Search results for "NEUROBIOLOGY"

showing 10 items of 125 documents

Heat shock factor 2 is a stress-responsive mediator of neuronal migration defects in models of fetal alcohol syndrome

2014

Fetal alcohol spectrum disorder (FASD) is a frequent cause of mental retardation. However, the molecular mechanisms underlying brain development defects induced by maternal alcohol consumption during pregnancy are unclear. We used normal and Hsf2-deficient mice and cell systems to uncover a pivotal role for heat shock factor 2 (HSF2) in radial neuronal migration defects in the cortex, a hallmark of fetal alcohol exposure. Upon fetal alcohol exposure, HSF2 is essential for the triggering of HSF1 activation, which is accompanied by distinctive post-translational modifications, and HSF2 steers the formation of atypical alcohol-specific HSF1–HSF2 heterocomplexes. This perturbs the in vivo bindi…

[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyMice0302 clinical medicineradial neuronal migrationHeat Shock Transcription FactorsHSF1[SDV.BDD]Life Sciences [q-bio]/Development BiologyResearch ArticlesHeat-Shock ProteinsComputingMilieux_MISCELLANEOUSRegulation of gene expressionCerebral CortexMice Knockout0303 health sciences[SDV.BDD.EO] Life Sciences [q-bio]/Development Biology/Embryology and OrganogenesisCell biologyheat shock factorsDNA-Binding Proteins[SDV.TOX] Life Sciences [q-bio]/Toxicologymedicine.anatomical_structureCerebral cortexFetal Alcohol Spectrum Disorders[SDV.TOX]Life Sciences [q-bio]/Toxicology[ SDV.NEU.NB ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyMolecular MedicinetranscriptionProtein BindingDoublecortin ProteinFetal alcohol syndromeBiology03 medical and health sciencesMediatorStress PhysiologicalHeat shock protein[SDV.BDD] Life Sciences [q-bio]/Development BiologymedicineAnimals[ SDV.BDD ] Life Sciences [q-bio]/Development Biologymicrotubule‐associated proteinsTranscription factor030304 developmental biologymicrotubule-associated proteins[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiologymedicine.diseaseHeat shock factorDisease Models Animal[SDV.BDD.EO]Life Sciences [q-bio]/Development Biology/Embryology and OrganogenesisGene Expression RegulationImmunologyfetal alcohol syndrome030217 neurology & neurosurgeryMalformations of Cortical Development Group IITranscription FactorsNeuroscience
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Evolution of vertebrate survival circuits

2018

Evolution selects those adaptive features that increase reproductive probabilities and facilitate survival. Analysing the brain circuits mediating risk-avoidance (e.g. defense) and those allowing reward-seeking (motivated) behaviours in different vertebrates leads to several main conclusions. First, circuits mediating risk-avoidance are similar in all studied vertebrates, where they include amygdala homologues located in the posterior half of the cerebral hemispheres, in close relationship with the chemosensory systems. Second, in all vertebrates, reward-seeking behaviours involve the activity of tegmento-striatal dopaminergic pathways, plus other inputs to the ventral striatum, including a…

avoidance0301 basic medicineCognitive NeuroscienceAmygdalareproduction03 medical and health sciencesBehavioral NeuroscienceGlutamatergic0302 clinical medicinemotivationbiology.animalventral striatummedicinecomparative neurobiologyrewardbiologyVentral striatumVertebrateamygdalabiology.organism_classificationdefensePsychiatry and Mental health030104 developmental biologymedicine.anatomical_structureAnamniotesDopaminergic pathwaysneural circuitryForebrainAmnioteNeuroscience030217 neurology & neurosurgeryCurrent Opinion in Behavioral Sciences
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Open science as a better gatekeeper for science and society: a perspective from neurolaw

2018

Results from cognitive neuroscience have been cited as evidence in courtrooms around the world, and their admissibility has been a challenge for the legal system. Unfortunately, the recent reproducibility crisis in cognitive neuroscience, showing that the published studies in cognitive neuroscience may not be as trustworthy as expected, has made the situation worse. Here we analysed how the irreproducible results in cognitive neuroscience literature could compromise the standards for admissibility of scientific evidence, and pointed out how the open science movement may help to alleviate these problems. We conclude that open science not only benefits the scientific community but also the le…

bepress|Life Sciences|Research Methods in Life SciencesOpen sciencePsyArXiv|Social and Behavioral Sciences|Forensic and Legal PsychologyMultidisciplinarybepress|Law|Law and Psychology05 social sciencesPerspective (graphical)NeurolawPsyArXiv|Meta-science050105 experimental psychologyScientific evidencebepress|Life Sciences|Neuroscience and NeurobiologyPsyArXiv|Neuroscience|Cognitive NeurosciencePsyArXiv|Social and Behavioral Sciences03 medical and health sciences0302 clinical medicinePsyArXiv|Neurosciencebepress|Social and Behavioral Sciences0501 psychology and cognitive sciencesEngineering ethicsSociologybepress|Life Sciences|Neuroscience and Neurobiology|Cognitive Neuroscience030217 neurology & neurosurgeryScience Bulletin
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The burden of mood-disorder/cerebrovascular disease comorbidity: essential neurobiology, psychopharmacology, and physical activity interventions

