Search results for "Nicotinic acetylcholine receptor"

showing 10 items of 50 documents

Nicotine enhances antisaccade performance in schizophrenia patients and healthy controls

2013

Abstract Nicotine has been proposed to be a cognitive enhancer, particularly in schizophrenia patients. So far, the published studies of nicotine effects on antisaccade performance in schizophrenia patients only tested participants who were deprived smokers. Thus, we aimed to test both smoking and non-smoking patients as well as healthy controls in order to extend previous findings. Moreover, we employed a paradigm using standard and delayed trials. We hypothesized that, if nicotine is a genuine cognitive enhancer, its administration would improve antisaccade performance both in smoking and non-smoking participants. A total of 22 patients with schizophrenia (12 smokers and 10 non-smokers) a…

Malemedicine.medical_treatmentStatistics as TopicNicotine2738 Psychiatry and Mental Health0302 clinical medicine2736 Pharmacology (medical)Pharmacology (medical)Nicotinic AgonistsCotinineCross-Over StudiesSmokingCognitionMiddle Aged16. Peace & justice3. Good healthPsychiatry and Mental healthNicotinic acetylcholine receptor3004 PharmacologySchizophreniaFemaleSmoking statusPsychologymedicine.drugClinical psychologyAdultNicotinemedicine.medical_specialtyAdolescentNicotine patch610 Medicine & healthAdministration CutaneousYoung Adult03 medical and health sciencesDouble-Blind MethodInternal medicineReaction TimeSaccadesmedicineHumansEffects of sleep deprivation on cognitive performanceBeneficial effectsPharmacologyAnalysis of Variancemedicine.disease030227 psychiatryElectrooculography10054 Clinic for Psychiatry Psychotherapy and PsychosomaticsSchizophrenia030217 neurology & neurosurgery
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A Structural Model of the Human α7 Nicotinic Receptor in an Open Conformation

2015

International audience; Nicotinic acetylcholine receptors (nAchRs) are ligand-gated ion channels that regulate chemical transmission at the neuromuscular junction. Structural information is available at low resolution from open and closed forms of an eukaryotic receptor, and at high resolution from other members of the same structural family, two prokaryotic orthologs and an eukary- otic GluCl channel. Structures of human channels however are still lacking. Homology modeling and Molecular Dynamics simulations are valuable tools to predict structures of unknown proteins, however, for the case of human nAchRs, they have been unsuccessful in providing a stable open structure so far. This is du…

Models MolecularHydrogen bondingalpha7 Nicotinic Acetylcholine ReceptorProtein ConformationMolecular Sequence DataMESH: Sequence Alignmentligand gated ion channles molecular dynamics simulation epibatidine waterlcsh:MedicineSequence alignmentMESH: Amino Acid SequenceMolecular Dynamics SimulationMESH: Models Molecular*Molecular dynamicsProtein structureSequence alignmentCationsHumansMESH: Molecular Dynamics SimulationHomology modelingAmino Acid SequenceNicotinic Receptorlcsh:ScienceBiochemical simulationsIon channelAcetylcholine receptorIonsMESH: Protein Conformation*MultidisciplinaryMESH: HumansMESH: Molecular Sequence DataChemistryMESH: Protein Multimerizationlcsh:RMESH: alpha7 Nicotinic Acetylcholine Receptor/chemistry*[SDV.BIBS]Life Sciences [q-bio]/Quantitative Methods [q-bio.QM]Transmembrane proteinSimulation and modelingNicotinic agonistBiochemistryBiophysicsProtein structurelcsh:QProtein MultimerizationResearch ArticleStructural Model
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Mapping of a binding site for ATP within the extracellular region of the Torpedo nicotinic acetylcholine receptor beta-subunit.

1997

Using 2,8,5'-[H-3]ATP as a direct photoaffinity label for membrane-bound nicotinic acetylcholine receptor (nAChR) from Torpedo marmorata, we have identified a binding site for ATP in the extracellular region of the beta-subunit of the receptor. Photolabeling was completely inhibited in the presence of saturating concentrations of nonradioactive ATP, whereas neither the purinoreceptor antagonists suramin, theophyllin, and caffeine nor the nAChR antagonists alpha-bungarotoxin and d-tubocurarine affected the labeling reaction. Competitive and noncompetitive nicotinic agonists and Ca2+ increased the yield of the photoreaction by up to 50%, suggesting that the respective binding sites are allost…

