Search results for "N 1"

showing 10 items of 1053 documents

IL-23 receptor regulates unconventional IL-17-producing T cells that control bacterial infections.

2010

AbstractIL-23 plays an important role in autoimmune tissue inflammation and induces the generation of not fully characterized effector cells that mediate protection against pathogens. In this paper, we established the essential role of IL-23R in the host response against intracellular pathogens. IL-23 was critical for the expansion or maintenance of γδ and double negative (DN) αβ T cells. These cells were rapidly recruited to the site of infection and produced large amounts of IL-17, IFN-γ, and TNF-α. Notably, DN T cells transferred into L. monocytogenes-infected RAG2−/− mice prevented bacterial growth, confirming their protective role against intracellular pathogens. Our results show that …

T cellCD8 AntigensReceptors Antigen T-Cell alpha-betaImmunologyMice NudeMice TransgenicBiologyArticleImmunophenotypingInterferon-gammaMiceImmune systemAntigenCell MovementT-Lymphocyte SubsetsmedicineImmunology and AllergyAnimalsInterferon gammaListeriosisCells CulturedMice KnockoutEffectorTumor Necrosis Factor-alphaIntracellular parasiteInterleukin-17Receptors Antigen T-Cell gamma-deltaReceptors InterleukinCoculture TechniquesCell biologymedicine.anatomical_structureImmunologyCD4 AntigensInterleukin-23 Subunit p19Tumor necrosis factor alphaInterleukin 17Peritoneummedicine.drugJournal of immunology (Baltimore, Md. : 1950)
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Uptake of Leishmania major by dendritic cells is mediated by Fcγ receptors and facilitates acquisition of protective immunity

2006

Uptake of Leishmania major by dendritic cells (DCs) results in activation and interleukin (IL)-12 release. Infected DCs efficiently stimulate CD4- and CD8- T cells and vaccinate against leishmaniasis. In contrast, complement receptor 3-dependent phagocytosis of L. major by macrophages (MPhi) leads exclusively to MHC class II-restricted antigen presentation to primed, but not naive, T cells, and no IL-12 production. Herein, we demonstrate that uptake of L. major by DCs required parasite-reactive immunoglobulin (Ig)G and involved FcgammaRI and FcgammaRIII. In vivo, DC infiltration of L. major-infected skin lesions coincided with the appearance of antibodies in sera. Skin of infected B cell-de…

T cellImmunologyAntigen presentationLeishmaniasis CutaneousMacrophage-1 AntigenPriming (immunology)Complement receptorArticleImmunoglobulin GMicePhagocytosismedicineAnimalsImmunology and AllergyLeishmania majorCells CulturedLeishmania majorMice KnockoutB-LymphocytesMice Inbred BALB CbiologyMacrophagesReceptors IgGArticlesDendritic Cellsbiology.organism_classificationMice Inbred C57BLmedicine.anatomical_structureImmunoglobulin GImmunologybiology.proteinInterleukin 12CD8Journal of Experimental Medicine
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Active suppression induced by cutaneous exposure to bacterial superantigen is prevented by interleukin-12 treatmentin vivo

1998

Exposure to the bacterial superantigen staphylococcal enterotoxin B (SEB) leads to inhibition of several immune responses and the induction of regulatory cells. The aim of this study was to characterize these regulatory cells further and to investigate the effect of interleukin-12 (IL-12) on superantigen-induced suppression. For this purpose BALB/c mice were injected subcutaneously with low doses of SEB that did not deplete the SEB-reactive V beta T cells. Intravenous transfer of unseparated local-draining lymph node cells from these SEB-treated animals suppressed the proliferative response of mononuclear spleen cells of naive syngeneic recipients for at least 3 weeks. The regulatory cells …

T cellImmunologyhemic and immune systemschemical and pharmacologic phenomenaSpleenPharmacologyBiologybiological factorsmedicine.anatomical_structureImmune systemAntigenImmunologymedicineSuperantigenInterleukin 12Immunology and AllergyLymph nodeCD8Immunology
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T helper cell polarisation in coeliac disease: any (T-)bet ?