2017

Cardio-vascular diseases (CVDs) and CVD-related disorders (including cerebrovascular diseases; CBVDs) are a major public health concern as they represent the leading cause of mortality and morbidity in developed countries. Patients with CVDs and CBVDs co-morbid with mood disorders, especially bipolar disorder (BD) and major depressive disorder (MDD), suffer reduced quality-of-life and significant disability adjusted for years of life and mortality. The relationship between CVDs/CBVDs and mood disorders is likely to be bidirectional. Evidence for shared genetic risk of pathways involved in stress reaction, serotonin or dopamine signalling, circadian rhythms, and energy balance was reported i…

cognitionmedicine.medical_specialtyPsychological interventionphysical activity03 medical and health sciences0302 clinical medicineNeurobiologycardiovascular diseasemental disordersmedicineHumansBipolar disorderPsychiatrybipolar disorderpsychopharmacologyexerciseMood DisordersDepression; bipolar disorder; cardiovascular disease; cognition; comorbidity; exercise; physical activity; psychopharmacology; quality-of-life; strokeDepressionmedicine.diseaseComorbiditystroke030227 psychiatryCerebrovascular DisordersPsychiatry and Mental healthcomorbidityMoodHypomaniaMood disordersquality-of-lifeMajor depressive disordermedicine.symptomPsychologybipolar disorder; cardiovascular disease; cognition; comorbidity; Depression; exercise; physical activity; psychopharmacology; quality-of-life; strokeMania030217 neurology & neurosurgeryClinical psychology
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Biocontrol of drosophila suzukii by two fatty acids

2020

International audience; Olfaction is a crucial sense for insects to detect food, seek partners or escape dangers. Volatile molecules are mainly perceived through insects' antenna. Odors bind to specific olfactory receptors located in sensilla. Olfactory sensory neurons then synapse on corresponding specific glomeruli of the antennal lobe, the primary olfaction center. This olfactory information is processed in higher centers to trigger a behavioral response. Understanding how odors can influence insect behavior is fundamental in order to protect cultures from pests. In this work, 2 volatile fatty acids are shown to modify significantly Drosophila suzukii courtship and copulation at increasi…

copulation[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiologyfungi[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyDrosophila[SDV.AEN]Life Sciences [q-bio]/Food and Nutritionvolatile fatty acidolfaction
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Study of the evolution of terminal differentiation programs using C. elegans serotonergic neurons as a model

2019

Tesis, 407 páginas, figuras y tablas.

evodevoSerotoninc. elegansneurobiologyEvolution of cell typesC. angariaEvodevoc. angariacaenorhabditis:CIENCIAS DE LA VIDA [UNESCO]serotonincell differentiationserotonergic neuronsNeurobiologySerotonergic neuronsevolution of cell typesUNESCO::CIENCIAS DE LA VIDAC. elegansCell differentiationCaenorhabditisVC4 VC5 motorneurons
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The relationship between vigilance capacity and physical exercise: a mixed-effects multistudy analysis

2019

We thank to all the participants who took part in the experiment.

media_common.quotation_subjectPhysical fitnesslcsh:MedicinePoison controlPhysical exerciseFootballGeneral Biochemistry Genetics and Molecular BiologyCognitive demandsbepress|Life Sciences|Neuroscience and Neurobiology03 medical and health sciences0302 clinical medicineCognitionAgeVO2Sport contextbepress|Life Sciences|Neuroscience and Neurobiology|Cognitive NeuroscienceCardiovascular fitnessGeneralLiterature_REFERENCE(e.g.dictionariesencyclopediasglossaries)Cardiovascular fitnessExercisemedia_commonSport typePsychomotor learningPublic healthbusiness.industryGeneral Neurosciencelcsh:RComputingMethodologies_MISCELLANEOUSPsychomotorPsychomotor vigilance task030229 sport sciencesGeneral MedicineKinesiologySustained attentionPsyArXiv|Neuroscience|Cognitive NeurosciencePsyArXiv|Neurosciencebepress|Life Sciences|Neuroscience and Neurobiology|Behavioral NeurobiologyPublic HealthPsyArXiv|Neuroscience|Behavioral NeuroscienceGeneral Agricultural and Biological SciencesPsychologybusinesshuman activities030217 neurology & neurosurgeryClinical psychologyVigilance (psychology)NeurosciencePeerJ
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Serotonergic modulation of hippocampal acetylcholine release after long-term neuronal grafting

