Search results for "Neural"

showing 10 items of 2783 documents

Vascular‐derived TGF‐β increases in the stem cell niche and perturbs neurogenesis during aging and following irradiation in the adult mouse brain

2012

Neurogenesis decreases during aging and following cranial radiotherapy, causing a progressive cognitive decline that is currently untreatable. However, functional neural stem cells remained present in the subventricular zone of high dose-irradiated and aged mouse brains. We therefore investigated whether alterations in the neurogenic niches are perhaps responsible for the neurogenesis decline. This hypothesis was supported by the absence of proliferation of neural stem cells that were engrafted into the vascular niches of irradiated host brains. Moreover, we observed a marked increase in TGF-β1 production by endothelial cells in the stem cell niche in both middle-aged and irradiated mice. I…

MaleAgingNeurogenesisStem cell theory of agingSubventricular zoneBiologyMice03 medical and health sciences0302 clinical medicineNeural Stem CellsTransforming Growth Factor betamedicineAnimalsHumansTGF-betaStem Cell NicheProgenitor cellResearch ArticlesCell Proliferation030304 developmental biology0303 health sciencesirradiationNeurogenesisBrainEndothelial CellsNeural stem cellCell biologyMice Inbred C57BLEndothelial stem cellNeuroepithelial cellmedicine.anatomical_structureImmunologyMolecular Medicine030217 neurology & neurosurgerySignal TransductionAdult stem cellEMBO Molecular Medicine
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Cognitive reserve impacts on inter-individual variability in resting-state cerebral metabolism in normal aging

2012

There is a great deal of heterogeneity in the impact of aging on cognition and cerebral functioning. One potential factor contributing to individual differences among the elderly is the cognitive reserve, which designates the partial protection from the deleterious effects of aging that lifetime experience provides. Neuroimaging studies examining task-related activation in elderly people suggested that cognitive reserve takes the form of more efficient use of brain networks and/or greater ability to recruit alternative networks to compensate for age-related cerebral changes. In this exploratory multi-center study, we examined the relationships between cognitive reserve, as measured by educa…

MaleAgingRestCognitive NeuroscienceIntraparietal sulcusCognitive ReserveNeuroimagingFluorodeoxyglucose F18Task-positive networkImage Interpretation Computer-AssistedNeural PathwaysHumansAttentionDefault mode networkAgedCognitive reserveAged 80 and overResting state fMRIBrainCognitionMiddle AgedVerbal reasoningNeurologyPositron-Emission TomographyFemaleRadiopharmaceuticalsPsychologyCognitive psychologyNeuroImage
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Color-coded diffusion-tensor-imaging of posterior cingulate fiber tracts in mild cognitive impairment.

2005

Abstract Different processes like microvascular dysfunction, free radical toxicity, β-amyloid deposits, and Wallerian degeneration can cause functionally relevant disturbances of cerebral neuronal networks by myelin degeneration. Color-coded diffusion-tensor-imaging (ccDTI) allows the structural identification and quantification of myelinated fiber tracts. Particularly, posterior cingulate fiber tracts, which are regarded as important neuronal substrates of the network representing memory processing can be localized only imprecisely by conventional magnetic resonance imaging techniques. The posterior cingulate bundles were assessed by ccDTI in 17 patients with amnestic mild cognitive impair…

MaleAgingWallerian degenerationPathologymedicine.medical_specialtyNeuropsychological TestsGyrus CinguliHippocampusNerve Fibers MyelinatedWhite matterAlzheimer DiseasePredictive Value of Testsmental disordersFractional anisotropyNeural PathwaysmedicineImage Processing Computer-AssistedDementiaHumansAgedMemory Disordersmedicine.diagnostic_testGeneral NeuroscienceMagnetic resonance imagingMiddle Agedmedicine.diseasemedicine.anatomical_structureDiffusion Magnetic Resonance Imagingnervous systemPosterior cingulateAnisotropyFemaleNeurology (clinical)Geriatrics and GerontologyAlzheimer's diseasePsychologyCognition DisordersNeuroscienceDevelopmental BiologyDiffusion MRINeurobiology of aging
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Interaction between Angiotensin Type 1, Type 2, and Mas Receptors to Regulate Adult Neurogenesis in the Brain Ventricular–Subventricular Zone

2019

The renin&ndash

MaleAgingproliferationNeurogenesisProliferationSubventricular zoneventricular–subventricular zoneBiologyModels BiologicalReceptor Angiotensin Type 2ArticleReceptor Angiotensin Type 1MiceNeuroblastNeural Stem CellsLateral VentriclesmedicineneurospheresAT1 receptorsAnimalsReceptorNeural stem cellsMice KnockoutAngiotensin II receptor type 1Cell growthAngiotensin IINeurogenesisagingAge FactorsGeneral MedicineImmunohistochemistryNeural stem cellOlfactory bulbCell biologyRatsVentricular–subventricular zonemedicine.anatomical_structurenervous systemAT2 receptorscardiovascular systemNeurosphereshormones hormone substitutes and hormone antagonistscirculatory and respiratory physiologyProtein BindingCells
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Acute blockade of CB1 receptor leads to reinstatement of MDMA-induced conditioned place preference