2004

Recent data strongly support the view that coeliac disease is a Th1 mediated inflammatory disease as both interferon γ production and T-bet levels in gut infiltrating cells are upregulated The puzzling observation on high interferon γ (IFN-γ) but low interleukin (IL)-12 levels in coeliac disease (CD) has resulted in questions about the underlying principles of T helper cell polarisation. In this issue of G ut ,1 the molecular basis of T helper cell polarisation in CD has been illuminated by the finding that T-bet, the master transcription factor of T helper cell type 1 (Th1) cells, is upregulated in this disease [see page 1090] . The past decade has witnessed a dramatic improvement in our p…

T cellInterleukinsGastroenterologyT helper cellCoeliac DiseaseBiologyTh1 CellsGliadinUp-RegulationInterleukin 21Celiac DiseaseInterferon-gammaImmune systemmedicine.anatomical_structureInterleukin 25ImmunologyInterleukin 13medicineCytotoxic T cellCytokinesHumansIL-2 receptorT-Box Domain ProteinsTranscription FactorsGut
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Enhanced dendritic cell maturation by TNF-alpha or cytidine-phosphate-guanosine DNA drives T cell activation in vitro and therapeutic anti-tumor immu…

2000

Abstract Dendritic cells (DC) manipulated ex vivo can induce tumor immunity in experimental murine tumor models. To improve DC-based tumor vaccination, we studied whether DC maturation affects the T cell-activating potential in vitro and the induction of tumor immunity in vivo. Maturation of murine bone marrow-derived DC was induced by GM-CSF plus IL-4 alone or by further addition of TNF-α or a cytidine-phosphate-guanosine (CpG)-containing oligonucleotide (ODN-1826), which mimics the immunostimulatory effect of bacterial DNA. Flow cytometric analysis of costimulatory molecules and MHC class II showed that DC maturation was stimulated most by ODN-1826, whereas TNF-α had an intermediate effec…

T cellT-LymphocytesImmunologyAntineoplastic AgentsCell CommunicationBiologyLymphocyte ActivationImmunotherapy AdoptiveMiceImmune systemAdjuvants ImmunologicIn vivomedicineTumor Cells CulturedImmunology and AllergyAnimalsInterleukin 4Cells CulturedMice Inbred BALB CTumor Necrosis Factor-alphaCell DifferentiationDendritic cellDendritic CellsMolecular biologyInterleukin-12Coculture TechniquesGrowth InhibitorsMice Inbred C57BLmedicine.anatomical_structureOligodeoxyribonucleotidesColonic NeoplasmsInterleukin 12Cancer researchTumor necrosis factor alphaCpG IslandsFemaleInterleukin-4Ex vivoNeoplasm TransplantationJournal of immunology (Baltimore, Md. : 1950)
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New therapies for sepsis: focus on the interleukin (IL)12 family member IL27

2007

Sepsis is a severe complication of abdominal infections such as peritonitis and is associated with high mortality. Unfortunately, the molecular mechanisms controlling the development of sepsis are still incompletely understood. Interestingly, the interleukin (IL) 12 family member IL27 seems to play a key role in sepsis. In a murine model of septic peritonitis induced by caecal ligation and puncture (CLP), IL27 levels were found to be strongly induced. Furthermore, mice deficient for the EBI3 subunit of IL27 were resistant to CLP-induced septic peritonitis as compared to wild-type controls. This effect could be suppressed by injection of recombinant IL27. Further studies demonstrated that IL…

T-LymphocytesImmunologyPeritonitisPeritonitisGeneral Biochemistry Genetics and Molecular BiologySepsisMiceRheumatologySepsismedicineAnimalsHumansImmunology and AllergyInterleukin 27business.industryInterleukin-17InterleukinCell DifferentiationEBI3medicine.diseaseBlockadeDisease Models AnimalImmunologyInterleukin 12ImmunotherapyInterleukin 17Reactive Oxygen SpeciesbusinessSupplementAnnals of the Rheumatic Diseases
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Cutting Edge: IL-23 Cross-Regulates IL-12 Production in T Cell-Dependent Experimental Colitis

2006

Abstract Although IL-12 and IL-23 share the common p40 subunit, IL-23, rather than IL-12, seems to drive the pathogenesis of experimental autoimmune encephalomyelitis and arthritis, because IL-23/p19 knockout mice are protected from disease. In contrast, we describe in this study that newly created LacZ knockin mice deficient for IL-23 p19 were highly susceptible for the development of experimental T cell-mediated TNBS colitis and showed even more severe colitis than wild-type mice by endoscopic and histologic criteria. Subsequent studies revealed that dendritic cells from p19-deficient mice produce elevated levels of IL-12, and that IL-23 down-regulates IL-12 expression upon TLR ligation. …