Molecular Sequence DataPhotoaffinity LabelsReceptors NicotinicTorpedoTritiumBiochemistryPeptide Mappingchemistry.chemical_compoundGanglion type nicotinic receptorAdenosine TriphosphateAdenine nucleotideAnimalsChymotrypsinTrypsinAmino Acid SequenceBinding siteBinding SitesbiologyHydrolysisCell MembranePeptide FragmentsNicotinic acetylcholine receptorNicotinic agonistBiochemistrychemistrybiology.proteinAlpha-4 beta-2 nicotinic receptorExtracellular SpaceAdenosine triphosphateSequence AnalysisATP synthase alpha/beta subunitsBiochemistry
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Neuronal Nicotinic Receptors in the Locust Locusta migratoria

1998

We have identified five cDNA clones that encode nicotinic acetylcholine receptor (nAChR) subunits expressed in the nervous system of the locust Locusta migratoria. Four of the subunits are ligand-binding α subunits, and the other is a structural β subunit. The existence of at least one more nAChR gene, probably encoding a β subunit, is indicated. Based on Northern analysis and in situ hybridization, the five subunit genes are expressed. locα1, locα3, andlocβ1 are the most abundant subunits and are expressed in similar areas of the head ganglia and retina of the adult locust. Because Locα3 binds α-bungarotoxin with high affinity, it may form a homomeric nAChR subtype such as the mammalian α7…

Nervous systemGene isoformmedicine.medical_specialtyanimal structuresbiologyProtein subunitCell BiologyIn situ hybridizationbiology.organism_classificationBiochemistryCell biologyNicotinic acetylcholine receptormedicine.anatomical_structureEndocrinologynervous systemInternal medicineMushroom bodiesmedicineHomomericsense organsMolecular BiologyLocustJournal of Biological Chemistry
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Allosterically potentiating ligands of nicotinic receptors as a treatment strategy for Alzheimer's disease.

2000

Abstract One of the most prominent cholinergic deficit in Alzheimer’s disease (AD) is the reduced number of nicotinic acetylcholine receptors (nAChR) in the hippocampus and cortex of AD patients, as compared to age-matched controls. This deficit results in reduced nicotinic cholinergic excitation which may not only impair postsynaptic depolarization but also presynaptic neurotransmitter release and Ca 2+ -dependent intracellular signaling, including transcriptional activity. Presently, the most common approach to correct the nicotinic cholinergic deficit in AD is the application of cholinesterase inhibitors. Due to the resulting increase in synaptic acetylcholine levels, both in concentrati…

NeuronsPatch-Clamp TechniquesBiologyNeurotransmissionReceptors NicotinicSynaptic TransmissionCell LineBehavioral NeuroscienceNicotinic acetylcholine receptorMiceGanglion type nicotinic receptorNicotinic agonistAllosteric RegulationAlzheimer DiseaseMuscarinic acetylcholine receptormedicineAnimalsHumansNicotinic AgonistsAlpha-4 beta-2 nicotinic receptorNeuroscienceAcetylcholine5-HT receptorAllosteric Sitemedicine.drugBehavioural brain research
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Conformational changes in acetylcholine binding protein investigated by temperature accelerated molecular dynamics.

2014

Despite the large number of studies available on nicotinic acetylcholine receptors, a complete account of the mechanistic aspects of their gating transition in response to ligand binding still remains elusive. As a first step toward dissecting the transition mechanism by accelerated sampling techniques, we study the ligand-induced conformational changes of the acetylcholine binding protein (AChBP), a widely accepted model for the full receptor extracellular domain. Using unbiased Molecular Dynamics (MD) and Temperature Accelerated Molecular Dynamics (TAMD) simulations we investigate the AChBP transition between the apo and the agonist-bound state. In long standard MD simulations, both confo…

Nicotinic Acetylcholine ReceptorsProtein ConformationGatingMolecular DynamicsLigandsBiochemistryBiophysics SimulationsIon ChannelsMolecular dynamicsAcetylcholine bindingComputational ChemistryBiochemical SimulationsNicotinic AgonistsBiomacromolecule-Ligand InteractionsBiochemistry SimulationsMultidisciplinaryHydrogen bondChemistryPhysicsQTemperatureRLigand (biochemistry)nicotinic receptor molecular dynamics tamd acethylcholine binding proteinChemistryNicotinic agonistBiochemistryMedicineBiophysic Al SimulationsResearch ArticleProtein BindingProtein subunitScienceBiophysicsMolecular Dynamics SimulationProtein ChemistryStatistical MechanicsChemical BiologyAnimalsBiologyAcetylcholine receptorBinding SitesProteinsComputational BiologyHydrogen BondingSettore FIS/07 - Fisica Applicata(Beni Culturali Ambientali Biol.e Medicin)Protein SubunitsMolluscaAcetylcholine ReceptorsBiophysicsLobelineCarrier ProteinsPLoS ONE
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Molecular Histochemistry of Nicotinic Receptors in Human Brain

