Search results for "Neurite"

showing 8 items of 48 documents

CRMP-4 expression in the adult cerebral cortex and other telencephalic areas of the lizard Podarcis hispanica.

2002

The control of neuritogenesis is crucial for the development, maturation and regeneration of the nervous system. The collapsin response-mediated protein 4 (CRMP-4) is a member of a family of proteins that are involved in neuronal differentiation and axonal outgrowth. In rodents, this protein is expressed in recently generated neurons such as some granule neurons of the dentate gyrus, as well as in certain differentiated neurons undergoing neurite outgrowth or synaptogenesis during adulthood. Since CRMP-4 protein appears to be highly conserved throughout the evolutionary scale, we have used immunocytochemistry to study its distribution in the lizard cerebral cortex. We have found pronounced …

TelencephalonNeuriteMedial cortexGrowth ConesSynaptogenesisNerve Tissue ProteinsPodarcis hispanicaEvolution MolecularDevelopmental NeurosciencemedicineAnimalsCerebral CortexbiologyDentate gyrusStem CellsNeurogenesisCell DifferentiationLizardsbiology.organism_classificationImmunohistochemistrymedicine.anatomical_structurenervous systemBromodeoxyuridineCerebral cortexDentate GyrusNeuroscienceNucleusCell DivisionDevelopmental BiologyBrain research. Developmental brain research
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Detection of Fibroblast Growth Factor Receptor 1 (FGFR1) Transactivation by Muscarinic Acetylcholine Receptors (mAChRs) in Primary Neuronal Hippocamp…

2018

In addition to their canonical intracellular signals involved in the regulation of neuronal plasticity, G-protein coupled receptors can also rapidly transactivate tyrosine kinase receptors and their downstream intracellular signaling in absence of specific ligands. Here we describe our protocol for dissociating and maintaining hippocampal primary neurons in high- and low-density culture, followed by a description of methods employed to evaluate neurite outgrowth and protein phosphorylation associated with fibroblast growth factor receptor 1 transactivation by muscarinic acetylcholine receptors. Our goal was to provide the reader with detailed protocols of the abovementioned techniques and t…

TransactivationChemistryFibroblast growth factor receptor 1Tyrosine kinase receptorHippocampal formationHippocampusSettore BIO/09 - FisiologiaFibroblast growth factor receptorWestern blottingCell biologyMuscarinic acetylcholine receptorPrimary neuronal cultureTransactivationNeurite growthMuscarinic acetylcholine receptorPhosphorylationReceptor–receptor interactions
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Nerve growth factor and epidermal growth factor stimulate clusterin gene expression in PC12 cells

1999

Clusterin (apolipoprotein J) is an extracellular glycoprotein that might exert functions in development, cell death and lipid transport. Clusterin gene expression is elevated at sites of tissue remodelling, such as differentiation and apoptosis; however, the signals responsible for this regulation have not been identified. We use here the clusterin gene as a model system to examine expression in PC12 cells under the control of differentiation and proliferation signals produced by nerve growth factor (NGF) and by epidermal growth factor (EGF) respectively. NGF induced clusterin mRNA, which preceded neurite outgrowth typical of neuronal differentiation. EGF also activated the clusterin mRNA, …

Transcriptional ActivationProgrammed cell deathNeuriteMolecular Sequence DataResponse ElementsTransfectionBinding CompetitivePC12 CellsBiochemistryEpidermal growth factorConsensus SequenceNeuritesAnimalsNerve Growth FactorsRNA MessengerCloning MolecularPromoter Regions GeneticMolecular BiologyGlycoproteinsSequence DeletionNeuronsRegulation of gene expressionMessenger RNABase SequenceEpidermal Growth FactorClusterinbiologyKinaseCell DifferentiationDNACell BiologyMolecular biologyeye diseasesRatsTranscription Factor AP-1ClusterinNerve growth factorbiology.proteinsense organsCell DivisionMolecular ChaperonesSignal TransductionResearch ArticleBiochemical Journal
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The ribose methylation enzyme FTSJ1 has a conserved role in neuron morphology and learning performance

