Search results for "NB"

showing 10 items of 4401 documents

Le vignoble gallo-romain de Gevrey-Chambertin "Au-dessus de Bergis", Côte d'Or (Ier-IIe s. ap. J.-C.) : Modes de plantation et de conduite de vignes …

2011

Recent excavations at " Au-dessus de Bergis " in Gevrey-Chambertin (Burgundy, France) brought to light in 2008 and 2009 numerous, rectangular aligned pits interpreted as an ancient vineyard dated from the mid-first, mid-second century to the third century AD. The vineyard measured at least one hectare in area and was bounded to the south by a hedge or fence. It was composed of parallel rows oriented 6° N that may have been trained over and along high, inclined wooden frames. Vine stocks were renewed by layering, over several generations of plants. This vineyard attests to the spreading of vineyards from the mid-first century AD, as was initially supposed, and it documents some agricultural …

vignobleAbsenken[SHS.ARCHEO]Humanities and Social Sciences/Archaeology and PrehistoryterroirsprovignagepalissageplantationvineyardAntiquitéRebenBurgundBourgogneVigneAntiquityWeinberg[SHS.ARCHEO] Humanities and Social Sciences/Archaeology and PrehistorytrainingAntikeVineantiquityPflanzung[ SHS.ARCHEO ] Humanities and Social Sciences/Archaeology and PrehistoryvinevigneBurgundyprovining
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Microtubules and microfilaments in HSV-Infected rabbit-kidney cells.

1981

In rabbit kidney cells infected with strains of Herpes simplex virus producing either cell-rounding or polycaryocytosis. Vinblastine induced paracrystals. This could be shown by phase-contrast- and electron-microscopy. Infections were done under one-step-growth conditions or at low MOI. 90 per cent noninfected cells contained stress fibers as detected by Servablue R250-staining. Shortly after recruitment into polycaryocytes, stress fibres of normal length appearing in criss-cross arrangement can be seen in the periphery of these cells. Later they polymerize to very long fibers and finally they are partially destroyed. The time of destruction depends on the MOI employed. By using Actinomycin…

virusesBiologyCycloheximideMicrofilamentmedicine.disease_causeKidneyVinblastineMicrotubulesCell LineCell Fusionchemistry.chemical_compoundViral ProteinsCytopathogenic Effect ViralVirologymedicineAnimalsSimplexvirusCytoskeletonKidneyCell fusionGeneral MedicineVirologyVinblastinemedicine.anatomical_structureHerpes simplex viruschemistryGiant cellCell cultureDNA ViralRabbitsmedicine.drugArchives of virology
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Vaginal infection of mice with HSV type 2 variant ER−: A new animal model for human primary genital HSV type 2 infections

1992

Abstract Studying the pathogenesis of vaginal infections in mice with two variants of Herpes simplex virus type 2 (HSV-2) strain ER we observed that both variants ER+ and ER− caused severe vaginitis but only ER+ invaded the CNS leading to lethal neurological disease. In contrast, mice infected with ER− cleared the virus from the vagina and recovered from infection. ER+ and ER− expressed equal levels of thymidine kinase (TK) indicating a TK-independent difference in neurovirulence. Using the non-neurovirulent variant ER−, we were able to investigate humoral immune responses late after infection. Vaginal infection with ER− suppressed serum antibody formation after a secondary systemic HSV-1 i…

virusesBiologyVirus Replicationmedicine.disease_causeModels BiologicalVirusHerpesviridaePathogenesisMiceImmune systemVirologymedicineAnimalsSimplexvirusVaginitisMice Inbred BALB CHerpes GenitalisVirulencemedicine.diseaseVirologyMice Inbred C57BLDisease Models AnimalHerpes simplex virusmedicine.anatomical_structureAntibody FormationVaginaVaginabiology.proteinFemaleAntibodyJournal of Virological Methods
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Mouse models of cytomegalovirus latency: overview.

2002

Abstract Background: The molecular regulation of viral latency and reactivation is a central unsolved issue in the understanding of cytomegalovirus (CMV) biology. Like human CMV (hCMV), murine CMV (mCMV) can establish a latent infection in cells of the myeloid lineage. Since mCMV genome remains present in various organs after its clearance from hematopoietic cells first in bone marrow and much later in blood, there must exist one or more widely distributed cell type(s) representing the cellular site(s) of enduring mCMV latency in host tissues. Endothelial cells and histiocytes are candidates, but the question is not yet settled. Another long debated problem appears to be solved: mCMV establ…

virusesCytomegalovirusBiologymedicine.disease_causeVirusHerpesviridaeImmediate-Early ProteinsTransactivationMiceViral ProteinsVirologyVirus latencymedicineCytotoxic T cellAnimalsHumansLatency (engineering)GeneMice Inbred BALB Cvirus diseasesmedicine.diseaseVirologyVirus LatencyHaematopoiesisDisease Models AnimalInfectious DiseasesImmunologyCytomegalovirus InfectionsTrans-ActivatorsVirus ActivationJournal of clinical virology : the official publication of the Pan American Society for Clinical Virology
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Functional properties of a monoclonal antibody inhibiting the hepatitis C virus RNA-dependent RNA polymerase.

