Search results for "Nicotinic agonist"

showing 10 items of 118 documents

Nicotinic and muscarinic modulation of 5-hydroxytryptamine (5-HT) release from porcine and canine small intestine

1992

Strips of porcine and canine small intestine were incubated in vitro and the release of 5-hydroxytryptamine (5-HT) was determined by HPLC with electrochemical detection. The spontaneous outflow of 5-HT from the porcine and canine small intestine largely reflects calcium-dependent 5-HT secretion from enterochromaffin cells which are under a spontaneous neuronal, excitatory input as indicated by the inhibitory effect (30-40%) of tetrodotoxin. In both species, nicotine enhanced the release of 5-HT in a concentration-dependent manner by a maximum of about 50% at 100 microM. This effect was blocked by the nicotine receptor antagonist hexamethonium, but not by the subtype-selective nicotine recep…

MaleNicotineSerotoninmedicine.medical_specialtySwineScopolamineHexamethonium CompoundsTetrodotoxinReceptors NicotinicBiologyHexamethoniumNicotine03 medical and health scienceschemistry.chemical_compoundDogs0302 clinical medicineInternal medicineIntestine SmallDrug DiscoveryMuscarinic acetylcholine receptorEnterochromaffin CellsmedicineOxotremorineAnimalsGenetics (clinical)030304 developmental biology0303 health sciencesMuscarineOxotremorineParasympatholyticsGeneral MedicineHydroxyindoleacetic AcidBungarotoxinsReceptors MuscarinicAcetylcholine3. Good healthNicotinic agonistEndocrinologyParasympathomimeticschemistryEnterochromaffin cellMolecular MedicineCalciumFemaleHexamethoniumDimethylphenylpiperazinium Iodide030217 neurology & neurosurgeryAcetylcholinemedicine.drugThe Clinical Investigator
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Acute intermittent nicotine treatment induces fibroblast growth factor-2 in the subventricular zone of the adult rat brain and enhances neuronal prec…

2007

Abstract Over the past years, evidence has accumulated that stem cells are present in the adult brain, and generate neurons and/or glia from two active germinal zones: the subventricular zone (SVZ) of the lateral ventricles and the subgranular zone (SGZ) of the dentate gyrus of the hippocampus. This study shows that acute intermittent nicotine treatment significantly enhances neuronal precursor cell proliferation in the SVZ of adult rat brain, but not in the SGZ of the hippocampus, and pre-treatment with mecamylamine, a nonselective nAChR antagonist, blocks the enhanced precursor proliferation by nicotine. This effect is supported by up-regulation of fibroblast growth factor-2 (FGF-2) mRNA …

MaleNicotinemedicine.medical_specialtyBasic fibroblast growth factorSubventricular zoneNicotinic AntagonistsReceptors NicotinicBiologyFibroblast growth factorSettore BIO/09 - FisiologiaHippocampusSubgranular zonechemistry.chemical_compoundLateral VentriclesInternal medicinePrecursor cellmedicineAnimalsPyrrolesNicotinic AgonistsRNA MessengerReceptor Fibroblast Growth Factor Type 1Rats WistarCell ProliferationNeuronsNeuronal PlasticityStem CellsGeneral NeuroscienceFibroblast growth factor receptor 1Dentate gyrusNeurogenesisCell DifferentiationNerve RegenerationRatsUp-RegulationCell biologymedicine.anatomical_structureEndocrinologynervous systemchemistryneurogenesis FGF-2 FGFR-1 subventricular zone nicotineFibroblast Growth Factor 2Neuroscience
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Spatial learning in male mice with different levels of aggressiveness: effects of housing conditions and nicotine administration

2003

The main aim of the present investigation was to evaluate the possible modulation of spatial learning ability by housing conditions and level of aggressiveness in mice, also testing whether differences in locomotion and anxiety could influence this relationship. Additionally, we have examined effects of nicotine in the acquisition and retention of a spatial learning task in groups of mice differing in these variables. NMRI male mice were either group-housed or individually housed for 30 days and then classified into mice with short (SAL) and long (LAL) attack latency after a pre-screening agonistic encounter. Locomotor activity and baseline levels of anxiety of these groups were evaluated i…

MaleNicotinemedicine.medical_specialtyElevated plus mazeTime Factorsmedicine.drug_classSpatial BehaviorEscape responseWater mazeAnxietyMotor ActivitySocial EnvironmentAnxiolyticDevelopmental psychologyDiscrimination LearningNicotineMiceBehavioral NeuroscienceEscape ReactionInternal medicineReaction TimemedicineAnimalsNicotinic AgonistsMaze LearningAnalysis of VarianceBehavior AnimalDose-Response Relationship DrugHousing AnimalAggressionEndocrinologyNicotinic agonistSocial IsolationAnxiogenicAnalysis of variancePsychologymedicine.drugBehavioural Brain Research
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Regulation of α4β2α5 nicotinic acetylcholinergic receptors in rat cerebral cortex in early and late adolescence: Sex differences in response to chron…

