Search results for "Neurotransmitter"

showing 10 items of 275 documents

Effects of CGS 10746B on hyperactivity and place preference induced by morphine

2001

The effects of CGS 10746B, a dopamine release inhibitor, on spontaneous locomotor activity, morphine-induced hyperactivity, acquisition of conditioned place paradigm and morphine-induced conditioned place preference (CPP) was evaluated in male mice. In experiment 1, animals treated with CGS 10746B (0.5, 1, 2, 4, 8, 16, 24 and 32 mg/kg), morphine (40 mg/kg) or morphine (40 mg/kg) plus CGS 10746B (0.5, 1, 2, 4, 8, 16, 24 and 32 mg/kg) were placed in an actimeter during a period of 90 min. In experiment 2, animals treated with CGS 10746B (0.5, 1, 3 and 10 mg/kg), morphine (40 mg/kg) or morphine (40 mg/kg) plus CGS 10746B (0.5, 1, 3 and 10 mg/kg) were conditioned following a procedure unbiased …

Malemedicine.medical_specialtyThiazepinesDopamineConditioning ClassicalMale miceMice Inbred StrainsSocial EnvironmentChoice BehaviorMiceBehavioral Neurosciencechemistry.chemical_compoundDopamineInternal medicinemedicineAnimalsDrug InteractionsNeurotransmitterDose-Response Relationship DrugMorphineChemistryAssociation LearningBrainConditioned place preferenceDose–response relationshipEndocrinologyMorphineCatecholamineConditioningNeuroscienceAntipsychotic Agentsmedicine.drugBehavioural Brain Research
researchProduct

Pravastatin treatment causes a shift in the balance of hippocampal neurotransmitter binding densities towards inhibition

2009

Since pravastatin, a HMG-CoA reductase inhibitor, has recently been shown to reduce infarct volumes and glutamate release in a rat model of ischemic stroke, the aim of the present study was to investigate whether this neuroprotective effect may be due to a modulation of excitatory and inhibitory neurotransmitter receptors. Therefore, Wistar rats were treated six times in 4 days with pravastatin or saline and allowed to survive for 6 hours or 5 days (n=10 per time point and group), respectively. Using quantitative receptor autoradiography, ligand binding densities of [(3)H]MK-801, [(3)H]AMPA, and [(3)H]muscimol for labeling of NMDA, AMPA, and GABA(A) receptors were analyzed in sensorimotor c…

Malemedicine.medical_specialtyTime FactorsKainate receptorAMPA receptorBiologyPharmacologyHippocampusReceptors N-Methyl-D-AspartateNeurotransmitter bindingRandom Allocationchemistry.chemical_compoundInternal medicinemedicineAnimalsReceptors AMPARats WistarLong-term depressionMolecular Biology5-HT receptorPravastatinCerebral CortexNeurotransmitter AgentsGABAA receptorGeneral NeuroscienceGlutamate receptorReceptors GABA-ACorpus StriatumRatsNeuroprotective AgentsEndocrinologynervous systemMuscimolchemistryNeurology (clinical)Developmental BiologyBrain Research
researchProduct

Long-Term Effects of Dietary Sodium Intake on Cytokines and Neurohormonal Activation in Patients With Recently Compensated Congestive Heart Failure

2009

Abstract Background A growing body of evidence suggests that the fluid accumulation plays a key role in the pathophysiology of heart failure (HF) and that the inflammatory and neurohormonal activation contribute strongly to the progression of this disorder. Methods and Results The study evaluated the long-term effects of 2 different sodium diets on cytokines neurohormones, body hydration and clinical outcome in compensated HF outpatients (New York Heart Association Class II). A total of 173 patients (105 males, mean age 72.5±7) recently hospitalized for worsening advanced HF and discharged in normal hydration and in clinical compensation were randomized in 2 groups (double blind). In Group …

Malemedicine.medical_specialtyTime Factorsmedicine.medical_treatmentPlasma renin activityGastroenterologychemistry.chemical_compoundDouble-Blind MethodInternal medicinemedicineHumansAgedAged 80 and overHeart FailureNeurotransmitter AgentsAldosteronebusiness.industryFurosemideSodium Dietarymedicine.diseaseBrain natriuretic peptidePathophysiologyAldosterone BNP bioelectrical impedance congestive heart failure cytochines plasma renin activatityEndocrinologyCytokinechemistryHeart failureCytokinesFemaleDiureticCardiology and Cardiovascular MedicinebusinessBiomarkersmedicine.drugFollow-Up Studies
researchProduct

Wasp venom injected into the prey's brain modulates thoracic identified monoaminergic neurons.

