Search results for "Nystagmu"

showing 10 items of 50 documents

Dizziness and vertigo in a department of emergency medicine.

1995

Dizziness is a common and vexing diagnostic problem in emergency departments. The term is rather undefinite and often misused, but can in practice be classified into four categories: fainting, disequilibrium, vertigo and miscellaneous syndromes. Vertigo is the most common category of dizziness. Classification of vertigo can be based either on chronological criteria (acute, recurrent or chronic vertigo) or on topographical criteria (peripheral or central vertigo). Physicians working in emergency departments must be able to rapidly identify patients with potentially serious forms of vertigo, which could cause death or disability, and patients with mild conditions, that can be effectively trea…

Chronic vertigoMalemedicine.medical_specialtySedationPhysical examinationDiseaseNystagmusFaintingDizzinessDiagnosis DifferentialClinical historyVertigootorhinolaryngologic diseasesmedicineHumansSyphilisEar DiseasesPhysical Examinationbiologymedicine.diagnostic_testbusiness.industrybiology.organism_classificationEmergency medicineEmergency MedicineVertigoFemalemedicine.symptomNervous System DiseasesbusinessEmergency Service HospitalAlgorithmsEuropean journal of emergency medicine : official journal of the European Society for Emergency Medicine
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V11. Functional hierarchy within an overall network for visual motion processing and ocular-motor control at rest

2015

Introduction Visual motion processing on one hand and ocular motor functions on the other are rarely studied together in vivo in humans. The interrelation of these functional networks is rather unclear, even though their functional dependence seems obvious. In several fMRI studies the essential nodes of both networks could be localized using voluntary optokinetic ('look') nystagmus (OKN) in the horizontal plane incorporating visual motion tracking (Dieterich et al., 2009). Here, functional connectivity (FC) between these nodes representing both networks was studies using resting-state FC. Methods Resting-state fMRI data of 200 healthy adults (age 44.1±17.9; 79 male) were included in the cro…

Communicationbusiness.industrySuperior colliculusPattern recognitionNystagmusOptokinetic reflexHorizontal planecomputer.software_genreSensory SystemsHierarchical clusteringWhite mattermedicine.anatomical_structureNeurologyVoxelPhysiology (medical)medicineNeurology (clinical)Artificial intelligencemedicine.symptomPsychologybusinesscomputerPartial correlationClinical Neurophysiology
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EP 34. Functional hierarchy within the neural network for optokinetic ‘look’ nystagmus

2016

Item does not contain fulltext Key nodes of neural networks for ocular motor control and visual motion processing have been localized using saccades, smooth pursuit, and optokinetic nystagmus (OKN). Within the context of an independent fMRI study using OKN, 9 bilateral network nodes were localized comprising cortical eye fields in frontal (FEF), supplementary motor (SEF), cingulate (CEF) and parietal cortex (PEF), visual motion centers MT+ and V6, the superior colliculus (SC), the lateral geniculate nucleus (LGN) and the globus pallidus (GP). Here, we examined the network's functional hierarchy as present in the structural co-variation (SCoV) and resting-state (RS) fMRI, and the effect of R…

Communicationbusiness.industrySuperior colliculusPosterior parietal cortexCognitive artificial intelligenceOptokinetic reflexNystagmusLateral geniculate nucleuscomputer.software_genreSensory SystemsSmooth pursuitCorrelationBrain Networks and Neuronal Communication [DI-BCB_DCC_Theme 4]NeurologyVoxelPhysiology (medical)medicineNeurology (clinical)medicine.symptombusinessPsychologyNeurosciencecomputerClinical Neurophysiology
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Observational study on risk factors determining residual dizziness after successful benign paroxysmal positional vertigo treatment: The role of subcl…

2018

Studio osservazionale sui fattori di rischio che causano residual dizziness dopo il trattamento della vertigine parossistica posizionale benigna: il ruolo della VPPB subclinica.Alcuni pazienti, dopo il trattamento della vertigine parossistica posizionale benigna concluso con successo, possono lamentare un disequilibrio residuo. La possibile spiegazione potrebbe essere: la persistenza di otoliti canalari insufficienti a provocare un nistagmo clinicamente evidente, una disfunzione utriculare, coesistenza di altri disordini del sistema vestibolare. Abbiamo condotto uno studio osservazionale prospettico caso-controllo, focalizzando l’attenzione sul ruolo di fattori di rischio che possono causar…

