Search results for "Myositi"

showing 10 items of 59 documents

Expression of cell adhesion molecules in inflammatory myopathies.

1995

We examined the expression of cell adhesion molecules in 25 cases of inflammatory myopathies. Inflammatory myopathies showed upregulation of adhesion molecules. ICAM-1 was strongly expressed on endothelial cells as well as on fibroblasts and infiltrating leukocytes while the expression of VCAM-1, similar in its distribution, was much weaker. A few muscle fibers in polymyositis revealed sarcolemmal labeling for ICAM-1. ELAM-1 showed only weak expression on vessels. The inflammatory cellular infiltrates contained varying amounts of cells bearing the VCAM-1 ligand VLA-4 and the ELAM-1 ligand SLeX as well as large amounts of cells expressing LFA-1 alpha and beta, ligands of ICAM-1.

ImmunologyIntercellular Adhesion Molecule-1Lewis X AntigenVascular Cell Adhesion Molecule-1InflammationNectinReceptors Very Late AntigenE-selectinmedicineImmunology and AllergyHumansCell adhesionbiologyMyositisCell adhesion moleculeChemistrySoluble cell adhesion moleculesIntercellular Adhesion Molecule-1Lymphocyte Function-Associated Antigen-1Cell biologyNeurologycardiovascular systembiology.proteinNeural cell adhesion moleculeNeurology (clinical)medicine.symptomE-SelectinCell Adhesion MoleculesJournal of neuroimmunology
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Recent Advances in the Morphology of Myositis

1985

Summary Myositis in man may be divided into infectious and non-infectious forms. The myopathologist more often deals with the latter forms which comprise dermatomyositis/polymyositis, inclusion body myositis, mixed connective tissue disease/collagenoses, and granulomatous myopathies. Modern morphological techniques as enzyme-histochemistry, electron microscopy, immunohistology, and morphometry are of different value in various forms of myositis, but are often indispensable techniques in up-to-date diagnostic work up of a myositis.

Inclusion BodiesPathologymedicine.medical_specialtyGranulomaMyositisHistocytochemistrybusiness.industryImmunochemistryGranulomatous myositisCell BiologyDermatomyositismedicine.diseasePolymyositisDermatomyositisPathology and Forensic MedicineMixed connective tissue diseaseMuscular DiseasesVirus DiseasesmedicineHumansInclusion body myositisbusinessMyositisMixed Connective Tissue DiseasePathology - Research and Practice
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MITOCHONDRIAL DISORDER SECONDARY TO INFLAMMATION IN POLYMYOSITIS - 2 CASES

1992

National audience; Abstract: Two cases of polymyositis were followed using phosphorus nuclear magnetic resonance spectroscopy. The spectra recorded during remission were normal, but those collected from the gastrocnemius muscle during the active phase of the diseases showed an increased inorganic phosphate level or a decreased phosphocreatine content. The intracellular pH was normal. These findings may be related to an impairment in mitochondrial metabolism secondary to the inflammatory process. Moreover, the fact that the abnormalities observed disappeared after treatment suggests that phosphorus NMR spectroscopy could be used as a non-invasive method in the follow-up of polymyositis, but …

MYOPATHIES[ INFO.INFO-IM ] Computer Science [cs]/Medical ImagingINVIVO[INFO.INFO-IM] Computer Science [cs]/Medical ImagingMAGNETIC-RESONANCE SPECTROSCOPY[INFO.INFO-IM]Computer Science [cs]/Medical ImagingMUSCLEDERMATOMYOSITISDYSTROPHY
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Patients experiencing statin-induced myalgia exhibit a unique program of skeletal muscle gene expression following statin re-challenge

2017

Statins, the 3-hydroxy-3-methyl-glutaryl (HMG)-CoA reductase inhibitors, are widely prescribed for treatment of hypercholesterolemia. Although statins are generally well tolerated, up to ten percent of statin-treated patients experience myalgia symptoms, defined as muscle pain without elevated creatinine phosphokinase (CPK) levels. Myalgia is the most frequent reason for discontinuation of statin therapy. The mechanisms underlying statin myalgia are not clearly understood. To elucidate changes in gene expression associated with statin myalgia, we compared profiles of gene expression in skeletal muscle biopsies from patients with statin myalgia who were undergoing statin re-challenge (cases)…