2001

The hepatitis C virus (HCV) RNA-dependent RNA polymerase (RdRp), represented by nonstructural protein 5B (NS5B), has recently emerged as a promising target for antiviral intervention. Here, we describe the isolation, functional characterization, and molecular cloning of a monoclonal antibody (mAb) inhibiting the HCV RdRp. This mAb, designated 5B-12B7, binds with high affinity to a conformational epitope in the palm subdomain of the HCV RdRp and recognizes native NS5B expressed in the context of the entire HCV polyprotein or subgenomic replicons. Complete inhibition of RdRp activity in vitro was observed at equimolar concentrations of NS5B and mAb 5B-12B7, whereas RdRp activities of classica…

virusesHepatitis C virusMolecular Sequence DataBiologyViral Nonstructural Proteinsmedicine.disease_causeBiochemistryAntiviral AgentsViruschemistry.chemical_compoundMiceRNA polymerasemedicineAnimalsAmino Acid SequenceMolecular BiologyNS5BImmunoglobulin FragmentsPolymeraseSubgenomic mRNAMice Inbred BALB CBase Sequencevirus diseasesRNAAntibodies MonoclonalCell BiologyVirologyMolecular biologydigestive system diseasesEpitope mappingchemistrybiology.proteinFemaleEpitope MappingThe Journal of biological chemistry
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Replication of herpes simplex virus type 1 and 2 in the medulla of the adrenal gland after vaginal infection of mice.

1996

After vaginal infections of mice with neuroinvasive strains of herpes simplex virus type 1 and 2 (HSV-1, HSV-2) virus replicates in the epithelium of the vagina, in the paravaginal ganglia, in the spinal cord and finally in the brain and in the adrenal glands. However, viral antigens could be demonstrated only in the medulla of the adrenal glands but not in the cortex, as assessed by immunohistochemistry (IHC). HSV could not be isolated from liver, spleen, uterus, and ovaries. This contrasts to the intraperitoneal (i.p) route of infection with replication in different visceral organs including the adrenal gland's cortex.

virusesHerpesvirus 2 HumanUterusSpleenHerpesvirus 1 HumanBiologymedicine.disease_causeVirus ReplicationHerpesviridaeVirusMiceVirologyChlorocebus aethiopsmedicineAnimalsHumansAntigens ViralVero CellsMedullaCerebral CortexMice Inbred BALB CAdrenal glandGeneral MedicineVirologymedicine.anatomical_structureHerpes simplex virusSpinal CordAdrenal MedullaVaginaVaginaFemaleArchives of virology
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Rapid and sensitive detection of metapneumovirus in clinical specimens by indirect fluorescence assay using a monoclonal antibody.

2008

Human metapneumovirus, with two known genotypes named A and B, is associated with mild respiratory symptoms to severe LRTI in children, high-risk adults and the elderly. Rapid and reliable methods of hMPV detection in clinical samples are essential to implement appropriate care, to better understand the pathology of hMPV and to determine its epidemiology. Respiratory samples from 1,386 patients collected during 2 consecutive years were screened for hMPV using indirect immunofluorescence (IFA) assay with a monoclonal antibody. Forty-three patients tested positive for hMPV by the IFA method. In parallel, the samples were examined with RT-PCR on the F gene. Of these, 41 specimens were RT-PCR p…

virusesMESH : AgedMESH : Respiratory Tract InfectionsMESH : Fluorescent Antibody Technique IndirectFusion geneMiceMESH : ChildGenotypeMetapneumovirusRespiratory systemChildFluorescent Antibody Technique IndirectAntigens ViralRespiratory Tract InfectionsCells CulturedComputingMilieux_MISCELLANEOUS[SDV.MP.VIR] Life Sciences [q-bio]/Microbiology and Parasitology/VirologyMice Inbred BALB CParamyxoviridae Infectionsmedicine.diagnostic_testbiologyAntibodies Monoclonalvirus diseasesMESH : AdultInfectious DiseasesMESH : Antibodies MonoclonalMESH : Sensitivity and SpecificityAdultmedicine.drug_classMonoclonal antibodyImmunofluorescenceSensitivity and Specificity[ SDV.MP.VIR ] Life Sciences [q-bio]/Microbiology and Parasitology/VirologyVirusHuman metapneumovirusVirologyMESH : MiceMESH : Cells CulturedmedicineAnimalsHumansMESH : Mice Inbred BALB CAgedMESH : HumansMESH : Antigens ViralMESH : Paramyxoviridae Infectionsbiology.organism_classificationVirologyrespiratory tract diseasesMESH : MetapneumovirusMetapneumovirusMESH : Animals
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Evaluation of HBs, HBc, and frCP virus-like particles for expression of human papillomavirus 16 E7 oncoprotein epitopes.

