Search results for "NERVE"

showing 10 items of 1683 documents

Induction of identified mesodermal cells by CNS midline progenitors in Drosophila.

1997

ABSTRACT The Drosophila ventral midline cells generate a discrete set of CNS lineages, required for proper patterning of the ventral ectoderm. Here we provide the first evidence that the CNS midline cells also exert inductive effects on the mesoderm. Mesodermal progenitors adjacent to the midline progenitor cells give rise to ventral somatic mucles and a pair of unique cells that come to lie dorsomedially on top of the ventral nerve cord, the so-called DM cells. Cell ablation as well as cell transplantation experiments indicate that formation of the DM cells is induced by midline progenitors in the early embryo. These results are corroborated by genetic analyses. Mutant single minded embryo…

Central Nervous SystemMesodermanimal structuresSomatic cellCellEctodermNerve Tissue ProteinsBiologyMesodermCell MovementProto-Oncogene ProteinsmedicineMorphogenesisAnimalsDrosophila ProteinsProgenitor cellEye ProteinsMolecular BiologyEmbryonic InductionEpidermal Growth FactorCell growthGene Expression Regulation DevelopmentalMembrane ProteinsEmbryoAnatomyCell biologyDNA-Binding Proteinsmedicine.anatomical_structureDrosophila melanogasterVentral nerve cordembryonic structuresDevelopmental BiologySignal TransductionTranscription FactorsDevelopment (Cambridge, England)
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Molecular mechanisms linking neuroinflammation and neurodegeneration in MS.

2013

Multiple sclerosis (MS) is an inflammatory demyelinating autoimmune disorder of the central nervous system (CNS) and one of the leading causes of neurological deficits and disability in young adults in western countries. Current medical treatment mainly influences disease progression via immunomodulatory or immunosuppressive actions. Indeed, MS research has been foremost focused on inflammation in the CNS, but more recent evidence suggests that chronic disability in MS is caused by neurodegeneration. Imaging studies show an early involvement of neurodegeneration as brain atrophy and gray matter lesions can be observed at disease onset. Thus, neuroprotective treatment strategies and the eluc…

Central Nervous SystemMultiple SclerosisCentral nervous systemBiologyNeuroprotectionPathogenesisAtrophyDevelopmental NeurosciencemedicineAnimalsHumansImmunologic FactorsNeuroinflammationInflammationMultiple sclerosisExperimental autoimmune encephalomyelitisNeurodegenerationmedicine.diseaseDisease Models Animalmedicine.anatomical_structureNeurologyImmunologyNerve DegenerationDisease ProgressionCytokinesNeuroscienceExperimental neurology
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Morphological Characterization of the Entire Interneuron Population Reveals Principles of Neuromere Organization in the Ventral Nerve Cord ofDrosophi…

2011

Decisive contributions to our understanding of the mechanisms underlying the development of the nervous system have been made by studies performed at the level of single, identified cells in the fruit flyDrosophila. While all the motor neurons and glial cells in thoracic and abdominal segments of theDrosophilaembryo have been individually identified, few of the interneurons, which comprise the vast majority of cells in the CNS, have been characterized at this level. We have applied a single cell labeling technique to carry out a detailed morphological characterization of the entire population of interneurons in abdominal segments A1–A7. Based on the definition of a set of spatial parameters…

Central Nervous SystemNervous systemCell typeInterneuronCD8 AntigensGreen Fluorescent ProteinsLIM-Homeodomain ProteinsModels NeurologicalStatistics as TopicPopulationCell CountBiologyFunctional LateralityAnimals Genetically ModifiedInterneuronsNeural PathwaysmedicineAnimalsDrosophila ProteinsAmino Acidseducationeducation.field_of_studyGeneral NeurosciencefungiArticlesNeuromereAxonsmedicine.anatomical_structureVentral nerve cordDrosophilaAxon guidanceNeuroscienceDrosophila ProteinTranscription FactorsThe Journal of Neuroscience
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Hunchback is required for the specification of the early sublineage of neuroblast 7-3 in the Drosophila central nervous system.

