Search results for "NF-κB"

showing 10 items of 102 documents

The herpesviral antagonist m152 reveals differential activation of STING ‐dependent IRF and NF ‐κB signaling and STING 's dual role during MCMV infec…

2019

Cytomegaloviruses (CMVs) are master manipulators of the host immune response. Here, we reveal that the murine CMV (MCMV) protein m152 specifically targets the type I interferon (IFN) response by binding to stimulator of interferon genes (STING), thereby delaying its trafficking to the Golgi compartment from where STING initiates type I IFN signaling. Infection with an MCMV lacking m152 induced elevated type I IFN responses and this leads to reduced viral transcript levels both in vitro and in vivo This effect is ameliorated in the absence of STING Interestingly, while m152 inhibits STING-mediated IRF signaling, it did not affect STING-mediated NF-κB signaling. Analysis of how m152 targets S…

Innate immune systemGeneral Immunology and MicrobiologyGeneral NeuroscienceNF-κBBiologyeye diseasesGeneral Biochemistry Genetics and Molecular BiologyCell biologyStingchemistry.chemical_compoundImmune systemViral replicationchemistryInterferonStimulator of interferon genesmedicineIRF3Molecular Biologymedicine.drugThe EMBO Journal
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Effects of Flavonoids on Cancer, Cardiovascular and Neurodegenerative Diseases: Role of NF-κB Signaling Pathway

2023

Flavonoids are polyphenolic phytochemical compounds found in many plants, fruits, vegetables, and leaves. They have a multitude of medicinal applications due to their anti-inflammatory, antioxidative, antiviral, and anticarcinogenic properties. Furthermore, they also have neuroprotective and cardioprotective effects. Their biological properties depend on the chemical structure of flavonoids, their mechanism of action, and their bioavailability. The beneficial effects of flavonoids have been proven for a variety of diseases. In the last few years, it is demonstrated that the effects of flavonoids are mediated by inhibiting the NF-κB (Nuclear Factor-κB) pathway. In this review, we have summar…

Inorganic Chemistryneurodegenerative diseaseOrganic ChemistrycancerflavonoidGeneral MedicinePhysical and Theoretical ChemistryNF-κB (Nuclear Factor-κB)Molecular Biologycardiac diseasesSpectroscopyCatalysisComputer Science ApplicationsInternational Journal of Molecular Sciences
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A Combination of Celecoxib and Glucosamine Sulfate Has Anti-Inflammatory and Chondroprotective Effects: Results from an In Vitro Study on Human Osteo…

2021

This study investigated the possible anti-inflammatory and chondroprotective effects of a combination of celecoxib and prescription-grade glucosamine sulfate (GS) in human osteoarthritic (OA) chondrocytes and their possible mechanism of action. Chondrocytes were treated with celecoxib (1.85 µM) and GS (9 µM), alone or in combination with IL-1β (10 ng/mL) and a specific nuclear factor (NF)-κB inhibitor (BAY-11-7082, 1 µM). Gene expression and release of some pro-inflammatory mediators, metalloproteinases (MMPs), and type II collagen (Col2a1) were evaluated by qRT-PCR and ELISA

Interleukin-1betachondrocytesAnti-Inflammatory AgentsApoptosisPharmacologymedicine.disease_causeNF-κBchemistry.chemical_compoundchondroprotectionoxidative stressSulfonesBiology (General)SpectroscopyCells CulturedGlucosamineglucosamine sulfatebiologycelecoxibChemistrySuperoxideNF-kappa BGeneral MedicineComputer Science ApplicationsChemistrychondrocyteosteoarthritiDrug Therapy Combinationmedicine.symptomInflammation Mediatorsmedicine.drugSignal TransductionNF-BQH301-705.5Cell SurvivalGlucosamine SulfateCatalysisArticleInorganic ChemistrySuperoxide dismutaseNitrilesOsteoarthritismedicineHumansPhysical and Theoretical ChemistryQD1-999Molecular Biologyoxidative streOrganic ChemistryMechanism of actionApoptosisinflammationbiology.proteinCelecoxibCyclooxygenaseOxidative stressInternational Journal of Molecular Sciences
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NF-κB and STAT3 Inhibition as a Therapeutic Strategy in Psoriasis: In Vitro and In Vivo Effects of BTH

2013

Benzo[b]thiophen-2-yl-3-bromo-5-hydroxy-5H-furan-2-one (BTH) is a simple and interesting synthetic derivative of petrosaspongiolide M, a natural compound isolated from a sea sponge with demonstrated potent anti-inflammatory activity through inhibition of the NF-κB signaling pathway. In the present study, we report the in vitro and in vivo pharmacological effect of BTH on some parameters related to the innate and adaptive response in the pathogenesis of psoriasis. BTH inhibited the release of some of the key psoriatic cytokines such as tumor necrosis factor α, IL-8, IL-6, and CCL27 through the downregulation of NF-κB in normal human keratinocytes. Moreover, it impaired signal transducers and…

