Search results for "Neurotransmitter"

showing 10 items of 275 documents

Increase by 5-hydroxykynuramine of spontaneous acetylcholine release from myenteric neurons: mediated by serotonin M receptors

1987

The effects of 5-hydroxykynuramine (5-OH-K) and of 3-(2-amino-5-hydroxyphenyl)-propaneamine (AHPP) on spontaneous and electrically evoked release of [3H]acetylcholine were studied in the guinea-pig myenteric plexus longitudinal muscle preparation preincubated with [3H]choline. 5-OH-K caused a concentration-dependent increase in spontaneous [3H]acetylcholine release (EC50 5.3 microM). This effect was diminished in the presence of a desensitizing concentration of 5-hydroxytryptamine (5-HT). AHPP (1-100 microM) did not affect the spontaneous outflow of [3H]acetylcholine. The electrically evoked release of [3H]acetylcholine was significantly reduced in the presence of 100 microM of either 5-OH-…

MaleAgonistSerotoninmedicine.medical_specialtymedicine.drug_classKynuramineMethiothepinGuinea PigsMyenteric PlexusIn Vitro TechniquesBiologychemistry.chemical_compoundInternal medicinemedicineAnimalsCholineReceptorNeurotransmitterMyenteric plexusPharmacologyPropiophenonesPropylaminesAcetylcholineElectric StimulationEndocrinologychemistryReceptors SerotoninMetitepineFemaleSerotoninAcetylcholinemedicine.drugEuropean Journal of Pharmacology
researchProduct

Neurotransmitters involved in the fast inhibitory junction potentials in mouse distal colon

2003

We investigated, in murine colon circular muscle, the role of adenosine 5′-triphosphate (ATP) and pituitary adenylate cyclase activating peptide (PACAP) as inhibitory neurotransmitters of the fast component of nerve-evoked inhibitory junction potential (fast IJP). Fast IJP was antagonised by apamin or suramin, abolished by desensitisation with the P2Y receptor agonist, adenosine 5′-O-2-thiodiphosphate (ADPβS), unaffected by desensitisation with P2X receptor agonist, α,β-methylene ATP (α,β-meATP), and reduced by PACAP-(6-38), a PACAP receptor antagonist. ATP induced membrane hyperpolarization resistant to tetrodotoxin, Nω-nitro-L-arginine methyl ester (L-NAME) or PACAP-(6-38), but antagonise…

MaleAgonistendocrine systemmedicine.medical_specialtyP2Y receptorColonmedicine.drug_classPurinoceptorNeuromuscular JunctionSuraminTetrodotoxinBiologyApaminSettore BIO/09 - FisiologiaMembrane PotentialsCellular and Molecular NeuroscienceMicechemistry.chemical_compoundAdenosine TriphosphateInternal medicinemedicineAnimalsMurinePharmacologyNeurotransmitter AgentsDose-Response Relationship Drugmusculoskeletal neural and ocular physiologyNeuropeptidesMembrane hyperpolarizationThionucleotidesHyperpolarization (biology)Receptor antagonistAdenosinePeptide FragmentsATPAdenosine DiphosphatePituitary adenylate cyclase-activating peptideNG-Nitroarginine Methyl EsterEndocrinologyApaminchemistryPituitary Adenylate Cyclase-Activating PolypeptideFast inhibitory junction potentialPACAP (pituitary adenylate cyclase activating peptide)hormones hormone substitutes and hormone antagonistsmedicine.drugEuropean Journal of Pharmacology
researchProduct

Long-term effects on cortical glutamate release induced by prenatal exposure to the cannabinoid receptor agonist (r)-(+)-[2,3-dihydro-5-methyl-3-(4-m…

2003

The aim of the present in vivo microdialysis study was to investigate whether prenatal exposure to the CB1 receptor agonist WIN55,212-2 mesylate (WIN; (R)-()-(2,3- dihydro-5-methyl-3-(4-morpholinyl-methyl)pyrrolo(1,2,3-de)- 1,4-benzoxazin-6-yl)-1-naphthalenylmethanone), at a dose of 0.5 mg/kg (s.c. from the fifth to the 20th day of gestation), that causes neither malformations nor overt signs of toxicity, influences cortical glutamate extracellular levels in adult (90- day old) rats. Dam weight gain, pregnancy length and litter size at birth were not significantly affected by prenatal treatment with WIN. Basal and K-evoked dialysate glutamate levels were lower in the cerebral cortex of adul…

