Search results for "Nicotine"

showing 10 items of 145 documents

Association of a variant in the muscarinic acetylcholine receptor 2 gene (CHRM2 ) with nicotine addiction

2009

Genetic factors contribute to the overall risk of developing nicotine addiction, which is the major cause of preventable deaths in western countries. However, knowledge regarding specific polymorphisms influencing smoking phenotypes remains scarce. In the present study we provide evidence that a common single nucleotide polymorphism (SNP) in the 5′ untranslated region of CHRM2, the gene coding for the muscarinic acetylcholine receptor 2 is associated with nicotine addiction. CHRM2 was defined as a candidate gene for nicotine addiction based on previous evidence that linked variations in CHRM2 to alcohol and drug dependence. A total of more than 5,500 subjects representative of the German po…

AdultMaleNicotineCandidate geneAdolescentmedia_common.quotation_subjectSingle-nucleotide polymorphismBiologyBioinformaticsNicotineCellular and Molecular NeuroscienceMuscarinic acetylcholine receptormedicineHumansSNPGenetic Predisposition to DiseaseAlleleAllelesGenetics (clinical)Agedmedia_commonAged 80 and overGeneticsReceptor Muscarinic M2AddictionSmokingGenetic VariationTobacco Use DisorderOdds ratioMiddle AgedPsychiatry and Mental healthFemalemedicine.drugAmerican Journal of Medical Genetics Part B: Neuropsychiatric Genetics
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Differences in psychomotor reaction time in male monozygotic twins discordant for lifetime cigarette smoking.

1996

The effects of long-term cigarette smoking on psychomotor reaction time were investigated among 8 pairs of monozygotic male twins highly discordant for lifetime smoking (means 32.4 versus 0.6 pack-years). The men had no diagnosed cardiovascular disease or other major diseases, musculoskeletal complaints, or vision problems that might interfere with reaction time testing. The twins had similar education, work, and exercise histories; alcohol and coffee consumption and exposure to solvents were examined as possible confounds. Direct comparison of cotwins also controlled for age, genetics, and possible early environmental factors. Simple and choice reaction time were measured in the dominant h…

AdultMaleNicotineChoice reaction timebusiness.industrySmokingExperimental and Cognitive PsychologyCoffee consumptionDiseaseTwins MonozygoticMiddle AgedSensory SystemsCohort StudiesCigarette smokingReaction TimeMedicineHumansPsychomotor reaction timebusinessPsychomotor PerformanceDemographyPerceptual and motor skills
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Long-term effects of nicotine gum on weight gain after smoking cessation.

1999

Smoking cessation usually results in weight gain. Nicotine gum therapy has been found to reduce weight gain in the first months after cessation, but its long-term effects are not fully known. The present study randomly assigned 608 smokers to receive placebo, 2 or 4 mg nicotine gum. In a follow-up analysis to the short-term weight change results reported in a previous paper [Doherty, Militello, Kinnunen, & Garvey (1996), Journal of Consulting and Clinical Psychology 64:799-807], we examined the effects of the nicotine gum on weight change for 1 year after cessation among the 92 1-year abstainers. We found that weight change showed some variation with gum dose (active vs. placebo), but that …

AdultMaleNicotineNicotine Chewing Gummedicine.medical_treatmentGingivaPhysiologyWeight Gainchemistry.chemical_compoundDouble-Blind MethodMedicineHumansCotinineNicotine replacementbusiness.industryWeight changePublic Health Environmental and Occupational HealthTobacco Use DisorderMiddle AgedNicotine replacement therapyGanglionic StimulantschemistryNicotine gumAnesthesiaSmoking cessationFemaleSmoking Cessationmedicine.symptombusinessCotinineWeight gainNicotinetobacco research : official journal of the Society for Research on Nicotine and Tobacco
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Association of attentional network function with exon 5 variations of the CHRNA4 gene

2007

Mutational analyses in xenopus oocyte and mice models indicate that the positive effect of nicotine on attention may be modulated by genetic variations within exon 5 of the alpha4 subunit of the nicotinergic acetylcholine receptor gene CHRNA4. The potential relevance of exon 5 is further emphasized by two recent family-based association studies of nicotine dependence because subgroups of nicotine-dependent subjects are thought to 'self-medicate' attentional deficits with nicotine. We investigated a synonymous single nucleotide polymorphisms (SNP): rs1044396, which has recently been associated with nicotine-dependence, plus two adjacent synonymous SNPs rs1044394 and rs1044393 in exon 5 of n …