2021

ABSTRACTFTSJ1 is a conserved human 2’-O-methyltransferase (Nm-MTase) that modifies several transfer RNAs (tRNAs) at position 32 and the wobble position 34 in the AntiCodon Loop (ACL). Its loss of function has been linked to Non-Syndromic X-Linked Intellectual Disability (NSXLID), and more recently to cancers. However, the molecular mechanisms underlying these pathologies are currently unclear. Here we report a novelFTSJ1pathogenic variant from a NSXLID patient. Using blood cells derived from this patient and other affected individuals carryingFTSJ1mutations, we performed an unbiased and comprehensive RiboMethSeq analysis to map the ribose methylation (Nm) on all human tRNAs and identify nov…

Transcriptomeeducation.field_of_studyNeuriteTransfer RNAmicroRNAPopulationMethylationBiologyeducationGeneLoss functionCell biology
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Clustering transmembrane-agrin induces filopodia-like processes on axons and dendrites

2005

The transmembrane form of agrin (TM-agrin) is primarily expressed in the CNS, particularly on neurites. To analyze its function, we clustered TM-agrin on neurons using anti-agrin antibodies. On axons from the chick CNS and PNS as well as on axons and dendrites from mouse hippocampal neurons anti-agrin antibodies induced the dose- and time-dependent formation of numerous filopodia-like processes. The processes appeared within minutes after antibody addition and contained a complex cytoskeleton. Formation of processes required calcium, could be inhibited by cytochalasine D, but was not influenced by staurosporine, heparin or pervanadate. Time-lapse video microscopy revealed that the processes…

animal structuresDendritic spineTime FactorsNeuriteCytochalasin BGrowth ConesVideo microscopyChick EmbryoBiologyNervous SystemAntibodiesCellular and Molecular NeuroscienceMicemedicineNeuritesAnimalsAgrinPseudopodiaGrowth coneCytoskeletonMolecular BiologyCells CulturedCytoskeletonAgrinMicroscopy VideoDose-Response Relationship DrugCell MembraneCell DifferentiationCell BiologyDendritesCell biologymedicine.anatomical_structurenervous systemAnimals NewbornNeuronFilopodia
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Fabry Disease With Concomitant Lewy Body Disease

2019

AbstractAlthough Gaucher disease can be accompanied by Lewy pathology (LP) and extrapyramidal symptoms, it is unknown if LP exists in Fabry disease (FD), another progressive multisystem lysosomal storage disorder. We aimed to elucidate the distribution patterns of FD-related inclusions and LP in the brain of a 58-year-old cognitively unimpaired male FD patient suffering from predominant hypokinesia. Immunohistochemistry (CD77, α-synuclein, collagen IV) and neuropathological staging were performed on 100-µm sections. Tissue from the enteric or peripheral nervous system was unavailable. As controls, a second cognitively unimpaired 50-year-old male FD patient without LP or motor symptoms and 3…

complications [Lewy Body Disease]MalePathologyAutopsyDisease0302 clinical medicineHypokinesiapathology [Brain]Lysosomal storage diseasespathology [Neurons]metabolism [alpha-Synuclein]metabolism [Fabry Disease]pathology [Astrocytes]Neuronsα-Synuclein0303 health sciencesParkinsonismTrihexosylceramidesBrainGeneral MedicineMiddle AgedParkinson diseasecomplications [Fabry Disease]Neurologymetabolism [Neurons]alpha-Synucleinmedicine.symptomLewy Body Diseasemedicine.medical_specialtymetabolism [Lewy Body Disease]Context (language use)Substantia nigrametabolism [Trihexosylceramides]Pathology and Forensic Medicineblood supply [Brain]03 medical and health sciencesCellular and Molecular Neuroscienceα-Galactosidase AmedicineHumansddc:610030304 developmental biologypathology [Lewy Bodies]Fabry diseasebusiness.industryPars compactapathology [Lewy Body Disease]Lewy bodies/neuritesOriginal Articlesmetabolism [Lewy Bodies]medicine.diseaseFabry diseasemetabolism [Brain]AstrocytesLewy BodiesNeurology (clinical)CD77pathology [Fabry Disease]business030217 neurology & neurosurgeryJournal of Neuropathology and Experimental Neurology
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Overexpression of nerve growth factor in peritoneal fluid from women with endometriosis may promote neurite outgrowth in endometriotic lesions