2002

The Drosophila ventral nerve cord (VNC) derives from neuroblasts (NBs), which mostly divide in a stem cell mode and give rise to defined NB lineages characterized by specific sets of sequentially generated neurons and/or glia cells. To understand how different cell types are generated within a NB lineage, we have focused on the NB7-3 lineage as a model system. This NB gives rise to four individually identifiable neurons and we show that these cells are generated from three different ganglion mother cells (GMCs). The finding that the transcription factor Hunchback (Hb) is expressed in the early sublineage of NB7-3, which consists of the early NB and the first GMC (GMC7-3a) and its progeny (E…

Central Nervous SystemNeuronsCell typeLineage (genetic)biologyStem CellsGene ExpressionAnatomyCell fate determinationbiology.organism_classificationCell biologyDNA-Binding ProteinsDrosophila melanogasterNeuroblastMutagenesisVentral nerve cordAnimalsDrosophila ProteinsCell LineageStem cellDrosophila melanogasterMolecular BiologyDrosophila ProteinDevelopmental BiologyTranscription FactorsDevelopment (Cambridge, England)
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Neurotrophin secretion: current facts and future prospects

2003

The proteins of the mammalian neurotrophin family (nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), neurotrophin-3 (NT-3) and neurotrophin-4/5 (NT-4/5)) were originally identified as neuronal survival factors. During the last decade, evidence has accumulated implicating them (especially BDNF) in addition in the regulation of synaptic transmission and synaptogenesis in the CNS. However, a detailed understanding of the secretion of neurotrophins from neurons is required to delineate their role in regulating synaptic function. Some crucial questions that need to be addressed include the sites of neurotrophin secretion (i.e. axonal versus dendritic; synaptic versus extrasyna…

Central Nervous SystemNeuronsNeuronal PlasticityArc (protein)biologyCell SurvivalGeneral NeuroscienceSynaptogenesisLong-term potentiationAMPA receptorNeurotransmissionCell Linenervous systemNeurotrophic factorsTrk receptorbiology.proteinAnimalsHumansNerve Growth FactorsPeptidesNeuroscienceForecastingNeurotrophinProgress in Neurobiology
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Programmed cell death in the embryonic central nervous system of Drosophila melanogaster.

2006

Although programmed cell death (PCD) plays a crucial role throughout Drosophila CNS development, its pattern and incidence remain largely uninvestigated. We provide here a detailed analysis of the occurrence of PCD in the embryonic ventral nerve cord (VNC). We traced the spatio-temporal pattern of PCD and compared the appearance of, and total cell numbers in,thoracic and abdominal neuromeres of wild-type and PCD-deficient H99mutant embryos. Furthermore, we have examined the clonal origin and fate of superfluous cells in H99 mutants by DiI labeling almost all neuroblasts, with special attention to segment-specific differences within the individually identified neuroblast lineages. Our data r…

Central Nervous SystemProgrammed cell deathanimal structuresEmbryo NonmammalianApoptosisCell CountBiologyNeuroblastInterneuronsmedicineAnimalsCell LineageMolecular BiologyBody PatterningNeuronsGene Expression Regulation DevelopmentalAnatomyNeuromerebiology.organism_classificationEmbryonic stem cellImmunohistochemistryCell biologyClone Cellsmedicine.anatomical_structureDrosophila melanogasternervous systemVentral nerve cordMutationNeuronDrosophila melanogasterGanglion mother cellDevelopmental BiologyDevelopment (Cambridge, England)
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A critical role for the cholesterol-associated proteolipids PLP and M6B in myelination of the central nervous system.

2012

The formation of central nervous system myelin by oligodendrocytes requires sterol synthesis and is associated with a significant enrichment of cholesterol in the myelin membrane. However, it is unknown how oligodendrocytes concentrate cholesterol above the level found in nonmyelin membranes. Here, we demonstrate a critical role for proteolipids in cholesterol accumulation. Mice lacking the most abundant myelin protein, proteolipid protein (PLP), are fully myelinated, but PLP-deficient myelin exhibits a reduced cholesterol content. We therefore hypothesized that "high cholesterol" is not essential in the myelin sheath itself but is required for an earlier step of myelin biogenesis that is f…

Central Nervous SystemProteolipid protein 1Nerve Tissue ProteinsBiologyCell Line03 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compoundMyelinMice0302 clinical medicineimmune system diseasesmedicineEvoked Potentials Auditory Brain StemAnimalsMyelin Proteolipid ProteinMyelin Sheath030304 developmental biology0303 health sciencesMembrane GlycoproteinsCholesterolProteolipidsLeukodystrophyPelizaeus–Merzbacher diseasemedicine.diseaseOligodendrocytenervous system diseasesMyelin proteolipid proteinmedicine.anatomical_structureCholesterolnervous systemNeurologychemistryBiochemistryEvoked Potentials Visuallipids (amino acids peptides and proteins)Vomeronasal Organ030217 neurology & neurosurgeryGlia
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The muscleblind gene participates in the organization of Z-bands and epidermal attachments of Drosophila muscles and is regulated by Dmef2.

