Search results for "Neuronal"

showing 10 items of 556 documents

Study of molecular mechanism involved in neuronal plasticity induced by magnetic stimulation in cultured hippocampal neurons

2014

Although a large number of investigations have shown that transcranial magnetic stimulation, a non-invasive method of brain stimulation with minimal side effects, is able to induce neuronal synaptic plastic change, very few studies have examined the molecular mechanisms of magnetic stimulation involved in synaptic plasticity. Since it is well known that neurotrophins and their receptors regulate synaptic strength and thereby mediate plasticity, in this study we have investigated the effects of low-frequency (1 Hz) magnetic stimulation, at different intensities, on the activation of neurotrophic factors receptors and relative intracellular pathways in primary cultures of hippocampal neurons.…

Magnetic stimulation neuronal plasticity neurotrophic factors
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Sex Differences in the Neuroadaptations of Reward-related Circuits in Response to Subchronic Variable Stress

2018

Women are twice as likely to be diagnosed with major depressive disorder. However, fewer studies in rodent models of depression have used female animals, leading to a relative lack of understanding of the female brain’s response to stress, especially at a neural circuit level. In this study, we utilized a 6-day subchronic variable stress (SCVS) mouse model and measured novelty suppressed feeding as behavioral criteria to evaluate susceptibility to SCVS in male and female mice. First, we showed that SCVS induced a decrease in latency to eat (susceptible phenotype) in female mice, but not in males (resilient phenotype). After determining behavioral phenotypes, we investigated the firing activ…

Male0301 basic medicineAction Potentialsneuronal activityTissue Culture Techniques0302 clinical medicinePremovement neuronal activitylocus coeruleuNeuronsSex CharacteristicsNeuronal Plasticitymusculoskeletal neural and ocular physiologyGeneral NeuroscienceBrainPhenotypeVentral tegmental areamedicine.anatomical_structureMajor depressive disorderFemaleDisease Susceptibilitylateral habenulamedicine.drugmedicine.medical_specialtyCell typesex differenceventral tegmental areaBiologyArticle03 medical and health sciencesRewardDopamineInternal medicinemedicineAnimalsAction PotentialDepressive DisorderAnimalNeuronmedicine.diseaseMice Inbred C57BLElectrophysiology030104 developmental biologyEndocrinologynervous systemSettore BIO/14 - FarmacologiaLocus coeruleusTissue Culture Techniquemajor depressionStress Psychological030217 neurology & neurosurgeryNeuroscience
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Functional Plasticity after Unilateral Vestibular Midbrain Infarction in Human Positron Emission Tomography.

2016

The aim of the study was to uncover mechanisms of central compensation of vestibular function at brainstem, cerebellar, and cortical levels in patients with acute unilateral midbrain infarctions presenting with an acute vestibular tone imbalance. Eight out of 17 patients with unilateral midbrain infarctions were selected on the basis of signs of a vestibular tone imbalance, e.g., graviceptive (tilts of perceived verticality) and oculomotor dysfunction (skew deviation, ocular torsion) in F18-fluordeoxyglucose (FDG)-PET at two time points: A) in the acute stage, and B) after recovery 6 months later. Lesion-behavior mapping analyses with MRI verified the exact structural lesion sites. Group su…

Male0301 basic medicineBrain Stem Infarctionslcsh:MedicinePathology and Laboratory MedicineMidbrainDiagnostic Radiology0302 clinical medicineThalamusMesencephalonCortex (anatomy)Medicine and Health SciencesMedicinelcsh:ScienceTomographyPostural BalanceVestibular systemNeuronal PlasticityMultidisciplinaryRadiology and ImagingBrainAnatomyFrontal eye fieldsMagnetic Resonance Imagingmedicine.anatomical_structureVestibular DiseasesInfarctionThalamic NucleiFemaleBrainstemAnatomyBrainstemResearch ArticleImaging TechniquesThalamusNeuroimagingResearch and Analysis Methods03 medical and health sciencesSigns and SymptomsDiagnostic MedicineOcular SystemHumansSkew deviationAgedbusiness.industrylcsh:RBiology and Life SciencesVestibular cortex030104 developmental biologyVisual cortexCase-Control StudiesPositron-Emission TomographyLesionsEyeslcsh:QbusinessHeadNeurosciencePositron Emission Tomography030217 neurology & neurosurgeryNeurosciencePLoS ONE
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Memory-enhancing and brain protein expression-stimulating effects of novel calcium antagonist in Alzheimer’s disease transgenic female mice

