Search results for "Neuroblast"

showing 10 items of 348 documents

How to minimise the effect of tumour cell content in detection of aberrant genetic markers in neuroblastoma

2011

Background: Clinical heterogeneity reflects the complexity of genetic events associated with neuroblastoma (NB). To identify the status of all described genetic loci with possible prognostic interest, high-throughput approaches have been used, but only with tumour cell content >60%. In some tumours, necrotic, haemorrhagic and/or calcification areas influence the low amount of neuroblasts. We evaluated the effect of tumour cell content in the detection of relevant aberrant genetic markers (AGM) diagnosed by fluorescence in situ hybridisation (FISH) on tissue microarrays (TMA) in NB. Methods: Two hundred and thirty-three MYCN non-amplified primary NB included in 12 TMAs were analysed. Results…

Genetic MarkersMaleCancer ResearchPathologymedicine.medical_specialtyShort CommunicationCellBiologyneuroblastomaFISHaberrant genetic markersNeuroblastomatumour cell contentGene duplicationmedicineHumansNuclear proteinneoplasmsIn Situ Hybridization FluorescenceNeoplasm StagingOncogene ProteinsN-Myc Proto-Oncogene Proteinmedicine.diagnostic_testGene AmplificationChromosome MappingInfantNuclear Proteinsprognostic factorsCancerPrognosismedicine.diseaseSurvival Ratemedicine.anatomical_structureOncologyTissue Array AnalysisGenetic markerFemaleNeoplasm stagingFluorescence in situ hybridizationBritish Journal of Cancer
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Analysis of biological prognostic factors using tissue microarrays in neuroblastic tumors

2009

Background Neuroblastic tumors (NT) are pediatric neoplasms with a heterogeneous genetic profile. They present genotypic alterations of prognostic value, the study of which is mandatory in designing therapeutic management. Tissue microarrays (TMA) from paraffin material allow the analysis of a large number of cases with minimal costs. The main purpose of the present study is to analyze specific genetic markers of neuroblastic tumors included in TMAs and determine their prognostic value. We compare the results obtained by different molecular techniques at different substrates to evaluate the feasibility of these assays. Procedure One hundred thirty-nine samples were included in four differen…

Genetic MarkersMalePathologymedicine.medical_specialtyDiseaseN-Myc Proto-Oncogene ProteinNeuroblastomaRisk FactorsGenotypeHumansMedicineStage (cooking)ChildRetrospective StudiesOncogene ProteinsN-Myc Proto-Oncogene ProteinTissue microarraybusiness.industryAge FactorsNuclear ProteinsCell DifferentiationRetrospective cohort studyHematologyPrognosisNeuroblastic TumorTreatment OutcomeOncologyTissue Array AnalysisGenetic markerMutationPediatrics Perinatology and Child HealthFemalebusinessPediatric Blood & Cancer
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Intragenic anaplastic lymphoma kinase (ALK) rearrangements: Translocations as a novel mechanism ofALKactivation in neuroblastoma tumors

2014

Anaplastic lymphoma kinase (ALK) has been demonstrated to be deregulated in sporadic as well as in familiar cases of neuroblastoma (NB). Whereas ALK-fusion proteins are common in lymphoma and lung cancer, there are few reports of ALK rearrangements in NB indicating that ALK mainly exerts its oncogenic capacity via activating mutations and/or overexpression in this tumor type. In this study, 332 NB tumors and 13 cell lines were screened by high resolution single nucleotide polymorphism microarray. Gain of 2p was detected in 23% (60/332) of primary tumors and 46% (6/13) of cell lines, while breakpoints at the ALK locus were detected in four primary tumors and two cell lines. These were furthe…

GeneticsCancer ResearchKinaseGene rearrangementAmpliconBiologymedicine.diseasemedicine.disease_causeLymphomaExonhemic and lymphatic diseasesNeuroblastomaGeneticsmedicineCancer researchAnaplastic lymphoma kinaseCarcinogenesisGenes, Chromosomes and Cancer
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Adult Neurogenesis in Reptiles