Male0301 basic medicinemyalgiaGene Expressionlcsh:MedicineApoptosis030204 cardiovascular system & hematologyPathology and Laboratory MedicineBioinformaticsBiochemistry0302 clinical medicineMedicine and Health SciencesGene Regulatory Networkslcsh:ScienceMusculoskeletal SystemEnergy-Producing OrganellesMyositisRegulation of gene expressionMultidisciplinaryCell DeathbiologyMusclesDrugsMiddle AgedMitochondriaCell ProcessesHMG-CoA reductaseFemalelipids (amino acids peptides and proteins)AnatomyCellular Structures and Organellesmedicine.symptomResearch ArticleSenescencemedicine.medical_specialtyStatinmedicine.drug_classPainBioenergeticsPolymorphism Single Nucleotide03 medical and health sciencesSigns and SymptomsDiagnostic MedicineInternal medicineGeneticsmedicineHumansGene Regulationcardiovascular diseasesMuscle SkeletalAgedPharmacologybusiness.industrylcsh:RStatinsBiology and Life SciencesComputational Biologynutritional and metabolic diseasesMyalgiaCell Biologymedicine.disease030104 developmental biologyEndocrinologyGene Expression RegulationSkeletal MusclesLeukocytes Mononuclearbiology.proteinProtein prenylationlcsh:QHydroxymethylglutaryl-CoA Reductase InhibitorsSLCO1B1businessPLOS ONE
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Acute polymyositis during treatment of acute hepatitis C with pegylated interferon alpha-2b.

2005

Hepatitis C virus is not cleared after primary infection in 50-85% of subjects exposed to hepatitis C virus. Anti-viral treatment during the early phase of infection significantly enhances the likelihood of a sustained clearance of hepatitis C virus. Although, a variety of autoimmune-related side effects have been observed during interferon therapy for chronic hepatitis, immuno-mediated adverse reactions have not been reported during treatment of acute hepatitis C. We describe the case of a patient who developed acute hepatitis C virus infection and, while receiving pegylated interferon alpha-2b monotherapy, developed a severe polymyositis. This case illustrates the potential risk of autoim…

MaleHepatitis C virusHepacivirusAcute hepatitis CAlpha interferonAutoimmunityHepacivirusInterferon alpha-2medicine.disease_causeIFNPolymyositisPolyethylene GlycolAntiviral AgentsVirusPolyethylene GlycolsPegylated interferonInterferonmedicineHumansDrug CarrierCreatine KinasePolymyositiAntiviral AgentDrug CarriersHepaciviruHepatologybiologybusiness.industryGastroenterologyInterferon-alphaHepatitis CRecombinant ProteinMiddle Agedmedicine.diseasebiology.organism_classificationHepatitis CRecombinant ProteinsAcute hepatitis C; Hepatitis C virus; IFN; Polymyositis; Acute Disease; Antiviral Agents; Autoimmunity; Creatine Kinase; Drug Carriers; Hepacivirus; Hepatitis C; Humans; Interferon-alpha; Male; Middle Aged; Polyethylene Glycols; Polymyositis; RNA Viral; Recombinant Proteins; GastroenterologyPolymyositisImmunologyAcute DiseaseRNA ViralbusinessHepatitis C virumedicine.drugHumanDigestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
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Frontotemporal dementia: the post-tau era.

