Search results for "Notch"

showing 10 items of 93 documents

Expression analysis of jagged genes in zebrafish embryos

2005

The interaction of transmembrane Delta and Jagged/Serrate ligands with Notch receptors on neighboring cells is critically involved in cell specification during development. In zebrafish, the early expression of delta but not of jagged genes has been investigated in some detail. We have analyzed the sequence and embryonic expression pattern of the three zebrafish genes jagged1a, jagged1b, and jagged2. These genes, whose transcripts are detectable by in situ hybridization from early somitogenesis, are widely and dynamically expressed in embryos. Coexpression is limited, however, to the notochord and lens (jagged1a and jagged1b) and to the otic vesicle and pronephros (jagged1b and jagged2). Co…

Nervous systemanimal structuresNotchNotch signaling pathwayNotochordBiologystomatognathic systemSomitogenesisNotochordmedicineAnimalsPancreaSerrate-Jagged ProteinsSomitePlacodeZebrafishPhylogenyNotch signalingZebrafishGeneticsVertebrateCalcium-Binding ProteinsGene Expression Regulation DevelopmentalMembrane ProteinsCell BiologyZebrafish Proteinsbiology.organism_classificationCell biologyPronephrosmedicine.anatomical_structurezebrafish; Notch; JaggedEmbryoIntercellular Signaling Peptides and ProteinsPronephroOtic vesicleJaggedJagged-2 ProteinOtic PlacodesDevelopmental biologyIn situ hybridizationJagged/serrate geneEmbryo; In situ hybridization; Jagged/serrate genes; Nervous system; Notch signaling; Notochord; Pancreas; Placodes; Pronephros; Somites; Vertebrate; Zebrafish; Developmental Biology; Cell BiologyDevelopmental Biology
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The

2016

ABSTRACT Members of the Junctophilin (JPH) protein family have emerged as key actors in all excitable cells, with crucial implications for human pathophysiology. In mammals, this family consists of four members (JPH1-JPH4) that are differentially expressed throughout excitable cells. The analysis of knockout mice lacking JPH subtypes has demonstrated their essential contribution to physiological functions in skeletal and cardiac muscles and in neurons. Moreover, mutations in the human JPH2 gene are associated with hypertrophic and dilated cardiomyopathies; mutations in JPH3 are responsible for the neurodegenerative Huntington's disease-like-2 (HDL2), whereas JPH1 acts as a genetic modifier …

NotchGenotypeCardiomyopathyGenes InsectAnimals Genetically ModifiedAnimalsDrosophila ProteinsAllelesMammalsNeuronsHuntingtin ProteinReceptors NotchMusclesMyocardiumMembrane ProteinsReproducibility of ResultsDrosHuntington's diseaseDisease Models AnimalDrosophila melanogasterPhenotypeGene Knockdown TechniquesMutationNerve DegenerationPhotoreceptor Cells InvertebrateRNA InterferenceJunctophilinDrosophilaTrinucleotide Repeat ExpansionSignal TransductionResearch ArticleDisease modelsmechanisms
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The Endothelial Transcription Factor ERG Mediates a Differential Role in the Aneurysmatic Ascending Aorta with Bicuspid or Tricuspid Aorta Valve: A P…

2022

The pathobiology of ascending aorta aneurysms (AAA) onset and progression is not well understood and only partially characterized. AAA are also complicated in case of bicuspid aorta valve (BAV) anatomy. There is emerging evidence about the crucial role of endothelium-related pathways, which show in AAA an altered expression and function. Here, we examined the involvement of ERG-related pathways in the differential progression of disease in aortic tissues from patients having a BAV or tricuspid aorta valve (TAV) with or without AAA. Our findings identified ERG as a novel endothelial-specific regulator of TGF-β-SMAD, Notch, and NO pathways, by modulating a differential fibrotic or calcified A…

NotchHeart Valve DiseasesCatalysisInorganic ChemistryBicuspid Aortic Valve DiseaseTranscriptional Regulator ERGascending aorta aneurysm; bicuspid aorta valve; tricuspid aorta valve; ERG transcriptional factor pathway; TGF-β-SMAD; Notch; NO pathways modulationTransforming Growth Factor betaSettore MED/05 - Patologia ClinicaHumansTGF-β-SMADEndotheliumPhysical and Theoretical ChemistryMolecular BiologySpectroscopyAortabicuspid aorta valveOrganic ChemistryERG transcriptional factor pathwayGeneral Medicineascending aorta aneurysmComputer Science ApplicationsSettore MED/23Aortic Valvetricuspid aorta valveNO pathways modulationBiomarkersTranscription FactorsInternational Journal of Molecular Sciences; Volume 23; Issue 18; Pages: 10848
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Endothelial Wnt/β-catenin signaling inhibits glioma angiogenesis and normalizes tumor blood vessels by inducing PDGF-B expression

2012

Wnt modulates glioma vascularization by regulating PDGF-B expression.

