Search results for "NITRIC OXIDE SYNTHASE"

showing 10 items of 531 documents

Brain BDNF levels are dependent on cerebrovascular endothelium-derived nitric oxide

2016

International audience; Scientific evidence continues to demonstrate a link between endothelial function and cognition. Besides, several studies have identified a complex interplay between nitric oxide (NO) and brain-derived neurotrophic factor (BDNF), a neurotrophin largely involved in cognition. Therefore, this study investigated the link between cerebral endothelium-derived NO and BDNF signaling. For this purpose, levels of BDNF and the phosphorylated form of endothelial NO synthase at serine 1177 (p-eNOS) were simultaneously measured in the cortex and hippocampus of rats subjected to either bilateral common carotid occlusion (n=6), physical exercise (n=6) or a combination of both (n=6) …

Male0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIIEndotheliumHippocampusPhysical exerciseTropomyosin receptor kinase BHippocampusNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemphysical exercisenitric oxideNeurotrophic factorsPhysical Conditioning AnimalInternal medicinemedicineAnimalsReceptor trkBRats WistarCerebral CortexBrain-derived neurotrophic factorbiologyChemistry[SCCO.NEUR]Cognitive science/NeuroscienceGeneral Neurosciencebrain-derived neurotrophic factorTrkB[ SDV.MHEP.CSC ] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemRatsCerebrovascular Disorders030104 developmental biologyEndocrinologymedicine.anatomical_structurecarotid arteries occlusionnervous system[ SCCO.NEUR ] Cognitive science/Neurosciencebiology.proteinEndothelium VascularNeuroscience030217 neurology & neurosurgeryNeurotrophinEuropean Journal of Neuroscience
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Retinal arteriole reactivity in mice lacking the endothelial nitric oxide synthase (eNOS) gene

2018

Dysfunctional vascular endothelial nitric oxide synthase (eNOS) has been proposed to play a main pathophysiological role in various ocular diseases. The aim of the present study was to test the hypothesis that the chronic lack of eNOS impairs endothelium-dependent vasodilation in retinal arterioles. The relevance of eNOS for mediating vascular responses was studied in retinal vascular preparations from eNOS-deficient mice (eNOS-/-) and wild-type controls in vitro. Changes in luminal diameter in response to vasoactive agents were measured by videomicroscopy. The thromboxane mimetic, U46619, induced similar concentration-dependent constriction of retinal arterioles in eNOS-/- and wild-type mi…

Male0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIIEndotheliumRetinal ArteryThromboxaneVasodilationMice03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineRetinal DiseasesEnosInternal medicinemedicineAnimalsEndothelial dysfunctionbiologyChemistrybiology.organism_classificationmedicine.diseaseSensory SystemsMice Inbred C57BLVasodilationNitric oxide synthaseArteriolesDisease Models AnimalOphthalmology030104 developmental biologyEndocrinologymedicine.anatomical_structureGene Expression Regulation030221 ophthalmology & optometrybiology.proteinRNACholinergicEndothelium VascularAcetylcholinemedicine.drugExperimental Eye Research
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Uncoupling of Endothelial Nitric Oxide Synthase in Perivascular Adipose Tissue of Diet-Induced Obese Mice

2015

Objective— The present study was conducted to investigate the contribution of perivascular adipose tissue (PVAT) to vascular dysfunction in a mouse model of diet-induced obesity. Approach and Results— Obesity was induced in male C57BL/6J mice with a high-fat diet for 20 weeks, and vascular function was studied with myograph. In PVAT-free aortas isolated from obese mice, the endothelium-dependent, nitric oxide–mediated vasodilator response to acetylcholine remained normal. In contrast, a clear reduction in the vasodilator response to acetylcholine was observed in aortas from obese mice when PVAT was left in place. Adipocytes in PVAT were clearly positive in endothelial nitric oxide synthase…

Male0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIIVasodilator AgentsAdipose tissueAorta ThoracicVasodilation030204 cardiovascular system & hematologyArginineDiet High-FatNitric OxideNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAdipokinesSuperoxidesEnosInternal medicineParacrine CommunicationAdipocytesmedicineAnimalsObesityEnzyme InhibitorsPhosphorylationAdiposityArginaseDose-Response Relationship DrugbiologyNitric Oxide Synthase Type IIIbiology.organism_classificationMice Inbred C57BLVasodilationArginaseDisease Models Animal030104 developmental biologyEndocrinologyAdipose TissuechemistryCytokinesInflammation MediatorsCardiology and Cardiovascular MedicineDiet-induced obeseSignal TransductionMyographArteriosclerosis, Thrombosis, and Vascular Biology
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Lamotrigine differently modulates 7-Nitroindazole and L-Arginine influence on Rat Maximal Dentate Gyrus Activation

2007

The effects induced on the maximal dentate gyrus activation (MDA) by administering the anticonvulsant lamotrigine (LTG), the selective inhibitor of neuronal nitric oxide synthase 7-nitroindazole (7-NI) and the precursor of nitric oxide (NO) synthesis L-arginine, alone or in combination, were studied in urethane anaesthetized rats. Either 7-NI or LTG alone administration reduced the number of convulsing animals following angular bundle (AB) stimulation; their combined treatment induced a further increase of the anticonvulsant effect as also demonstrated by the decrease of MDA and afterdischarge (AD) durations in the animals still responding to AB stimulation. On the contrary, the injection o…