T-LymphocytesTransgeneT cellImmunologyDown-RegulationMice TransgenicInterleukin-23PathogenesisMiceInterleukin 23AnimalsImmunology and AllergyMedicineColitisCells Culturedbusiness.industryInterleukinsExperimental autoimmune encephalomyelitisColitismedicine.diseaseInterleukin-12Survival RateDisease Models AnimalProtein Subunitsmedicine.anatomical_structureImmunologyKnockout mouseInterleukin-23 Subunit p19Interleukin 12Disease SusceptibilitybusinessThe Journal of Immunology
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A Regulatory Mechanism Involving TBP-1/Tat-Binding Protein 1 and Akt/PKB in the Control of Cell Proliferation

2011

Abstract TBP-1 /Tat-Binding Protein 1 (also named Rpt-5, S6a or PSMC3) is a multifunctional protein, originally identified as a regulator of HIV-1-Tat mediated transcription. It is an AAA-ATPase component of the 19S regulative subunit of the proteasome and, as other members of this protein family, fulfils different cellular functions including proteolysis and transcriptional regulation. We and others reported that over expression of TBP-1 diminishes cell proliferation in different cellular contexts with mechanisms yet to be defined. Accordingly, we demonstrated that TBP-1 binds to and stabilizes the p14ARF oncosuppressor increasing its anti-oncogenic functions. However, TBP-1 restrains cell…

TBP-1/Tat-Binding Protein 1lcsh:Medicinemacromolecular substancesBiologymTORC2Cell GrowthTBP-1/Tat-Binding Protein 1; cell proliferationp14arfMolecular Cell BiologyGeneticsCancer GeneticsTranscriptional regulationGene Networkslcsh:ScienceBiologyProtein kinase BPI3K/AKT/mTOR pathwayMultidisciplinaryCell growthAKTBinding proteinlcsh:RMolecular biologySignaling CascadesCell biologyTBP-1enzymes and coenzymes (carbohydrates)cell proliferationbiology.proteinMdm2lcsh:QCell DivisionResearch ArticleSignal TransductionPLoS ONE
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Common gene expression strategies revealed by genome-wide analysis in yeast

2007

A comprehensive analysis of six variables characterizing gene expression in yeast, including transcription and translation, mRNA and protein amounts, reveals a general tendency for levels of mRNA and protein to be harmonized, and for functionally related genes to have similar values for these variables.

TBX1GeneticsRegulation of gene expressionResearchRNA StabilityStructural geneGenes FungalComputational BiologyGene ExpressionSaccharomyces cerevisiaeBiologyRetinoblastoma-like protein 1EIF4EBP1SaccharomycesGene Expression Regulation FungalMultiprotein ComplexesSNAP23Gene expressionExpressió genèticaCluster AnalysisGeneGenome Biology
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Focal adhesions are hotspots for keratin filament precursor formation

2006

Recent studies showed that keratin filament (KF) formation originates primarily from sites close to the actin-rich cell cortex. To further characterize these sites, we performed multicolor fluorescence imaging of living cells and found drastically increased KF assembly in regions of elevated actin turnover, i.e., in lamellipodia. Abundant KF precursors (KFPs) appeared within these areas at the distal tips of actin stress fibers, moving alongside the stress fibers until their integration into the peripheral KF network. The earliest KFPs were detected next to actin-anchoring focal adhesions (FAs) and were only seen after the establishment of FAs in emerging lamellipodia. Tight spatiotemporal …

TalinKeratin 14Intermediate Filamentsmacromolecular substancesBiologyTransfectionKeratin 18Cell LineFocal adhesionMiceReportStress FibersCell cortexMetalloproteinsAnimalsHumansRNA AntisensePseudopodiaCytoskeletonActinResearch ArticlesCell Line TransformedFocal AdhesionsKeratin FilamentKeratin-18Keratin-14Cell BiologyBridged Bicyclo Compounds HeterocyclicActinsZyxinCell biologyProtein TransportThiazolesBiochemistryEpidermolysis Bullosa SimplexMutationKeratinsThiazolidinesMarine ToxinsLamellipodiumPaxillinThe Journal of Cell Biology
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