2006

As scientists have begun to decipher the molecular genetic bases of hereditary frontotemporal dementia (FTD), it has become clear that the biology of these human neurodegenerative diseases has a complexity not previously suspected. FTD has been found to be linked to several chromosomal loci including those in chromosome 9, chromosome 17, and chromosome 3. The article by Guyant-Marechal et al. in this issue of Neurology reports the clinical, pathologic, and molecular characteristics of a form of FTD associated with inclusion body myopathy and Paget disease of the bone observed in members of two families and expands our knowledge on genetically determined FTD.1 The disorder is associated with…

MaleHeterozygoteMultiple Organ FailureDNA Mutational AnalysisChromosome 9Cell Cycle ProteinsChromosome Disorderstau ProteinsBiologyRisk AssessmentMyositis Inclusion BodyExonRisk FactorsValosin Containing ProteinmedicinePrevalenceHumansGenetic Predisposition to DiseaseGeneRetrospective StudiesGeneticsAdenosine TriphosphatasesIncidenceChromosomeSyndromeMiddle Agedmedicine.diseaseOsteitis DeformansPhenotypePedigreeChromosome 17 (human)Chromosome 3MutationDementiaFemaleNeurology (clinical)FranceFrontotemporal dementiaNeurology
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Clinical spectrum time course in anti jo-1 positive antisynthetase syndrome: Results from an international retrospective multicenter study

2015

Anti Jo-1 antibodies are the main markers of the antisynthetase syndrome (ASSD), an autoimmune disease clinically characterized by the occurrence of arthritis, myositis, and interstitial lung disease (ILD). These manifestations usually co-occur (for practical purpose complete forms) in the same patient, but cases with only 1 or 2 of these findings (for practical purpose incomplete forms) have been described. In incomplete forms, the ex novo occurrence of further manifestations is possible, although with frequencies and timing not still defined. The aim of this international, multicenter, retrospective study was to characterize the clinical time course of anti Jo-1 positive ASSD in a large c…

MalePathologyNeurologyAnti Jo-1:Analytical Diagnostic and Therapeutic Techniques and Equipment::Investigative Techniques::Epidemiologic Methods::Epidemiologic Study Characteristics as Topic::Epidemiologic Studies::Case-Control Studies::Retrospective Studies [Medical Subject Headings]MedizinArthritisAntisynthetase syndrome:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]AntinuclearMasculinoMyositis:Persons::Persons::Age Groups::Adult::Aged [Medical Subject Headings]Medicine (all)Interstitial lung diseaseFemeninoGeneral MedicineMiddle Aged:Diseases::Musculoskeletal Diseases::Muscular Diseases::Myositis [Medical Subject Headings]HumanosAnticuerpos antinuclearesAntibodies Antinuclear:Diseases::Musculoskeletal Diseases::Joint Diseases::Arthritis [Medical Subject Headings]Female:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Blood Proteins::Immunoproteins::Immunoglobulins::Antibodies::Autoantibodies::Antibodies Antinuclear [Medical Subject Headings]Adultmedicine.medical_specialty:Check Tags::Male [Medical Subject Headings]AntibodiesNOEstudios retrospectivosInternal medicinemedicineHumansRisk factorAdult; Aged; Antibodies Antinuclear; Arthritis; Female; Humans; Male; Middle Aged; Myositis; Retrospective Studies; Medicine (all):Persons::Persons::Age Groups::Adult [Medical Subject Headings]AgedRetrospective StudiesArtritisMyositisbusiness.industryArthritisRetrospective cohort study:Persons::Persons::Age Groups::Adult::Middle Aged [Medical Subject Headings]Anti Jo-1 Antisynthetase Syndromemedicine.diseaseDermatologyRheumatology:Check Tags::Female [Medical Subject Headings]Miositisantisynthetase syndromebusiness
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Myofibrillar disorganization characterizes myopathy of camptocormia in Parkinson’s disease

2011

Camptocormia is a highly disabling syndrome that occurs in various diseases but is particularly associated with Parkinson’s disease (PD). Although first described nearly 200 years ago, the morphological changes associated with camptocormia are still under debate and the pathophysiology is unknown. We analyzed paraspinal muscle biopsies of 14 PD patients with camptocormia and compared the findings to sex-matched postmortem controls of comparable age to exclude biopsy site-specific changes. Camptocormia in PD showed a consistent lesion pattern composed of myopathic changes with type-1 fiber hypertrophy, loss of type-2 fibers, loss of oxidative enzyme activity, and acid phosphatase reactivity …