1997

Only a decade ago the existence and functional significance of central nervous nicotinic acetylcholine receptors (nAChR) was still a subject of controversy. Today, the importance of this receptor class for signal transduction in human brain in normal and pathological conditions has become quite evident. nAChRs have turned out to be important pharmacological targets in disorders like Alzheimer’s disease (AD) (Arneric et al., 1994). One prerequisite to understand nAChR function is a detailed study of the cellular distribution of nAChR subtypes. In recent years several human-specific data have been made available. This paper attempts to show actual developments in this field, summarizing the e…

Nicotinic Receptorsmusculoskeletal neural and ocular physiologyHuman brainBiologyNicotinic acetylcholine receptormedicine.anatomical_structureNicotinic agonistnervous systemmedicineImmunohistochemistrysense organsSignal transductionReceptorNeuroscienceAcetylcholine receptor
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Desensitization is a property of the cholinergic binding region of the nicotinic acetylcholine receptor, not of the receptor-integral ion channel

1991

AbstractThe reversible acetylcholine esterase inhibitor (−)-physostigmine (eserine) is the prototype of a new class of nicotinic acetylcholine receptor (nAChR) activating ligands: it induces cation fluxes into nAChR-rich membrane vesicles from Torpedo marmorala electric tissue even under conditions of antagonist blocked acetylcholine binding sites (Okonjo, Kuhlmann, Maclicke, Neuron, in press). This suggests that eserine exerts its channel-activating property via binding sites at the nAChR separate from those of the natural transmitter. We now report that eserine can activate the channel even when the receptor has been preincubated (desensitized) with elevated concentrations of acetylcholin…

Nicotinic acetylcholine receptorStereochemistryAcetylcholine-gated cation channelPhysostigmineBiophysicsCesiumIon fluxDesensitizationIn Vitro TechniquesReceptors NicotinicTorpedoBiochemistryIon ChannelsAnticholinesteraseAcetylcholine bindingGanglion type nicotinic receptorStructural BiologyMuscarinic acetylcholine receptor M5GeneticsmedicineAnimalsMolecular BiologyAcetylcholine receptorBinding SitesChemistryCell BiologyBungarotoxinsAcetylcholineNicotinic acetylcholine receptorNicotinic agonistCarbamateBiophysicsCholinergicAcetylcholineEserinemedicine.drugFEBS Letters
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Biochemical Characterization of a Novel Channel-Activating Site on Nicotinic Acetylcholine Receptors

1993

We have studied the interaction of the reversible acetylcholine esterase inhibitor (-)physostigmine and several structurally related compounds with the nicotinic acetylcholine receptor (nAChR) from Torpedo marmorata electric tissue by means of ligand-induced ion flux into nAChR-rich membrane vesicles, direct binding studies and photoaffinity labeling. (-)Physostigmine acts as a channel-activating ligand at low concentrations and as a direct channel blocker at elevated concentrations. Channel activation is not inhibited by desensitizing concentrations of ACh or ACh-competitive ligands (including alpha-bungarotoxin and D-tubocurarine) but is inhibited by antibody FK1 and several other compoun…

PharmacologyPhotoaffinity labelingChemistryPhysostigmineMolecular Sequence DataIn Vitro TechniquesReceptors NicotinicTorpedoIon ChannelsAcetylcholine bindingNicotinic acetylcholine receptorNicotinic agonistnervous systemBiochemistrymedicineAnimalsChannel blockerAmino Acid SequenceBinding siteAcetylcholineAcetylcholine receptormedicine.drugJournal of Receptor Research
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Nicotinic acetylcholine receptors have ligand-specific attachment point patterns.

1991

Employing a panel of synthetic peptides as representative structural elements of the nicotinic acetylcholine receptor from Torpedo electric organ, we recently identified three sequence regions of the receptor (alpha 55-74, alpha 134-153 and alpha 181-200) serving as subsites for the binding of high molecular weight antagonists of acetylcholine (Conti-Tronconi et al. 1990). The relative binding affinities to these subsites of alpha-bungarotoxin and three competitive antibodies varied in a ligand-specific fashion. Employing a set of homologous synthetic peptides differing from alpha 181-200 by the exchange of single amino acid residues along the sequence, we now find that ligand binding cruci…

Pharmacologychemistry.chemical_classificationChemistryStereochemistryMolecular Sequence DataAntibodies MonoclonalReceptors NicotinicLigand (biochemistry)LigandsTorpedolaw.inventionAmino acidNicotinic acetylcholine receptorNicotinic agonistlawmedicineAnimalsAmino Acid SequenceReceptorAcetylcholineTorpedoAcetylcholine receptormedicine.drugJournal of receptor research
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