2013

Chronic nicotine administration in animals, and smoking in humans, causes up-regulation of α4β2* neuronal nicotinic receptors (nAChRs), which has been hypothesized to contribute to the addictive actions of nicotine. We used a rat model to test whether such up-regulatory effects differ in adolescents versus adults, and in males versus females. Following chronic treatment with nicotine or saline via subcutaneous osmotic minipumps, we measured α4β2 and α4β2α5 nAChRs in cerebral cortex using [3H]epibatidine to label assembled nAChRs, and selective antibodies to measure the individual subunits via immunoprecipitation. For the first time, we provide a detailed characterization of the response of …

MaleNicotinemedicine.medical_specialtyPyridinesmedia_common.quotation_subjectReceptors NicotinicTritiumArticleRats Sprague-DawleyNicotineCellular and Molecular NeuroscienceDownregulation and upregulationInternal medicinemedicineAnimalsImmunoprecipitationNicotinic AgonistsReceptormedia_commonCerebral CortexPharmacologySex CharacteristicsBinding SitesAddictionBridged Bicyclo Compounds HeterocyclicUp-RegulationNicotinic agonistEndocrinologymedicine.anatomical_structureCerebral cortexEpibatidineFemalePsychologymedicine.drugSex characteristicsNeuropharmacology
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Nicotinic drugs and postganglionic sympathetic transmission

1970

1. Isolated rabbit hearts with the sympathetic nerves attached were perfused with Tyrode solution. The noradrenaline output into the perfusate was measured fluorimetrioally. 2. When the niootinic autoinhibition produced by infusions of nicotine, DMPP, or acetylcholine (in the presence of atropine) was fully developed, the output of noradrenaline evoked by electrical stimulation of the postganglionic sympathetic nerves was not depressed. 3. Acetylcholine in the presence of atropine produced a transitory facilitation of the noradrenaline output evoked by sympathetic nerve stimulation. 4. Prolonged infusion of DMPP caused an adrenergic neurone block which was not observed after nicotine, or ac…

MaleNicotinemedicine.medical_specialtySympathetic nervous systemSympathetic Nervous SystemReceptors DrugAdrenergicStimulationIn Vitro TechniquesSynaptic TransmissionPiperazinesNicotineNorepinephrineNorepinephrineInternal medicinemedicineAnimalsFluorometryGanglia AutonomicNerve EndingsPharmacologyChemistryHeartGeneral MedicineAcetylcholineElectric StimulationPerfusionAtropineEndocrinologyNicotinic agonistmedicine.anatomical_structureDepression ChemicalFemaleRabbitsAcetylcholinemedicine.drugNaunyn-Schmiedebergs Archiv f�r Pharmakologie
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Behavioral effects of combined environmental enrichment and chronic nicotine administration in male NMRI mice

2013

Abstract Environmental enrichment (EE) is an experimental paradigm which provides sensory, social, physical and cognitive stimulation for rodents. Experimental evidence indicates that this type of housing induces different neurobiological and behavioral changes. However, few studies have evaluated the consequences of combined exposure to an enriched environment and nicotine administration during a critical period of development such as adolescence. Taking into account previous studies, it can be hypothesized that a chronic treatment with nicotine would modulate the effects of rearing animals in enriched environments. In the current study, our main aim was to evaluate the effects of EE and c…

MaleNicotinemedicine.medical_specialtyTime FactorsDrinkingExperimental and Cognitive PsychologyEnvironmentInhibitory postsynaptic potentialDrug Administration ScheduleNicotineMiceBehavioral Neurosciencechemistry.chemical_compoundInternal medicineAnimals Outbred StrainsAvoidance LearningmedicineAnimalsNicotinic AgonistsCotinineMaze LearningAnalysis of VarianceEnvironmental enrichmentBehavior AnimalBody WeightInhibition PsychologicalEndocrinologyChronic nicotinechemistryNmri miceAnesthesiaExploratory BehaviorAnxietyCholinergicmedicine.symptomPsychologyCotinineLocomotionmedicine.drugPhysiology & Behavior
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Expression of α4-1 and α5 Nicotinic Cholinoceptor mRNA in the Aging Rat Cerebral Cortex

1997

Although important in neurodegeneration, systematic studies of nicotinic acetylcholine receptor expression in normal aging human brains are difficult to perform. We have studied the expression of nicotinic receptor alpha 4-1 and alpha 5 mRNA in the frontal and parietal isocortex of 3- (young adult), 24- (late middle aged), and 33-month-old (old) rats by nonisotopic in situ hybridization. In all groups transcripts were mainly present in layer II/III and V pyramidal neurons. The numerical densities of alpha 4-1 mRNA-containing neurons with respect to those of cresyl violet-stained neurons decreased with aging in the rat frontal and parietal cortex, while those of alpha 5 mRNA-containing neuro…

MaleSenescenceAgingmedicine.medical_specialtyPosterior parietal cortexReceptors NicotinicBiologyNicotineInternal medicinemedicineAnimalsRNA MessengerRats WistarIn Situ HybridizationAcetylcholine receptorCerebral CortexGeneral NeuroscienceNeurodegenerationmedicine.diseaseImmunohistochemistryRatsNicotinic acetylcholine receptorNicotinic agonistEndocrinologymedicine.anatomical_structureCerebral cortexNeurology (clinical)Geriatrics and GerontologyDevelopmental Biologymedicine.drugNeurobiology of Aging
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Nicotinic cholinoceptive neurons of the frontal cortex are reduced in Alzheimer's disease.