MaleAgonistmedicine.medical_specialtyMicrodialysisTime FactorsCannabinoid receptormedicine.drug_classMicrodialysisMorpholinesGlutamic Acidmaternal marijuana consumptionNaphthalenesBiologyTimechemistry.chemical_compoundGlutamatergicPiperidinesPregnancyInternal medicinebasal and K -evoked glutamate levelsmedicineAnimalsDrug InteractionsWakefulnessNeurotransmitterReceptorSR141716A; basal and K+-evoked glutamate levels; maternal marijuana consumptionCerebral CortexAnalysis of VarianceDose-Response Relationship DrugCannabinoidsGeneral NeuroscienceGlutamate receptorBenzoxazinesRatsEndocrinologyAnimals NewbornchemistryPrenatal Exposure Delayed EffectsSR141716AToxicityPotassiumPyrazolesSR141716A; basal and K -evoked glutamate levels; maternal marijuana consumption.CalciumFemaleRimonabantExtracellular Space
researchProduct

The effects of several muscarinic antagonists on pre- and postsynaptic receptors in the isolated rabbit heart

1981

In order to reveal possible differences between pre- and postsynaptic muscarine receptors, seven antagonists were tested for their affinities on these receptor sites in the rabbit isolated perfused heart. Methacholine was used as an agonist to inhibit the noradrenaline overflow evoked by electrical stimulation (3 Hz, 3 min) of the sympathetic nerves (presynaptic parameter) and to decrease the systolic tension development of the right atrium (postsynaptic parameter). The affinity of an antagonist was expressed as pA2. A decreasing order of potency was obtained with ipratropium, scopolamine, atropine, trihexyphenidyl, amitriptyline, and gallamine, both for pre- and postsynaptic responses. The…

MaleAgonistmedicine.medical_specialtymedicine.drug_classPopulationStimulationIn Vitro TechniquesNorepinephrinechemistry.chemical_compoundPostsynaptic potentialInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsReceptors CholinergicReceptoreducationPharmacologyeducation.field_of_studyBinding SitesMuscarineDose-Response Relationship DrugGallamine TriethiodideMyocardiumParasympatholyticsGeneral MedicineReceptors MuscarinicReceptors NeurotransmitterEndocrinologychemistryFemaleMethacholineRabbitsmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
researchProduct

Rewarding effects and reinstatement of MDMA-induced CPP in adolescent mice.

2007

Although the rewarding effects of 3,4-methylenedioxy-metamphetamine (MDMA) have been demonstrated in self-administration and conditioned place preference (CPP) procedures, its addictive potential (ie, the vulnerability to relapse, measured by its ability to induce reinstatement of an extinguished response), remains poorly understood. In this study, the effects of MDMA (5, 10, and 20 mg/kg) on the acquisition, extinction and reinstatement of CPP were evaluated in mice, using two different protocols during acquisition of CPP. In the first experiment, animals were trained using a two-session/day schedule (MDMA and saline for 4 consecutive days), whereas in the second experiment, they were trai…

MaleBiogenic AminesReinforcement ScheduleN-Methyl-34-methylenedioxyamphetamineStriatumPharmacologyExtinction Psychologicalchemistry.chemical_compoundMiceRewardDopaminemental disordersmedicineAnimalsNeurotransmitterPharmacologyBrain ChemistryAnalysis of VarianceBehavior AnimalDose-Response Relationship DrugMDMAExtinction (psychology)Conditioned place preferenceRatsPsychiatry and Mental healthchemistryAnimals NewbornAnesthesiaHallucinogensConditioning OperantSerotoninAnalysis of variancePsychologypsychological phenomena and processesmedicine.drugNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
researchProduct

Glia talk back.

2014

This study shows that the activity of neurons can trigger shedding of a protein, NG2, from the surface of oligodendrocyte precursor cells; this protein in turn modulates synaptic transmission, revealing a two-way conversation between neurons and glia.

MaleCell signalingNeural NetworksQH301-705.5AMPA receptorBiologyNeurotransmissionResearch and Analysis MethodsMechanical Treatment of SpecimensGeneral Biochemistry Genetics and Molecular BiologyBehavioral NeuroscienceNeurotransmitter receptormedicineBiological neural networkPremovement neuronal activityAnimalsBiology (General)AntigensNeuronsGeneral Immunology and MicrobiologyGeneral NeuroscienceMembrane ProteinsBiology and Life SciencesLong-term potentiationADAM ProteinsOligodendrogliamedicine.anatomical_structureElectroporationnervous systemSpecimen DisruptionSpecimen Preparation and TreatmentCellular NeuroscienceImmunologyProteoglycansNeuronAmyloid Precursor Protein SecretasesMolecular NeuroscienceGeneral Agricultural and Biological SciencesNeuroscienceResearch ArticleNeurosciencePLoS biology
researchProduct

Release of [3H]acetylcholine from a modified rat phrenic nerve-hemidiaphragm preparation

1986

Two different preparations of the rat phrenic nerve-hemidiaphragm (whole nerve-muscle preparation, end-plate preparation) were used for studying synthesis and release of radioactive acetylcholine in the absence and presence of cholinesterase inhibitors. When the whole nerve-muscle preparation (110-180 mg) was incubated with [3H]choline, only small amounts of radioactive acetylcholine were synthesized within the tissue. Electrical nerve stimulation of the whole nerve-muscle preparation produced no increase in tritium outflow. Incubation of the end-plate preparation (16-29 mg) which was obtained after removal of most of the muscle mass led to the formation of large amounts of [3H]acetylcholin…