2016

The prevalence of Alzheimer's disease (AD) is higher in females than in males, and causes more severe cognitive, memory and behavioral impairments. Previously, in male transgenic (Tg) APPSweDI mice, we reported that the novel lipophilic 1,4-dihydropyridine (DHP) derivative AP-12 crossed the blood-brain barrier, blocked neuronal and vascular calcium channels, changed brain protein expression and improved behavior. In this study, we used female Tg APPSweDI mice to assess the effects of AP-12 on behavior, and brain protein expression, with a particular focus on those of the GABAergic system. The results showed that in female Tg mice, similar to male Tg mice, AP-12 improved spatial learning/mem…

Male0301 basic medicineCingulate cortexDihydropyridinesmedicine.medical_specialtyElevated plus mazeVesicular Inhibitory Amino Acid Transport ProteinsHippocampusMice TransgenicWater mazeBiologyHippocampal formationGyrus CinguliHippocampusArticleAmyloid beta-Protein PrecursorMice03 medical and health sciences0302 clinical medicineAlzheimer DiseaseMemoryInternal medicineNeuroplasticitymedicineAnimalsGABAergic NeuronsMaze LearningPharmacologyAmyloid beta-PeptidesNeuronal PlasticityGlutamate DecarboxylaseCalcium Channel BlockersUp-RegulationDisease Models Animal030104 developmental biologyEndocrinologyAnti-Anxiety AgentsBlood-Brain BarrierSynaptic plasticityGABAergicCalciumFemale030217 neurology & neurosurgeryPharmacological Research
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Effects of the Antidepressant Fluoxetine on the Somatostatin Interneurons in the Basolateral Amygdala

2018

Although the precise mechanism of action of antidepressant drugs remains elusive, the neuroplastic hypothesis has gained acceptance during the last two decades. Several studies have shown that treatment with antidepressants such as Fluoxetine is associated with enhanced plasticity in control animals, especially in regions such as the visual cortex, the hippocampus and the medial prefrontal cortex. More recently, the basolateral amygdala has been shown to be affected by Fluoxetine leading to a reopening of critical period-like plasticity in the fear and aggression circuits. One of the key elements triggering this type of brain plasticity are inhibitory networks, especially parvalbumin intern…

Male0301 basic medicineDendritic spinegenetic structuresInterneuronHippocampusMice TransgenicMice03 medical and health sciences0302 clinical medicineInterneuronsFluoxetineNeuroplasticitymedicineAnimalsPrefrontal cortexNeuronal PlasticitybiologyBasolateral Nuclear ComplexGeneral NeuroscienceAntidepressive Agents030104 developmental biologymedicine.anatomical_structureSomatostatinnervous systembiology.proteinSomatostatinNeuroscience030217 neurology & neurosurgeryParvalbuminBasolateral amygdalaNeuroscience
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Arachidonate 5-lipoxygenase (ALOX5) gene polymorphism is associated with Alzheimer's disease and body mass index

2016

IF 2.126; International audience; Dementias of old age, in particular Alzheimer's disease (AD), pose a growing threat to the longevity and quality of life of individuals as well as whole societies world-wide. The risk factors are both genetic and environmental (lifestyle) and there is an overlap with similar factors predisposing to cardiovascular diseases (CVD). Using a case control genetic approach, we have identified a SNP (rs10507391) in ALOX5 gene, previously associated with an increased risk of stroke, as a novel genetic risk factor for AD. ALOX5 gene encodes a 5'-lipoxygenase (5'-LO) activating protein (FLAP), a crucial component of the arachidonic acid/leukotriene inflammatory cascad…

