10-week supervised lower extremity progressive resistance training program. Before and after the intervention biopsies from the vastus lateralis muscle and the patellar tendon we…

estrogeenitmedicine.medical_specialtyestradioliTESTOSTERONE LEVELStendonPhysiologyVastus lateralis muscleUrologyIsometric exerciselcsh:PhysiologyMuscle hypertrophy03 medical and health sciences0302 clinical medicinePhysiology (medical)estradiolmedicineestrogenmuscle hypertrophyIN-VIVOHUMAN ACHILLES-TENDONOriginal Researchlcsh:QP1-981ehkäisypilleritexerciseMUSCLE-FIBER SIZETENDON COLLAGEN-SYNTHESISbusiness.industryResistance training030229 sport sciencesMECHANICAL-PROPERTIESTendonmedicine.anatomical_structureContraceptive uselihasmassaBIOMECHANICAL PROPERTIESMuscle strengthmuscle strengthPROTEIN-SYNTHESISSKELETAL-MUSCLEvoimaharjoitteluwomenMENSTRUAL-CYCLEbusinessMyofibril030217 neurology & neurosurgerylihasvoima
researchProduct

Goodpasture antigen-binding protein (GPBP) y su efector GPBP-interacting protein de 130-kDa (GIP130) dirigen la formación de miofibrillas

2014

Goodpasture antigen-binding protein (GPBP) es una proteína cinasa no convencional que regula la organización del colágeno IV de la membrana basal glomerular, y de la que existen al menos tres isoformas: GPBP-1 que es soluble y exportable; GPBP-2 que es citosólica y se genera por procesamiento alternativo de exones; y GPBP-3 que está asociada a membranas y regula la secreción de GPBP-1. El gen que expresa las proteínas GPBP (COL4A3BP) tiene notable expresión en tejido muscular, en el que GPBP-1 es la isoforma mayoritaria y se encuentra en la línea M de la banda A de las miofibrillas. Durante la miogénesis de mioblastos C2C12 en cultivo hay una progresiva disminución de la expresión de GPBP-2…

gpbpUNESCO::CIENCIAS MÉDICASUNESCO::CIENCIAS DE LA VIDAtubular aggregates:CIENCIAS MÉDICAS [UNESCO]myofibrilgip130:CIENCIAS DE LA VIDA [UNESCO]
researchProduct

M-cadherin and its sisters in development of striated muscle

1999

Cadherins are calcium-dependent, transmembrane intercellular adhesion proteins with morphoregulatory functions in the development and maintenance of tissues. In the development of striated muscle, the expression and function of mainly M-, N-, and R-cadherin has been studied so far. While these three cadherins are expressed in skeletal muscle cells, of these only N-cadherin is expressed in cardiac muscle. In this review, M-, N-, and R-cadherin are discussed as important players in the terminal differentiation and possibly also in the commitment of skeletal muscle cells. Furthermore, reports are described which evaluate the essential role of N-cadherin in the formation of heart tissue.

medicine.medical_specialtyMyofilamentHistologyBiologyMuscle DevelopmentSarcomerePathology and Forensic MedicineEmbryonic and Fetal DevelopmentMiceInternal medicineMyosinmedicineAnimalsHumansMyocyteMuscle SkeletalCardiac muscleGene Expression Regulation DevelopmentalSkeletal muscleCell DifferentiationHeartCell BiologyCadherinsCell biologyEndocrinologymedicine.anatomical_structureITGA7MyofibrilCell and Tissue Research
researchProduct

Morphometry of myocardial apex in endurance-trained mice of different ages

1979

Mitochondrial volume density, surface density of the outer mitochondrial membrane, the mean number and size of mitochondria, and the mean surface density of crista membranes together with the volume densities of myofibrils and sarcoplasmic space were morpho-metrically analyzed in cardiac muscle of two groups of sedentary control mice aged 3 and 7 months, and in two groups of mice trained either 1 month rather intensely or 4 months moderately. Of the calculated mitochondrial variables only the surface density of the outer mitochondrial membrane differed between the older controls and the older trained animals, the density being slightly smaller in the trained group. The myofibrillar volume d…

medicine.medical_specialtyPhysical Education and TrainingTime FactorsPhysiologyMyocardiumSarcoplasmAge FactorsCardiac muscleStereologyOrgan SizeBiologyMitochondria HeartMiceCristamedicine.anatomical_structureCardiac muscle hypertrophyEndocrinologyEndurance trainingInternal medicinePhysical EndurancemedicineAnimalsMyocyteMyofibrilActa Physiologica Scandinavica
researchProduct

Gender differences in skeletal muscle fibre damage after eccentrically biased downhill running in rats

1999

Specific antibodies against structural proteins of muscle fibres (actin, desmin, dystrophin) and extracellular matrix (fibronectin) were used to study the effect of eccentrically biased downhill running exercise (13,5 degrees, 17 m min(-1), 130 min) on the magnitude and properties of myofibre injury in the quadriceps femoris muscle of male and female rats. Muscle beta-glucuronidase activity, a quantitative indicator of muscle damage, showed clearly smaller increase in female than in male rats during the 4-day period following exercise. A similar course of histopathological changes was observed in both sexes, although females showed slower and less marked changes than males. In males, discon…

medicine.medical_specialtySarcolemmabiologyPhysiologyAnatomyQuadriceps femoris muscleLesionEndocrinologyInternal medicinemedicinebiology.proteinDesminmedicine.symptomCytoskeletonDystrophinMyofibrilActinActa Physiologica Scandinavica
researchProduct