2011

Adult neurogenesis in reptiles is a well-documented phenomenon and exists in many telencephalic areas. The newly generated neurons originate along the walls of the lateral ventricles, mainly in the sulci. The putative neural progenitors are radial glial cells. These glial cells give rise to neuroblasts that migrate to their final destination. In general, the new neurons are born in the portion of the ventricular zone (VZ) adjacent to the telencephalic area where they will be recruited and migrate radially through the brain parenchyma along the processes of radial glial cells to their final destination, although migration to the olfactory bulbs (OB) is different. Specifically, it seems that …

Glial fibrillary acidic proteinNeurogenesisBiologyOlfactory bulbLateral ventriclesmedicine.anatomical_structurenervous systemNeuroblastParenchymamedicinebiology.proteinSomaNeuroscienceGliogenesis
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Migrating neuroblasts of the rostral migratory stream are putative targets for the action of nitric oxide

2007

It has been demonstrated that the gaseous messenger nitric oxide influences cell proliferation and cell migration, and therefore affects adult neurogenesis in mammals. Here, we investigated the putative targets for this action in the rostral migratory stream of the rat. We used immunocytochemical detection of the beta1 subunit of the enzyme soluble guanylyl cyclase, which can be activated by nitric oxide. Our results under light and electron microscopy demonstrated that the migrating neuroblasts (type A cells) were beta1-immunopositive. The astrocytes (type B cells), immature precursors (type C cells) and ependymal cells (type E cells) were beta1-immunonegative. The neurochemical characteri…

Glial fibrillary acidic proteinbiologyRostral migratory streamGeneral NeuroscienceNeurogenesisCell migrationCell biologyNitric oxidechemistry.chemical_compoundNeuroblastchemistryBiochemistrySecond messenger systembiology.proteinSoluble guanylyl cyclaseEuropean Journal of Neuroscience
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Immunohistochemical Study as a Tool in Differential Diagnosis of Pediatric Malignant Rhabdoid Tumor

2010

Malignant rhabdoid tumors (MRTs) are aggressive childhood neoplasms, occurring mainly in the kidney and brain. We describe 2 unusual cases of extrarenal and noncranial location (liver and soft tissue with dissemination) mimicking hepatoblastoma, neuroblastoma or Ewing sarcoma. Both cases revealed a polyphenotypic profile, combined with cytokeratin, vimentin, and CD99 expression. INI1/BAF-47 showed negative protein nuclear expression in both cases, suggesting a diagnosis of MRT. An extensive immunohistochemical panel was performed to exclude pediatric tumors reminiscent of MRT. The genetic studies failed to detected MYCN amplification, 11q23 deletion, and EWS break-apart positivity. No alter…

HepatoblastomaPathologymedicine.medical_specialtySkin NeoplasmsHistologyDesmoplastic small-round-cell tumorChromosomal Proteins Non-HistoneCD9912E7 AntigenN-Myc Proto-Oncogene ProteinPathology and Forensic MedicineDiagnosis DifferentialNeoplasms Multiple PrimaryFatal OutcomeAntigens CDNeuroblastomaAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansVimentinRhabdoid TumorChromosome AberrationsOncogene ProteinsN-Myc Proto-Oncogene Proteinbusiness.industryLiver NeoplasmsInfant NewbornInfantNuclear ProteinsWilms' tumorSMARCB1 Proteinmedicine.diseaseImmunohistochemistryDNA-Binding ProteinsMedical Laboratory TechnologyDrug Resistance NeoplasmKeratinsFemaleSarcomaRNA-Binding Protein EWSDifferential diagnosisbusinessCell Adhesion MoleculesTranscription FactorsApplied Immunohistochemistry & Molecular Morphology
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Analysis of Complete Neuroblast Cell Lineages in the Drosophila Embryonic Brain via DiI Labeling

2013

Proper functioning of the brain relies on an enormous diversity of neural cells generated by neural stem cell-like neuroblasts (NBs). Each of the about 100 NBs in each side of brain generates a nearly invariant and unique cell lineage, consisting of specific neural cell types that develop in defined time periods. In this chapter we describe a method that labels entire NB lineages in the embryonic brain. Clonal DiI labeling allows us to follow the development of an NB lineage starting from the neuroectodermal precursor cell up to the fully developed cell clone in the first larval instar brain. We also show how to ablate individual cells within an NB clone, which reveals information about the…