2011

To investigate the role of the nerve growth factor (NGF) in the endometriosis-associated innervation in the development of endometriosis- associated symptoms, 41 peritoneal fluid samples (PF) from patients with surgically and histologically proven endometriosis and 20 PF from patients with other gynecologic conditions were analyzed with Western blot and a novel in vitro model using dorsal root ganglia (DRG) to show neuronal outgrowth; endometrial cells also were analyzed. The results suggest that the PF of endometriosis patients and endometriotic lesions have neurotropic properties, because the Western blot analysis and the cell culture stainings showed NGF expression, and the neurite outgr…

medicine.medical_specialtyPathologySensory Receptor CellsNeuriteBlotting WesternEndometriosisEndometriosisFluorescent Antibody TechniqueNerve fiberNeuriteChick Embryosensory nerve fiberPelvic PainEndometriumSensory Receptor Cellsensory nerveEndometriumpain conductionDysmenorrheaWestern blotGanglia SpinalNerve Growth FactorNeuritesmedicineAnimalsAscitic FluidHumansEndometriosiCells Culturedpathophysiology of endometriosis-related painGynecologymedicine.diagnostic_testAnimalbusiness.industryPeritoneal fluidObstetrics and Gynecologymedicine.diseaseEndometriosis; pain conduction; sensory nervemedicine.anatomical_structureNerve growth factornervous systemReproductive MedicineFemalebusinessHumanSensory nerveFertility and Sterility
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Tiam1 as a Signaling Mediator of Nerve Growth Factor-Dependent Neurite Outgrowth

2010

Nerve Growth Factor (NGF)-induced neuronal differentiation requires the activation of members of the Rho family of small GTPases. However, the molecular mechanisms through which NGF regulates cytoskeletal changes and neurite outgrowth are not totally understood. In this work, we identify the Rac1-specific guanine exchange factor (GEF) Tiam1 as a novel mediator of NGF/TrkA-dependent neurite elongation. In particular, we report that knockdown of Tiam1 causes a significant reduction in Rac1 activity and neurite outgrowth induced by NGF. Physical interaction between Tiam1 and active Ras (Ras- GTP), but not tyrosine phosphorylation of Tiam1, plays a central role in Rac1 activation by NGF. In add…

rac1 GTP-Binding ProteinTiam1; Nerve growth factor (NGF)GTPaseTropomyosin receptor kinase ABiochemistryPC12 CellsCell Biology/Cell Signalingchemistry.chemical_compoundChlorocebus aethiopsNerve Growth FactorTiam1Guanine Nucleotide Exchange FactorsT-Lymphoma Invasion and Metastasis-inducing Protein 1NGFNeuronsMultidisciplinaryUNESCO::CIENCIAS DE LA VIDA::Biología molecularQOtras Medicina BásicaRCell Differentiation//purl.org/becyt/ford/3.1 [https]Cell biologyNeoplasm ProteinsMedicina BásicaNeuronal differentiationNerve growth factor (NGF)COS CellsMedicine//purl.org/becyt/ford/3 [https]Guanine nucleotide exchange factorSignal transductionResearch ArticleSignal TransductionCIENCIAS MÉDICAS Y DE LA SALUDNeuriteScienceCell Biology/Neuronal Signaling MechanismsRAC1Biology:CIENCIAS DE LA VIDA::Biología molecular [UNESCO]Neuroscience/Neuronal Signaling MechanismsNeuritesAnimalsHumansReceptor trkATyrosine phosphorylationMolecular biologyRatsNerve growth factorchemistrynervous systemras ProteinsRac1 GTPasePLoS ONE
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