KeratinocytesMaleSTAT3 Transcription FactorForeskinPrimary Cell CultureDermatitisInflammationDermatologyPharmacologyBiochemistryMicechemistry.chemical_compoundIn vivoPsoriasisThiadiazolesmedicineAnimalsHumansPsoriasisSTAT3Molecular BiologyCell ProliferationMice Inbred BALB CbiologyNF-kappa BNF-κBCell Biologymedicine.diseaseDisease Models Animalmedicine.anatomical_structurechemistrybiology.proteinCytokinesFemaleCCL27Signal transductionmedicine.symptomKeratinocyteJournal of Investigative Dermatology
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Potential antipsoriatic effect of chondroitin sulfate through inhibition of NF-κB and STAT3 in human keratinocytes

2012

Abstract Chondroitin sulfate (CS) is a natural glycosaminoglycan, formed by the 1–3 linkage of d -glucuronic acid to N-acetylgalactosamine, present in the extracellular matrix. It is used as a slow acting disease modifying agent in the treatment of osteoarthritis, and part of its beneficial effects are due to its antiinflammatory properties that result from an inhibitory effect on NF-κB signaling pathway. This ability raises the hypothesis that CS might be effective in other chronic inflammatory processes such as psoriasis, in which a deregulation of NF-κB is a key feature. In addition, psoriasis is characterized by an upregulation of STAT3 signaling pathway that is related to the epidermal…

KeratinocytesSTAT3 Transcription FactorBlotting WesternPrimary Cell CultureAnti-Inflammatory AgentsDermoscopyElectrophoretic Mobility Shift AssayPharmacologyStat3 Signaling Pathwaychemistry.chemical_compoundDownregulation and upregulationPsoriasismedicineHumansPsoriasisChondroitin sulfateCells CulturedPharmacologyChemistryChondroitin SulfatesNF-kappa BNF-κBmedicine.diseaseMicroscopy FluorescenceImmunologyPhosphorylationTumor necrosis factor alphaSignal transductionProtein BindingPharmacological Research
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Avarol inhibits TNF-a generation and NF-kB activation in human cells and in animal models

2007

Avarol is a marine sesquiterpenoid hydroquinone with interesting pharmacological properties including anti-inflammatory and antipsoriatic effects. In the present study we evaluated the pharmacological effect of avarol on some inflammatory parameters related to the pathogenesis of psoriasis. Avarol inhibited tumor necrosis factor-alpha (TNF-alpha) generation in stimulated human monocytes (IC(50) 1 microM) and TNF-alpha-induced activation of nuclear factor-kappaB (NF-kappaB)-DNA binding in keratinocytes. In the mouse air pouch model, administration of avarol produced a dose-dependent reduction of TNF-alpha generation (ED(50) 9.2 nmol/pouch) as well as of interleukin (IL)-1beta, prostaglandin …

Keratinocytesmedicine.medical_specialtymedicine.medical_treatmentAnti-Inflammatory AgentsAntineoplastic AgentsBiologyPharmacologyMonocytesGeneral Biochemistry Genetics and Molecular BiologyCell LineMicechemistry.chemical_compoundDownregulation and upregulationIn vivoInternal medicinemedicineAnimalsHumansPsoriasisGeneral Pharmacology Toxicology and PharmaceuticsPeroxidaseInflammationHyperplasiaDose-Response Relationship DrugTumor Necrosis Factor-alphaNF-kappa B p50 SubunitInterleukinNF-κBGeneral MedicineDisease Models AnimalEndocrinologymedicine.anatomical_structureEicosanoidchemistryTetradecanoylphorbol AcetateFemaleTumor necrosis factor alphaEpidermisInflammation MediatorsKeratinocyteSesquiterpenesProstaglandin E
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β - glikānu pretiekaisuma efektu pārbaude in vitro

2020

β – glikāni ir dabiski polisaharīdi un ir atrodami baktēriju un sēnīšu šūnu sieniņās. Tie ir sastopami dažādos avotos, kā, piemērām, sēnēs, baktērijās un graudaugos. Tieši dažādo avotu strukturālās atšķirības ir saistītas ar dažādām farmakoloģiskām īpašībām un bioloģiskām aktivitātēm. Visi β – glikāni ir glikozes polimēri, kas savienoti ar 1 → 3 lineāru β – glikozīdisku ķēdi, taču sānu ķēdes saites katram β – glikānam ir atsķirīgas un specifiskas. Uzskata, ka uzturā bieži lietotajiem un labi zināmiem β – glikāniem, kas ir iegūti no rauga un auzām, piemīt imūnsistēmu stiprinoša darbība un tie aktivizē imūnsistēmas šūnas organismā. Darba mērķis bija noskaidrot vai dabīgajiem rauga un auzu β –…

LPSTHP-1 monocītu šūnu līnijaβ – glikāniFarmācijapretiekaisuma efektsNF-κB
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Essential oil of Cyphostemma juttae (Vitaceae): Chemical composition and antitumor mechanism in triple negative breast cancer cells