2000

Adult female rats sustained aspirative fimbria-fornix lesions and, 2 weeks later, received intrahippocampal grafts of fetal septal or mixed septal-raphe cell suspensions. Twenty-four months later, the extracellular concentration of hippocampal acetylcholine (ACh) was determined by microdialysis. Basal ACh levels (5-65 fmol/5 microl sham-operated rats) were strongly reduced after lesioning (3-7 fmol/5 microl). In septally transplanted and septal-raphe co-transplanted rats, hippocampal ACh concentrations were restored to near-normal levels (15-25 fmol/5 microl), indicating long-term functional survival of hippocampal transplants. After administration of citalopram (100 microM by infusion) and…

medicine.medical_specialtyMicrodialysisSerotoninTime FactorsFenfluramine[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyFornix BrainHippocampal formationBiologySerotonergicHippocampus03 medical and health sciences0302 clinical medicineFetusInternal medicineExtracellularmedicineAnimalsBrain Tissue TransplantationRats Long-EvansComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciencesFetusNeuronal PlasticityRapheGeneral NeuroscienceGraft SurvivalAcetylcholineRatsEndocrinologyCholinergic FibersRaphe NucleiSeptal Nuclei[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]030217 neurology & neurosurgeryAcetylcholinemedicine.drug
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Modulation of hippocampal acetylcholine release after fimbria-fornix lesions and septal transplantation in rats

1997

Abstract Female Long–Evans rats sustained electrolytic lesions of the fimbria and the dorsal fornix causing a partial lesion of the septohippocampal pathway. Two weeks later, the rats received intra-hippocampal grafts of fetal septal cell suspensions. Nine to twelve months later, the release of acetylcholine (ACh) in the hippocampus of sham-operated, lesion-only and grafted rats was measured by microdialysis. The extent of cholinergic (re)innervation was determined by acetylcholinesterase (AChE) staining and densitometry. In both lesion-only and grafted rats, the ratio of ACh release to AChE staining intensity was increased as compared to sham-operated rats, indicating a loss of endogenous …

medicine.medical_specialtySciences du Vivant [q-bio]/Neurosciences [q-bio.NC]Microdialysis[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyScopolamineMuscarinic AntagonistsHippocampal formationBiologySerotonergicHippocampus03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineNeural PathwaysmedicineAnimalsBrain Tissue TransplantationCholinergic neuronNeurotransmitterComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciences8-Hydroxy-2-(di-n-propylamino)tetralinGeneral NeuroscienceFornixMuscarinic antagonistRats Inbred StrainsAcetylcholineRatsEndocrinologychemistryCholinergic FibersAnesthesiaReceptors SerotoninCholinergicRaphe NucleiFemaleSeptal Nuclei[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Receptors Serotonin 5-HT1030217 neurology & neurosurgeryAcetylcholinemedicine.drug
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Activation of mGlu3 Receptors Stimulates the Production of GDNF in Striatal Neurons

2009

Metabotropic glutamate (mGlu) receptors have been considered potential targets for the therapy of experimental parkinsonism. One hypothetical advantage associated with the use of mGlu receptor ligands is the lack of the adverse effects typically induced by ionotropic glutamate receptor antagonists, such as sedation, ataxia, and severe learning impairment. Low doses of the mGlu2/3 metabotropic glutamate receptor agonist, LY379268 (0.25-3 mg/kg, i.p.) increased glial cell line-derived neurotrophic factor (GDNF) mRNA and protein levels in the mouse brain, as assessed by in situ hybridization, real-time PCR, immunoblotting, and immunohistochemistry. This increase was prominent in the striatum, …

medicine.medical_specialtymedicine.drug_classlcsh:MedicineSubstantia nigraReceptors Metabotropic GlutamateSettore BIO/09 - FisiologiaPolymerase Chain ReactionMiceNeurotrophic factorsInternal medicinemedicineGlial cell line-derived neurotrophic factorAnimalsGlial Cell Line-Derived Neurotrophic FactorRNA MessengerAmino Acidslcsh:ScienceReceptorIn Situ HybridizationNeurological Disorders/Movement DisordersNeuronsMultidisciplinarybiologyNeuroscience/Neuronal and Glial Cell Biologylcsh:RGlutamate receptorBridged Bicyclo Compounds HeterocyclicReceptor antagonistCorpus StriatumEndocrinologyMetabotropic receptornervous systemMetabotropic glutamate receptorSettore BIO/14 - Farmacologiabiology.proteinlcsh:QNeuroscience/Neurobiology of Disease and RegenerationReceptors Metabotropic Glutamate/agonists Glial Cell Line-Derived Neurotrophic FactorResearch Article
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