Search results for "Non alcoholic"

showing 9 items of 39 documents

P1013 : Chronic intermittent hypoxia is associated with liver damage and atherosclerosis in patients with non-alcoholic fatty liver disease

2015

all the steatosis grades, except S2 vs S3. The diagnostic performances of CAP in quantifying each steatosis grade was: for S ≥1 AUC=0.813 (cutoff 260dB/m, Se = 64.84%, Sp =87.27%, PPV=80.8%, NPV=75%, +LR =5.09, −LR =0.40, DA=76.11%); for S ≥2 AUC=0.822 (cutoff 285dB/m, Se = 69.70%, Sp =85.12%, PPV=47.9%, NPV=93.5%, +LR =4.68, −LR =0.36, DA=82.08%); for S ≥3 AUC=0.838 (cutoff 294dB/m, Se =83.33%, Sp =82.54%, PPV=23.3%, NPV=98.7%, +LR =4.77, −LR =0.20, DA=81.59%). AUCs calculated between two steatosis grades only were: 0.772 (for S0 vs S1), 0.874 (S0 vs S2), 0.904 (S0 vs S3), 0.659 (S1 vs S2), 0.777 (S1 vs S3), and 0.665 (S2 vs S3) respectively. Conclusions: Maximal diagnostic accuracy could …

medicine.medical_specialtyHepatologybusiness.industryFatty liverNon alcoholicDiseasemedicine.diseaseGastroenterologyInternal medicinemedicineCutoffChronic intermittent hypoxiaIn patientLiver damageSteatosisbusinessJournal of Hepatology
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Letter: coronary atherosclerosis in patients with significant hepatic fibrosis in non‐alcoholic fatty liver disease—the role for non‐invasive testing

2021

medicine.medical_specialtyHepatologybusiness.industryFatty liverNon invasiveGastroenterologyNon alcoholicDiseasemedicine.diseaseGastroenterologyText miningInternal medicinemedicinePharmacology (medical)In patientbusinessHepatic fibrosisCoronary atherosclerosisAlimentary Pharmacology & Therapeutics
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OC.03.2 THE PRESENCE OF WHITE MATTER LESIONS IS ASSOCIATED WITH THE HISTOLOGICAL SEVERITY OF NON-ALCOHOLIC FATTY LIVER DISEASE

2016

medicine.medical_specialtyHepatologybusiness.industryInternal medicineFatty liverGastroenterologyMedicineNon alcoholicDiseasebusinessmedicine.diseaseGastroenterologyHyperintensityDigestive and Liver Disease
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Early menopausal status is associated with the severity of liver fibrosis in Italian patients with non-alcoholic fatty liver disease

2014

medicine.medical_specialtyHepatologybusiness.industrySettore MED/12 - GASTROENTEROLOGIALiver fibrosisFatty liverGastroenterologyNon alcoholicDiseasemedicine.diseaseGastroenterologyInternal medicinemedicinebusinessDigestive and Liver Disease
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Visceral adiposity index and exercise in non-alcoholic fatty liver disease: authors’ reply

2012

We thank Prof. Filik for his interest in our recent article. 2 He points to the fact that the interpretation of our results could be affected by lack of data on physical activity and diet. In response to this issue, we are aware that both physical activity and diet are able to affect not only anthropometric and metabolic parameters of visceral adiposity index (VAI) but also the severity of liver disease. Unfortunately, data on both physical activity and diet in our patients with biopsy-proven non-alcoholic fatty liver disease (NAFLD) are not available, even if we are confident that their variations should not significantly affect our results. In fact, in our study, we evaluated histological…

medicine.medical_specialtyHepatologybusiness.industrySurrogate endpointFatty liverGastroenterologyNon alcoholicDiseaseAnthropometrymedicine.diseaseGastroenterologyLiver diseaseEndocrinologyInternal medicineMedicinePharmacology (medical)Liver damagebusinessMETABOLIC FEATURESAlimentary Pharmacology & Therapeutics
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Steatohepatitis and type 2 diabetes mellitus are influenced by genetic susceptibility to increased intestinal permeability in patients with non-alcoh…

2017

medicine.medical_specialtyIntestinal permeabilityHepatologybusiness.industrySettore MED/12 - GASTROENTEROLOGIAFatty liverGastroenterologyType 2 Diabetes MellitusNon alcoholicDiseasemedicine.diseaseGastroenterologyN/AInternal medicinemedicineGenetic predispositionIn patientSteatohepatitisbusinessDigestive and Liver Disease
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GEMS, a GEnetic and Metabolic Staging predicting the outcome of non-alcoholic fatty liver disease

2021

Background and Aim: Non-alcoholic fatty liver disease(NAFLD) is an emergent cause of liver-related events(LRE). We have assessed the ability of a composite score based on clinical features, metabolic comorbidities and genetic background, to predict LRE. Methods: 546 consecutive patients with NAFLD were recruited and stratified according to FIB-4(low risk <1.3;intermedium-high risk ≥1.3). LRE were defined as occurrence of HCC or hepatic decompensation. Cox regression multivariate analysis was used to identify baseline variables associated with LRE. Results: Over a median follow-up of 73.8 months, 58 patients experienced LRE(only 1 in the cohort of patients with FIB-4 65 years(HR 17.96), plat…

medicine.medical_specialtyMultivariate analysisHepatologyProportional hazards modelbusiness.industryFatty liverGastroenterologyNon alcoholicDiseasemedicine.diseaseOutcome (game theory)GastroenterologyHelsinki declarationHepatocellular carcinomaDiabetes mellitusInternal medicineCohortMedicinebusinessTM6SF2Digestive and Liver Disease
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HEPATIC EXPRESSION OF CYCLOOXYGENASE-2 IN NON-ALCOHOLIC FATTY LIVER DISEASE

2008

medicine.medical_specialtybiologybusiness.industryFatty liverNon alcoholicDiseasemedicine.diseaseEndocrinologyInternal medicineInternal Medicinemedicinebiology.proteinCyclooxygenasebusinessEuropean Journal of Internal Medicine
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Involvement of B cells in Non-Alcoholic Steatohepatitis (NASH)

2019

medicine.medical_specialtybusiness.industryInternal medicinemedicineNon alcoholicSteatohepatitismedicine.diseasebusinessGastroenterology35. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber
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