2005

The wasp Ampulex compressa injects a cocktail of neurotoxins into the brain of its cockroach prey to induce an enduring change in the execution of locomotory behaviors. Our hypothesis is that the venom injected into the brain indirectly alters the activity of monoaminergic neurons, thus changing the levels of monoamines that tune the central synapses of locomotory circuits. The purpose of the present investigation was to establish whether the venom alters the descending control, from the brain, of octopaminergic neurons in the thorax. This question was approached by recording the activity of specific identified octopaminergic neurons after removing the input from the brain or after a wasp s…

Malemedicine.medical_treatmentWaspsVenomSensory systemWasp VenomsMotor ActivityMembrane PotentialsCellular and Molecular Neurosciencebiology.animalMonoaminergicmedicineAnimalsPeriplanetaOctopamineNeuronsCockroachbiologyGeneral NeuroscienceBrainInsect Bites and StingsAxotomyThoraxElectrophysiologyElectrophysiologyMonoamine neurotransmittermedicine.anatomical_structurenervous systemNeuronAxotomyNeuroscienceJournal of neurobiology
researchProduct

The contribution of acetylcholine and dopamine to subprocesses of visual working memory--what patients with amnestic mild cognitive impairment and Pa…

2014

Attentional selection, i.e. filtering out of irrelevant sensory input and information storage are two crucial components of working memory (WM). It has been proposed that the two processes are mediated by different neurotransmitters, namely acetylcholine for attentional selection and dopamine for memory storage. However, this hypothesis has been challenged by others, who for example linked a lack in dopamine levels in the brain to filtering deficits. Here we tested the above mentioned hypothesis in two patient cohorts which either served as a proxy for a cholinergic or a dopaminergic deficit. The first group comprised 18 patients with amnestic mild cognitive impairment (aMCI), the second 22…

Malephysiopathology [Cognitive Dysfunction]Parkinson's diseaseCognitive NeuroscienceDopamineModels NeurologicalExperimental and Cognitive Psychologyphysiopathology [Brain]Neuropsychological TestsCohort StudiesBehavioral Neurosciencechemistry.chemical_compoundDopaminemedicineHumansAttentionCognitive Dysfunctionddc:610metabolism [Dopamine]NeurotransmitterAgedWorking memoryDopaminergicBrainCognitionParkinson Diseasephysiopathology [Amnesia]Middle Agedphysiology [Visual Perception]medicine.diseaseAcetylcholineMemory Short-Termchemistryphysiology [Memory Short-Term]physiology [Attention]Visual PerceptionCholinergicFemalephysiopathology [Parkinson Disease]AmnesiaPsychologyNeurosciencemetabolism [Acetylcholine]Acetylcholinemedicine.drugNeuropsychologia
researchProduct

Maize based diets and possible neurobehavioural after-effects among some populations in the world

1996

Maize is a cereal particularly lacking in tryptophan, which is the precursor of serotonin, an important neurotransmitter. Altough complementary foods may eliminate tryptophan deficiency, serotonin deficiency may often continue to exist because of competition made by other Large Neutral Amino Acids (LNAA) against tryptophan for neuron access, since they use the same carrier to cross the blood-brain barrier. Thus serotonin synthesis depends on two variables: the amount of tryptophan and the trp/LNAA ratio (R). “R” is lowest for common maize, low for beans and, as a rule, for most vegetable foods, higher for meat. So, when maize is the preponderant food in the meal, the “R” value lowers and so…

MealSerotonin synthesisNative americanbusiness.industrymedia_common.quotation_subjectTryptophanBiologyCompetition (biology)BiotechnologyNeutral Amino Acidschemistry.chemical_compoundchemistryAnthropologyFood scienceSerotoninbusinessNeurotransmittermedia_commonHuman Evolution
researchProduct

Stimulation of hippocampal acetylcholine release by hyperforin, a constituent of St. John’s Wort

2004

Abstract Extracts of the medicinal plant St. John’s Wort ( Hypericum perforatum ) are widely used in the therapy of affective disorders and have been reported to exert antidepressant, anxiolytic, and cognitive effects in experimental and clinical studies. We here report that hyperforin, the major active constituent of the extract, increases the release of acetylcholine from rat hippocampus in vivo as determined by microdialysis. Hippocampal acetylcholine levels were increased by 50–100% following the systemic administration of pure hyperforin at doses of 1 and 10 mg/kg. The effect was almost completely suppressed by local perfusion with calcium-free buffer or with tetrodotoxin (1 μM). We co…

