Search results for "NSC"

showing 10 items of 5810 documents

Molecular Pathways Mediating Immunosuppression in Response to Prolonged Intensive Physical Training, Low-Energy Availability, and Intensive Weight Lo…

2019

Exercise and exercise-induced weight loss have a beneficial effect on overall health, including positive effects on molecular pathways associated with immune function, especially in overweight individuals. The main aim of our study was to assess how energy deprivation (i.e., "semi-starvation") leading to substantial fat mass loss affects the immune system and immunosuppression in previously normal weight individuals. Thus, to address this hypothesis, we applied a high-throughput systems biology approach to better characterize potential key pathways associated with immune system modulation during intensive weight loss and subsequent weight regain. We examined 42 healthy female physique athle…

0301 basic medicinemedicine.medical_treatmentPhysiologyliikuntaOverweightSystemic inflammationLeukocyte Countphysical training0302 clinical medicineWeight lossLeukocytesImmunology and AllergyMedicineOXIDATIVE STRESSta315DIETARY RESTRICTIONSport and Fitness SciencesOriginal Research2. Zero hungerimmunosuppressionIdrottsvetenskapbioinformatiikkaImmunosuppressionbioinformaticslow energy availability3. Good healthimmuunivasteIMMUNE FUNCTIONOBESITYChemokine secretionFemalemedicine.symptomfyysinen aktiivisuusAdultlcsh:Immunologic diseases. AllergyImmunologyEXERCISEInflammationYoung Adult03 medical and health sciencesLEPTINImmune systemINFLAMMATIONImmune ToleranceHumansimmunosuppression ; low energy availability ; physical training ; bioinformatics ; weight lossCell Proliferationbusiness.industrylaihdutusCYTOKINESmedicine.diseaseObesityDietenergiansaanti030104 developmental biologyHEMATOPOIETIC STEMImmunoglobulin G3121 General medicine internal medicine and other clinical medicineCELLS3111 Biomedicineweight lossEnergy IntakeTranscriptomelcsh:RC581-607business030215 immunologyFrontiers in Immunology
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Interleukin-25-mediated resistance against intestinal trematodes does not depend on the generation of Th2 responses

2020

AbstractInterleukin-25 (IL-25) is recognized as the most relevant initiator of protective Th2 responses in intestinal helminth infections. It is well known that IL-25 induces resistance against several species of intestinal helminths, including the trematode Echinostoma caproni. E. caproni has been extensively used as an experimental model to study the factors determining the resistance to intestinal infections. Herein, we assessed the role of IL-25 in the generation of resistance in mice to E. caproni infections. ICR mice are permissive hosts for E. caproni in which chronic infections are developed in relation to the lack of IL-25 production in response to primary infection and the consequ…

0301 basic medicinemedicine.medical_treatmentResistanceDrug ResistanceHelminthiasisGene ExpressionEchinostoma caproniMiceTh20302 clinical medicineEchinostomaInterleukin 25Intestinal Diseases ParasiticSTAT6EchinostomiasisInterleukin-17PhenotypeIntestinesInfectious DiseasesCytokineCytokinesTrematodaSecondary infection030231 tropical medicineAntibodies HelminthTrematode InfectionsBiologyIntestinal helminthlcsh:Infectious and parasitic diseases03 medical and health sciencesTh2 CellsImmune systemMemory cellImmunitymedicineHelminthsAnimalslcsh:RC109-216RNA MessengerPermissiveResistance (ecology)ResearchImmunityDisease Models Animal030104 developmental biologyImmunoglobulin GImmunologySTAT proteinParasitologySTAT6 Transcription FactorInterleuquin-25
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Discovery of Natural Products as Novel and Potent FXR Antagonists by Virtual Screening

2018

Farnesoid X receptor (FXR) is a member of nuclear receptor family involved in multiple physiological processes through regulating specific target genes. The critical role of FXR as a transcriptional regulator makes it a promising target for diverse diseases, especially those related to metabolic disorders such as diabetes and cholestasis. However, the underlying activation mechanism of FXR is still a blur owing to the absence of proper FXR modulators. To identify potential FXR modulators, an in-house natural product database (NPD) containing over 4,000 compounds was screened by structure-based virtual screening strategy and subsequent hit-based similarity searching method. After the yeast t…

0301 basic medicinenatural product01 natural scienceslcsh:Chemistry03 medical and health scienceschemistry.chemical_compoundTranscriptional regulationGeneIC50Original ResearchVirtual screeningNatural productantagonistmolecular dockingsimilarity searchingGeneral Chemistryvirtual screening0104 chemical sciencesChemistry010404 medicinal & biomolecular chemistry030104 developmental biologyFXRlcsh:QD1-999Nuclear receptorBiochemistrychemistryFarnesoid X receptorGuggulsteroneFrontiers in Chemistry
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Transcranial Static Magnetic Field Stimulation over the Primary Motor Cortex Induces Plastic Changes in Cortical Nociceptive Processing.