MalePediatricsNistagmoNystagmusResidualSettore MED/42 - Igiene Generale E ApplicataDisequilibrio residuoNystagmus0302 clinical medicineRisk FactorsMedicine030223 otorhinolaryngologyResidual dizzinessSubclinical infectionAged 80 and overHigh prevalenceDizzineMiddle AgedGeneral Energy030220 oncology & carcinogenesisCohortVPPB soggettivaFemaleBenign paroxysmal positional vertigomedicine.symptomAdultmedicine.medical_specialtyBenign paroxysmal positional vertigoAdolescentVertigine parossistica posizionale benignaResidual dizzineDizzinessNystagmuVestibologyYoung Adult03 medical and health sciencesDisequilibriootorhinolaryngologic diseasesHumansIn patientAgedbusiness.industrymedicine.diseaseLogistic ModelsOtorhinolaryngologyCase-Control StudiesObservational studysense organsbusinessSubjective BPPV
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Two distinct phenotypes, hemiplegic migraine and episodic Ataxia type 2, caused by a novel common CACNA1A variant

2020

Abstract Background To investigate the genetic and environmental factors responsible for phenotype variability in a family carrying a novel CACNA1A missense mutation. Mutations in the CACNA1A gene were identified as responsible for at least three autosomal dominant disorders: FHM1 (Familial Hemiplegic Migraine), EA2 (Episodic Ataxia type 2), and SCA6 (Spinocerebellar Ataxia type 6). Overlapping clinical features within individuals of some families sharing the same CACNA1A mutation are not infrequent. Conversely, reports with distinct phenotypes within the same family associated with a common CACNA1A mutation are very rare. Case presentation A clinical, molecular, neuroradiological, neuropsy…

MaleProbandmedicine.medical_specialtyNeurologyMigraine with AuraFamilial hemiplegic migraine type 1Mutation MissenseneuropsychologyCase Reportmedicine.disease_causeNystagmus Pathologiclcsh:RC346-42903 medical and health sciences0302 clinical medicinemedicineHumansSpinocerebellar ataxia type 6Missense mutationFamilyChildFamilial hemiplegic migrainelcsh:Neurology. Diseases of the nervous system030304 developmental biologyEpisodic ataxiaGenetics0303 health sciencesMutationbusiness.industryCACNA1A geneEpisodic ataxia type2Cognitive affective syndromeGeneral Medicinemedicine.diseasePhenotypePhenotypeAtaxiaCalcium ChannelsNeurology (clinical)businessCognitive affective syndrome neuropsychology.030217 neurology & neurosurgeryBMC Neurology
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Effects of electrical stimulation in vestibular cortex areas in humans.

2010

The case of a patient with focal epilepsy is reported who underwent presurgical evaluation by stereotactic intracranial electroencephalographic (EEG) recording. A subdural semi-grid electrode, consisting of three multi-channel strip electrodes, was implanted over the temporal lobe and temporo-occipital region; one multi-channel depth electrode was applied towards the posterior insular cortex. During electrical stimulation and EEG monitoring eye movements were recorded by 3-D video-oculography. Stimulation of the medial temporal gyrus induced blurring of vision and horizontal nystagmus. Stimulation of the superior temporal gyrus with low intensities also induced blurring of vision and a simi…

Malegenetic structuresEye MovementsNystagmusElectroencephalographyInsular cortexTemporal lobeSuperior temporal gyrusYoung AdultGyrusNystagmus PhysiologicMonitoring IntraoperativePreoperative CaremedicineHumansPostural BalanceBrain MappingEpilepsymedicine.diagnostic_testEye movementElectroencephalographyReflex Vestibulo-OcularVestibular cortexeye diseasesElectric StimulationTemporal LobeElectrophysiologymedicine.anatomical_structureNeurologyNeurology (clinical)Vestibule Labyrinthmedicine.symptomPsychologyNeurosciencePsychomotor PerformanceJournal of the neurological sciences
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17. Masseter reflex abnormalities with meso-diencephalic lesions

2012

different papers and some eponyms used to characterize a certain neurological state differ from the original description. Except for Wallenberg‘s syndrome, classical brainstem syndromes are rarely seen in clinical practice. Lacunar brainstem syndromes are the consequence of infarcts, which involve long tracts and spare intra-axial cranial nerve segments. This group includes pure motor or pure sensory stroke, dysarthriaclumsy hand syndrome, or ataxic hemiparesis. Such infarcts may also cause body lateropulsion with or without limb ataxia, internuclear ophthalmoplgia, skew-torsion sign, or ocular tilt reaction. Small deep infarcts, which solely affect certain nuclei, may be followed by horizo…

Palsybusiness.industryLimb ataxiaSensory systemAnatomymedicine.diseaseSensory SystemsNeurologyPhysiology (medical)VomitingMedicinecardiovascular diseasesNeurology (clinical)BrainstemUpbeat nystagmusmedicine.symptombusinessStrokeJaw jerk reflexClinical Neurophysiology
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18F-fluorodeoxyglucose hypometabolism in cerebellar tonsil and flocculus in downbeat nystagmus.