AdultMaleNicotineXenopusPosterior parietal cortexSingle-nucleotide polymorphismReceptors NicotinicBiologyModels BiologicalWhite PeopleMiceExonParietal LobeGeneticsmedicineAnimalsHumansAttentionMolecular BiologyOddball paradigmGenetics (clinical)Genetic associationGeneticsSupplementary motor areamedicine.diagnostic_testParietal lobeExonsTobacco Use DisorderGeneral MedicineMagnetic Resonance ImagingRadiographymedicine.anatomical_structureAttention Deficit Disorder with HyperactivityFemaleFunctional magnetic resonance imagingHuman Molecular Genetics
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Schizophrenic patients who smoke have a faster finger tapping rate than non-smokers

2002

The increased rate of smoking in schizophrenia patients remains unexplained and may reflect attempts at self-treatment. The effect sought from smoking may be related to nicotine's stimulating action. We tested this hypothesis by examining the relationship between smoking status and finger tapping rate, a measure of central processing, in schizophrenia patients treated with atypical antipsychotics. Smokers showed significantly faster finger tapping rates than non-smokers. This was not related to clinical state, illness chronicity, medication side-effects, antipsychotic dose or plasma concentrations. Nicotine can improve central processing in medicated schizophrenia patients and this may cons…

AdultMaleNicotinemedicine.medical_specialtyPatientsMovementmedicine.medical_treatmentClinical stateFingersNicotineInternal medicinemedicineHumansPharmacology (medical)Nicotinic AgonistsAntipsychoticPsychiatryBiological PsychiatryPharmacologySmokeAnalysis of VarianceSmokingMiddle Agedmedicine.diseasePsychiatry and Mental healthNeurologySchizophreniaFinger tappingPlasma concentrationSchizophreniaSmoking statusNeurology (clinical)PsychologyPsychomotor Performancemedicine.drugEuropean Neuropsychopharmacology
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Cytotoxic and genotoxic effects of electronic cigarette liquids on human mucosal tissue cultures of the oropharynx

2016

The popularity of electronic cigarettes (ECs) is rapidly growing and ECs are claimed to be an uncritically regarded alternative to conventional cigarettes. The mucosal tissue of the upper aerodigestive tract (UADT) is the first contact organ for xenobiotics such as liquids of ECs. The aim of this study is to investigate the bimolecular effects of e-liquids on human pharyngeal tissue cultures to evaluate whether e-liquids and their components present a risk factor for head and neck squamous cell carcinoma. Fresh tissue samples of healthy oropharyngeal mucosa were assembled into mucosal tissue cultures. Two fruit-flavored liquids (FLs), one tobacco-flavored liquid (TL) (all containing nicotin…

AdultMalePathologymedicine.medical_specialtyOropharyngeal TissueDNA damageCell SurvivalHealth Toxicology and MutagenesisOropharynxElectronic Nicotine Delivery SystemsToxicologyPathology and Forensic Medicine03 medical and health sciencesTissue cultureYoung Adult0302 clinical medicineFresh TissueRisk FactorsGermanyMedicineHumansCarcinogenMucous Membranebusiness.industryCytotoxinsSquamous Cell Carcinoma of Head and NeckGeneral Medicinemedicine.diseaseHead and neck squamous-cell carcinoma3. Good health030228 respiratory systemHead and Neck NeoplasmsToxicityCancer researchCarcinoma Squamous CellDNA fragmentationFemalebusiness030217 neurology & neurosurgeryDNA DamageMutagens
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Tobacco smoking and nicotine dependence in first episode and established psychosis

2019

Aim\ud People with psychotic disorders have increased premature mortality in comparison with the general population, with high rates of cigarette use a contributing factor. We aimed to describe the prevalence of cigarette use and nicotine dependence (ND) in first episode psychosis (FEP), and established psychosis; and to investigate associations between clinical symptoms and ND.\ud \ud Methodology\ud Smoking and clinical data were collected from two cohorts: 181 people with FEP recruited as part of the Physical Health and Substance Use Measures in First Onset Psychosis (PUMP) study and from 432 people with established psychosis recruited as part of the Improving\ud physical health and reduc…

AdultMalePsychosismedicine.medical_specialtyAdolescentmedia_common.quotation_subjectmedicine.medical_treatmentPopulationAddictionPhysical healthComorbiditySubstance useCigarette SmokingNicotineYoung AdultmedicineHumanseducationPsychiatryPsychoticGeneral PsychologyRandomized Controlled Trials as Topicmedia_commonFirst episodeeducation.field_of_studybusiness.industryAddictionSmokingTobacco Use DisorderGeneral Medicinemedicine.diseaseUnited KingdomPsychiatry and Mental healthPsychotic DisordersSchizophreniaCohortSchizophreniaSmoking cessationFemalebusinessmedicine.drug
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Risk gene variants for nicotine dependence in the CHRNA5-CHRNA3-CHRNB4 cluster are associated with cognitive performance