Lineage (genetic)Cell divisionNeuroblastPrecursor cellCell CloneBiologyClone (B-cell biology)Molecular biologyNeural cellNeural stem cellCell biology
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On the roles of Notch, Delta, kuzbanian, and inscuteable during the development of Drosophila embryonic neuroblast lineages

2009

AbstractThe generation of cellular diversity in the nervous system involves the mechanism of asymmetric cell division. Besides an array of molecules, including the Par protein cassette, a heterotrimeric G protein signalling complex, Inscuteable plays a major role in controlling asymmetric cell division, which ultimately leads to differential activation of the Notch signalling pathway and correct specification of the two daughter cells. In this context, Notch is required to be active in one sibling and inactive in the other. Here, we investigated the requirement of genes previously known to play key roles in sibling cell fate specification such as members of the Notch signalling pathway, e.g…

Lineage (genetic)Embryo NonmammalianNotchCell divisionCell fate specificationDisintegrinsNeurogenesisContext (language use)BiologyCell fate determinationPolymerase Chain Reaction03 medical and health sciences0302 clinical medicineNeuroblastAsymmetric cell divisionAnimalsDrosophila ProteinsCell LineageMolecular Biology030304 developmental biologyDNA PrimersGeneticsNeurons0303 health sciencesBase SequenceReceptors NotchNeurogenesisIntracellular Signaling Peptides and ProteinsMembrane ProteinsMetalloendopeptidasesCell BiologyEmbryonic stem cellImmunohistochemistryCytoskeletal ProteinsAsymmetric cell divisionDrosophilakuzbanian030217 neurology & neurosurgerySignal TransductionDevelopmental BiologyDevelopmental Biology
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Chemoprotective effect of carotenoids from Lycium barbarum L. on SH-SY5Y neuroblastoma cells treated with beauvericin

2020

Goji berry has recently been introduced in Mediterranean diet and its consumption is increasing. This study aims to determine cytoprotection of lutein (LUT), zeaxanthin (ZEAX) and goji berry extract (GBE) rich in carotenoids against Beauvericin (BEA)-induced cytotoxicity on SH-SY5Y neuroblastoma cells. Both carotenoids and GBE showed cytoprotective effects. Cytoprotection was evaluated by simultaneous combination of the two xanthophylls LUT and ZEAX with BEA, as well as using pre-treatment assays. The highest protective effect occurred in 16%, 24% and 12% respectively for LUT, ZEAX and LUT + ZEAX incubating simultaneously with BEA, while by pre-treatment assay LUT showed a cytoprotection ef…

LuteinToxicologyNeuroblastoma03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyfoodZeaxanthinsCell Line TumorDepsipeptidesHumansFood scienceCarotenoid030304 developmental biologychemistry.chemical_classification0303 health sciencesbiologyPlant ExtractsChemistryLuteinGoji berry04 agricultural and veterinary sciencesGeneral MedicineLyciumbiology.organism_classificationCarotenoids040401 food scienceCytoprotectionfood.foodBeauvericinZeaxanthinCytoprotectionXanthophyllLyciumFood ScienceFood and Chemical Toxicology
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Abstract A292: Salinomycin, an anti-cancer stem cell antibiotic, overcomes acquired resistance to BRAF inhibitors in BRAF-mutant human melanoma cell …

2013

Abstract Advanced malignant melanoma is one of the most lethal cancers, because it is highly metastatic and refractory to conventional chemotherapy. About 60% of melanomas harbor oncogenic BRAF mutations which aberrantly activate MEK/ERK signaling pathway. BRAF and MEK inhibitors have been shown efficacious in patients with BRAF-mutant melanoma, but there is not effective target therapy for BRAF wild type melanomas. Unfortunately acquired resistance to BRAF targeted therapies is a common event: 50% of treated patients progressed within 6 to 7 months after the initiation of treatment. Resistance is associated with reactivation of the MAPK pathway (through development of de novo NRAS, NF1 or …

MAPK/ERK pathwayNeuroblastoma RAS viral oncogene homologCancer ResearchMelanomaWnt signaling pathwayCancerBiologymedicine.diseaseOncologyCancer stem cellImmunologymedicineCancer researchVemurafenibneoplasmsPI3K/AKT/mTOR pathwaymedicine.drugMolecular Cancer Therapeutics
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