2006

A patient with downbeat nystagmus was examined by F-fluorodeoxyglucose-positron emission tomography once while off and twice while on successful treatment with 4-aminopyridine. All positron emission tomography scans of the patient showed a reduced cerebral glucose metabolism bilaterally in the region of the cerebellar tonsil and flocculus/paraflocculus when compared with a normal database of the whole brain. An additional region-of-interest analysis revealed that 4-aminopyridine treatment lessened the hypometabolism. This finding supports the hypothesis that the cerebellar tonsil and (para-) flocculus play a crucial role in downbeat nystagmus. The hypometabolism might reflect reduced inhibi…

Pathologymedicine.medical_specialtyCerebellumgenetic structuresEye MovementsNystagmusFlocculusNystagmus PathologicDownbeat nystagmusImaging Three-DimensionalVestibular nucleiFluorodeoxyglucose F18CerebellummedicinePotassium Channel BlockersHumans4-AminopyridineAgedFluorodeoxyglucosemedicine.diagnostic_testbusiness.industryGeneral Neurosciencemedicine.anatomical_structurePositron emission tomographyPositron-Emission TomographyCerebellar tonsilFemalemedicine.symptombusinessNeurosciencemedicine.drugNeuroreport
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Ocular Motor Paroxysmal Events in Neonates and Infants: A Review of the Literature

2021

Abstract Background Ocular paroxysmal events can accompany a variety of neurological disorders. Particularly in infants, ocular paroxysmal events often represent a diagnostic challenge. Distinguishing between epileptic and nonepileptic events or between physiological and pathologic paroxysmal events can be challenging at this age because the clinical evaluation and physical examination are often limited. Continuous polygraphic video-electroencephalography (EEG) monitoring can be helpful in these situations. Methods We review ocular paroxysmal events in newborns and infants. The aim is to improve clinical recognition of ocular paroxysmal events and provide a guide to further management. Usin…

Pediatricsmedicine.medical_specialtygenetic structuresOcular motorSocio-culturaleEpileptic phenomenaPhysical examinationNystagmusElectroencephalographyOpsoclonus.NystagmuDiagnosis DifferentialNystagmus03 medical and health sciencesEpileptic phenomena; Newborn; Nonepileptic phenomena; Nystagmus; Opsoclonus0302 clinical medicineOcular Motility DisordersDevelopmental NeuroscienceNeuroimaging030225 pediatricsmedicineHumansNonepileptic phenomenaEpilepsymedicine.diagnostic_testbusiness.industryInfant NewbornInfantMultimodal therapyElectroencephalographyOpsoclonusNewborneye diseasesNeurologyPediatrics Perinatology and Child HealthEtiologyNeurology (clinical)medicine.symptombusinessClinical evaluation030217 neurology & neurosurgery
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Vestibular adaptation to centrifugation does not transfer across planes of head rotation

2008

Out-of-plane head movements performed during fast rotation produce non-compensatory nystagmus, sensations of illusory motion, and often motion sickness. Adaptation to this cross-coupled Coriolis stimulus has previously been demonstrated for head turns made in the yaw (transverse) plane of motion, during supine head-on-axis rotation. An open question, however, is if adaptation to head movements in one plane of motion transfers to head movements performed in a new, unpracticed plane of motion. Evidence of transfer would imply the brain builds up a generalized model of the vestibular sensory-motor system, instead of learning a variety of individual input/output relations separately. To investi…

PhysicsVestibular systemmedicine.medical_specialtySupine positionGeneral NeuroscienceAcousticsNystagmusAudiologymedicine.diseaseSensory SystemsSagittal planeTransverse planeIllusory motionmedicine.anatomical_structureMotion sicknessOtorhinolaryngologymedicineReflexNeurology (clinical)medicine.symptomJournal of Vestibular Research
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