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showing 10 items of 2571 documents

Evidence of oxidative stress in very long chain fatty acid--treated oligodendrocytes and potentialization of ROS production using RNA interference-di…

2011

X-linked adrenoleukodystrophy (X-ALD) and pseudo neonatal adrenoleukodystrophy (P-NALD) are neurodegenerative demyelinating diseases resulting from the functional loss of the peroxisomal ATP-binding cassette transporter D (ABCD1) and from single peroxisomal enzyme deficiency (Acyl-CoA oxidase1: ACOX1), respectively. As these proteins are involved in the catabolism of very long chain fatty acids (VLCFA: C24:0, C26:0), X-ALD and P-NALD patients are characterized by the accumulation of VLCFA in plasma and tissues. Since peroxisomes are involved in the metabolism of reactive oxygen species (ROS) and nitrogen species (RNS), we examined the impact of VLCFA on the oxidative status of 158N murine o…

congenital hereditary and neonatal diseases and abnormalitiesendocrine systemendocrine system diseasesVery long chain fatty acidBlotting Westernmedicine.disease_causeReal-Time Polymerase Chain ReactionTransfectionATP Binding Cassette Transporter Subfamily D Member 1Gas Chromatography-Mass SpectrometrySuperoxide dismutaseLipid peroxidationchemistry.chemical_compoundMicemedicinePeroxisomesAnimalsAdrenoleukodystrophyCells Culturedchemistry.chemical_classificationReactive oxygen speciesbiologyReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceFatty Acidsnutritional and metabolic diseasesPeroxisomemedicine.diseaseFlow CytometryOligodendrogliaOxidative StressBiochemistrychemistryGene Knockdown Techniquesbiology.proteinACOX1AdrenoleukodystrophyATP-Binding Cassette TransportersRNA InterferenceAcyl-CoA OxidaseReactive Oxygen SpeciesOxidation-ReductionOxidative stressNeuroscience
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Follow-up in transthyretin familial amyloid polyneuropathy: Useful investigations

2020

Patients with transthyretin amyloid polyneuropathy (TTR-FAP) and asymptomatic mutation-carriers have to be regularly followed-up in order to identify disease progression and the time point for starting or modifying therapy. In this case series we describe the potential suitability of different variables as progression markers. We retrospectively analyzed the follow-up charts of 10 TTR-FAP patients. Clinical examination included the Neuropathy Impairment Score of Lower Limb (NIS-LL), temperature perception thresholds, nerve conduction and autonomic function tests. The NIS-LL had the greatest value for a sensitive and correct follow-up for all TTR-FAP stages. All other examinations provided u…

congenital hereditary and neonatal diseases and abnormalitiesendocrine systemmedicine.medical_specialtyNeural ConductionPhysical examinationAsymptomatic03 medical and health sciences0302 clinical medicineClinical investigationInternal medicinemedicineHumansPrealbumin030212 general & internal medicineRetrospective StudiesAmyloid Neuropathies Familialbiologymedicine.diagnostic_testbusiness.industryAmyloidosisDisease progressionnutritional and metabolic diseasesmedicine.diseasedigestive system diseasesTransthyretinNeurologybiology.proteinAmyloid polyneuropathyNeurology (clinical)medicine.symptombusinessPolyneuropathy030217 neurology & neurosurgeryFollow-Up StudiesJournal of the Neurological Sciences
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Echocardiographic diagnosis of Candida endocarditis of the tricuspid valve and of the right atrium in a young infant.

1983

Systemic candidiasis developed in a seven-week-old premature baby after 6 weeks treatment with antibiotics for suspected septicemia. At that time the echocardiogram showed a dense layer of echoes posteriorly to the anterior tricuspid leaflet during atrial systole. The diagnosis of Candida endocarditis with vegetations on the tricuspid valve and with right atrial thrombus secondary to the Candida infection was verified by autopsy.

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyAutopsyInfant Premature DiseasesInternal medicinemedicineEndocarditisHumanscardiovascular diseasesHeart AtriaTricuspid valveCardiac cycleEndocarditisbusiness.industryCandidiasisInfant NewbornInfantVascular surgerymedicine.diseaseCardiac surgeryAnti-Bacterial AgentsRight Atrial Thrombusmedicine.anatomical_structureEchocardiographyPediatrics Perinatology and Child Healthcardiovascular systemCardiologySystemic candidiasisTricuspid ValveCardiology and Cardiovascular MedicinebusinessPediatric cardiology
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Langer-Giedion syndrome with interstitial 8q-deletion.