MalePathologymedicine.medical_specialtyParkinson's diseaseMyopathyClinical Neurology610BiologySpinal CurvaturesMuscle hypertrophyPathology and Forensic MedicineLesionMuscular Atrophy Spinal03 medical and health sciencesCamptocormiaMyofibrillar disorganizationCellular and Molecular Neuroscience0302 clinical medicineMyofibrilsBiopsymedicineHumansProspective StudiesMyopathyMuscle SkeletalParkinson’s disease; Camptocormia; Myopathy; Myofibrillar disorganization; ProprioceptionMyositis030304 developmental biologyAgedAged 80 and over0303 health sciencesOriginal Papermedicine.diagnostic_testParkinson DiseaseMiddle Agedmedicine.diseaseProprioceptionPathophysiologyCamptocormiaParkinson’s diseaseFemaleNeurology (clinical)medicine.symptomMedicine & Public Health; Neurosciences; Pathology030217 neurology & neurosurgeryActa Neuropathologica
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New-onset third-degree atrioventricular block because of autoimmune-induced myositis under treatment with anti-programmed cell death-1 (nivolumab) fo…

2017

There has been considerable progress in treating malignant melanoma over the last few years. The immune-checkpoint-inhibitors nivolumab and pembrolizumab have been approved by the Food and Drug Administration in 2014 for the therapy of metastatic melanoma. Anti-programmed cell death-1-blocking antibodies are known to cause immune-related adverse events. Physicians should be aware of common and rare side effects and pay attention to new ones. We therefore report a severe and life-threatening side effect of anti-programmed cell death-1 immunotherapy with nivolumab that has not been previously reported: the development of a third-degree atrioventricular block. After a second infusion with nivo…

MaleUveal NeoplasmsOncologyCancer Researchmedicine.medical_specialtyMyocarditisSide effectDermatologyPembrolizumab030204 cardiovascular system & hematologyAutoimmune Diseases03 medical and health sciencesAntineoplastic Agents ImmunologicalFatal Outcome0302 clinical medicineInternal medicineHumansMedicineAtrioventricular BlockMelanomaMyositisMyositisbusiness.industryThird-degree atrioventricular blockMelanomaAntibodies MonoclonalMiddle Agedmedicine.diseaseNivolumabOncology030220 oncology & carcinogenesisImmunologyNivolumabbusinessAtrioventricular blockMelanoma Research
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Prednisolone decreases exercise-induced acid hydrolase response in mouse skeletal muscle.

1984

Male NMRI-mice were subjected to exhaustive treadmill exercise. 3 and 6 days after the exertion, quadriceps femoris muscles were examined histologically and analyzed for acid hydrolases in order to follow the degree and progress of injuries. Prednisolone (PRED), an anti-inflammatory corticosteroid, was given to some of the animals in order to modify the exercise response. The PRED administration began 14 h before exercise and continued until the end of the experiment (6 days). The doses were 25 and 50 mg . kg-1 i.p. twice a day. The activities of both arylsulphatase and beta-glucuronidase increased significantly in the exercise control group after 3 and 6 days. The increase in activity corr…

Malemedicine.medical_specialtyNecrosisPhysiologymedicine.drug_classPrednisolonePhysical ExertionPhysical exerciseInflammationMice Inbred StrainsBiologyMiceMuscular DiseasesPhysiology (medical)Internal medicinemedicineAnimalsRegenerationOrthopedics and Sports MedicineExertionArylsulfatasesGlucuronidaseMyositisMusclesPublic Health Environmental and Occupational HealthSkeletal muscleGeneral Medicinemedicine.anatomical_structureEndocrinologyGlucoseDepression ChemicalPrednisolonebiology.proteinExercise TestCorticosteroidmedicine.symptomSulfatasesAcid hydrolasemedicine.drugEuropean journal of applied physiology and occupational physiology
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