Search results for "Nephritis"

showing 10 items of 101 documents

Acute human pyelonephritis: Leukocytic infiltration of tubules and localization of bacteria

1988

The fine structural details of how leukocytes appear in the lumen of tubules and the localization of bacteria in the tubulo-interstitial space were studied by light and electronmicroscopy in renal cortical biopsy specimens from three patients with acute pyelonephritis. The cells of interstitial infiltrates infiltrated and sometimes disrupted the cortical collecting tubules preferentially, while inflammatory infiltration of the proximal and distal convoluted tubules occurred more rarely. Since the emigration of tubular wall-localized individual leukocytes into the lumen was not observed even in long series of thin sections, focal inflammatory disruption of the uriniferous ducts was considere…

Pathologymedicine.medical_specialtyKidney CortexNeutrophilsLumen (anatomy)BiologyPathology and Forensic MedicineBiopsyLeukocytesmedicineHumansInflammatory infiltrationReceptorMolecular BiologyBacteriaPyelonephritismedicine.diagnostic_testMacrophagesCell BiologyGeneral MedicineInterstitial infiltratesmedicine.diseasebiology.organism_classificationMicroscopy ElectronKidney TubulesAcute DiseaseInfiltration (medical)BacteriaVirchows Archiv A Pathological Anatomy and Histopathology
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Alport's syndrome: specificity and pathogenesis of glomerular basement membrane alterations.

1987

In Alport's syndrome (AS) thinning and splitting of the glomerular basement membrane (GBM) are assumed to be characteristic ultrastructural alterations. Both lesions are, however, non-specific because they can occur in other glomerulopathies. In addition, splitting may be found in non-glomerular structures. It should be emphasized that the characteristic lesion in AS is a result of the widespread combination of thin and split GBM in the same biopsy specimen. In our opinion the basic lesion is the thin GBM, which is characterized by a lamina densa (measuring 50-150 nm in thickness) which may begin to split as a result of focal detachment of podocyte pedicles (spacing) and repeated subepithel…

Pathologymedicine.medical_specialtyKidney GlomerulusNephritis HereditaryBiologyurologic and male genital diseasesBasement MembranePodocyteLesionPathogenesisBiopsymedicineHumansmedicine.diagnostic_testurogenital systemGlomerular basement membraneAnatomymedicine.diseasefemale genital diseases and pregnancy complicationsmedicine.anatomical_structureNephrologyPediatrics Perinatology and Child HealthUltrastructureLamina densamedicine.symptomNephritisPediatric nephrology (Berlin, Germany)
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Interleukin 6 (IL-6) deficiency delays lupus nephritis in MRL-Faslpr mice: the IL-6 pathway as a new therapeutic target in treatment of autoimmune ki…

2009

Objective.To investigate the pathophysiological effect of interleukin 6 (IL-6) on lupus nephritis in MRL-Faslprmice.Methods.We generated IL-6-deficient MRL-Faslprmice using a backcross/intercross breeding scheme. Renal pathology was evaluated using immunohistochemistry detection for macrophages, lymphocytes, vascular cell adhesion molecule-1 (VCAM-1), and TUNEL (terminal deoxynucleotide transferase-mediated dUTP nick end-labeling) for apoptotic cells, and renal IgG and C3 deposition by immunofluorescence staining. Expression of inflammatory markers in the spleen was analyzed by quantitative real-time reverse transcription-polymerase chain reaction. Serum cytokine concentrations were detecte…

Pathologymedicine.medical_specialtyMice Inbred MRL lprImmunologyLupus nephritisVascular Cell Adhesion Molecule-1SpleenKidneyInterferon-gammaMiceLife ExpectancyRheumatologyImmunology and AllergyMedicineAnimalsHumansLupus Erythematosus Systemicfas ReceptorInterleukin 6InflammationMice KnockoutKidneyMice Inbred BALB CLupus erythematosusbiologybusiness.industryInterleukin-6Kidney metabolismmedicine.diseaseLupus NephritisInterleukin-10Proteinuriamedicine.anatomical_structureRenal pathologyImmunologybiology.proteinFemalebusinessBiomarkersSpleenKidney diseaseSignal TransductionThe Journal of rheumatology
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Metalloprotease meprin beta in rat kidney: glomerular localization and differential expression in glomerulonephritis