1982

We describe a 12-year-old girl with Langer-Giedion syndrome (tricho-rhino-phalangeal syndrome type II) who also had vertebral malformations. Chromosome analysis identified an interstitial del(8q): 46,XX,del(8)(pter leads to q22::q234 leads to qter) as a cause of this syndrome.

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyChromosome DisordersNoseBone and BonesLanger–Giedion syndromeFingersChromosome analysisInternal medicineIntellectual Disabilityotorhinolaryngologic diseasesMedicineHumansAbnormalities MultipleChildGenetics (clinical)Chromosome AberrationsChromosomes Human 6-12 and XSyndrome typebusiness.industryAnatomySyndromemedicine.diseaseSpineChromosome BandingEndocrinologyKaryotypingFemaleChromosome DeletionbusinessAmerican journal of medical genetics
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Musculoskeletal ultrasound in hemophilia: Results and recommendations from a global survey and consensus meeting

2021

Abstract Introduction For persons with hemophilia, optimization of joint outcomes is an important unmet need. The aim of this initiative was to determine use of ultrasound in evaluating arthropathy in persons with hemophilia, and to move toward consensus among hemophilia care providers regarding the preferred ultrasound protocols for global adaptation. Methods A global survey of hemophilia treatment centers was conducted that focused on understanding how and why ultrasound was being used and endeavored to move toward consensus definitions of both point‐of‐care musculoskeletal ultrasound (POC‐MSKUS) and full diagnostic ultrasound, terminology to describe structures being assessed by ultrasou…

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyDiagnostic ultrasoundMusculoskeletal ultrasoundUnmet needsTerminologysurveyshemic and lymphatic diseaseshemophiliaHealth careMedicineImage acquisitionMedical physicsDiseases of the blood and blood-forming organsClinical caremusculoskeletalbusiness.industryOriginal ArticlesHematologyultrasonographyconsensus hemophilia musculoskeletal surveys ultrasonographyconsensusOriginal ArticlePatient representativesRC633-647.5businessResearch and Practice in Thrombosis and Haemostasis
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Increased susceptibility of microcytic red blood cells to in vitro oxidative stress.

1995

Oxidative damage to erythrocytes in thalassaemia has been related to generation of free radicals by an excess of denaturated alpha- or beta-globin chains, intracellular iron overload and low concentration of normal haemoglobin (HGB). Two good indicators of such oxidative damage are the high red blood cell (RBC) malonyldialdehyde (MDA) production detected following exogenous oxidant stress and the decrease of pyrimidine 5'-nucleotidase (P5N), the most sensitive enzyme to SH-group damage in vivo. Conflicting data, however, have so far accumulated in the literature concerning differences in oxidative damage between the different forms of thalassaemia and iron deficiency anaemia (IDA). In the p…

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyErythrocytes Abnormalmedicine.disease_causeSuperoxide dismutasechemistry.chemical_compoundhemic and lymphatic diseasesInternal medicineMalondialdehydemedicineHumans5'-Nucleotidasechemistry.chemical_classificationAnemia HypochromicGlutathione PeroxidasebiologySuperoxide DismutaseGlutathione peroxidaseMicrocytosisHematologyGeneral MedicineGlutathioneIron deficiencymedicine.diseaseGlutathioneRed blood cellOxidative StressEndocrinologymedicine.anatomical_structurechemistryIron-deficiency anemiaImmunologybiology.proteinThalassemiaReactive Oxygen SpeciesOxidative stressEuropean journal of haematology
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Evaluation of the risk factors of asymptomatic vertebral fractures in postmenopausal women with osteopenia at the femoral neck

2016

To identify risk factors of asymptomatic vertebral fracture (aVF) in postmenopausal women with osteopenia at the femoral neck and to evaluate the association between the number of aVFs and the risk of major and hip osteoporotic fracture calculated with the FRAX(®) algorithm.Epidemiological case-series study with data collected transversally.728 postmenopausal women with osteopenia were included: 284 (39.0%) had aVF, of whom 200 (70.4%) had prior fragility fractures (FF). The likelihood of having an osteoporotic fracture in the next 10 years increased significantly with the number of aVF. The percentage of women with height loss, which was assessed as the difference between the greatest heig…