1998

We report the embryonic phenotype of muscleblind (mbl), a recently described Drosophila gene involved in terminal differentiation of adult ommatidia. mbl is a nuclear protein expressed late in the embryo in pharyngeal, visceral, and somatic muscles, the ventral nerve cord, and the larval photoreceptor system. All three mbl alleles studied exhibit a lethal phenotype and die as stage 17 embryos or first instar larvae. These larvae are partially paralyzed, show a characteristically contracted abdomen, and lack striation of muscles. Our analysis of the somatic musculature shows that the pattern of muscles is established correctly, and they form morphologically normal synapses. Ultrastructural a…

Central Nervous SystemSomatic cellMuscle Fibers SkeletalNeuromuscular JunctionMuscle ProteinsGenes InsectBiologymuscle attachmentsmuscleblindMesodermTendonsEctodermAnimalsDrosophila ProteinsConnectinRNA MessengerNuclear proteinMuscle SkeletalMolecular BiologyZ-bandsCell NucleusEpidermis (botany)MyogenesisMEF2 Transcription FactorsDrosophila.Gene Expression Regulation DevelopmentalNuclear ProteinsEmbryoCell DifferentiationCell BiologyAnatomybacterial infections and mycosesEmbryonic stem cellPhenotypeCell biologyDNA-Binding ProteinsMyogenic Regulatory FactorsVentral nerve cordMutationInsect ProteinsDrosophilaPhotoreceptor Cells InvertebratemyogenesisDevelopmental BiologyTranscription FactorsDevelopmental biology
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Neuronal Activity Drives Localized Blood-Brain-Barrier Transport of Serum Insulin-like Growth Factor-I into the CNS

2010

Upon entry into the central nervous system (CNS), serum insulin-like growth factor-1 (IGF-I) modulates neuronal growth, survival, and excitability. Yet mechanisms that trigger IGF-I entry across the blood-brain barrier remain unclear. We show that neuronal activity elicited by electrical, sensory, or behavioral stimulation increases IGF-I input in activated regions. Entrance of serum IGF-I is triggered by diffusible messengers (i.e., ATP, arachidonic acid derivatives) released during neurovascular coupling. These messengers stimulate matrix metalloproteinase-9, leading to cleavage of the IGF binding protein-3 (IGFBP-3). Cleavage of IGFBP-3 allows the passage of serum IGF-I into the CNS thro…

Central Nervous SystemTime FactorsMicrodialysismedicine.medical_treatmentAction PotentialsStimulationFunctional LateralityBody TemperatureReceptor IGF Type 1chemistry.chemical_compoundNeural PathwaysPremovement neuronal activityDrug InteractionsInsulin-Like Growth Factor IMicroscopy ImmunoelectronReceptorCells CulturedNeuronsGeneral NeuroscienceSysneuro//purl.org/becyt/ford/3.1 [https]Protein TransportMedicina Básicamedicine.anatomical_structureMatrix Metalloproteinase 9Blood-Brain BarrierSIGNALING//purl.org/becyt/ford/3 [https]Arachidonic acidNeurogliaLow Density Lipoprotein Receptor-Related Protein-1CIENCIAS MÉDICAS Y DE LA SALUDNeuroscience(all)Central nervous systemNeurocienciasBiophysicsGlutamic AcidEnzyme-Linked Immunosorbent AssayNerve Tissue ProteinsBiologyBlood–brain barrierMOLNEUROmedicineAnimalsHumansImmunoprecipitationRats WistarAnalysis of VarianceGrowth factorEndothelial CellsTransporterCoculture TechniquesElectric StimulationSignalingRatsMolneurochemistryRegional Blood FlowVibrissaeSYSNEURODigoxigeninExcitatory Amino Acid AntagonistsNeuroscience
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Mice lacking Plexin-B3 display normal CNS morphology and behaviour

2009

Semaphorins and their receptors, plexins, have emerged as important regulators of a multitude of biological processes. Plexin-B3 has been shown to be selectively expressed in postnatal oligodendrocytes. In contrast to the well-characterized Plexin-A family and the Plexin-B family members Plexin-B1 and -B2, no data are available on the functional role of Plexin-B3 in the central nervous system in vivo. Here we have elucidated the functional significance of Plexin-B3 by generating and analyzing constitutive knock-out mice. Plexin-B3-deficient mice were found to be viable and fertile. A systematic histological analysis revealed no morphological defects in the brain or spinal cord of mutant ani…

Central Nervous Systemanimal structuresCentral nervous systemNerve Tissue ProteinsReceptors Cell SurfaceAnxietyMotor ActivityNeuropsychological TestsBiologyMiceCellular and Molecular NeuroscienceSemaphorinmedicineAnimalsReceptorMolecular BiologyCells CulturedMice KnockoutBehavior AnimalPlexinAge FactorsCell BiologySpinal cordMotor coordinationOligodendrogliamedicine.anatomical_structureSpinal Cordembryonic structuresbiology.proteinMotor learningNeuroscienceBiomarkersFunction (biology)Molecular and Cellular Neuroscience
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