1991

The cellular distribution of nicotinic acetylcholine receptors was studied in the frontal cortex (area 10) of 1) Alzheimer patients and compared to 2) age-matched and 3) middle-aged controls using the monoclonal antibody WF 6 and an immunoperoxidase protocol. Statistical analysis revealed significant differences between the number of labeled neurons among all three groups tested (middle-aged controls greater than aged controls greater than Alzheimer cases). No differences were seen for cresyl violet-stained samples. These findings underline that the nicotinic receptor decrease found with radioligand binding may reflect a postsynaptic in addition to a presynaptic component.

Malemedicine.medical_specialtyAgingImmunocytochemistryBiologyReceptors NicotinicImmunoenzyme TechniquesPostsynaptic potentialAlzheimer DiseaseInternal medicineOxazinesmedicineHumansReceptorAcetylcholine receptorAgedNeuronsImmunoperoxidaseGeneral NeuroscienceAntibodies Monoclonalmedicine.diseaseBenzoxazinesFrontal Lobemedicine.anatomical_structureEndocrinologyNicotinic agonistCerebral cortexFemaleNeurology (clinical)Geriatrics and GerontologyAlzheimer's diseaseDevelopmental BiologyNeurobiology of aging
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Nicotine-induced fibroblast growth factor-2 restores the age-related decline of precursor cell proliferation in the subventricular zone of rat brain.

2007

Precursor cell proliferation is present in the subventricular zone (SVZ) of the lateral ventricles and the subgranular zone (SGZ) of the dentate gyrus of the hippocampus of adult rat and persists during aging although at reduced levels. Previous studies have shown that acute intermittent nicotine treatment significantly increases fibroblast growth factor-2 (FGF-2) expression in several brain regions of aged rats. The aim of the present investigation was to test the hypothesis that nicotine-induced expression of FGF-2 may restore the age-related decline of precursor cell proliferation. It was first demonstrated that nicotine treatment increases both mRNA and protein FGF-2 in the SVZ of aged …

Malemedicine.medical_specialtyAgingNicotineBasic fibroblast growth factorSubventricular zoneCell CountNerve Tissue ProteinsBiologyFibroblast growth factorSettore BIO/09 - FisiologiaAntibodiesSubgranular zoneNestinchemistry.chemical_compoundIntermediate Filament ProteinsInternal medicinePrecursor cellLateral VentriclesGlial Fibrillary Acidic ProteinmedicineAnimalsNicotinic AgonistsRats WistarMolecular BiologyCell ProliferationAnalysis of VarianceCell growthGeneral NeuroscienceDentate gyrusFibroblast growth factor receptor 1BrainPrecursor proliferationFGF-2 FGFR1 SVZ Nicotine AgedRatsAdult Stem CellsEndocrinologymedicine.anatomical_structurenervous systemchemistryBromodeoxyuridineGene Expression RegulationFibroblast Growth Factor 2Neurology (clinical)Developmental BiologyBrain research
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Nicotinic cholinoceptors in the rat pineal gland as analyzed by Western blot, light- and electron microscopy

1992

Abstract The monoclonal antibody WF6, raised against purified Torpedo nicotinic acetylcholine receptor (nAChR) was used to study the distribution of cholinoceptors in the rat pineal gland by means of Western blot analysis, light- and electron microscopy. The immunoblot analysis using homogenized pineal gland revealed a labeled protein band of apparent molecular weight 40 kDa which was identified as α-subunits of a nAChR. In the light microscope, approximately one-fourth of the pinealocytes exhibited cytoplasmic immunoreactivity (IR) of varying density. In the electron microscope, IR was seen as patchy staining of cell membranes of pinealocyte somata and processes. Presynaptic IR material wa…

Malemedicine.medical_specialtyBlotting WesternImmunocytochemistryReceptors NicotinicBiologyPineal GlandPinealocytelaw.inventionPineal glandWestern blotAntibody SpecificitylawInternal medicinemedicineAnimalsMolecular BiologyCerebral Cortexmedicine.diagnostic_testGeneral NeuroscienceAntibodies MonoclonalRats Inbred StrainsImmunohistochemistryMolecular biologyRatsMicroscopy ElectronNicotinic acetylcholine receptormedicine.anatomical_structureNicotinic agonistEndocrinologynervous systemElectrophoresis Polyacrylamide GelFemaleNeurology (clinical)Electron microscopeDevelopmental BiologyEndocrine glandBrain Research
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