2011

Cannabis is one of the drugs most commonly consumed in combination with ecstasy (3,4-methylenedioxymethamphetamine, MDMA). Although numerous studies have attempted to further our understanding of the role of the cannabinoid system in drug abuse, few have focused on how it influences the rewarding effects of MDMA. The aim of the present study was to evaluate the role of the CB1 cannabinoid receptor in vulnerability to reinstatement of a MDMA-induced conditioned place preference (CPP). Mice were first conditioned with 5mg/kg of MDMA. Once the preference had been extinguished, a priming dose of MDMA, alone or plus the CB1 cannabinoid agonist WIN 55,212-2 (0.1 and 0.5mg/kg) or the CB1 cannabino…

MaleAgonistCannabinoid receptormedicine.drug_classMorpholinesN-Methyl-34-methylenedioxyamphetaminemedicine.medical_treatmentDrug-Seeking BehaviorClinical BiochemistryEcstasyNaphthalenesPharmacologyToxicologyBiochemistryMiceBehavioral NeuroscienceReceptor Cannabinoid CB1Conditioning Psychologicalmental disordersmedicineAnimalsBiological PsychiatryPharmacologyDose-Response Relationship DrugCannabinoidsbusiness.industrymusculoskeletal neural and ocular physiologyAntagonistMDMAEndocannabinoid systemConditioned place preferenceBenzoxazinesnervous systemlipids (amino acids peptides and proteins)Cannabinoidbusinesspsychological phenomena and processesmedicine.drugPharmacology Biochemistry and Behavior
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Selective activation of 5-HT(2C) receptors stimulates GABA-ergic function in the rat substantia nigra pars reticulata: a combined in vivo electrophys…

2007

In vivo electrophysiology and microdialysis were used to investigate the physiological role of 5-HT(2C) receptors in the control of substantia nigra pars reticulata (SNr) function. Extracellular single-unit recordings were performed from putative GABA-containing neurons in the SNr of anesthetized rats, and local GABA release was studied by in vivo microdialysis in the SNr of awake freely-moving rats. Systemic administration of the selective 5-HT(2C) receptor agonist (S)-2-(chloro-5-fluoro-indol-1-yl)-1-methylethylamine 1:1 C(4)H(4)O(4) (RO 60-0175) caused a dose-dependent excitation of about 30% of the SNr neurons recorded. However, the remaining neurons were either inhibited or unaffected …

MaleAgonistSerotoninMicrodialysismedicine.drug_classMicrodialysisAction PotentialsBiologyPharmacologyInhibitory postsynaptic potentialSynaptic TransmissionRats Sprague-Dawleychemistry.chemical_compoundReceptor Serotonin 5-HT2CmedicineAnimalsDrug InteractionsNeurotransmittergamma-Aminobutyric Acid5-HT receptorNeuronsDose-Response Relationship DrugGeneral NeuroscienceExcitatory Postsynaptic PotentialsExtracellular FluidNeural InhibitionReceptor antagonistRatsSerotonin Receptor AgonistsUp-RegulationElectrophysiologySubstantia Nigranervous systemchemistrySB-243213Serotonin 5-HT2 Receptor AntagonistsSystemic administrationSerotonin AntagonistsNeuroscienceSerotonin 5-HT2 Receptor Agonists
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Neurotransmitters involved in the fast inhibitory junction potentials in mouse distal colon

2003

We investigated, in murine colon circular muscle, the role of adenosine 5′-triphosphate (ATP) and pituitary adenylate cyclase activating peptide (PACAP) as inhibitory neurotransmitters of the fast component of nerve-evoked inhibitory junction potential (fast IJP). Fast IJP was antagonised by apamin or suramin, abolished by desensitisation with the P2Y receptor agonist, adenosine 5′-O-2-thiodiphosphate (ADPβS), unaffected by desensitisation with P2X receptor agonist, α,β-methylene ATP (α,β-meATP), and reduced by PACAP-(6-38), a PACAP receptor antagonist. ATP induced membrane hyperpolarization resistant to tetrodotoxin, Nω-nitro-L-arginine methyl ester (L-NAME) or PACAP-(6-38), but antagonise…

