Search results for "NECROSIS"

showing 10 items of 1354 documents

Major histocompatibility complex regulation of cytokine production.

1996

This review describes the phenomenon of the major histocompatibility complex (MHC) control of cytokine production both in experimental animals and in humans. H-2 (mouse MHC) regulates which type of cytokine is selectively produced in response to the hapten trinitrophenyl (TNP). T cells from TNP-immune H-2k mice produce interferon-gamma (IFN-gamma), interleukin-2 (IL-2), IL-3, IL-5, tumor necrosis factor-alpha (TNF-alpha), IL-10, and very low levels of IL-4 on reexposure to the specific antigen in vitro. By contrast, T cells from H-2d mice produce IL-3, TNF-alpha, IL-10, and IL-4 but very low levels of IL-2, IL-5 and IFN-gamma. As MHC-congenic matched strains (BALB/k and BALB/c) are used, th…

medicine.medical_treatmentImmunologychemical and pharmacologic phenomenaMajor histocompatibility complexPeripheral blood mononuclear cellMajor Histocompatibility ComplexInterferon-gammaMiceImmune systemAntigenVirologyImmunopathologymedicineAnimalsHumansbiologyTumor Necrosis Factor-alphaInterleukinsH-2 AntigensCell BiologyCytokineImmunologyAntibody Formationbiology.proteinCytokinesTumor necrosis factor alphaHapten
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INFLAMMATORY CYTOKINES IN ACUTE ISCHEMIC STROKE.

2008

Three major cytokines, namely, tumor necrosis factor (TNF-alpha), interleukin (IL)-1, and IL-6 are produced by cultured brain cells after various stimuli such as ischemia. Neurones, astrocytes, microglia and oligodendrocytes can produce inflammatory mediators, and cytokine receptors are expressed constitutionally throughout the Central Nervous System (CNS), albeit at low levels. Cytokines are involved in virtually every facet of stroke and they have numerous pro-inflammatory and pro-coagulant effects on endothelium. TNF-alpha expression after stroke stimulates expression of tissue factor and adhesion molecules for leukocytes, release of interleukin-1 (IL-1), nitric oxide, factor VIII/von Wi…

medicine.medical_treatmentInflammationProinflammatory cytokineBrain IschemiaBrain ischemiaTissue factorDrug DiscoverymedicineHumansStrokePharmacologyInflammationMicrogliabusiness.industrymedicine.diseasePrognosisStrokeCytokinemedicine.anatomical_structureImmunologyAcute DiseaseCytokinesTumor necrosis factor alphamedicine.symptomStroke cytokinesbusinessBiomarkers
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Interaction Between Cytokines and Oxidative Stress in Acute Pancreatitis

2006

Acute pancreatitis is an inflammation initially localized in the pancreatic gland which may lead to local and systemic complications. The development of severe acute pancreatitis is mediated by pathophysiological mechanisms involved in the systemic inflammatory response, cytokines and oxidative stress being their components of major importance. Nevertheless, it is still unknown why an episode of acute pancreatitis remains mild or progresses to a severe form. Activated leukocytes are the main source of cytokines. Interleukin 1beta and tumor necrosis factor alpha (TNF-alpha) initiate and propagate almost all the consequences of the systemic inflammatory response syndrome, leading to amplifica…

medicine.medical_treatmentInflammationmedicine.disease_causeBiochemistryProinflammatory cytokineDrug DiscoverymedicineAnimalsHumansPharmacologyChemistryOrganic ChemistryModels Immunologicalmedicine.diseaseSystemic inflammatory response syndromeOxidative StressCytokinePancreatitisAcute DiseaseImmunologyCytokinesMolecular MedicineAcute pancreatitisPancreatitisTumor necrosis factor alphamedicine.symptomOxidative stressCurrent Medicinal Chemistry
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Regulation of cytochrome P450 IID by acute phase mediators in C3H/HeJ mice.

1992

Abstract Cytochrome P450 IID6 is a drug metabolizing enzyme and the major target antigen in LKM-1 antibody positive chronic active hepatitis. The histological hallmark of chronic active hepatitis is a lymphocytic infiltrate in the liver. It is unknown whether and how cytokines produced and secreted by these tissue infiltrating mononuclear cells regulate the cellular expression of cytochrome P450 IID6. To study the effect of interleukin 1, tumor necrosis factor and interleukin 6 on the hepatocellular RNA expression of cytochrome P450 IID, we injected each of the cytokines in C3H HeJ mice. We found a time-dependent suppression of the cytochrome in the liver. Six hours after the intraperitonea…

medicine.medical_treatmentIntraperitoneal injectionBiophysicsBiochemistryMixed Function OxygenasesMiceCytochrome P-450 Enzyme SystemmedicineEscherichia coliAnimalsHumansInterleukin 6Molecular BiologyHepatitisMice Inbred C3HbiologyInterleukin-6Tumor Necrosis Factor-alphaCytochrome P450InterleukinCell Biologymedicine.diseaseBlotting NorthernRecombinant ProteinsEndotoxinsCytokineCytochrome P-450 CYP2D6LiverImmunologybiology.proteinRNATumor necrosis factor alphaFemaleAntibodyInterleukin-1Biochemical and biophysical research communications
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Ex vivoevidence for PGE2and LTB4involvement in cutaneous leishmaniasis: relation with infection status and cytokine production