PathologyAngiogenesisCentral Nervous System NeoplasmsMice0302 clinical medicineImmunology and AllergyWnt Signaling Pathwaybeta Catenin0303 health sciencesbiologyNeovascularization PathologicBrain NeoplasmsWnt signaling pathwayIntracellular Signaling Peptides and ProteinsForkhead Transcription FactorsGliomaProto-Oncogene Proteins c-sis3. Good healthmedicine.anatomical_structureBlood-Brain Barrier030220 oncology & carcinogenesiscardiovascular systemIntercellular Signaling Peptides and ProteinsFemalemedicine.medical_specialtyBeta-cateninEndotheliumImmunologyNotch signaling pathwayMice NudeWnt1 ProteinMural cellArticle03 medical and health sciencesGliomamedicineAnimalsHumansddc:610neoplasms030304 developmental biologyAdaptor Proteins Signal TransducingCalcium-Binding ProteinsMembrane Proteinsmedicine.diseaseXenograft Model Antitumor Assaysnervous system diseasesDKK1Cancer researchbiology.proteinEndothelium VascularNeoplasm Grading
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Notch signalling is off and is uncoupled from HES1 expression in Ewing's sarcoma

2010

Notch can act as an oncogene or as a tumour suppressor and thus can either promote or inhibit tumour cell growth. To establish Notch status in Ewing's sarcoma family of tumours (ESFT), we investigated the Notch pathway by gene expression profiling meta-analysis or immunohistochemistry in samples obtained from 96 and 24 ESFT patients, respectively. We found that although Notch receptors were highly expressed, Notch did not appear to be active, as evidenced by the absence of Notch receptors in cell nuclei. In contrast, we show that Notch receptors known to be active in colon adenocarcinoma, hepatocarcinoma, and pancreatic carcinoma stain cell nuclei in these tumours. High expression of the No…

Pathologymedicine.medical_specialtyCellNotch signaling pathwayBone NeoplasmsSarcoma EwingBiologyPathology and Forensic MedicineBasic Helix-Loop-Helix Transcription FactorsTumor Cells CulturedmedicineHumansHES1HEY1Transcription factorCell ProliferationCell NucleusHomeodomain ProteinsRegulation of gene expressionReceptors NotchCell growthGene Expression ProfilingNeoplasm ProteinsGene Expression Regulation Neoplasticmedicine.anatomical_structureNeoplastic Stem CellsCancer researchTranscription Factor HES-1Cyclin-dependent kinase 8Signal TransductionThe Journal of Pathology
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Factores pronósticos del resultado clínico de las prótesis de cabeza de radio

2019

espanolObjetivo. El sobredimensionamiento de las protesis de cabeza del radio se ha asociado a resultados clinicos no satisfactorios junto aerosiones de capitellum. El objetivo del estudio es identificar los factores, tanto clinicos como radiologicos que han influido en nuestra serie. Material y metodo. Estudio analitico transversal de una muestra de 31 pacientes entre 2005 y 2017 con un seguimiento medio de 5 anos. Analisis de variables cualititativas de edad, sexo, tipo de lesion y tipo de tratamiento utilizado y analisis del tamano del implante, mediante mediciones radiologicas (implante y margen proximal de la fosa sigmoidea cubital y angulo ulnohumeral lateral) realizadas por evaluador…

Pharmacologybusiness.industryProximal marginUNESCO::CIENCIAS MÉDICASMedicineRadial head:CIENCIAS MÉDICAS [UNESCO]businessNuclear medicineSigmoid notch
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Selective Modulation of Aβ42 Production in Alzheimers Disease: Non-Steroidal Anti-Inflammatory Drugs and Beyond