MaleAgonistendocrine systemmedicine.medical_specialtyP2Y receptorColonmedicine.drug_classPurinoceptorNeuromuscular JunctionSuraminTetrodotoxinBiologyApaminSettore BIO/09 - FisiologiaMembrane PotentialsCellular and Molecular NeuroscienceMicechemistry.chemical_compoundAdenosine TriphosphateInternal medicinemedicineAnimalsMurinePharmacologyNeurotransmitter AgentsDose-Response Relationship Drugmusculoskeletal neural and ocular physiologyNeuropeptidesMembrane hyperpolarizationThionucleotidesHyperpolarization (biology)Receptor antagonistAdenosinePeptide FragmentsATPAdenosine DiphosphatePituitary adenylate cyclase-activating peptideNG-Nitroarginine Methyl EsterEndocrinologyApaminchemistryPituitary Adenylate Cyclase-Activating PolypeptideFast inhibitory junction potentialPACAP (pituitary adenylate cyclase activating peptide)hormones hormone substitutes and hormone antagonistsmedicine.drugEuropean Journal of Pharmacology
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Obese Rats Exhibit High Levels of Fat Necrosis and Isoprostanes in Taurocholate-Induced Acute Pancreatitis

2012

BACKGROUND: Obesity is a prognostic factor for severity in acute pancreatitis in humans. Our aim was to assess the role of oxidative stress and abdominal fat in the increased severity of acute pancreatitis in obese rats. METHODOLOGY: Taurocholate-induced acute pancreatitis was performed in lean and obese Zucker rats. Levels of reduced glutathione, oxidized glutathione, L-cysteine, cystine, and S-adenosylmethionine were measured in pancreas as well as the activities of serine/threonine protein phosphatases PP1 and PP2A and tyrosin phosphatases. Isoprostane, malondialdehyde, triglyceride, and free fatty acid levels and lipase activity were measured in plasma and ascites. Lipase activity was m…

MaleAnatomy and PhysiologyNecrosislcsh:MedicineAdipose tissueIsoprostanesmedicine.disease_causeBiochemistrychemistry.chemical_compoundMalondialdehydeMolecular Cell Biologylcsh:ScienceMultidisciplinaryPancreatitis Acute Necrotizingmusculoskeletal neural and ocular physiologyAnimal ModelsMalondialdehydeGlutathioneLipidsEnzymesBlood ChemistryMedicineAcute pancreatitismedicine.symptomResearch ArticleTaurocholic AcidCell Physiologymedicine.medical_specialtyBlotting WesternImmunologyGastroenterology and Hepatologymacromolecular substancesModel OrganismsInternal medicineChemical BiologymedicineAnimalsFat necrosisObesityPancreasBiologyTriglyceridesbusiness.industrylcsh:Rmedicine.diseaseObesityRatsRats ZuckerOxidative StressMetabolismEndocrinologyPancreatitisnervous systemchemistrySmall MoleculesRatPancreatitislcsh:QbusinessOxidative stressPLoS ONE
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Specific Hippocampal Interneurons Shape Consolidation of Recognition Memory

2020

Summary A complex array of inhibitory interneurons tightly controls hippocampal activity, but how such diversity specifically affects memory processes is not well understood. We find that a small subclass of type 1 cannabinoid receptor (CB1R)-expressing hippocampal interneurons determines episodic-like memory consolidation by linking dopamine D1 receptor (D1R) signaling to GABAergic transmission. Mice lacking CB1Rs in D1-positive cells (D1-CB1-KO) display impairment in long-term, but not short-term, novel object recognition memory (NOR). Re-expression of CB1Rs in hippocampal D1R-positive cells rescues this NOR deficit. Learning induces an enhancement of in vivo hippocampal long-term potenti…

MaleAnimals CB1 receptor D1 receptor Dopamine Endocannabinoid system GABA Hippocampus Interneurons Long-term potentiation Male Memory Mice Novel object recognition Recognition PsychologyCB1 receptorCB1 cannabinoid receptorsD(1) receptorhippocampus[SDV]Life Sciences [q-bio]D1 receptorneuronsCB(1) receptorArticleCA1informationMiceGABAMemoryAnimalsendocannabinoid systemlong-term potentiationinterneuronsmusculoskeletal neural and ocular physiologyRecognition Psychologyepisodic memoryinhibition[SDV] Life Sciences [q-bio]modulationnervous systemdopamineLTPnovel object recognition memory
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Anophthalmia-Waardenburg syndrome with expanding phenotype: does neural crest play a role?

2007

We describe a child with bilateral anophthalmia, limb anomalies, skin lesions, cerebral malformations, epilepsy, and mental retardation. This patient, according to eponymous classification, should fit into the Anophthalmia-Waardenburg syndrome, although he also presents cutaneous and cerebral manifestations never reported in this syndrome until now. These clinical findings could be explained by the new classification of brain malformations, which takes into account the role of neural crest in Waardenburg syndrome.

MaleAnophthalmia Waardenburg syndromeElectroencephalography03 medical and health sciencesEpilepsy0302 clinical medicine030225 pediatricsAnophthalmosmedicineHumansWaardenburg SyndromeChildAnophthalmiamedicine.diagnostic_testWaardenburg syndromeNeural crestAnophthalmosElectroencephalographyAnatomymedicine.diseasePhenotypePhenotypeNeural CrestPediatrics Perinatology and Child HealthNeurology (clinical)Psychology030217 neurology & neurosurgeryJournal of child neurology
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