1996

SUMMARYEx vivoculture of spleen cells from BALB/c mice infected with 2 × 106Leishmania major(L.major) promastigotes were cultured with ConcanavalinA (ConA) or leishmanial antigen (L. Ag) and tested for prostaglandin E2(PGE2) and for leukotriene B4(LTB4), in order to study their involvement in the evolution of cutaneous leishmaniasis and the connexion with lymphokine-mediated responses. The data were compared with those obtained in BALB/c mice protected againstL. majorby sublethal irradiation (550 rad; cured mice). In the unprotected BALB/c mice the levels of PGE2that were responsible for the depression of interferon-γ (IFN-γ) and tumour necrosis factor-α (TNFα) Th1-associated cytokines and …

medicine.medical_treatmentLeishmaniasis CutaneousInflammationLeukotriene B4DinoprostoneInterferon-gammaMiceTh2 CellsImmune systemCutaneous leishmaniasismedicineAnimalsLeishmania majorInterferon gammaCells CulturedLeishmania majorMice Inbred BALB CbiologyTumor Necrosis Factor-alphaTh1 Cellsbiology.organism_classificationmedicine.diseaseInfectious DiseasesCytokineImmunologyFemaleAnimal Science and ZoologyParasitologyTumor necrosis factor alphaInterleukin-4medicine.symptomSpleenWhole-Body IrradiationEx vivomedicine.drugParasitology
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Cytokine expression profile in idiopathic inflammatory myopathies.

1996

Cytokines have been shown to be potent inducers of major histocompatibility complexes (MHC) class I and II as well as of cell adhesion molecules in muscle tissue cultures, indicating that cytokines may play a role in mediating muscle fiber damage in inflammatory myopathies. We found in 21 cases of autoimmune myositis various amounts of inflammatory cells expressing interleukin (IL)-1 alpha and -beta, IL-2, IL-4, tumor necrosis factor (TNF) -alpha and -beta, and interferon (IFN)-gamma and its receptor. Muscle fibers displayed enhanced expression of IL-1 alpha and -beta, IL-2, and TNF-alpha. Upregulation of cytokines was strongest at sites of cellular infiltration typical for the respective m…

medicine.medical_treatmentMuscle Fibers SkeletalInflammationCytokine Expression ProfileBiologyMuscular DystrophiesPathology and Forensic MedicineCellular and Molecular NeuroscienceInterferonmedicineCytotoxic T cellHumansInterleukin 4InflammationInterleukinGeneral MedicineImmunohistochemistryPolymyositisCytokineNeurologyImmunologyCytokinesTumor necrosis factor alphaNeurology (clinical)medicine.symptommedicine.drugJournal of neuropathology and experimental neurology
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Differential requirements for antigen or homeostatic cytokines for proliferation and differentiation of human Vgamma9Vdelta2 naive, memory and effect…

2005

We have compared four human subsets of Vgamma9Vdelta2 T cells, naive (T(naive), CD45RA(+)CD27(+)), central memory (T(CM), CD45RA(-)CD27(+)), effector memory (T(EM), CD45RA(-)CD27(-)) and terminally differentiated (T(EMRA), CD45RA(+)CD27(-)), for their capacity to proliferate and differentiate in response to antigen or homeostatic cytokines. Cytokine responsiveness and IL-15R expression were low in T(naive) cells and progressively increased from T(CM) to T(EM) and T(EMRA) cells. In contrast, the capacity to expand in response to antigen or cytokine stimulation showed a reciprocal pattern and was associated with resistance to cell death and Bcl-2 expression. Whereas antigen-stimulated cells a…

medicine.medical_treatmentT cellCellular differentiationImmunologychemical and pharmacologic phenomenaBiologyLymphocyte ActivationAntigenimmune system diseasesT-Lymphocyte SubsetsmedicineImmunology and AllergyHomeostasisHumansAntigensReceptorCells CulturedInterleukin-15Receptors Interleukin-15virus diseaseshemic and immune systemsCell DifferentiationReceptors Antigen T-Cell gamma-deltaReceptors Interleukin-2In vitroCell biologyTumor Necrosis Factor Receptor Superfamily Member 7Cytokinemedicine.anatomical_structureInterleukin 15CytokinesLeukocyte Common AntigensImmunologic MemoryEx vivoEuropean journal of immunology
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The Italian Society of Gastroenterology (SIGE) and the Italian Group for the study of Inflammatory Bowel Disease (IG-IBD) Clinical Practice Guideline…