2010

Recent studies strongly support an association of the nicotinic acetylcholine receptor gene cluster CHRNA5-CHRNA3-CHRNB4 with nicotine dependence (ND). However, the precise genotype-phenotype relationship is still unknown. Clinical and epidemiological data on smoking behavior raise the possibility that the relevant gene variants may indirectly contribute to the development of ND by affecting cognitive performance in some smokers who consume nicotine for reasons of "cognition enhancement." Here, we tested seven single nucleotide polymorphisms (SNPs) rs684513, rs637137, rs16969968, rs578776, rs1051730, rs3743078, rs3813567 from the CHRNA5-CHRNA3-CHRNB4 gene cluster for association with ND, me…

AdultMaleRiskGenotypeGene ExpressionNerve Tissue ProteinsSingle-nucleotide polymorphismReceptors NicotinicBiologyBioinformaticsPolymorphism Single NucleotideNicotineCellular and Molecular NeuroscienceCognitionGene clustermedicineHumansGenetic Predisposition to DiseaseRNA MessengerRisk factorAlleleGenetic Association StudiesGenetics (clinical)AgedGeneticsChromosomes Human Pair 15Gene Expression ProfilingCHRNA5HaplotypeWechsler ScalesGenetic VariationCognitionTobacco Use DisorderMiddle AgedPsychiatry and Mental healthMultigene Familybiology.proteinFemalemedicine.drugAmerican Journal of Medical Genetics Part B: Neuropsychiatric Genetics
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Increased sensitivity of the neuronal nicotinic receptor alpha-2 subunit causes familial epilepsy with nocturnal wandering and ictal fear

2006

Sleep has traditionally been recognized as a precipitating factor for some forms of epilepsy, although differential diagnosis between some seizure types and parasomnias may be difficult. Autosomal dominant frontal lobe epilepsy is characterized by nocturnal seizures with hyperkinetic automatisms and poorly organized stereotyped movements and has been associated with mutations of the α4 and β2 subunits of the neuronal nicotinic acetylcholine receptor. We performed a clinical and molecular genetic study of a large pedigree segregating sleep-related epilepsy in which seizures are associated with fear sensation, tongue movements, and nocturnal wandering, closely resembling nightmares and sleep …

AdultMalemedicine.medical_specialtyAdolescentSomnambulismMolecular Sequence DataMutation MissenseAutosomal dominant nocturnal frontal lobe epilepsyReceptors NicotinicBiologymedicine.disease_causeLigandsNicotinicArticleEpilepsyBIO/09 - FISIOLOGIAInternal medicineAcetylcholine; Adolescent; Adult; Aged; Aged 80 and over; Amino Acid Sequence; Epilepsy; Female; Humans; Ligands; Male; Molecular Sequence Data; Mutation Missense; Neurons; Pedigree; Receptors Nicotinic; Somnambulism; FearReceptorsmedicine80 and overGeneticsHumansIctalGenetics(clinical)Amino Acid SequenceGenetics (clinical)Acetylcholine receptorAgedAged 80 and overNeuronsMutationEpilepsySeizure typesFearmedicine.diseaseAcetylcholinePedigreeNicotinic acetylcholine receptorNicotinic agonistEndocrinologyMutationnAChR patch-clamp ADNFLE sleep-related epilepsy M1 TM1 ACh nicotineSettore MED/26 - NeurologiaFemaleMissense
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The P300 event-related potential and smoking--a population-based case-control study.

2009

A better understanding of the factors underlying habitual tobacco smoking may further new strategies to go about this major health problem. The P300 event-related potential (ERP) has emerged as a valuable (endo)phenotype in neuropsychiatric research. Previous studies suggested the P300 ERP to be reduced in smokers. The main purpose of the present study was to provide an in-depth description of smoking-related behavioral, biological and electrophysiological phenotypes with an emphasis on the P300 ERP and its mutual relationship with other smoking-related parameters. In this case-control study N=1318 participants (smokers and never-smoking controls) were investigated at 6 German academic inst…

AdultMalemedicine.medical_specialtyPopulationNicotineYoung AdultEvent-related potentialPhysiology (medical)medicineHumansYoung adulteducationPsychiatryeducation.field_of_studyGeneral NeuroscienceConfoundingSmokingCase-control studyCognitionElectroencephalographyMiddle Agedmedicine.diseaseEvent-Related Potentials P300Substance abuseNeuropsychology and Physiological PsychologyCase-Control StudiesPopulation SurveillanceFemalePsychologymedicine.drugInternational journal of psychophysiology : official journal of the International Organization of Psychophysiology
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