2019

The genus Cyphostemma (Planch.) Alston (Vitaceae) includes about 150 species distrib- uted in eastern and southern Africa and Madagascar. Some species are used in traditional medicine and their biological activities, including antiproliferative effects against cancer cell lines, have been demonstrated. To date no investigations on Cyphostemma essential oils have been carried out. Essential oils, which play important roles in plant defenses have been demonstrated to be active in the treatment of several human diseases and to enhance bioavability of other drugs. The aim of this paper was to identify the chemical composition of the essential oil of the leaves of Cyphostemma juttae (Dinter &amp…

LeavesChemical CompositionTriple Negative Breast NeoplasmsPlant ScienceBiochemistryNF-κBAntioxidantsMass Spectrometrylaw.inventionAnalytical ChemistryTerpenechemistry.chemical_compound0302 clinical medicineSpectrum Analysis Techniquespro-oxidantlawBreast TumorsPlant defense against herbivoryMedicine and Health Sciencesantitumor0303 health sciencesMultidisciplinarybiologyTraditional medicineOrganic CompoundsPlant AnatomyQChromatographic TechniquesCell CycleRNF-kappa BLipidsChemistryOncologyVitaceaeCell Processes030220 oncology & carcinogenesisCyphostemmaPhysical SciencesMedicinecytotoxic effectterpenoidResearch ArticleCell SurvivalScienceVitaceaeResearch and Analysis Methodsessential oilGas Chromatography-Mass SpectrometryCell Growthphytol03 medical and health sciencesPhytolCyphostemma juttaeCell Line TumorBreast CancerOils VolatileHumansEssential oil030304 developmental biologyCell ProliferationCell growthTerpenesOrganic ChemistryChemical CompoundsBiology and Life SciencesCancers and NeoplasmsCell Biologybiology.organism_classificationAntineoplastic Agents PhytogenicPlant LeaveschemistrySettore BIO/03 - Botanica Ambientale E ApplicataSettore BIO/14 - FarmacologiaReactive Oxygen SpeciesOilsPLoS ONE
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Beta-MSH inhibits brain inflammation via MC(3)/(4) receptors and impaired NF-kappaB signaling.

2005

The anti-inflammatory effects of melanocortin peptides have been demonstrated in different inflammation models. This is the first report describing the molecular mechanisms for the beta-MSH-induced suppression of bacterial lipopolisaccharide (LPS)-caused brain inflammation. We found that beta-MSH suppresses LPS-induced nuclear translocation of the transcription factor NF-kappaB, and inhibits the expression of inducible nitric oxide synthase, and the following nitric oxide overproduction in the brain, in vivo. Moreover, administering the preferentially MC(4) receptor selective antagonist HS014 blocked completely these effects, suggesting a tentative MC(4) receptor mediated mechanism of actio…

LipopolysaccharidesMalemedicine.medical_specialtyImmunologyNitric Oxide Synthase Type IIInflammationElectrophoretic Mobility Shift AssayNitric OxidePeptides CyclicNitric oxidechemistry.chemical_compoundMiceInternal medicinebeta-MSHmedicineImmunology and AllergyAnimalsDrug InteractionsReceptorBrain ChemistryMice Inbred ICRbiologyDose-Response Relationship DrugImmunochemistryElectron Spin Resonance SpectroscopyNF-kappa BNF-κBHormonesCell biologyNitric oxide synthaseDisease Models AnimalEndocrinologyNeurologyMechanism of actionchemistrybiology.proteinEncephalitisReceptor Melanocortin Type 4Neurology (clinical)medicine.symptomMelanocortinSignal transductionhormones hormone substitutes and hormone antagonistsReceptor Melanocortin Type 3Signal TransductionJournal of neuroimmunology
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Phenolic Extract from Sonchus oleraceus L. Protects Diabetes-related Liver Injury in Rats through TLR4/NF-κB Signaling Pathway

2019

Sonchus oleraceus Linn. is of great interest in scientific platform because it exhibits significant biological activities. This study investigated the liver protective effects and mechanism of phenolic extract from S. oleraceus (SOL) on Streptozotocin (STZ)-induced diabetic rats. SOL significantly increased both superoxide dismutase activity and glutathione level, while causing a reduction of malondialdehyde level in the liver. Moreover, SOL ameliorates STZ-induced liver function and pathological damages. Diabetic rats fed with S. oleraceus daily for 6 weeks showed significantly decreased levels of tumor necrosis factor alpha and interleukin-1β in the liver. Sonchus oleraceus decreased the …

Liver injurybiologylcsh:TP368-456ChemistryAnti inflammationPharmacologybiology.organism_classificationmedicine.diseaseSonchus oleraceusin vivoSonchus oleraceuslcsh:Food processing and manufactureIn vivolcsh:RA1190-1270Diabetes mellitusAnti-inflammationTlr4 nf κbmedicineSignal transductionNF-κB signaling pathwayliver injurylcsh:Toxicology. PoisonseFood
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