2010

Biological therapies are an important step in the management of Inflammatory Bowel Diseases. In consideration of high cost and safety issues there is the need to have clear recommendations for their use. Despite the American Gastroenterological Association and the European Crohn's and Colitis Organisation have published exhaustive Inflammatory Bowel Disease guidelines, national guidelines may be necessary as cultural values, economical and legal issues may differ between countries. For these reasons the Italian Society of Gastroenterology and the Italian Group for the study of Inflammatory Bowel Disease have decided to elaborate the Italian guidelines on the use of biologics in Inflammatory…

methods Tumor Necrosis Factor-alphaAnti-Inflammatory AgentsUlcerativeDiseaseGUIDELINESHumanized Antibodieetiology Pregnancy Pregnancy ComplicationGastroenterologyInflammatory bowel diseaseetiology Opportunistic InfectionCrohn DiseasePregnancyNeoplasmsMonoclonaldrug therapy Remission Inductionantagonists /&/ inhibitorsSettore MED/12 - GastroenterologiaRemission InductionGastroenterologyAntibodies MonoclonalUlcerative colitisAnti-Inflammatory AgentItalyadverse effects/therapeutic use Intestinal FistulaTumor necrosis factor alphaFemaleImmunosuppressive Agentsmedicine.drugbiological drugsmedicine.medical_specialtyIBDOpportunistic InfectionsAntibodies Monoclonal HumanizedAutoimmune Diseasesadverse effects/therapeutic use Autoimmune DiseaseInternal medicinemedicineAdalimumabIntestinal FistulaHumansColitisdiagnosis/drug therapy/surgery Italy Neoplasmadverse effects/therapeutic use AntibodieHepatologydrug therapy Female Humans Immunosuppressive Agentbusiness.industryTumor Necrosis Factor-alphaAdalimumabCancermedicine.diseaseInfliximabInfliximabdigestive system diseasesdrug therapy Crohn Diseaseetiology ColitiPregnancy ComplicationsColitis Ulcerativebusiness
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Consequences of analgesics use in early pregnancy: Results of tests on mice.

2019

International audience; Self-medication during pregnancy continues to be an issue in developing countries due topoor medical education. The most commonly used drugs are analgesics, mainlyacetaminophen (paracetamol, APAP) and, to a lesser extent, ketoprofen (KPF). The aim of thestudy was to establish whether there are consequences of accidental use of these two drugsduring early embryogenesis. The experimental study was performed on 30 pregnant whitemice, divided into three groups: a first group to which APAP was administered, a secondgroup to which KPF was administered, and group 3 as a control group. At delivery, the babymice were examined, and after their first parturition, they were take…

mice embryogenesisEnvironmental EngineeringNecrosisketoprofen010504 meteorology & atmospheric sciencesparacetamolUrinary systemPhysiology010501 environmental sciencesKidney01 natural sciencesliver.MiceNecrosisPregnancyEctasiamedicineEnvironmental ChemistryAnimalsWaste Management and Disposal0105 earth and related environmental sciencesAcetaminophenPregnancyKidneybusiness.industrydigestive oral and skin physiologyAnalgesics Non-Narcoticmedicine.diseasePollution3. Good healthAcetaminophenmedicine.anatomical_structureLiverHemosiderinToxicity[SDE]Environmental SciencesFemalemicroscopic changesmedicine.symptomChemical and Drug Induced Liver Injurybusinessmedicine.drugThe Science of the total environment
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Anti-senescence and Anti-inflammatory Effects of the C-terminal Moiety of PTHrP Peptides in OA Osteoblasts.

2016

Osteoarthritis (OA) is characterized by degenerative changes in the whole joint leading to physical disability in the elderly population. This condition is associated with altered bone metabolism in subchondral areas suggesting that therapeutic strategies aimed at modifying bone cell metabolism may be of interest. We have investigated the effects of several parathyroid hormone-related protein (PTHrP)-derived peptides (1-37): (N-terminal), (107-111) and (107-139) (C-terminal) on senescence features induced by inflammatory stress in human OA osteoblasts. Incubation of these primary cells with interleukin(IL)-1β led to an increased expression of senescence markers senescence-associated-β-galac…

musculoskeletal diseases0301 basic medicineSenescenceMaleAgingmedicine.medical_specialtyInterleukin-1betaParathyroid hormoneFluorescent Antibody TechniqueReal-Time Polymerase Chain ReactionDinoprostone03 medical and health sciencesDownregulation and upregulationInternal medicineBone cellOsteoarthritismedicineHumansProstaglandin E2Cells CulturedCellular SenescenceAgedOsteoblastsParathyroid hormone-related proteinbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaParathyroid Hormone-Related ProteinPeptide Fragments030104 developmental biologyEndocrinologyTumor necrosis factor alphaFemaleGeriatrics and GerontologyInflammation MediatorsbusinessCell aginghormones hormone substitutes and hormone antagonistsmedicine.drugThe journals of gerontology. Series A, Biological sciences and medical sciences
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