2008

Meprin (EC 3.4.24.18) is an oligomeric metalloendopeptidase found in microvillar membranes of kidney proximal tubular epithelial cells. Here, we present the first report on the expression of meprin beta in rat glomerular epithelial cells and suggest a potential involvement in experimental glomerular disease. We detected meprin beta in glomeruli of immunostained rat kidney sections on the protein level and by quantitative RT-PCR of laser-capture microdissected glomeruli on the mRNA level. Using immuno-gold staining we identified the membrane of podocyte foot processes as the main site of meprin beta expression. The glomerular meprin beta expression pattern was altered in anti-Thy 1.1 and pas…

Pathologymedicine.medical_specialtyNephrology/Acute Renal Failure10039 Institute of Medical GeneticsKidney GlomerulusFluorescent Antibody Techniquelcsh:MedicinePodocyte foot610 Medicine & health1100 General Agricultural and Biological SciencesBiologyurologic and male genital diseasesHeymann NephritisGlomerulonephritisWestern blot1300 General Biochemistry Genetics and Molecular BiologymedicineAnimalsRNA MessengerMicroscopy Immunoelectronlcsh:ScienceKidneyMetalloproteinase1000 MultidisciplinaryMultidisciplinarymedicine.diagnostic_testPodocytesReverse Transcriptase Polymerase Chain Reactionurogenital systemImmune Seralcsh:RNephrology/Chronic Kidney DiseaseMetalloendopeptidasesGlomerulonephritismedicine.diseaseMolecular biologyRats Inbred F344Ratsmedicine.anatomical_structureRats Inbred Lew570 Life sciences; biologylcsh:QNephritisImmunostainingResearch Article
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Acanthocyturia—A characteristic marker for glomerular bleeding

1991

Acanthocyturia—A characteristic marker for glomerular bleeding. Erythrocyte morphology by phase contrast microscopic examination (PCM) of the urine is widely employed in distinguishing glomerular from nonglomerular bleeding. The proposed percentages of dysmorphic red cells are significant for glomerular bleeding in the range of 10 to 80% in the literature, because there is no clear cut definition of “dysmor-phism.” In the present study midstream urine samples of 351 patients with hematuria (> 8 erythrocytes/μl) and of 33 healthy controls were examined. The various dysmorphic red cells were analyzed by PCM according to a detailed hematological classification. Most of the dysmorphic red cells…

Pathologymedicine.medical_specialtyRenal glomerulusKidney GlomerulusEchinocyteAcanthocytesHemorrhageUrineurologic and male genital diseasesBiopsymedicineHumansUrine cytologyKidneymedicine.diagnostic_testRed Cellurogenital systembusiness.industryOsmolar ConcentrationGlomerulonephritisHydrogen-Ion Concentrationmedicine.diseasefemale genital diseases and pregnancy complicationsProteinuriamedicine.anatomical_structureNephrologyKidney DiseasesAcanthocytebusinessBiomarkersKidney International
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Endocytotic activity in epitheloid and Langhans’ giant cells

1984

In the experimental tubulo-interstitial (anti-basement membrane) nephritis in the rat, electron microscopic studies after the in vivo microinjection of native ferritin in areas of granulomatous inflammation near the surface of the kidney indicate that epitheloid and multinucleate Langhans’ giant cells are capable of endocytosis and particularly of micropinocytosis. This suggests the possibility that endocytotic activities as well as secretion phenomena are important in the immune defense mechanisms linked with these “specifically” developed cells.

Pathologymedicine.medical_specialtybiologyLanghans giant cellmedicine.diseaseEndocytosisFerritinMultinucleateGiant cellbiology.proteinmedicineSecretionMicropinocytosisNephritisVirchows Archiv B Cell Pathology Including Molecular Pathology
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New developments in IL-6 dependent biology and therapy: where do we stand and what are the options?