Male0301 basic medicineDiseaseBioinformaticsBody Mass Index0302 clinical medicinePutative roleSurveys and QuestionnairesGenotypeMedicineCzech RepublicAged 80 and over2. Zero hungerGeneticsbiologyAlzheimer's disease3. Good healthRisk-factorsArachidonic acidNeurologyArachidonate 5-lipoxygenaseActivating proteinFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neuronal 5-LipoxygenaseLeukotrienesCurcuminGenotypeDna methylationFLAPPolymorphism Single NucleotideMouse modelAssociation03 medical and health sciencesMessenger-RnaAlzheimer DiseaseGeneticsHumansSNPPolymorphismSingle-Nucleotide polymorphisms5-lipoxygenase-activating proteinLife StyleGenetic Association StudiesAgedAmyloidotic phenotypeInflammationCaffeic acidArachidonate 5-Lipoxygenasebusiness.industryBody WeightOdds ratio030104 developmental biology[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Relative riskbiology.proteinNeurology (clinical)businessBody mass index030217 neurology & neurosurgeryJournal of the Neurological Sciences
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Neurovascular EGFL7 regulates adult neurogenesis in the subventricular zone and thereby affects olfactory perception

2016

Adult neural stem cells reside in a specialized niche in the subventricular zone (SVZ). Throughout life they give rise to adult-born neurons in the olfactory bulb (OB), thus contributing to neural plasticity and pattern discrimination. Here, we show that the neurovascular protein EGFL7 is secreted by endothelial cells and neural stem cells (NSCs) of the SVZ to shape the vascular stem-cell niche. Loss of EGFL7 causes an accumulation of activated NSCs, which display enhanced activity and re-entry into the cell cycle. EGFL7 pushes activated NSCs towards quiescence and neuronal progeny towards differentiation. This is achieved by promoting Dll4-induced Notch signalling at the blood vessel-stem …

Male0301 basic medicineGeneral Physics and AstronomyNEURAL STEM-CELLSMOUSEMiceSUBEPENDYMAL ZONENeural Stem CellsLateral VentriclesLINEAGE PROGRESSIONBRAININ-VIVOMice KnockoutNeuronal PlasticityMultidisciplinaryCell CycleQNeurogenesisNICHEAnatomyNeural stem cellCell biologyAdult Stem Cellsmedicine.anatomical_structureSignal TransductionSTIMULATES NEUROGENESISEGF Family of ProteinsNeurogenesisScienceNotch signaling pathwaySubventricular zoneBiologyInhibitory postsynaptic potentialArticleGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesNeuroplasticitymedicineBiological neural networkAnimalsCalcium-Binding ProteinsProteinsGeneral ChemistryOlfactory PerceptionENDOTHELIAL-CELLSnervous system diseasesOlfactory bulbMice Inbred C57BLSELF-RENEWAL030104 developmental biologynervous system
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Existence of muscarinic acetylcholine receptor (mAChR) and fibroblast growth factor receptor (FGFR) heteroreceptor complexes and their enhancement of…

2017

Abstract Background Recently, it was demonstrated that G-protein-coupled receptors (GPCRs) can transactivate tyrosine kinase receptors in absence of their ligands. In this work, driven by the observation that mAChRs and fibroblast growth factor receptors (FGFRs) share signalling pathways and regulation of brain functions, it was decided to explore whether mAChRs activation may transactivate FGFRs and, if so, to characterize the related trophic effects in cultured hippocampal neurons. Methods Oxotremorine-M transactivation of FGFRs and related trophic effects were tested in primary hippocampal neurons. Western blotting and in situ proximity ligation assay (PLA) were used to detect FGFR phosp…