MaleDiaphragmNeuromuscular JunctionStimulationIn Vitro TechniquesNeuromuscular junctionCholinechemistry.chemical_compoundmedicineAnimalsCholinePhrenic nerveCholinesterasePharmacologyNeurotransmitter AgentsbiologyRats Inbred StrainsHemicholinium 3General MedicineAcetylcholineMuscle DenervationRatsPhrenic Nervemedicine.anatomical_structurechemistryAnesthesiaTetrodotoxinbiology.proteinBiophysicsLiberationAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
researchProduct

Assessment of the abuse potential of MDMA in the conditioned place preference paradigm: Role of CB1 receptors

2013

Numerous reports have highlighted the role of the endocannabinoid system in the addictive potential of MDMA (3,4-methylenedioxy-methamphetamine). A previous report showed that CB1 knockout (KOCB1) mice do not acquire MDMA self-administration, despite developing conditioned place preference (CPP). This contradiction could be due to the particular procedure of place conditioning used. The present work compares MDMA-induced CPP in KOCB1 mice using unbiased and biased procedures of place conditioning. In the unbiased procedure, MDMA induced CPP and reinstatement of the extinguished preference in wild type (WT) mice, but not in KOCB1 mice. In contrast, in a biased protocol of CPP, MDMA produced …

MaleElevated plus mazeTime FactorsSubstance-Related Disordersmedicine.drug_classDopamineN-Methyl-34-methylenedioxyamphetamineNucleus accumbensPharmacologyAnxiolyticDevelopmental psychologyMiceNeurochemicalReceptor Cannabinoid CB1mental disordersmedicineAnimalsMaze LearningBiological PsychiatryMice KnockoutPharmacologyAnalysis of VarianceDose-Response Relationship DrugBrainHomovanillic AcidMDMAConditioned place preferenceDisease Models AnimalMonoamine neurotransmitternervous systemHallucinogens34-Dihydroxyphenylacetic AcidConditioning OperantSerotoninPsychologyReinforcement Psychologypsychological phenomena and processesmedicine.drugProgress in Neuro-Psychopharmacology and Biological Psychiatry
researchProduct

Tiagabine, a gamma-amino-butyric acid transporter inhibitor impairs spatial learning of rats in the Morris water-maze.

2002

Abstract γ-Amino-butyric acid (GABA) is cleaved from the synaptic cleft by uptake via specific transporters. Inhibition of such transporters increases the effectiveness of physiologically released GABA. Increased GABAergic neurotransmission has an impact on learning and memory. Therefore, effects of tiagabine, a GABA-transporter inhibitor, were investigated on spatial orientation in the Morris water-maze. Rats were given four training trials per day for 4 days and a probe trial without platform on the 5th day. Compared to saline treated rats, rats treated daily with 20 mg/kg tiagabine showed impaired learning during the acquisition trials. Retrieval was impaired in rats treated only at the …

MaleGABA Plasma Membrane Transport ProteinsSynaptic cleftTiagabinemedicine.medical_treatmentNipecotic AcidsMorris water navigation taskOrganic Anion TransportersPharmacologyBehavioral Neurosciencechemistry.chemical_compoundMemorymedicineGABA transporterAnimalsNeurotransmitterMaze LearningTiagabineSalineGABA AgonistsSwimmingbiologyMembrane ProteinsMembrane Transport ProteinsTransporterRats Inbred StrainsReceptors GABA-ARatschemistrybiology.proteinReuptake inhibitorCarrier ProteinsNeurosciencemedicine.drugBehavioural brain research
researchProduct

Cocaine enhances the conditioned rewarding effects of MDMA in adolescent mice.

2015

Although the consumption of cocaine is frequent in young users of MDMA (3,4-methylenedioxymethamphetamine), the influence of exposure to cocaine on the rewarding effects of MDMA in adolescents has not been studied. The purpose of the present work was to evaluate the effect of co-administration of cocaine (1 and 10 mg/kg) and a sub-threshold dose of MDMA (1.25 mg/kg) on the acquisition of conditioned place preference (CPP) (experiment 1). In addition, the effect of pre-treatment with cocaine on MDMA-induced CPP was evaluated (experiment 2). Levels of monoamines in striatum, hippocampus and cortex were measured in both experiments. Our hypotheses were that cocaine co-administration or pre-tre…

MaleGeneral NeuroscienceN-Methyl-34-methylenedioxyamphetamineDopaminergicHippocampusMDMADrug SynergismStriatumPharmacologyConditioned place preferenceDrug synergismMiceMonoamine neurotransmitterCocaineRewardmental disordersConditioning PsychologicalmedicineConditioningAnimalsBiogenic MonoaminesPsychologypsychological phenomena and processesmedicine.drugBrain research bulletin
researchProduct