2005

Interleukin-6 (IL-6) is a four-helical protein which, on target cells, binds to a specific IL-6-receptor and two molecules of the promiscuous signal transducing protein gp130. Structure-function analysis defined three molecular contact sites between IL-6 and its receptor subunits. Using this information, competitive antagonistic proteins as well as hyperagonistic proteins were developed. Possible therapeutic applications of IL-6 antagonists are in IL-6 dependent haematological disorders (Castleman's disease, POEMS syndrome, multiple myeloma) and bone diseases (Paget's disease, osteoporosis). Designer IL-6 antagonists could suppress inflammatory activity in rheumatic and autoimmune diseases …

PharmacologySystemic lupus erythematosusbiologybusiness.industryUnstable anginamedicine.medical_treatmentGlomerulonephritisGeneral Medicinemedicine.diseaseGlycoprotein 130CytokineImmunologymedicinebiology.proteinPharmacology (medical)businessInterleukin 6ReceptorMultiple myelomaExpert opinion on investigational drugs
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UN ERRORE DIAGNOSTICO IN CHIRURGIA PEDIATRICA: La pielonefrite grantogranulomatosa ed il Tumore di Wilms

1993

The Authors report a case of Xanthogranulomatous Pyelonephritis characterized by a torpid course and clinical presentation of renal mass.

Settore MED/20 - Chirurgia Pediatrica E Infantilerenal masses Xanthogranulomatous Pyelonephritis Wilms' tumor
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Segmented relationships to model erosion of regression effect in Cox regression

2010

In this article we propose a parsimonious parameterisation to model the so-called erosion of the covariate effect in the Cox model, namely a covariate effect approaching to zero as the follow-up time increases. The proposed parameterisation is based on the segmented relationship where proper constraints are set to accomodate for the erosion. Relevant hypothesis testing is discussed. The approach is illustrated on two historical datasets in the survival analysis literature, and some simulation studies are presented to show how the proposed framework leads to a test for a global effect with good power as compared with alternative procedures. Finally, possible generalisations are also present…

Statistics and ProbabilitybreakpointEpidemiologyProportional hazards modelLiver Cirrhosis BiliaryErosion (morphology)Lupus NephritisSet (abstract data type)Segmented regressionHealth Information ManagementNonlinear DynamicsRegression toward the meanCox modelCovariateStatisticsEconometricsHumansComputer SimulationSettore SECS-S/05 - Statistica SocialeSettore SECS-S/01 - Statisticaerosion of effectStatistical hypothesis testingMathematicsFollow-Up StudiesProportional Hazards Models
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Autoreactivity to mouse C1q in a murine model of SLE.

1995

A large proportion of systemic lupus erythematosus (SLE) patients develop glomerulonephritis, coincident with the appearance of autoantibodies to C1q, the Fc-recognizing collagen-like subcomponent of the first component of complement, C1. The MRL/lpr/lpr mouse is an established model for SLE, developing both antinuclear and anti-type II collagen autoantibodies, and rheumatoid factors(s), exhibiting reduced complement levels and later on developing glomerulonephritis and often arthritis. We report here an age-dependent decrease in serum C1q levels coincident with the development of IgG2b autoantibodies reactive with mouse C1q in MRL/lpr/lpr mice. Unlike IgG2b, although high levels of IgM, Ig…

Systemic diseaseImmunologyArthritischemical and pharmacologic phenomenaEnzyme-Linked Immunosorbent Assayurologic and male genital diseasesmedicine.disease_causeAutoimmunityMiceRheumatologyimmune system diseasesImmunology and AllergyMedicineAnimalsLupus Erythematosus Systemicskin and connective tissue diseasesAutoantibodiesLupus erythematosusbusiness.industryComplement C1qAutoantibodyGlomerulonephritismedicine.diseaseConnective tissue diseaseLupus NephritisDisease Models AnimalImmunologybusinessAnti-SSA/Ro autoantibodiesRheumatology international
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