Male0301 basic medicineHippocampusBiochemistryReceptor tyrosine kinaseReceptors G-Protein-CoupledRats Sprague-DawleyTransactivation0302 clinical medicineMuscarinic acetylcholine receptorNeural plasticityNeuronsNeuronal PlasticitybiologyReceptors MuscarinicCell biologyFibroblast growth factor receptorFibroblast Growth Factor 2Signal TransductionProto-oncogene tyrosine-protein kinase Srcmedicine.medical_specialtyNeuriteNeuronal OutgrowthBiophysicsHeteroreceptor03 medical and health sciencesHippocampuInternal medicinemedicineAnimalsReceptor Fibroblast Growth Factor Type 1Rats WistarMolecular BiologyTransactivationAnimalOxotremorineFibroblast growth factor receptor 1Receptor Muscarinic M1NeuronReceptors Fibroblast Growth FactorRatsFGFR1030104 developmental biologyEndocrinologyM1receptorBiophysicHeteroreceptor complexebiology.proteinRat030217 neurology & neurosurgeryBiochimica et Biophysica Acta (BBA) - General Subjects
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Perineuronal Nets Regulate the Inhibitory Perisomatic Input onto Parvalbumin Interneurons and γ Activity in the Prefrontal Cortex

2020

Parvalbumin-expressing (PV+) interneurons play a key role in the maturation and synchronization of cortical circuitry and alterations in these inhibitory neurons, especially in the medial prefrontal cortex (mPFC), have been found in different psychiatric disorders. The formation of perineuronal nets (PNNs) around many of these interneurons at the end of the critical periods reduces their plasticity and sets their connectivity. Consequently, the presence of PNNs must have an important impact on the synaptic input and the physiology of PV+ cells. In the present study, we have found that in adult male mice, prefrontocortical PV+ cells surrounded by PNNs show higher density of perisomatic excit…

Male0301 basic medicineInterneuronPrefrontal CortexInhibitory postsynaptic potentialMice03 medical and health sciences0302 clinical medicineInterneuronsBasket cellmedicineExtracellularAnimalsGamma RhythmPrefrontal cortexResearch ArticlesNeuronal PlasticitybiologyChemistryGeneral NeurosciencePerineuronal netExtracellular MatrixMice Inbred C57BLParvalbumins030104 developmental biologymedicine.anatomical_structurenervous systembiology.proteinExcitatory postsynaptic potentialNeuroscience030217 neurology & neurosurgeryParvalbuminThe Journal of Neuroscience
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Protective effects of mirtazapine in mice lacking the Mbnl2 gene in forebrain glutamatergic neurons: Relevance for myotonic dystrophy 1

2019

Myotonic dystrophy type 1 (DM1) is a multisystemic disorder characterized by muscle weakness and wasting and by important central nervous system-related symptoms including impairments in executive functions, spatial abilities and increased anxiety and depression. The Mbnl2 gene has been implicated in several phenotypes consistent with DM1 neuropathology. In this study, we developed a tissue-specific knockout mouse model lacking the Mbnl2 gene in forebrain glutamatergic neurons to examine its specific contribution to the neurobiological perturbations related to DM1. We found that these mice exhibit long-term cognitive deficits and a depressive-like state associated with neuronal loss, increa…

Male0301 basic medicineMirtazapineGlutamic AcidHippocampusMice TransgenicMirtazapineMyotonic dystrophyAnimals Genetically ModifiedMice03 medical and health sciencesCellular and Molecular NeuroscienceGlutamatergicProsencephalon0302 clinical medicinemedicineAnimalsMyotonic DystrophyDentate gyrusInflammationMice KnockoutNeuronsPharmacologyDepressionbusiness.industryCognitive deficitsDentate gyrusNeurogenesisRNA-Binding Proteinsmedicine.disease3. Good healthMice Inbred C57BLNeuroprotective Agents030104 developmental biologynervous systemKnockout mouseForebrainNeuronal lossDrosophilaFemaleDM1businessNeuroscience030217 neurology & neurosurgerymedicine.drugNeuropharmacology
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