2018

Transcranial static magnetic field stimulation (tSMS) is a novel and inexpensive, non-invasive brain stimulation (NIBS) technique. Here, we performed non-invasive modulation of intra-epidermal electrical stimulation-evoked potentials (IES-EPs) by applying tSMS or sham stimulation over the primary motor (M1) and somatosensory (S1) cortices in 18 healthy volunteers for 15 min. We recorded EPs after IES before, right after, and 10 min after tSMS. The IES-EP amplitude was significantly reduced immediately after tSMS over M1, whereas tSMS over S1 and sham stimulation did not affect the IES-EP amplitude. Thus, tSMS may affect cortical nociceptive processing. Although the results of intervention f…

0301 basic medicinenon-invasive brain stimulationStandard of carenociceptive processingStimulationSomatosensory systemlcsh:RC321-57103 medical and health sciencesBehavioral Neuroscience0302 clinical medicineintra-epidermal electrical stimulationMedicinepainlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryBiological PsychiatryOriginal Researchbusiness.industryChronic painHealthy subjectsmedicine.diseaseNociceptive processingPsychiatry and Mental health030104 developmental biologyNeuropsychology and Physiological PsychologyNeurologyBrain stimulationtranscranial static magnetic field stimulationPrimary motor cortexbusinessNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in human neuroscience
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Non-invasive Brain Stimulation in Pediatric Migraine: A Perspective From Evidence in Adult Migraine

2019

Pediatric migraine remains still a challenge for the headache specialists as concerns both diagnostic and therapeutic aspects. The less ability of children to describe the exact features of their migraines and the lack of reliable biomarker for migraine contribute to complicate the diagnostic process. Therefore, there's need for new effective tools for supporting diagnostic and therapeutic approach in children with migraine. Recently, promising results have been obtained in adult headache by means of application of neurostimulation techniques both for investigating pathophysiological mechanisms and also for therapeutical applications. Non-invasive brain stimulation (NIBS) techniques like tr…

0301 basic medicinenon-invasive brain stimulationmedicine.medical_specialtyTMS tDCS migraine pediatric populationMini Reviewmedicine.medical_treatmentSettore BIO/09 - Fisiologialcsh:RC346-42903 medical and health sciencesTherapeutic approach0302 clinical medicinetranscranial magnetic stimulationtherapeuticsMedicineIntensive care medicineNeurostimulationlcsh:Neurology. Diseases of the nervous systemTranscranial direct-current stimulationbusiness.industrypediatric migrainemedicine.diseaseSettore MED/39 - Neuropsichiatria InfantileBiomarker (cell)Transcranial magnetic stimulation030104 developmental biologyNeurologyMigraineBrain stimulationSettore MED/26 - NeurologiaNeurology (clinical)transcranial direct current stimulationHeadachesmedicine.symptombusiness030217 neurology & neurosurgeryFrontiers in Neurology
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Genomic and non-genomic mechanisms of action of thyroid hormones and their catabolite 3,5-diiodo-l-thyronine in Mammals

2020

Since the realization that the cellular homologs of a gene found in the retrovirus that contributes to erythroblastosis in birds (v-erbA), i.e. the proto-oncogene c-erbA encodes the nuclear receptors for thyroid hormones (THs), most of the interest for THs focalized on their ability to control gene transcription. It was found, indeed, that, by regulating gene expression in many tissues, these hormones could mediate critical events both in development and in adult organisms. Among their effects, much attention was given to their ability to increase energy expenditure, and they were early proposed as anti-obesity drugs. However, their clinical use has been strongly challenged by the concomita…

0301 basic medicinenonalcoholic fatty liver diseaseobesityDiiodothyroninesEndogenyReviewthyroid hormone metabolism and transportMitochondrionmedicine.disease_causeProto-Oncogene Maslcsh:Chemistry0302 clinical medicineTranscription (biology)Settore BIO/10 - BiochimicaGene expressionSettore BIO/06 - Anatomia Comparata E CitologiaSettore MED/49 - Scienze Tecniche Dietetiche Applicatelcsh:QH301-705.5SpectroscopyMammalsReceptors Thyroid Hormonehepatic steatosisthyroid hormone mechanisms of actionGeneral Medicineresistance to thyroid hormones (RTH)Computer Science ApplicationsCell biology35-diiodo-L-thyronineThyroid Hormones030209 endocrinology & metabolismBiologyIodide PeroxidaseCatalysisInorganic Chemistry03 medical and health sciencesmedicineAnimalsHumansPhysical and Theoretical ChemistryMolecular BiologyGeneOrganic ChemistryBiological TransportLipid Metabolismhepatic steatosi030104 developmental biologyNuclear receptorlcsh:Biology (General)lcsh:QD1-999MutationBasal MetabolismLipid PeroxidationOxidative stressHormone
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Microbiota-Mitochondria Inter-Talk: A Potential Therapeutic Strategy in Obesity and Type 2 Diabetes