2002

<i>Objectives:</i> In an attempt to develop virus-like particles (VLPs) as experimental vaccine against human papilloma virus (HPV)-induced tumours, the HPV16 E7 oncoprotein epitopes spanning amino acid (aa) residues 35–98 were expressed on three proteins capable of VLP formation: hepatitis B virus (HBV) surface (HBs) and core (HBc) antigens, and RNA phage fr coats (frCP). <i>Methods:</i> The profile of immunoglobulin isotypes induced in Balb/C mice after immunization with purified chimeric proteins was studied. <i>Results:</i> The HBs*-E7(35–54) protein expressing E7 residues 35–54 between residues 139 and 142 of the HBs carrier formed HBs-like particles…

virusesPapillomavirus E7 ProteinsRecombinant Fusion ProteinsMolecular Sequence DataRNA PhagesAntibodies ViralEpitopeVirusEpitopesMiceHpv16 e7Immune systemCapsidPapillomavirus E7 ProteinsVirologyAnimalsHumansAmino Acid SequenceHuman papillomavirusneoplasmsMice Inbred BALB CHepatitis B Surface AntigensbiologyVirionvirus diseasesOncogene Proteins ViralVirologyHepatitis B Core Antigensfemale genital diseases and pregnancy complicationsImmunoglobulin IsotypesInfectious DiseasesImmunizationbiology.proteinFemaleImmunizationAntibodyIntervirology
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PTC124-mediated translational readthrough of a nonsense mutation causing Usher syndrome type 1C.

2011

We investigated the therapeutic potential of the premature termination codon (PTC) readthrough-inducing drug PTC124 in treating the retinal phenotype of Usher syndrome, caused by a nonsense mutation in the USH1C gene. Applications in cell culture, organotypic retina cultures, and mice in vivo revealed significant readthrough and the recovery of protein function. In comparison with other readthrough drugs, namely the clinically approved readthrough-inducing aminoglycoside gentamicin, PTC124 exhibits significant better retinal biocompatibility. Its high readthrough efficiency in combination with excellent biocompatibility makes PTC124 a promising therapeutic agent for PTCs in USH1C, as well a…

virusesUsher syndromeGenetic enhancementNonsense mutationGenetic VectorsCell Cycle ProteinsRetina03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineIn vivootorhinolaryngologic diseasesGeneticsmedicineAnimalsHumansMolecular BiologyCells Cultured030304 developmental biologyAdaptor Proteins Signal TransducingGenetics0303 health sciencesOxadiazolesbusiness.industryfungiAminoglycosideTranslational readthroughmedicine.diseasePhenotype3. Good healthAtalurenMice Inbred C57BLCytoskeletal ProteinsLuminescent ProteinsElectroporationchemistryMicroscopy FluorescenceCodon NonsenseCancer researchMolecular MedicineGentamicinsbusinessUsher Syndromes030217 neurology & neurosurgeryHuman gene therapy
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Enhancement by TNF-alpha of reactivation and replication of latent herpes simplex virus from trigeminal ganglia of mice.

1995

The influence of tumor-necrosis-factor-alpha (TNF-alpha), granulocyte-macrophage colony-stimulating factor (GM-CSF), interleukine-1 (IL-1) and IL-3 on the in vitro reactivation frequency and replication rate of trigeminal ganglia of mice latently infected with herpes simplex virus (HSV) strain KOS was studied. It could be demonstrated that TNF-alpha and possibility GM-CSF, but not IL-1 and IL-3, enhanced the reactivation frequency and replication of HSV. Interferon alpha/beta (IFN alpha/beta) prevented reactivation and replication.

virusesmedicine.medical_treatmentHerpesvirus 1 HumanBiologymedicine.disease_causeVirus ReplicationVirusHerpesviridaeMiceInterferonVirologyAlphaherpesvirinaeChlorocebus aethiopsmedicineAnimalsHumansVero CellsMice Inbred BALB CTumor Necrosis Factor-alphaGranulocyte-Macrophage Colony-Stimulating FactorInterferon-alphaGeneral MedicineInterferon-betabiology.organism_classificationVirologyIn vitroVirus LatencyCytokineHerpes simplex virusViral replicationTrigeminal GanglionInterleukin-3Virus Activationmedicine.drugInterleukin-1Archives of virology
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