2006

The amyloid-β (Aβ) peptides and in particular the longer, highly amyloidogenic isoform Aβ42 are believed by many to be the central disease-causing agents in Alzheimers disease (AD). Consequently, academic and pharmaceutical laboratories have focused on elucidating the mechanisms of Aβ production and developing strategies to diminish Aβ formation for treatment or prevention of AD. The most substantial advances have been made with respect to inhibitors of the γ-secretase enzyme, which catalyzes the final step in the generation of Aβ from the amyloid precursor protein (APP). Highly potent γ-secretase inhibitors which suppress production of all Aβ peptides are available today. However, due to t…

Pharmacologychemistry.chemical_classificationGene isoformbiologybusiness.industryNotch signaling pathwayPharmacologymedicine.diseaseSmall moleculePathogenesisEnzymechemistryMechanism of actionDrug DiscoveryImmunologymedicineAmyloid precursor proteinbiology.proteinAlzheimer's diseasemedicine.symptombusinessCurrent Pharmaceutical Design
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Functional antagonism between histone H3K4 demethylases in vivo

2011

Dynamic regulation of histone modifications is critical during development, and aberrant activity of chromatin-modifying enzymes has been associated with diseases such as cancer. Histone demethylases have been shown to play a key role in eukaryotic gene transcription; however, little is known about how their activities are coordinated in vivo to regulate specific biological processes. In Drosophila, two enzymes, dLsd1 (Drosophila ortholog of lysine-specific demethylase 1) and Lid (little imaginal discs), demethylate histone H3 at Lys 4 (H3K4), a residue whose methylation is associated with actively transcribed genes. Our studies show that compound mutation of Lid and dLsd1 results in increa…

Settore BIO/11 - Biologia MolecolareBiologyMethylationHistoneshistone demethylasesHistone H3HeterochromatinHistone H2AHistone methylationGeneticsAnimalsDrosophila ProteinsHistone codeGeneticsReceptors NotchEZH2Oxidoreductases N-DemethylatingHistone-Lysine N-MethyltransferaseSettore BIO/18 - GeneticaDrosophila melanogasterPhenotypeGene Expression RegulationHistone methyltransferaseMutationHeterochromatin protein 1Histone DemethylasesSignal TransductionResearch PaperDevelopmental Biology
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Quasi-Ductile Behaviour of Carbon-Reinforced Carbon

1991

Firstly a brief review of the problem of a “ductilization” of ceramic materials by endless-fiber reinforcement is given. The problems, especially the influence of the frictional stress in the fiber-matrix interface, are pointed out for the unidirectionally reinforced ceramic matrix composite (CMC) system LAS/SiC, which is well documented in the literature.

Stress (mechanics)Notch rootMaterials sciencechemistrychemistry.chemical_elementComposite materialReinforcementCeramic matrix compositeCarbon
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Efficient differentiation of embryonic stem cells into mesodermal precursors by BMP, retinoic acid and Notch signalling

2012

The ability to direct differentiation of mouse embryonic stem (ES) cells into specific lineages not only provides new insights into the pathways that regulate lineage selection but also has translational applications, for example in drug discovery. We set out to develop a method of differentiating ES cells into mesodermal cells at high efficiency without first having to induce embryoid body formation. ES cells were plated on a feeder layer of PA6 cells, which have membrane-associated stromal-derived inducing activity (SDIA), the molecular basis of which is currently unknown. Stimulation of ES/PA6 co-cultures with Bone Morphogenetic Protein 4 (BMP4) both favoured self-renewal of ES cells and…

Stromal cellCellular differentiationMyocytes Smooth MuscleNotch signaling pathwaylcsh:MedicineDevelopmental SignalingTretinoinEmbryoid bodyBiologyCell LineMesoderm03 medical and health sciencesMice0302 clinical medicineRetinoic Acid Signaling CascadeMolecular Cell BiologyExpressió genèticaAnimalslcsh:ScienceBiologyEmbryonic Stem Cells030304 developmental biology0303 health sciencesMultidisciplinaryReceptors NotchStem Cellslcsh:RComputational BiologyCell DifferentiationNestinSignaling in Selected DisciplinesMolecular biologyEmbryonic stem cellSignaling CascadesSignaling NetworksP19 cellBone morphogenetic protein 4embryonic structuresBone Morphogenetic Proteinslcsh:QCellular TypesStromal CellsTranscriptomeCèl·lules mare030217 neurology & neurosurgeryResearch ArticleDevelopmental BiologySignal Transduction
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