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyFRAXBone density030209 endocrinology & metabolismRisk AssessmentAsymptomaticGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineBone DensityRisk FactorsEpidemiologymedicineHumanscardiovascular diseases030212 general & internal medicineAgedRetrospective StudiesFemoral neckFemur NeckHip Fracturesbusiness.industryObstetrics and GynecologyRetrospective cohort studyMiddle Agedmedicine.diseaseSurgeryPostmenopauseOsteopeniaBone Diseases Metabolicmedicine.anatomical_structureSpainMultivariate AnalysisSpinal FracturesFemalemedicine.symptombusinessRisk assessmentAlgorithmsOsteoporotic Fractures
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A systematic overview of the first pasteurised VWF/FVIII medicinal product, Haemate P/Humate -P: history and clinical performance

2008

Patients with von Willebrand disease (VWD) and haemophilia A (HA) lack, to varying degrees, the von Willebrand factor (VWF) and coagulation factor VIII (FVIII) that are critical for normal haemostasis. These conditions in turn make patients prone to uncontrolled bleeding. Historically, patients with severe forms of VWD or HA were crippled before adulthood and their life expectancy was significantly reduced. Over the past decades, specific coagulation factor replacement therapies including Haemate P, have been developed to help patients achieve and maintain normal haemostasis. Haemate P is a human, plasma-derived VWF/FVIII medicinal product, which was first licensed in Germany in 1981 for th…

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyHaemophilia AHemophilia AHaemophiliaHaemate P; Humate-P; von Willebrand disease; von Willebrand factor; haemophilia; factor VIIIVon Willebrand factorhemic and lymphatic diseasesInternal medicinevon Willebrand FactormedicineVon Willebrand diseaseHumansDosingDesmopressinHematologybiologybusiness.industryHematologyGeneral Medicinemedicine.diseasevon Willebrand DiseasesCoagulationImmunologybiology.proteinSafetybusinessSettore MED/15 - Malattie del Sanguemedicine.drug
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Photoplethysmography Analysis of Artery Properties in Patients with Cardiovascular Diseases

2008

In this study arterial parameters of healthy subjects were compared to those of patients with cardiovascular diseases. The photoplethysmography (PPG) measurements of blood volume pulsations have been performed. Using a novel algorithm for analysis of simultaneously measured ear and finger PPG signals, arterial parameters were evaluated in representative groups of healthy subjects and patients with cardiovascular diseases. Digital volume pulse (DVP), pulse cycle duration (T), augmentation index (AIx), reflection index (RI) and transit time of reflected wave (RTT) were evaluated in every heartbeat cycle. Correlations between the AIx and RI, T and RTT, AIx and standard deviation of AIx, RTT an…

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyHeartbeatPulse (signal processing)business.industryTransit timeBlood volumemedicine.diseasemedicine.anatomical_structureInternal medicinePhotoplethysmogrammedicineArterial stiffnessCardiologyIn patientbusinessArtery
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Genetische Hämochromatose und das HFE-Gen: von der Molekulargenetik zur klinischen Diagnostik

2000

More than 90% of patients with genetic hemochromatosis carry a characteristic mutation in the HFE-gene (C282Y). HFE modulates the iron uptake by the transferrin receptor. Duodenal crypt cells of HFE-knockout mice show low intracellular iron concentrations which lead to an upregulation of the divalent metal transporter and enhanced iron uptake by duodenal enterocytes. Heterozygosity for the C282Y mutation appears to alter the course of other liver diseases like porphyria cutanea tarda and nonalcoholic steatohepatitis.

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyMutationdigestive oral and skin physiologyGastroenterologynutritional and metabolic diseasesTransferrin receptorBiologymedicine.diseasemedicine.disease_causedigestive systemPathogenesisLoss of heterozygosityEndocrinologyDownregulation and upregulationInternal medicineMolecular geneticsmedicinePorphyria cutanea tardaskin and connective tissue diseasesHemochromatosisZeitschrift für Gastroenterologie
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