2020

The rising prevalence of obesity and type 2 diabetes (T2D) is a growing concern worldwide. New discoveries in the field of metagenomics and clinical research have revealed that the gut microbiota plays a key role in these metabolic disorders. The mechanisms regulating microbiota composition are multifactorial and include resistance to stress, presence of pathogens, diet, cultural habits and general health conditions. Recent evidence has shed light on the influence of microbiota quality and diversity on mitochondrial functions. Of note, the gut microbiota has been shown to regulate crucial transcription factors, coactivators, as well as enzymes implicated in mitochondrial biogenesis and meta…

0301 basic medicineobesitymitochondrial oxidative/nitrosative stressPhysiologyClinical BiochemistryInflammationReviewType 2 diabetesMitochondrionGut floraBioinformaticsdigestive systemBiochemistryProinflammatory cytokine03 medical and health sciences0302 clinical medicinemedicineMolecular BiologyTranscription factorgut microbiotabiologylcsh:RM1-950InflammasomeCell Biologybiology.organism_classificationmedicine.diseasemitochondrialcsh:Therapeutics. Pharmacology030104 developmental biologyMitochondrial biogenesisinflammation030220 oncology & carcinogenesistype 2 diabetesmedicine.symptommedicine.drugAntioxidants
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Role of p27Kip1 as a transcriptional regulator

2018

The protein p27Kip1 is a member of the Cip/Kip family of cyclin-dependent kinase (Cdk) inhibitors. It interacts with both the catalytic and the regulatory subunit (cyclin) and introduces a region into the catalytic cleave of the Cdk inducing its inactivation. Its inhibitory capacity can be modulated by specific tyrosine phosphorylations. p27Kip1 also behaves as a transcriptional regulator. It associates with specific chromatin domains through different transcription factors. ChIP on chip, ChIP-seq and expression microarray analysis allowed the identification of the transcriptional programs regulated by p27Kip1. Thus, important cellular functions as cell division cycle, respiration, RNA proc…

0301 basic medicinep27Kip1Review03 medical and health sciencesTranscriptional regulationCyclin-dependent kinaseTranscription (biology)Gene expressionTranscriptional regulationcancertranscriptional regulationNeurodegenerationCàncerTranscription factorE2F4CancerbiologyChemistryMalalties neurodegenerativesneurodegenerationNeurodegenerative DiseasesChIP-on-chipExpressió gènicaCell biologyChromatin030104 developmental biologyOncologybiology.proteinGene expressionOncotarget
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p27Kip1 regulates alpha-synuclein expression

2018

Alpha-synuclein (α-SYN) is the main component of anomalous protein aggregates (Lewy bodies) that play a crucial role in several neurodegenerative diseases (synucleinopathies) like Parkinson’s disease and multiple system atrophy. However, the mechanisms involved in its transcriptional regulation are poorly understood. We investigated here the role of the cyclin-dependent kinase (Cdk) inhibitor and transcriptional regulator p27Kip1 (p27) in the regulation of α-SYN expression. We observed that selective deletion of p27 by CRISPR/Cas9 technology in neural cells resulted in increased levels of α-SYN. Knock-down of the member of the same family p21Cip1 (p21) also led to increased α-SYN levels, in…

0301 basic medicinep27Kip1[SDV]Life Sciences [q-bio]03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCyclin-dependent kinaseTranscriptional regulationalpha synucleinAlpha synucleinPsychological repressionE2F4Alpha-synucleinSynucleinopathiesbiologyPromoterEnzyme inhibitorsMolecular biologyExpressió gènica3. Good healthnervous system diseases030104 developmental biologyOncologychemistryInhibidors enzimàticsnervous systemE2F4biology.proteinGene expressionTranscription Factor E2F4transcriptionp21Cip1Transcription030217 neurology & neurosurgeryResearch Paper
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The MID1 protein is a central player during development and in disease.

2015

Loss-of-function mutations in the MID1 gene cause a rare monogenic disorder, Opitz BBB/G syndrome (OS), which is characterized by malformations of the ventral midline. The MID1 gene encodes the MID1 protein, which assembles a large microtubule-associated protein complex. Intensive research over the past several years has shed light on the function of the MID1 protein as a ubiquitin ligase and regulator of mTOR signalling and translational activator. As a central player in the cell MID1 has been implicated in the pathogenesis of various other disorders in addition to OS including cancer and neurodegenerative diseases. Influencing the activity of the MID1 protein complex is a promising new st…

0301 basic medicinephysiopathology [Huntington Disease]CarcinogenesisUbiquitin-Protein LigasesRegulatorDiseaseBiologyBioinformaticsmedicine.disease_causephysiopathology [Alzheimer Disease]Congenital AbnormalitiesPathogenesis03 medical and health sciencesMiceAlzheimer Diseasephysiology [Nuclear Proteins]medicineAnimalsHumansgenetics [Microtubule Proteins]ddc:610GenePI3K/AKT/mTOR pathwayActivator (genetics)Nuclear Proteinsgenetics [Nuclear Proteins]genetics [Transcription Factors]physiology [Transcription Factors]Ubiquitin ligase030104 developmental biologyHuntington DiseaseMutationbiology.proteinMicrotubule Proteinsphysiology [Microtubule Proteins]CarcinogenesisMid1 protein humanTranscription FactorsFrontiers in bioscience (Landmark edition)
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