Male7-NitroindazoleIndazolesArgininemedicine.medical_treatmentStimulationPharmacologyLamotrigineArginineLamotrigineNitric OxideSettore BIO/09 - FisiologiaNitric oxidechemistry.chemical_compoundSeizuresmedicineAnimalsEnzyme InhibitorsRats WistarBiological PsychiatryTriazinesDentate gyrusElectric StimulationRatsPsychiatry and Mental healthAnticonvulsantNeurologychemistryDentate GyrusAnticonvulsantsNeurology (clinical)Neuronal Nitric Oxide Synthasemedicine.drugLAMOTRIGINE NITRIC OXIDE EPILEPSY CONTROL
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In the rat maximal dentate activation model of partial complex epilepsy, the anticonvulsant activity of levetiracetam is modulated by nitric oxide-ac…

2009

The effects of nitric oxide-active drugs on the anticonvulsant action of the antiepileptic drug levetiracetam in an experimental model of partial complex seizures named maximal dentate gyrus activation were studied in rats. Levetiracetam was given alone or in combination with 7-nitroindazole, a preferential inhibitor of neuronal nitric oxide synthase, or with L: -arginine, the precursor of nitric oxide synthesis. The maximal dentate activation parameters were the time of latency and the durations of maximal dentate activation and afterdischarge responses. The administration of levetiracetam showed an anticonvulsant effect that was increased when given in combination with 7-nitroindazole. Th…

Male7-NitroindazoleIndazolesLevetiracetamMaximal dentate activation - Nitric oxide - Levetiracetam - Modulation - 7-Nitroindazolemedicine.medical_treatmentNitric Oxide Synthase Type IPharmacologyArginineNitric OxideSettore BIO/09 - FisiologiaNitric oxideEpilepsychemistry.chemical_compoundEpilepsy Complex PartialmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats WistarMaximal dentate activation Nitric oxide Levetiracetam Modulation 7-NitroindazoleBiological PsychiatryDose-Response Relationship DrugChemistryDentate gyrusPiracetammedicine.diseaseEffective dose (pharmacology)PiracetamRatsPsychiatry and Mental healthDisease Models AnimalDrug CombinationsAnticonvulsantNeurologyDentate GyrusAnticonvulsantsNeurology (clinical)Levetiracetammedicine.drugJournal of neural transmission (Vienna, Austria : 1996)
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7-Nitroindazole blocks conditioned place preference but not hyperactivity induced by morphine.

2003

The effects of 7-nitroindazole (7-NI), a neural nitric oxide synthase (nNOS) inhibitor, on spontaneous locomotor activity, morphine-induced hyperactivity, acquisition of place conditioning and morphine-induced conditioned place preference (CPP) were evaluated in male mice. In experiment 1, animals treated with 7-NI (25, 50 and 100 mg/kg), morphine (40 mg/kg) or morphine (40 mg/kg) plus 7-NI (25, 50 or 100 mg/kg) were placed in an actimeter for 3 h. In experiment 2, animals treated with the same drugs and doses were conditioned following an unbiased procedure. 7-NI did not affect the spontaneous locomotor activity or hyperactivity induced by morphine. However, the moderate and high doses of …

Male7-NitroindazoleIndazolesRatónMale miceNitric Oxide Synthase Type IPharmacologyHyperkinesisMotor ActivityNitric oxideDevelopmental psychologyBehavioral Neurosciencechemistry.chemical_compoundMiceRewardmedicineAnimalsEnzyme InhibitorsbiologyDose-Response Relationship DrugMorphineConditioned place preferenceNitric oxide synthaseAnalgesics OpioidchemistryMorphinebiology.proteinConditioningConditioning OperantNitric Oxide Synthasemedicine.drugBehavioural brain research
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Nitric oxide modulates striatal neuronal activity via soluble guanylyl cyclase: an in vivo microiontophoretic study in rats.

2003

It is now well established that nitric oxide (NO) acts as a neuromodulator in the central nervous system. To assess the role of NO in modulating striatal activity, single-unit recording was combined with iontophoresis to study presumed spiny projection neurons in urethane-anesthetized male rats. Striatal neurons recorded were essentially quiescent and were therefore activated to fire by the iontophoretic administration of glutamate, pulsed in cycles of 30 sec on and 40 sec off. In this study, iontophoresis of 3-morpholinosydnonimine hydrochloride (SIN 1), a nitric oxide donor, produced reproducible, current-dependent inhibition of glutamate-induced excitation in 12 of 15 striatal neurons, r…

MaleAction PotentialsReceptors Cytoplasmic and NuclearPharmacologyMedium spiny neuronNitric OxideNitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundSoluble Guanylyl CyclasePremovement neuronal activityAnimalsRats WistarCyclic guanosine monophosphateNeuronsbiologyIontophoresisGlutamate receptorIontophoresisCorpus StriatumRatsNitric oxide synthasenervous systemchemistryBiochemistrySolubilityGuanylate CyclaseMolsidominebiology.proteinSoluble guanylyl cyclaseSynapse (New York, N.Y.)
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Inhibitory responses to exogenous adenosine in murine proximal and distal colon”