Microdialysismedicine.drug_classMicrodialysisTetrodotoxinPhloroglucinolPharmacologyHippocampusAnxiolyticRats Sprague-DawleyBridged Bicyclo Compoundschemistry.chemical_compoundmedicineAnimalsAnesthetics LocalNeurotransmitterPlant ExtractsTerpenesGeneral NeuroscienceHypericum perforatumAcetylcholineAnti-Bacterial AgentsRatsHyperforinchemistryAntidepressantCholinergicHypericumAcetylcholinemedicine.drugNeuroscience Letters
researchProduct

Synthesis, in vitro activity, and three-dimensional quantitative structure-activity relationship of novel hydrazine inhibitors of human vascular adhe…

2010

Vascular adhesion protein-1 (VAP-1) belongs to the semicarbazide-sensitive amine oxidases (SSAOs) that convert amines into aldehydes. SSAOs are distinct from the mammalian monoamine oxidases (MAOs), but their substrate specificities are partly overlapping. VAP-1 has been proposed as a target for anti-inflammatory drug therapy because of its role in leukocyte adhesion to endothelium. Here, we describe the synthesis and in vitro activities of novel series of VAP-1 selective inhibitors. In addition, the molecular dynamics simulations performed for VAP-1 reveal that the movements of Met211, Ser496, and especially Leu469 can enlarge the ligand-binding pocket, allowing larger ligands than those s…

Models MolecularSubstrate SpecificitiesQuantitative structure–activity relationshipMolecular ConformationQuantitative Structure-Activity RelationshipMolecular Dynamics SimulationLigandsMolecular dynamicsCricetulusCricetinaeDrug DiscoveryAnimalsHumansMonoamine OxidaseBinding SitesChemistryStereoisomerismIn vitrorespiratory tract diseasesRatsMonoamine neurotransmitterHydrazinesBiochemistryDocking (molecular)Molecular MedicineAmine gas treatingAmine Oxidase (Copper-Containing)Cell Adhesion MoleculesVASCULAR ADHESION PROTEIN 1Protein BindingJournal of medicinal chemistry
researchProduct

Studies of the Hypothalamus-Pituitary-Adrenocortical System: An Example of Progress in Psychoneuroendocrinology

1986

Psychobiological study of affective disorders has passed through several phases during the last three decades. With the discovery of thymoleptic drugs and our partial understanding of their pharmacological properties, a dominant theme in psychiatric research came to be the pathophysiology underlying depressive illness. Since enhancement of monoamine neurotransmission was found to be a common characteristic of most antidepressants, several attempts were made to test the hypothesis of a defective cerebral monoamine transmission as the prime cause of depression. In this context, neuroendocrinology became an area that was of particular interest to investigators, for several reasons. Basic resea…

Monoamine neurotransmitterHypothalamusbusiness.industryEndocrine systemMedicineContext (language use)NeuroendocrinologybusinessNeuroscienceProlactinHormonePsychoneuroendocrinology
researchProduct

Animal Models of Stress - Current Knowledge and Potential Directions

2021

Finding new therapies and new antidepressant agents is of high clinical priority given that many cases of depressive disorder do not respond to conventional monoaminergic antidepressants such as selective serotonin reuptake inhibitors, tricyclic antidepressants, and monoamine oxidase inhibitors The authors demonstrated that electroacupuncture and fluoxetine, a second-generation antidepressant categorized as a selective serotonin reuptake inhibitor (Perez-Caballero et al , 2014), regulate the expression of key proteins in the calmodulin kinase (CAMK) signaling pathway, which are related to depression in the hippocampi of rats (Takemoto-Kimura et al , 2017;Xie et al , 2019) In a paper on “Sho…

Monoamine oxidaseCognitive NeuroscienceSerotonin reuptake inhibitorContext (language use)Pharmacologyalternative therapylcsh:RC321-571stress03 medical and health sciencesBehavioral Neurosciencechemistry.chemical_compound0302 clinical medicineorganoselenium compoundsMonoaminergicmedicineNeurotransmitterlcsh:Neurosciences. Biological psychiatry. Neuropsychiatry030304 developmental biologychemistry.chemical_classification0303 health sciencesFluoxetinebusiness.industryanimal modelsEditorialNeuropsychology and Physiological PsychologychemistryAntidepressantmajor depressionbusiness030217 neurology & neurosurgeryTricyclicmedicine.drugFrontiers in Behavioral Neuroscience
researchProduct