2006

The aims of the present study were firstly, to characterize pharmacologically the subtypes of P(1) purinoreceptors involved in the inhibitory effects induced by exogenous adenosine in longitudinal smooth muscle of mouse colon, and secondly, to examine differences in the function and distribution of these receptors between proximal and distal colon. Adenosine (100 microM-3 mM) caused a concentration-dependent reduction of the amplitude of spontaneous contractions in the proximal colon, and muscular relaxation in the distal colon. In the proximal colon, adenosine effects were antagonized by a selective A(1) receptor antagonist, 1,3-dipropyl-8-cyclopentylxanthine (DPCPX, 10 nM), but were not m…

MaleAdenosineNitric Oxide Synthase Type IIIColonmouse colonadenosine A2B receptorNitric OxideSettore BIO/09 - FisiologiaMiceP1 purinoreceptorAnimalsadenosine A3 receptorEnzyme InhibitorsDose-Response Relationship Drugadenosine A1 receptorReceptors Purinergic P1Muscle SmoothTriazolesnitrergic nervesMice Inbred C57BLNG-Nitroarginine Methyl Esteradenosine A2 receptorPurinergic P1 Receptor AntagonistsXanthinesPapersQuinazolinesTheobrominemechanical activityMuscle ContractionSignal Transduction
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Glutathione Peroxidase-1 Deficiency Potentiates Dysregulatory Modifications of Endothelial Nitric Oxide Synthase and Vascular Dysfunction in Aging

2014

Recently, we demonstrated that gene ablation of mitochondrial manganese superoxide dismutase and aldehyde dehydrogenase-2 markedly contributed to age-related vascular dysfunction and mitochondrial oxidative stress. The present study has sought to investigate the extent of vascular dysfunction and oxidant formation in glutathione peroxidase-1–deficient ( GPx-1 −/− ) mice during the aging process with special emphasis on dysregulation (uncoupling) of the endothelial NO synthase. GPx-1 −/− mice on a C57 black 6 (C57BL/6) background at 2, 6, and 12 months of age were used. Vascular function was significantly impaired in 12-month-old GPx-1 −/− -mice as compared with age-matched controls. Oxidan…

MaleAgingmedicine.medical_specialtyGPX1Nitric Oxide Synthase Type IIIBiologymedicine.disease_causeMicechemistry.chemical_compoundGlutathione Peroxidase GPX1Internal medicineLeukocytesInternal MedicinemedicineAnimalsHumansPhosphorylationEndothelial dysfunctionProtein kinase ACells CulturedAgedMice Knockoutchemistry.chemical_classificationGlutathione PeroxidaseGlutathione peroxidaseEndothelial CellsNitric Oxide Synthase Type IIIGlutathioneOxidantsmedicine.diseaseMice Inbred C57BLOxidative StressEndocrinologychemistryImmunologyPhosphorylationEndothelium VascularOxidative stressHypertension
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Altered tachykinergic influence on gastric mechanical activity in mdx mice

2006

Abstract This study investigated whether alterationsin gastric activity in dystrophic mdx mouse can beattributed to dysfunctions of tachykinins. Endolumi-nal pressure was recorded and the expression ofneuronal nitric oxide synthase (nNOS), NK1 and NK2neurokinin receptors was investigated by immunoh-istochemistry. SR48968, NK2 receptor antagonist, butnot SR140333, NK1 receptor antagonist, decreased thetone only in mdx gastric preparations. In the presenceof N x -nitro- L -arginine methyl ester ( L -NAME), inhib-itor of NOS, SR48968 reduced the tone also in normalstomach. [Sar 9 , Met(O 2 ) 11 ]-SP, agonist of NK1 recep-tors, caused tetrodotoxin-sensitive relaxations, antag-onized by SR140333…

MaleAgonistQuinuclidinesmedicine.medical_specialtymdx mouseManometryPhysiologymedicine.drug_classNitric Oxide Synthase Type ISettore BIO/09 - FisiologiaNitric oxideMicechemistry.chemical_compoundimmunohistochemistry mdx mouse nitric oxide stomach tachykininsOrgan Culture TechniquesNeurokinin-1 Receptor AntagonistsPiperidinesTachykininsInternal medicinemedicineAnimalsEnzyme InhibitorsReceptorbiologyEndocrine and Autonomic SystemsStomachStomachGastroenterologyAntagonistMuscle SmoothReceptors Neurokinin-2Receptors Neurokinin-1musculoskeletal systemImmunohistochemistryMuscular Dystrophy DuchenneNitric oxide synthaseDisease Models AnimalNG-Nitroarginine Methyl Estermedicine.anatomical_structureEndocrinologychemistryMuscle TonusBenzamidesMice Inbred mdxbiology.proteinNK1 receptor antagonistGastrointestinal MotilityMuscle Contraction
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